ライフサイエンスコーパス: CCI

1 CCI (at hospital discharge and after 6 months) was the r

2 CCI data were prospectively collected over a 1-year peri

3 CCI induced up-regulation of MyD88 and chemokine C-C mot

4 CCI injury strongly induced Runx1 expression in activate

5 CCI significantly up-regulated GFAP, V1aR and AQP4 prote

6 CCI was not accurate in calculating the severity of a co

7 CCI, registered during 90 days after pouch construction,

8 CCI-induced mechanical hyperalgesia was reduced in IL-33

9 CCI-ION caused orofacial hypersensitivity that correlate

10 CCI scores were as follows: CCI 2, n = 589; CCI 3 or 4, n = 599; CCI 5 or 6, n = 229; and CCI >/= 7,

11 ollows: CCI 2, n = 589; CCI 3 or 4, n = 599; CCI 5 or 6, n = 229; and CCI >/= 7, n = 102.

12 ed that the G(1)-S cell-cycle inhibitor-779 (CCI-779), an mTOR inhibitor, inhibited UBE2C mRNA and pr

13 ard ratio [HR] = 1.89; 95% CI, 1.24 to 2.88; CCI >or= 1 (HR = 1.38; 95% CI, 1.01 to 1.88), functional

14 subhazard ratio [SHR] 1.4; 95% CI, 1.1-1.9), CCI score higher than 3 (SHR 2.8; 95% CI, 2.1-3.7), a ce

15 y, among patients with pT1Nx-0 disease and a CCI </= 1, risk of non-RCC death exceeded that of abdomi

16 For patients with pT1Nx-0 disease but a CCI >/= 2, the risk of non-RCC death exceeded that of ab

17 ndings suggest that AdGDNF alone following a CCI was not therapeutic and although combining it with l

18 200-12.767; P = 0.024) as risk factors for a CCI >60.

19 ssion and estimated the odds ratio (OR) of a CCI score >/= 4 using generalized estimating equations.

20 havioural testing showed that animals with a CCI exhibited heat hyperalgesia within 4 days of the inj

21 ents 75 years and older, and patients with a CCI score of 3 or greater.

22 Age-adjusted CCI (hazard ratio [HR] = 1.36, P < .001), L5HU (HR = 2.8

23 oncancer death versus 0.718 for age-adjusted CCI alone.

24 ng, androgen deprivation, race, age-adjusted CCI, L5HU, and PsoasL4-5 were included in a multivariabl

25 After CCI, upregulation of CD11b in sciatic nerve was less in

26 After CCI, we observed galanin upregulation in DRG and sciatic

27 of YAP and TAZ was on day 14 and day 1 after CCI, respectively; (3) there were also unique topographi

28 brain resident microglia prior to and after CCI, in order to confirm that modulations of HP [1-(13)C

29 ut changes in the number of astrocytes after CCI injury.

30 the beneficial effects of candesartan after CCI, suggesting that PPARgamma activation may contribute

31 mido adenosine HCl (CGS21680), 10-14 d after CCI versus sham surgery, produced a long-duration revers

32 ection in cortex and hippocampus 1 day after CCI and glucose administration.

33 transient impairment in motor function after CCI in rats and mice.

34 ing the CA1 and CA3 of the hippocampus after CCI.

35 tment was delivered for the first hour after CCI.

36 at 2 g/kg, i.p.) initiated immediately after CCI was found to significantly attenuate a TBI-induced d

37 expression was significantly increased after CCI, indicating translocation of TAZ from the cytoplasma

38 ollowed two distinct temporal patterns after CCI, such that the highest expression of YAP and TAZ was

39 ne-synthesizing enzyme serine racemase after CCI injury improved synaptic plasticity, brain oscillati

40 an treatment reduced the lesion volume after CCI injury by approximately 50%, decreased the number of

41 llodynia) were established at one week after CCI.

42 r cells with WYE-132 or the rapamycin analog CCI-779 led to a rapid loss of the phosphorylation at Se

43 In multivariate analysis, CCI was the most powerful predictor of OS, which was sti

44 In-hospital mortality was 1%, and CCI was 21 +/- 19.

45 CI 3 or 4, n = 599; CCI 5 or 6, n = 229; and CCI >/= 7, n = 102.

46 inical interactions between lenalidomide and CCI-779 seemed to occur, with in vitro data indicating l

47 ent patterns of correlations between NAA and CCI were found in higher verbal ability versus lower ver

48 In CCI-vehicle, sham and CCI-SR49059 groups, fluorescence intensity of GFAP was 3

49 In CCI-vehicle, sham and CCI-SR49059 groups, GFAP was 1.58+/-0.04, 0.47+/-0.02, a

50 atched with CSS alone in six, CSS as well as CCI in one, and neither CSS or CCI in two scenarios.

51 ATL313 attenuated CCI-induced upregulation of spinal cord activation marke

52 ed candesartan or vehicle to mice 5 h before CCI injury.

53 o significant differences in binding between CCI and sham mice.

54 CI showed no significant differences between CCI and sham mice.

55 ificant differences in B(max) values between CCI and sham-operated mice, indicating that the differen

56 %; 9.5+/-0.9 microm(2)), and SR49059 blunted CCI-induced increases in brain edema (79.0+/-0.2%; 9.4+/

57 Morris water maze test showed that both CCI and S-CCI produced persisting memory deficits.

58 a randomized trial (effect size detected by CCI vs conventional standardized morbidity outcomes).

59 e YAP expression were selectively induced by CCI but not CFA-induced hindpaw inflammation; and (5) th

60 val after resection of CLM was stratified by CCI (high, >/=26.2; low, <26.2).

61 ewer patients with morbidity, but comparable CCI when morbidity is present.

62 ociated with a complex postoperative course (CCI at 6 months >60).

63 Lower HRR scores and higher D15 CCI (both indicating worse color vision) were associated

64 and Lanthony D15 color confusion index (D15 CCI).

65 Despite CCI, the HP [1-(13)C] lactate-to-pyruvate ratio at the i

66 The recently developed CCI for measuring complications is a step forward in thi

67 e observed between patients in the different CCI groups.

68 IL-33-mediated hyperalgesia during CCI was dependent on a reciprocal relationship with TNF-

69 ons, intrathecal injection of IL-33 enhanced CCI hyperalgesia and induced hyperalgesia in naive mice.

70 st but not Lanthony's D-15d test [Farnsworth CCI mean difference = 0.05, 95% confidence interval (CI)

71 Following CCI injury, hippocampal neurons downregulated d-serine l

72 ally contributed to pain behaviors following CCI.

73 t edema was predominantly cellular following CCI and documented that V1aR inhibition with SR49059 sup

74 sions and increased motor deficits following CCI injury.

75 ministration of AdGDNF immediately following CCI.

76 idonoylglycerol (2-AG) in the rACC following CCI showed no significant differences between CCI and sh

77 At earlier times following CCI, ultrastructural studies reveal that microglial cell

78 Resulting CCI scores were as follows: CCI 2, n = 589; CCI 3 or 4, n = 599; CCI 5 or 6, n = 229

79 Similar and significant changes for CCI-779 pharmacokinetics were also observed with increas

80 The molecular mechanisms for CCI-779 inhibition of UBE2C gene expression involved a d

81 The corresponding OR for CCI >/= 4 was 2.79 (95% CI: 1.02, 7.59; p-trend = 0.05).

82 However, T cells from CCI patients exhibited no suppressed TCR response at the

83 We found that T cells from CCI patients featured higher basal levels of activation

84 strate an augmented response in T cells from CCI patients in response to TCR/coreceptor (CD3/CD28) ch

85 sity was reduced in high-threshold CSNs from CCI mice compared with sham animals, with no differences

86 ociceptive-like phenotype among neurons from CCI animals compared with sham mice.

87 220 (35%) were age >/=65 years, and 92% had CCI > 0, indicating >/=1 comorbid conditions.

88 High CCI (odds ratio 3.99, P <0.001) was associated with high

89 High CCI is a potent predictor of worse RFS and CSS after res

90 c resection were worse in patients with high CCI than in patients with low CCI (RFS at 3 yrs 26% vs.

91 ort, LCT-EOS cutoff best discriminating high-CCI, 90-day-mortality and severe-morbidity were 3, 3 and

92 nd 17.7% the accuracy of the models for high-CCI, 90-day-mortality and severe-morbidity, respectively

93 Higher CCI was significantly associated with lower OS probabili

94 Median in-hospital CCI was comparable for both groups (DCD 38.2; DBD 36.7;

95 clinical study of chronic critical illness (CCI) patients aimed at assessing the long-term consequen

96 In the controlled cortical impact (CCI) animal model of pediatric TBI (postnatal day 16-17)

97 also occur post controlled cortical impact (CCI) demonstrating a reduction in catalytic subunit dist

98 s produced using controlled cortical impact (CCI) in a rat model, and TNS treatment was delivered for

99 veral days after controlled cortical impact (CCI) in mice.

100 damage caused by controlled cortical impact (CCI) injury in mice results in a switch from neuronal to

101 to TBI using the controlled cortical impact (CCI) injury model.

102 1(-/-) mice in a controlled cortical impact (CCI) injury murine model of traumatic brain injury (TBI)

103 refore performed controlled cortical impact (CCI) injury on mice to investigate whether the ARB cande

104 lly prepared for controlled cortical impact (CCI) injury or sham surgery.

105 at 7 days after controlled cortical impact (CCI) injury were examined.

106 ed adult mice to controlled cortical impact (CCI) injury, and isolated RNA from the SVZ and DG at dif

107 el of unilateral controlled cortical impact (CCI) injury.

108 The controlled cortical impact (CCI) model is one of the most commonly used models of co

109 s study, using a controlled cortical impact (CCI) model of head injury, we show a large increase in t

110 injury (CHI) and controlled cortical impact (CCI) models, we developed a bilateral head injury model

111 um recovery in a controlled cortical impact (CCI) mouse model of TBI.

112 week prior to a controlled cortical impact (CCI) over the forelimb sensorimotor cortex of the rat (F

113 Using a controlled cortical impact (CCI) procedure in rats, we show that traumatic brain inj

114 after digitally controlled cortical impact (CCI) to produce traumatic brain injury (TBI).

115 Following controlled cortical impact (CCI), bolus administrations of NAM (500 mg/kg), 7.5% HTS

116 at 6 weeks after controlled cortical impact (CCI).

117 In CCI-vehicle, sham and CCI-SR49059 groups, fluorescence i

118 In CCI-vehicle, sham and CCI-SR49059 groups, GFAP was 1.58+

119 ded are data from 2862 participants (1337 in CCI's, 1425 in HH's, and 100 street youth).

120 n in HH reported an adequate diet vs. 95% in CCI's and 99% among street youth.

121 ent pain relief after oral administration in CCI rats (ED(50) = 19.6 (+/- 1.2) mumol/kg), which was c

122 ent, also diminishes mechanical allodynia in CCI in female mice.

123 : 3.6-9.5) were more likely than children in CCI's to be low height-for-age.

124 y low height-for-age compared to children in CCI's, suggesting chronic malnutrition among them.

125 ave an adequate diet compared to children in CCI's.

126 neuropathic painful conditions evaluated in CCI and STZ murine model.

127 rea (n=3/group) were significantly higher in CCI-vehicle (80.5+/-0.3%; 18.0+/-1.4 microm(2)) versus s

128 Globally, national level increases in CCI were 17.5% faster than they would have been without

129 ons of lamina I spinoparabrachial neurons in CCI animals co-varied significantly (P < 0.03, ANCOVA) w

130 analgesia was associated with reductions in CCI-related GAD65 and GAT-1 serine dephosphorylation as

131 o have an adequate diet compared to those in CCI's (AOR 0.4, 95% CI 0.2-1.0).

132 urvival (OS) and crude cumulative incidence (CCI) of local recurrence (LR) and distant metastasis (DM

133 02) were independent predictors of increased CCI.

134 ces; clinical by Charlson comorbidity index (CCI) and body mass index; function by number of physical

135 plication of the Charlson comorbidity index (CCI) and other uremia-related comorbidities, not include

136 ith age-adjusted Charlson Comorbidity Index (CCI) by using the Harrell c statistic.

137 the MoRT and the Charlson Comorbidity Index (CCI) for discharge and 1-year mortality were estimated u

138 ities, including Charlson Comorbidity Index (CCI) score; ocular comorbidities; cataract severity; and

139 The Charlson Comorbidity Index (CCI) was developed in 1987 and is the most used comorbid

140 s were age, sex, Charlson Comorbidity Index (CCI), radiation and chemotherapy, immunomodulator medica

141 y Index, and the Charlson Comorbidity Index (CCI).

142 ailty score, and Charlson comorbidity index (CCI).

143 gnosis using the Charlson Comorbidity Index (CCI); 511 indexed comorbidities were reported in 1519 CM

144 = 80 years), and Charlson comorbidity index (CCI; </= 1 and >/= 2).

145 ell as the Comprehensive Complication Index (CCI) (Ann Surg 258(1):1-7, 2013).

146 ints (high comprehensive complication index (CCI) scores, 90-day-mortality and severe-morbidity), pro

147 lue of the comprehensive complication index (CCI) to standard assessment of postoperative morbidity,

148 (CSS) with comprehensive complication index (CCI) using a questionnaire-based survey of experienced g

149 Mean comprehensive complication index (CCI) was 26.0 (IQR, 8.7-36.2).

150 d with the comprehensive complication index (CCI), and the neutrophil-to-lymphocyte ratio (NLR) was u

151 the novel Comprehensive Complication Index (CCI).

152 using the comprehensive complication index (CCI).

153 including comprehensive complications index (CCI), Clavien-Dindo complication (CDC), and pulmonary co

154 obic anxiety scale of the Crown-Crisp Index (CCI) assessment at mean +/- SD age of 59 +/- 9 years (ra

155 leading to a "chlorophyll/carotenoid index" (CCI) that tracks evergreen photosynthesis at multiple sp

156 , from which a "Composite Creativity Index" (CCI) was created.

157 ed trends in a composite coverage indicator (CCI) based on eight reproductive, maternal, newborn, and

158 difference in mean color confusion indices (CCI) was statistically significant for the Farnsworth te

159 al root ganglion (DRG) neurons and inhibited CCI-evoked neuroinflammation in DRGs and spinal cord tis

160 tion of lenalidomide with the mTOR inhibitor CCI-779 has displayed synergy in vitro and represents a

161 weeks in murine chronic constriction injury (CCI) and spared nerve injury models.

162 neurons in the chronic constriction injury (CCI) model of neuropathic pain were compared to those of

163 but not in the chronic constriction injury (CCI) model of the sciatic nerve.

164 pain using the chronic constriction injury (CCI) model.

165 pain using the chronic constriction injury (CCI) model.

166 Chronic constriction injury (CCI) of the left sciatic nerve was performed on wild typ

167 ynia induced by chronic constriction injury (CCI) of the sciatic nerve in mice, was related to both a

168 Chronic constriction injury (CCI) of the sciatic nerve induced IL-33 production in th

169 Chronic constriction injury (CCI) produced a significant decrease in WIN 55,212-2-sti

170 nistration in a chronic constriction injury (CCI) rat model of neuropathic pain with an ED(50) of 8.3

171 Chronic constriction injury (CCI) reduced paw withdrawal latency, which was maximal a

172 pain following chronic constriction injury (CCI), were reduced in CKO mice.

173 d rat models of chronic constriction injury (CCI)-induced neuropathic pain.

174 group of rats to cortical contusion injury (CCI) and another to transient middle cerebral artery occ

175 brain edema after cortical contusion injury (CCI) in rat 5h post-injury.

176 bolic changes in controlled cortical injury (CCI) mice (n = 57).

177 y chronic constriction sciatic nerve injury (CCI).

178 model of constriction sciatic nerve injury (CCI).

179 h chronic constriction sciatic nerve injury (CCI).

180 n chronic constriction sciatic nerve injury (CCI).

181 (HH), 19 Charitable Children's Institutions (CCIs), and 100 street-involved children.

182 fidence interval (CI): 0.003, 0.10; Lanthony CCI mean difference = 0.07, 95% CI: -0.02, 0.15].

183 Eighty-eight patients had high and 487 low CCI.

184 ents with high CCI than in patients with low CCI (RFS at 3 yrs 26% vs. 41%, P = 0.003; CSS at 5 yrs 4

185 Overall, ta-IPAA led to lower CCI scores.

186 Blood was drawn from 12 male CCI patients (median age 67 y, range 48-79 y) receiving

187 In patients with morbidity, mean CCI of the transanal approach was 2.23 points lower than

188 Six-month postoperative median CCI was significantly higher for DCD grafts (53.4 vs 47.

189 on regimen was 25 mg lenalidomide with 15 mg CCI-779, with toxicities of fatigue, neutropenia, and el

190 termined to be 25 mg lenalidomide with 15 mg CCI-779.

191 ith 15 to 25 mg lenalidomide and 15 to 20 mg CCI-779.

192 ffect of a controlled cortical impact model (CCI) of traumatic brain injury (TBI) on their distributi

193 The effect of moderate CCI on the expression and distribution of calcineurin B

194 nstriction injury to the infraorbital nerve (CCI-ION) to study whether CCI-ION caused calcium channel

195 nstriction injury to the infraorbital nerve (CCI-ION).

196 latory complex-complex interaction networks (CCIs).

197 Healthy non-CCI rats (n = 5), were also employed.

198 which contributes to antitumor activities of CCI-779 in UBE2C overexpressed AR-positive CRPC.

199 inetic analysis indicated increased doses of CCI-779 resulted in statistically significant changes in

200 GTPgammaS binding in the thalamus and PAG of CCI mice, with no change in the rACC.

201 ), thalamus and periaqueductal grey (PAG) of CCI and sham-operated mice.

202 rostral anterior cingulate cortex (rACC) of CCI mice when compared to sham-operated controls.

203 Mice were subjected to S-CCI or CCI using an electromagnetic impactor (Impactor One, MyN

204 SS as well as CCI in one, and neither CSS or CCI in two scenarios.

205 ective pharmacologic inhibitor, Ku0063794 or CCI-779, caused the up-regulation of nuclear phospho-Mst

206 Multivariate analyses for OS, CCI of LR, and DM were performed.

207 ical and behavioral outcome in the pediatric CCI model.

208 the DRG increased transiently on day 1 post-CCI in WT but not in GFAP-IkappaBalpha-dn mice, and no e

209 de amnesia test was conducted on day 12 post-CCI and showed that all groups had significant retention

210 ations (2 g/kg, i.p.) at 0, 1, 3 and 6h post-CCI.

211 rats treated with AdGDNF and l-arginine post-CCI had a significantly smaller contusion than injured r

212 ing mitochondrial cyt c release at 24 h post-CCI and -MCAO.

213 urons in cortex and hippocampus at 24 h post-CCI.

214 .0 mL/kg) were given at 2, 24, and 48 h post-CCI.

215 hermal hyperalgesia compared to WT mice post-CCI.

216 e of CGRP upregulation in sciatic nerve post-CCI was found.

217 erficial dorsal horn (lamina I) 3 weeks post-CCI-ION.

218 In nerve injury models, AXO, PSNL, CCI, and SNL caused changes to the largest number of gai

219 t indices with the rank order being AXO>PSNL=CCI >> SNL.

220 e, MyNeuroLab; semicircular tip: 3mm radius; CCI tip diameter: 3mm).

221 o specific allosteric inhibitors (rapamycin, CCI-779, and RAD001) of the pivotal cell growth regulato

222 Moreover, i.t. BMSCs reduced CCI-induced spontaneous pain and axonal injury of dorsal

223 ptor (IL-33 decoy receptor) markedly reduced CCI-induced hyperalgesia.

224 Resulting CCI scores were as follows: CCI 2, n = 589; CCI 3 or 4,

225 ver operating characteristic curves revealed CCI to be a more sensitive, specific, and accurate predi

226 s water maze test showed that both CCI and S-CCI produced persisting memory deficits.

227 Here we report a new semicircular CCI (S-CCI) model by increasing the impact tip area to cover bo

228 We showed that S-CCI, at two injury severities, significantly decreased t

229 matosensory and motor deficits only in the S-CCI groups.

230 ages after the injury, suggesting that the S-CCI model produces long-lasting motor deficits.

231 otor cortex, and hippocampal damage in the S-CCI.

232 Mice were subjected to S-CCI or CCI using an electromagnetic impactor (Impactor O

233 Here we report a new semicircular CCI (S-CCI) model by increasing the impact tip area to c

234 ator and rapidly (within minutes) suppressed CCI-evoked spinal synaptic plasticity and DRG neuronal h

235 DA-approved rapamycin analogue temsirolimus (CCI-779) blocks ANDV protein expression and virion relea

236 particular the mTOR inhibitor temsirolimus (CCI-779), induce autophagy, which can promote tumor surv

237 Compared with the rapalog temsirolimus/CCI-779, WYE-132 elicited a substantially stronger inhib

238 Thus, the present findings reveal that CCI sepsis patients feature signs of immune suppression

239 These results suggest that CCI induced a region-specific adaptation of mu-opioid re

240 These data suggest that CCI produced desensitization of the cannabinoid 1 recept

241 The CCI adds information on postoperative morbidity in almos

242 The CCI can be readily computed on the basis of tabulated co

243 The CCI in the RCT on colon resections confirmed the absence

244 The CCI is calculated as the sum of all complications that a

245 The CCI may serve as an appealing endpoint for future RCTs a

246 The CCI summarizes all postoperative complications and is mo

247 The CCI used was unadjusted for recipient age.

248 The CCI was highly sensitive in detecting treatment effect d

249 The CCI was introduced about 3 years ago as a novel metric o

250 The CCI was tested in 3 published RCTs from European centers

251 The CCI, integrating all complications including their sever

252 From 1994 to 2014, the CCI increased by 0.82 percent points a year across all c

253 the MoRT (0.59; 95% CI: 0.56, 0.62) and the CCI (0.59; 0.56, 0.62).

254 the MoRT (0.77, 95% CI: 0.74, 0.79) and the CCI (0.77, 95% CI: 0.75, 0.80).

255 postoperative events were assessed, and the CCI calculated.

256 obtained were compared with the CSS and the CCI for these patients.

257 d to separate comorbidity covariates and the CCI.

258 had a similar overall discrimination as the CCI for mortality at hospital discharge in injured adult

259 ex hospital stay for DCD and DBD LT, but the CCI increases significantly for DCD recipients in 6 mont

260 Information gained by the CCI increased with the complexity of surgery and observa

261 using any or most severe complications, the CCI revealed significant differences between treatment g

262 In contrast, the CCI induced motor deficits only at early stages after th

263 es in trials are up to 9 times lower for the CCI than for traditional morbidity endpoints.

264 inter-connection, should be included in the CCI calculation to best mirror the patients' postoperati

265 n IOF rate experienced a 43% increase in the CCI compared with patients who received less than the me

266 ore responsive to graded heat stimuli in the CCI, compared to controls (P < 0.02, 2-factor repeated-m

267 In the CCI, nociceptive lamina I spinoparabrachial neurons had

268 a-related comorbidities, not included in the CCI, were associated with mortality in these patients.

269 pain behavior in the oxaliplatin but not the CCI model.

270 Within 30min of the CCI and AdGDNF injections, some animals were injected wi

271 sed to determine the additional value of the CCI compared to the Clavien-Dindo classification.

272 This study demonstrates superiority of the CCI to traditional endpoints.

273 ity to treatment effects and validity of the CCI was performed by 4 different approaches, based on 12

274 To compare the sensitivity of the CCI with traditional morbidity endpoints, for example, p

275 suggest solutions for consistent use of the CCI.

276 It remains unclear, how often the CCI adds to standard reporting of complications and how

277 ging Spectroradiometer satellite sensor, the CCI closely follows the seasonal patterns of daily gross

278 lly in hippocampal tissue ipsilateral to the CCI injury.

279 loped, and its influence was compared to the CCI.

280 le size calculations were compared using the CCI and traditional endpoints.

281 Besides, we wondered whether the CCI was best to study the influence of comorbidity in ki

282 ex that incorporated these measures with the CCI was associated with improved accuracy for prediction

283 ctive of mortality had some overlap with the CCI, but this study identified 2 novel predictors: cardi

284 cation by the time of discharge, and thereby CCI added information to the standard grading system of

285 Furthermore, these CCI-induced behavioral and cellular responses were dimin

286 Thus, CCI-ION-induced Cavalpha2delta1 up-regulation may contri

287 addition, exposure of abl and C4-2B cells to CCI-779 also decreased UBE2C-dependent cell invasion.

288 block cell-cycle G(1) to S-phase transition, CCI-779 causes a cell-cycle G(2)-M accumulation and an i

289 f the rat immediately following a unilateral CCI over the FL-SMC.

290 tomoses less than 10 cm from the anal verge, CCI of 3 or more, high inferior mesenteric artery ligati

291 ed with poor treatment tolerance; these were CCI >or= 1 (odds ratio [OR] = 2.49; 95% CI, 1.18 to 5.25

292 nfraorbital nerve (CCI-ION) to study whether CCI-ION caused calcium channel alpha2delta1 (Cavalpha2de

293 preoperative sarcopenia was associated with CCI (P = 0.043), and CDC >/=IIIb (P = 0.003).

294 r calculating morbidity and compared it with CCI.

295 281 matched with CSS while 143 matched with CCI (P = 0.0001, odds ratio: 3.7; 95% CI 2.8-4.8).

296 %, 89.4%, 77.6%, and 46.4% for patients with CCI 2, 3 to 4, 5 to 6, and >/=7, respectively.

297 tor hydroxychloroquine (HCQ) synergizes with CCI-779 and led to melanoma cell death via apoptosis.

298 Combination treatment with CCI-779 and HCQ suppressed melanoma growth and induced c

299 Treatment with CCI-779 significantly decreased abl cell proliferation i

300 The 5, 8, and 10-year CCI of LR and DM were 25.9 (95% CI, 23.1, 29.1), 31.3 (9