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1 les, as well as the subsequent activation of CD4-positive T lymphocytes.
2 ass II (MHC II) molecules for recognition by CD4-positive T lymphocytes.
3                           Infection of human CD4-positive T lymphocytes by human immunodeficiency vir
4                  The change in percentage of CD4-positive T lymphocytes (CD4%) was investigated in 10
5 troviral therapy (HAART) on the evolution of CD4-positive T-lymphocyte (CD4 cell) count among human i
6 s evaluated population- and individual-level CD4-positive T-lymphocyte (CD4 cell) count trajectories
7 with 77 nonusers on age, race, and pre-HAART CD4-positive T-lymphocyte (CD4+ cell) count and viral lo
8  tested semiannually to identify their first CD4-positive T-lymphocyte cell count below 200/microl; t
9 equency of measurement of HIV RNA levels and CD4-positive T-lymphocyte cell counts introduce a possib
10                                   The annual CD4-positive T-lymphocyte cell decline was less in untre
11 e (p = 0.10), low hemoglobin (p = 0.11), and CD4-positive T-lymphocyte count (p = 0.04).
12 men and how this effect differs depending on CD4-positive T-lymphocyte count.
13         Flow cytometric analysis showed that CD4-positive T lymphocyte division was specifically supp
14 creased on mononuclear leukocytes, including CD4-positive T lymphocytes from HIV-positive patients, c
15 essor or cytotoxic subset) predominated over CD4-positive T lymphocytes (helper cells) surrounding th
16 lecule (VCAM-1), and MHC class II as well as CD4-positive T lymphocyte infiltration was detected by i
17 ex (MHC) class II-restricted presentation to CD4-positive T lymphocytes occurs after they are interna
18 ion aged 5.7-12.0 weeks with a percentage of CD4-positive T lymphocytes of at least 25% were randomly
19 acterial lipopeptides that are recognized by CD4-positive T lymphocytes of Mycobacterium tuberculosis

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