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1 CDAD pathology is induced by two exotoxins, toxin A and
2 CDAD predominated in general hospitals versus other faci
4 dium difficile colitis (CCDC) was defined as CDAD associated with graft loss, total colectomy, or dea
5 with a 5% incidence of antibiotic-associated CDAD (median control group risk), probiotic prophylaxis
6 on model, antibodies to both toxins A and B (CDAD antitoxin) were required to prevent morbidity and m
8 s treated with a carbapenem were followed by CDAD, whereas only 2.1% of infections treated without ca
10 c strains of C difficile isolates that cause CDAD were also recovered from stools of symptom-free pat
11 tomy in the treatment of severe, complicated CDAD resulting in reduced morbidity and preservation of
12 omy improves survival in severe, complicated CDAD, outcomes remain poor with associated mortality rat
13 symptom-free carriers (1.5 [1.5]) developed CDAD (pooled risk difference -2.3% [95% CI 0.3-4.3], p=0
14 -up 1.7 weeks [SD 1.3]), 22 (3.6%) developed CDAD, whereas only two (1.0%) of 192 primary symptom-fre
16 illness are at greatest risk for developing CDAD following treatment of polymicrobial infections.
18 Clostridium difficile-associated diarrhea (CDAD) is an increasingly important diagnosis in solid or
19 f Clostridium difficile-associated diarrhea (CDAD) is highly desirable in the setting of hospital cos
20 f Clostridium difficile-associated diarrhea (CDAD) occurs in 15 to 20% of patients after discontinuat
21 l Clostridium difficile-associated diarrhea (CDAD), a series of recommendations and a pathway to prev
25 Clostridium-difficile-associated diarrhoea (CDAD) are culture-positive or culture-negative before il
26 iated diarrhea due to Clostridium difficile (CDAD) is thought to reflect colonization of a disrupted
27 luation for C. difficile-associated disease (CDAD) among 864 patients was based on clinical criteria
28 Clostridium difficile-associated disease (CDAD) constitutes a large majority of nosocomial diarrhe
29 ed Clostridium difficile-associated disease (CDAD) data from the intensive care unit (ICU) and hospit
30 Clostridium difficile-associated disease (CDAD) due to toxigenic strains is prevented in hamsters
34 PARgamma in C. difficile-associated disease (CDAD), immunity and gut pathology, we used a mouse model
35 tients with C. difficile-associated disease (CDAD), one of whom died from extensive pseudomembranous
41 umber of patients treated with colectomy for CDAD preceding the initiation of this current treatment
44 pecific objectives: (1) conduct a review for CDAD and prevention; (2) develop statements based upon p
45 uggests that use of a probiotic strategy for CDAD prevention in humans receiving antibiotics might be
46 nosed with severe, complicated ("fulminant") CDAD and were treated at the University of Pittsburgh Me
50 light of the recent substantial increases in CDAD incidence and severity, this model will be valuable
51 prophylaxis results in a large reduction in CDAD without an increase in clinically important adverse
54 t disinfection (7/8); (8) early detection of CDAD in symptomatic patients (7/8); (9) usage of protect
55 re known to contribute to the development of CDAD, the role of preventive antibiotics is unproven.
58 animals that survived an initial episode of CDAD showed no evidence of diarrhea or colitis after sub
59 assays positive or chart review evidence of CDAD, 97 (14.8%) stool specimens were positive by one or
65 nly carbapenem use affected the incidence of CDAD: 3.5% of infections treated with a carbapenem were
73 itivity of each test method for screening of CDAD was as follows: bacterial culture, 95%; culture wit
76 sistant nontoxigenic C. difficile to prevent CDAD must be balanced against the risk that resistance m
81 CD strains is highly effective in preventing CDAD in hamsters challenged with toxigenic CD strains, w
83 es for prevention and treatment of recurrent CDAD, which is often recalcitrant to existing therapies.
84 fecal communities in patients with recurrent CDAD were highly variable in bacterial composition and w
85 rgrowth (8/8); (4) staff education regarding CDAD preventive measures (8/8); (5) appropriate hand hyg
91 ofiled the fecal microbiota of patients with CDAD (both initial and recurrent episodes) by culture-in
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