コーパス検索結果 (1語後でソート)
通し番号をクリックするとPubMedの該当ページを表示します
1 es the regulation of alternative splicing by CELF proteins.
2 o antagonistic factor families, the MBNL and CELF proteins.
3 tive exons, both of which are antagonized by CELF proteins.
4 important to identify downstream targets of CELF proteins.
9 nding and splicing analyses demonstrate that CELF proteins block splicing through interfering with bi
13 the two motifs were responsive to two other CELF proteins (CUG-BP1 and CELF4), indicating that diffe
14 is analysis was performed in parallel on two CELF proteins, ETR-3 (CUG-BP2, NAPOR, BRUNOL3) and CELF4
15 CUGBP2, the 2 CUGBP1 and ETR-3 like factor (CELF) proteins expressed in heart, demonstrated elevated
16 Members of the CUG-BP and ETR-3 like factor (CELF) protein family bind within conserved intronic elem
20 s type I (NF1) exon 23a is a novel target of CELF protein-mediated splicing regulation in neuron-like
22 n of different splicing events by individual CELF proteins requires separable regions, implying the n
23 ith mice overexpressing CUG-BP1, a wild-type CELF protein, rescues defects in alternative splicing, t
25 ice that express a nuclear dominant-negative CELF protein specifically in the heart (MHC-CELFDelta) d
27 runcation mutants to identify the regions of CELF proteins that are required to activate and to repre
29 n-regulation of CUGBP and ETR-3-like factor (CELF) proteins that regulate nearly half of developmenta
30 ia expression of a nuclear dominant negative CELF protein under an alpha-myosin heavy chain promoter.
31 tive cTNT and IR exons are also regulated by CELF proteins, which were previously implicated in DM pa
WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。