ライフサイエンスコーパス: CFU-E

1 e majority of cattle excrete less than 10(2) CFU E. coli O157/g feces, most studies, including those

2 No rescue of BFU-E and CFU-E growth was observed when NmU peptide was exogenous

3 The binding of BFU-E and CFU-E to the RGDS and CS-1 sites was blocked by beta1 in

4 and erythroid colony-forming unit (BFU-E and CFU-E) colonies, the clonogenic assays that quantify ear

5 tion to promoting adhesion of both BFU-E and CFU-E, supported the highest levels of CFU-E migration (

6 ing erythroid burst-forming unit (BFU-E) and CFU-E progenitors purified by a new technique, we demons

7 ematopoietic progenitors (CFU-GM, BFU-E, and CFU-E colonies).

8 Bone marrow CFU-GM, BFU-E, and CFU-E colony formation was significantly reduced while p

9 ed while peripheral blood CFU-GM, BFU-E, and CFU-E was increased in the trauma patients compared with

10 t-forming unit-erythroid were increased, and CFU-E displayed increased sensitivity to suboptimal EPO

11 In contrast, CFU-E colony formation in vitro by normal fetal liver pr

12 e increased the number of one marrow-derived CFU-E from Wv/+ mice.

13 nly at a stage of erythroid differentiation (CFU-E) preceding the activation of beta-globin genes, co

14 n 1A expression also increases 3-fold during CFU-E maturation may be attributable to the action of NF

15 -forming unit and colony-forming unit (BFU-E/CFU-E) activities were significantly reduced in the homo

16 g unit-erythroid (BFU-E), and CFU-erythroid (CFU-E).

17 lopment of later stage definitive erythroid (CFU-E), mast cell and bipotential granulocyte/macrophage

18 n of human and rabbit bone marrow erythroid (CFU-E, BFU-E) and myeloid (CFU-GM) colony growth.

19 and inhibited colony-forming unit erythroid (CFU-E) and CFU granulocyte-monocyte formation in BM cult

20 (SCF) to form colony-forming unit erythroid (CFU-E) colonies.

21 While colony-forming unit-erythroid (CFU-E) and burst-forming unit-erythroid (BFU-E) colonies

22 nts decreased colony-forming unit-erythroid (CFU-E) and colony-forming unit-granulocyte macrophage (C

23 d (BFU-E) and colony-forming unit-erythroid (CFU-E) formation compared with control.

24 al numbers of colony-forming unit-erythroid (CFU-E) progenitors and erythroid cells that are generate

25 ntiation from colony-forming unit-erythroid (CFU-E) progenitors in fetal liver cells.

26 cellularity, colony forming unit-erythroid (CFU-E) progenitors, and a total absence of germ cells.

27 le numbers of colony-forming unit-erythroid (CFU-E), colony-forming unit-granulocyte (CFU-G), and col

28 des inhibited colony-forming unit-erythroid (CFU-E)-derived colony growth in a concentration-dependen

29 re BFU-E, and colony-forming unit-erythroid (CFU-E).

30 lly increased colony-forming unit-erythroid (CFU-E).

31 se in marrow colony-forming units-erythroid (CFU-E).

32 enitor cells (colony-forming unit-erythroid [CFU-E], burst-forming unit [BFU]-E, and CFU-granulocyte-

33 progenitors (colony-forming units-erythroid, CFU-E) and severe anemia in cats.

34 ntities of the ACK2 antibody reduced femoral CFU-E, BFU-E, and CFU-GM content to less than half that

35 , different H-ras proteins were expressed in CFU-E progenitors and early erythroblasts with the use o

36 he majority of microRNAs (miRNAs) present in CFU-E erythroid progenitors are down-regulated during te

37 er GM-CSF or IL-3 also leads to reduction in CFU-E numbers and hematocrits but does not significantly

38 o induce strong expression of a transgene in CFU-E stage cells.

39 mechanism by which glucocorticoids increase CFU-E formation.

40 mately 50% to 70% of BFU-E and 60% to 80% of CFU-E bound to the carboxy-terminal HBD and to the CS-1

41 e RGDS sequence in contrast to 75% to 85% of CFU-E.

42 BD alone, although they promoted adhesion of CFU-E, failed to support significant levels of migration

43 d self-renewal, which increases formation of CFU-E cells > 20-fold.

44 ured cells, is essential for the function of CFU-E progenitors.

45 .25 to 1 mmol/L, SNP inhibited the growth of CFU-E by 30% to 75%.

46 25 to 1 mmol/L, SNAP inhibited the growth of CFU-E by 33% to 100%.

47 E and CFU-E, supported the highest levels of CFU-E migration (11-fold above background).

48 We measured migration of CFU-E on fragments of FN containing each cell binding re

49 escued BFU-E and yielded a greater number of CFU-E than observed with control.

50 Maximal numbers of CFU-E and burst-forming unit-erythroid were increased, a

51 The numbers of CFU-E increased modestly in the femur, and approximately

52 nic H-ras promotes abnormal proliferation of CFU-E progenitors and early erythroblasts and supports t

53 onic retroviral system, and their effects on CFU-E colony formation and erythroid differentiation wer

54 FLVCR is upregulated on CFU-E, indicating that heme export is important in prima

55 T-HSC, MPP, premegakaryocytic/erythroid, Pre CFU-E, Pre GM, MkP, and granulocyte-macrophage compartme

56 e erythropoiesis beyond the late progenitor (CFU-E) stage was drastically inhibited by the EpoR mutat

57 H-ras, not dominant-negative H-ras, reduced CFU-E colony formation.

58 and enhances the formation of Epo-sensitive CFU-E progenitors.

59 must have occurred in vivo before or at the CFU-E progenitor stage.

60 iesis failure occurs in these animals at the CFU-E/proerythroblast stage, a point at which the transf

61 rovide evidence that the transition from the CFU-E to erythroblasts is critically dependent on c-Myb

62 From the CFU-E/proerythroblast (CD71(+) Ter119(-) cells) stage on

63 se expression is confined exclusively to the CFU-E erythroid precursor cells, but not in mature eryth

64 )CD105(+)CD36(+) cells as LEP giving rise to CFU-E, in a hierarchical fashion.

65 ent growth of erythroid colony-forming unit (CFU-E) and erythropoietin hypersensitivity, and Southern

66 conventional erythroid colony-forming unit (CFU-E) assays have been obtained concerning the role of

67 t (BFU-E) and erythroid colony-forming unit (CFU-E) numbers only in the adult bone marrow of the trip

68 that enhance erythroid colony-forming unit (CFU-E) production, we studied the mechanism by which glu

69 re, EpoR(+/-) erythroid colony-forming unit (CFU-E) progenitors are reduced both in frequency and in

70 myb knockdown erythroid colony-forming unit (CFU-E) stage progenitors displayed an immature phenotype

71 unit (BFU-E), erythroid colony-forming unit (CFU-E), and colony-forming unit-granulocyte macrophage-e

72 development (erythroid colony-forming unit [CFU-E] stage).

73 progenitors (erythroid colony-forming unit [CFU-E]) are reduced in p85alpha-/- fetal livers compared

74 progenitors (erythroid colony-forming unit [CFU-E]) were reduced in K-Ras(-/-) fetal livers compared