ライフサイエンスコーパス: CHO cell

1 lular assay overexpressing human arginase I (CHO cells).

2 ere measured in human receptors expressed in CHO cells.

3 ed at high levels as a secreted protein from CHO cells.

4 interacts with LYVE-1 when overexpressed in CHO cells.

5 e biosynthetic enzymes to produce heparin in CHO cells.

6 anced by the expression of H type II HBGA in CHO cells.

7 the lack of a unifying genomic resource for CHO cells.

8 activation of SM synthesis in OSBP-deficient CHO cells.

9 rotein, as well as Thy-1(+)-activated EC and CHO cells.

10 an observed in mtDNA isolated from wild-type CHO cells.

11 YFP-coupled LH receptors stably expressed on CHO cells.

12 und to the SOAT1 protein prepared from SOAT1-CHO cells.

13 the G(1196)|G(1197) dipeptide in transfected CHO cells.

14 o the nsp2 product identified in transfected CHO cells.

15 IC2 fragments were detected in oocytes or in CHO cells.

16 teins were expressed transiently in PC12 and CHO cells.

17 in hagfish plasma and in stably transfected CHO cells.

18 AM17 activity in both murine fibroblasts and CHO cells.

19 ells expressing EGFR and quiescent wild-type CHO cells.

20 five residues in GH4C1 pituitary cells as in CHO cells.

21 ntact ER fractions prepared from SOAT1/SOAT2-CHO cells.

22 t mediates efficient processing of nsp2-3 in CHO cells.

23 by B. thailandensis in transfected HEK293 or CHO cells.

24 AT1 in a cell-based assay using SOAT1-/SOAT2-CHO cells.

25 decreased CatL activity in integrin-negative CHO cells.

26 ant human erythropoietin (rhEPO) produced in CHO cells.

27 M) using flow cytometry of P2Y14R-expressing CHO cells.

28 rms diffusing in the plasma membrane of live CHO cells.

29 maintained their activity on CMG2-expressing CHO cells.

30 II, and sLRP1-IV) recombinantly expressed in CHO cells.

31 ysiological properties of Kv2.1 expressed in CHO cells.

32 embrane patches from transiently transfected CHO cells.

33 of cytoplasmic CRT and R-CRT in NIH 3T3 and CHO cells.

34 h effector functions superior to PG9 made in CHO cells.

35 s and inhibits Kv7.4 currents in transfected CHO cells.

36 he plasma membrane of Chinese hamster ovary (CHO) cells.

37 kidney (HEK 293) and Chinese hamster ovary (CHO) cells.

38 d in tyrosine-limited Chinese hamster ovary (CHO) cells.

39 (rHuEPO) expressed in Chinese hamster ovary (CHO) cells.

40 ovary cells that express alphavbeta3 (beta3-CHO) cells.

41 ioreactors, including Chinese Hamster Ovary (CHO) cells.

42 antibody expressed in Chinese hamster ovary (CHO) cells.

43 ae were isolated from Chinese hamster ovary (CHO) cells.

44 growth inhibition in Chinese hamster ovary (CHO) cells.

45 w cytotoxicity toward Chinese hamster ovary (CHO) cells.

46 cancer cell lines and Chinese hamster ovary (CHO) cells.

47 (cAMP) production by Chinese hamster ovary (CHO) cells.

48 are reconstituted in Chinese hamster ovary (CHO) cells.

49 opically expressed in Chinese hamster ovary (CHO) cells.

50 n (EGFP) plasmid into Chinese hamster ovary (CHO) cells.

51 cell lines including Chinese hamster ovary (CHO) cells.

52 e generated rhC7 from Chinese hamster ovary (CHO) cells.

53 (FR) alpha-expressing Chinese hamster ovary (CHO) cells.

54 almost identical effect on alpha2-expressing CHO cell adhesion to collagen I, but only BTT-3033 block

55 at least 80% knockdown of a GFP reporter in CHO cells after 72h.

56 EGFR expressed in CHO cells also became activated in quiescent cells but n

57 of RNA copy number in both HT-1080 cells and CHO cells also suggests that RBMBs can be used to image

58 orter activity to 3.6%+/-0.3% of baseline in CHO cells and 16%+/-3% in myocytes (both P<0.05), and mu

59 added exogenously is rapidly internalized by CHO cells and accumulates in nuclei in an NLS-dependent

60 localized to mitochondria when expressed in CHO cells and behaves as a peripheral membrane protein,

61 ovides valuable insights into translation in CHO cells and can guide efforts to enhance protein produ

62 steine-substituted mutants were expressed in CHO cells and covalently labeled with the sulfhydryl-rea

63 Only DCs bound PSGL-1 CHO cells and did so in a PSGL-1-blocking antibody-inhib

64 ted HS3st1 is broadly distributed throughout CHO cells and forms no detectable AT-binding sites, wher

65 Using transfected CHO cells and gene-targeted mice, we observed that CL40

66 In study 2, gD2 was produced in CHO cells and given intramuscularly with monophosphoryl

67 between P11L and G170R in stably transformed CHO cells and have studied for the first time whether a

68 exhibited prolonged transgene expression in CHO cells and HSCs up to 10 days and 14 days, in the res

69 1, CysLT2 and P2Y12 overexpressed in HEK293, CHO cells and human platelets were used and responsivene

70 cyclic AMP production in beta2AR-transfected CHO cells and induced potent dilation of isolated rat cr

71 e" effects over native hERG channels in both CHO cells and mouse atrial-derived HL-1 cells.

72 e rank order was similar for genotoxicity in CHO cells and mutagenicity in S. typhimurium, the Salmon

73 vity to 62%+/-4% and 57%+/-2% of baseline in CHO cells and myocytes, respectively (both P<0.05).

74 einizing hormone (LH) receptors expressed on CHO cells and native LH receptors on both KGN human gran

75 ly labeled lipoproteins in ILDR1-transfected CHO cells and release of CCK from isolated intestinal ce

76 of nonintegrin, non-HS receptors for FMDV on CHO cells and revealed a novel, non-RGD-dependent use of

77 nts were defective in Yop translocation into CHO cells and splenocyte-derived neutrophils and macroph

78 rified from wild-type Chinese hamster ovary (CHO) cells and HepG2 cells converted dUMP to dTMP in the

79 (Platyhelminthes), in Chinese hamster ovary (CHO) cells and use fluorescence-based assays to examine

80 age endogenous H2S in Chinese hamster ovary (CHO) cells and use the developed constructs to report on

81 ions of Q1 and E1 Cys-substituted mutants in CHO cells, and determined the extents of spontaneous dis

82 CHO Eogt reduced binding of CTD110.6 to Lec1 CHO cells, and expression of a human EOGT cDNA increased

83 Thy-1(+)-activated human dermal EC, Thy-1(+) CHO cells, and immobilized Thy-1 protein.

84 increased upon metaphase arrest in COS-1 and CHO cells, and in a pancreatic beta cell line that expre

85 ated complement activation on the surface of CHO cells, and it protected complement-sensitive intrace

86 recipitated with PP2A upon transfection into CHO cells, and PP2A/Aalpha knockdown recapitulated the i

87 ing to SRs was confirmed using SR-expressing CHO cells, and this binding was blocked by competitive i

88 ins were expressed in Chinese hamster ovary (CHO) cells, and their expression level on the cell surfa

89 CHO cell- and E. coli-derived rhBMP-2 in an ACS carrier

90 CHO cell- and E. coli-derived rhBMP-2 supported comparab

91 use retina, 2. Labeling of TRPM1-transfected CHO cells; and 3. Attenuation of the ERG b-wave followin

92 ore virologically relevant T lymphocytes, in CHO cells; and purifying Env with different chromatograp

93 rn analysis, HAT-L4 expressed in transfected CHO cells appeared as a 48-kDa protein.

94 While parental Chinese hamster ovary (CHO) cells are permissive for both strains, neither stra

95 Chinese hamster ovary (CHO) cells are widely used for the production of biother

96 d the ability to infect CHO and HS-deficient CHO cells as a result of a single glutamine (Q)-to-lysin

97 as quantified using a Chinese hamster ovary (CHO) cell assay, and the descending rank order for cytot

98 fering RNA (siRNA) in Chinese hamster ovary (CHO) cells augments M3-MR signaling.

99 e Rasa3 in integrin alphaIIbbeta3-expressing CHO cells blocked Rap1 activity and integrin alphaIIbbet

100 82) results in efficient infection of mutant CHO cells but a decrease in heparin binding, whereas Arg

101 mutation (RRR to AAA) was able to penetrate CHO cells but did not translocate to the nucleus.

102 ts or suspended alpha(IIb)beta(3)-expressing CHO cells but is recruited to integrin during cell adhes

103 ficantly reduced activity on TEM8-expressing CHO cells but maintained their activity on CMG2-expressi

104 when transfected into Chinese hamster ovary (CHO) cells but, surprisingly, exerted "chaperone-like" e

105 ion of 14-3-3beta increased ENaC activity in CHO cells, but concomitant expression of beta1Pix attenu

106 so resulted in an acquired ability to infect CHO cells by type O and Asia-1 FMDV.

107 solution images of lipid-binding proteins on CHO cells can be acquired with NanoSIMS imaging.

108 he therapeutic efficacy of rhEPO produced in CHO cells can be improved by shunting intracellular CMP-

109 e hamster genome as the reference upon which CHO cells can be studied and engineered for protein prod

110 In CHO cells, CDK1/2-dependent phosphorylation of Ubc2/Rad6

111 CHO cell chronic cytotoxicity tests indicate that chlori

112 In Chinese hamster ovary (CHO cells), coexpression of rat Oatp1a1 and human ABCG2

113 tibodies from typical contaminants including CHO cell conditioned media, ascites fluid, DNA, and othe

114 somes, and inhibited the proliferation of WT CHO cells, confirming that it is an essential protein fo

115 ered efficiently into Chinese hamster ovary (CHO) cells constitutively expressing HSV-1 human recepto

116 This protective response in CHO cells correlated with the ability of the receptor to

117 was implemented for monitoring variations in CHO cell culture media upon exposure to high temperature

118 FN-gamma) produced in Chinese hamster ovary (CHO) cell culture.

119 focus here is on the extracellular milieu of CHO cell cultures, this methodology is generally applica

120 xture extracted from HTL-WW expressed potent CHO cell cytotoxic activity, with a LC(50) at 7.5% of HT

121 en represent those most commonly reported on CHO cell derived therapeutic antibodies.

122 ived rhBMP-2 displays comparable efficacy to CHO cell-derived rhBMP-2 in vitro and in small-animal mo

123 li-derived rhBMP-2 compared to the benchmark CHO cell-derived rhBMP-2 using an established large-anim

124 Chinese hamster ovary (CHO) cell-derived recombinant human bone morphogenetic p

125 rain efficiently bound to or infected mutant CHO cells devoid of GAG expression.

126 elemin-like protein present in platelets and CHO cells does not associate with alpha(IIb)beta(3) in r

127 pling to cAMP production in HEK293 cells and CHO cells ectopically expressing the receptor and in Mad

128 hly optimized growth conditions, recombinant CHO cells engineered to produce a model human IgG clone

129 content cell-based assay in the presence of CHO cells engineered with GFP-PDK1.

130 CHO cells express two RGD-binding integrins (alpha5beta1

131 In addition, when CHO cells expressed gamma318, gamma323, or gammaFL with

132 light chain (LC) of a Chinese hamster ovary (CHO) cell-expressed monoclonal antibody (mAb).

133 and did not increase further in MV-infected CHO cells expressing >/=4,620 CD46 copies/cell, there wa

134 CHO cells expressing CD36 with mutated Lys-164 had impai

135 in DAT-positive immortalized DA neurons and CHO cells expressing DAT decreased the magnitude and rat

136 genic death compared with both proliferating CHO cells expressing EGFR and quiescent wild-type CHO ce

137 ing [(35)S]GTPgammaS binding was assessed in CHO cells expressing either human D2 or D3 receptors.

138 interact with itself in trans by binding to CHO cells expressing full-length LINGO-1.

139 T6, and JAK3 phosphorylation was observed in CHO cells expressing gamma323 and gammaFL but not in gam

140 occurred for K(+) currents representing IKr (CHO cells expressing hERG; IC50=219+/-21 mumol/L) and IK

141 ac and McK(gKDelta31-68) viruses entered all CHO cells expressing HSV-1 receptors via a pH-independen

142 000 synthetic small molecules was done using CHO cells expressing human AQP4 and a human NMO recombin

143 c binding to beta2AR in H9c2 cardiomyocytes, CHO cells expressing human beta2AR, and rat aorta.

144 CHO cells expressing human CD36 were SSO-treated, and th

145 capsaicin and acid-induced calcium influx in CHO cells expressing human TRPV1.

146 r into CHO-PILRalpha cells, while it entered CHO cells expressing HVEM and nectin-1 more efficiently

147 In CHO cells expressing integrin alphaIIbbeta3, co-expressi

148 essing hERG; IC50=219+/-21 mumol/L) and IKs (CHO cells expressing KCNQ1+KCNE1; IC50=184+/-12 mumol/L)

149 Here, using CHO cells expressing mGlu1a receptors, we show that the

150 X-1, whereas little increase was observed in CHO cells expressing only LOX-1.

151 CHO cells expressing rbMate1 truncated at residue Gly-54

152 nhibitor of icilin-induced calcium influx in CHO cells expressing recombinant rat TRPM8.

153 Membrane vesicles from stably transfected CHO cells expressing recombinant SMDR2 show significant

154 Using CHO cells expressing the MR, we confirmed that the GPLs

155 Furthermore, when GalNAc-T3 was expressed in CHO cells expressing the MUC2 C terminus, the second thr

156 ciation (k(off)) of prebound (125)I-TSH from CHO cells expressing the TSH holoreceptor.

157 ranslocation assay in Chinese hamster ovary (CHO) cells expressing an NFkappaB-EGFP reporter.

158 -EA to membranes from Chinese hamster ovary (CHO) cells expressing either recombinant human CB1 or CB

159 e cardiac myocytes or Chinese hamster ovary (CHO) cells expressing the mouse or human subunits.

160 om both E. coli and Chinese hamster ovaries (CHO) cell expression platforms; however, isotopic labeli

161 gp120-alpha4beta7 interactions similarly to CHO cell fibronectin.

162 CHO cell fibronectins were able to mediate the binding o

163 Chinese hamster ovary (CHO) cells, first isolated in 1957, are the preferred pr

164 membrane of isolated Chinese Hamster Ovary (CHO) cells following exposure to nsPEF.

165 oxP site of the alphoid(tetO)-HAC in hamster CHO cells from where the HAC may be MMCT-transferred to

166 profiles observed offer potential to direct CHO cell function during culture through medium design a

167 parental CHO cells over the course of three CHO cell generations.

168 Chlorination of wastewaters produced CHO cell genotoxicity comparable to chloramination, 3.9

169 The estimated demand of NSs towards CHO cell glycosylation can be used to rationally design

170 tric coefficients for NS consumption towards CHO cell glycosylation.

171 lipids to estimate the demand of NSs towards CHO cell glycosylation.

172 eptors in the membrane of stably transfected CHO cells has been observed.

173 cosylation in multiple cell lines, including CHO cells, HeLa cells, normal and patient fibroblasts, i

174 targeted by AKAP79 in Chinese hamster ovary (CHO) cells heterologously expressing KCNQ1-5 subunits an

175 o cell lines and to primary cells, including CHO cells, hiPSC-CMs, and human astrocytes derived in 3D

176 er gene expression in Chinese Hamster Ovary (CHO) cells, Human Immortalized Myelogenous Leukemia (K56

177 hen Env trimers are truncated or produced in CHO cells.IMPORTANCE A protective HIV-1 vaccine will lik

178 lating cAMP accumulation in receptor-bearing CHO cells in a concentration-dependent manner.

179 CdtA subunit remains on the cell surface of CHO cells in association with cholesterol-containing and

180 s stably expressed in Chinese hamster ovary (CHO) cells increased AMPK activity and AMPK phosphorylat

181 Parallel studies in transfected CHO cells indicate that Kv1.1, Kv1.2, Kv7.2/7.3, and mBK

182 d epitopes Lewis X and sialylated Lewis X in CHO cells, indicating competition with Slc35c1.

183 d the genotoxicity to Chinese hamster ovary (CHO) cells induced by municipal secondary wastewater eff

184 residue of most repeats, which in wild-type CHO cells is glycosylated with the typical sialylated co

185 In CHO cells, Kv1.1, Kv1.2, and Kv7.2/7.3 K(+) currents wer

186 ein Dendra2 in epithelial A-431 cells and in CHO cells lacking endogenous cadherin.

187 i expression system or from an overexpressed CHO cell line (disulfide scrambling), is often a great c

188 be expressed from this construct in a stable CHO cell line and purified at an acceptable yield using

189 2A peptides to generate a stably transfected CHO cell line constitutively secreting high levels of re

190 trates, the expression of the Fc domain in a CHO cell line in the presence of an alpha-mannosidase in

191 We designed a CHO cell line overexpressing the London mutated betaAPP

192 Then, we established a stable CHO cell line that produces rhEPO in the context of repr

193 Conversely, virions derived from a CHO cell line that was engineered to overexpress human C

194 Samples of r-alpha hCG obtained from a CHO cell line were also analyzed and briefly shown to be

195 produced in stable cell lines derived from a CHO cell line with titers over 1 g/L.

196 onsible for the repair defects of the EM-C11 CHO cell line, caused protein instability; a V86R mutati

197 w of protein translation in an IgG-producing CHO cell line, measured with ribosome profiling.

198 or protein (APP) in a Chinese hamster ovary (CHO) cell line by cleaving APP at the alpha-secretase si

199 of an IgG1-producing Chinese hamster ovary (CHO) cell line for 18-25 days.

200 forebrain neurons when they are cultured on CHO cell lines expressing DIgLON:CEPU-1-OBCAM and DIgLON

201 We prepared CHO cell lines expressing nonfluorescent halves (YN and

202 resequenced and analyzed the genomes of six CHO cell lines from the CHO-K1, DG44 and CHO-S lineages.

203 Although genetic heterogeneity among CHO cell lines has been well documented, a systematic, n

204 Using CHO cell lines stably expressing human ABCA1 or ABCG1, w

205 Here we used wild-type and mutant CHO cell lines with alterations in tubulin to demonstrat

206 dentified hamster genes missing in different CHO cell lines, and detected >3.7 million single-nucleot

207 ins to citrullinated collagen was studied by CHO cell lines, each overexpressing 1 of the 4 human col

208 A protein kinase catalytic subunit-deficient CHO cell lines, which are defective for the nonhomologou

209 sphatidylcholine (LPC) compared to SP2/0 and CHO cell lines.

210 lycan (GAG)-deficient Chinese hamster ovary (CHO) cell lines and soluble GAGs, we found that an argin

211 tion assays on mutant Chinese hamster ovary (CHO) cell lines defective in various stages of glycan ch

212 expressed in a set of Chinese hamster ovary (CHO) cell lines under conditions of full glycosylation,

213 racterized a panel of Chinese hamster ovary (CHO) cell lines with inducible transgenes encoding tagge

214 For Chinese Hamster ovary (CHO) cell lines, key indicators of metabolic events link

215 In contrast to the whole CHO cell lysate which contained a variety of fatty acids

216 P2Y(12) variant showed surface expression in CHO cells, markedly reduced binding to [(3)H]2MeS-ADP, a

217 e attachment were significantly decreased in CHO cell mutants defective in glycosaminoglycan synthesi

218 ntibodies produced in Chinese hamster ovary (CHO) cells often exhibit a slight yellow-brown color, bu

219 integrin alphaIIbbeta3-mediated spreading of CHO cells on fibrinogen.

220 luorescently labeled PSA or PSA-NCAM to live CHO cells or hippocampal neurons expressing MARCKS as a

221 tic cell immunoreceptor (DCIR) constructs in CHO cells or on DCs were examined by using fluorescent m

222 lt male Hound Labrador mongrel dogs received CHO cell- or E. coli-derived rhBMP-2 (0.2 mg/mL) in an a

223 e doxycycline-induced cells or from parental CHO cells over the course of three CHO cell generations.

224 invading and damaging endothelial cells and CHO cells overexpressing GRP78.

225 ibits glucagon-stimulated cAMP production in CHO cells overexpressing the human glucagon receptor wit

226 CHO cells overexpressing the human insulin receptor and

227 A production is enhanced more than 2-fold by CHO cell overexpression of integrin beta(3).

228 Inside living CHO cells, PhDY-Chol mimics the behavior of cholesterol,

229 CHO cells preadapted to oxidative stress resist the effe

230 In semi-intact SOAT1-/SOAT2-CHO cells prepared by a treatment with digitonin (plasma

231 integrin activation, ATX generates LPA from CHO cells primed with bee venom phospholipase A(2), and

232 unction, and overexpression of N-cadherin in CHO cells promoted barrier function.

233 Expression of M3R in CHO cells promoted plasma membrane localization of YFP-P

234 N-linked and O-GalNAc glycosites across the CHO cell proteome, and have derived stoichiometric coeff

235 gamma transactivation activities in a Tet-on CHO cell reporter system, RORalpha co-activator assays a

236 However, LRP6 knockdown in wild type CHO cells resulted in a much greater decline in LDL bind

237 dase-treated or sialic acid-deficient mutant CHO cells revealed a 3-15-fold increase in relative bind

238 itu proximity ligation assay (PLA) assays in CHO cells revealed the presence of cell-surface complexe

239 lity of this approach through an analysis of CHO cell samples.

240 (50) values determined on human recombinant (CHO) cells showed very similar inhibitory activities alb

241 in as well as elevated enzymatic activity in CHO cells stably expressing CGalT.

242 CHO cells stably expressing human CD36 released severalf

243 In parallel with these results, CHO cells stably expressing M(1) receptors studied under

244 SR48692 (5a) using a FLIPR calcium assay in CHO cells stably expressing rat NTS2.

245 CHO cells stably expressing rbSGLT1 were probed by using

246 forskolin-stimulated accumulation of cAMP in CHO cells stably expressing the CB2 receptor (IC(50) = 9

247 Knockdown of SET by siRNA in CHO cells stably expressing the M3-MR did not alter agon

248 forded complement resistance, we grew NDV in CHO cells stably transfected with CD46 or HeLa cells, wh

249 Chinese hamster ovary (CHO) cells stably expressing a Tet-on RORalpha or RORgam

250 ) 293, C6 glioma, and Chinese hamster ovary (CHO) cells stably expressing this receptor.

251 In CHO cells, stably expressing alphaIIbbeta3 integrins, vi

252 Moreover, expression of FcgammaRI in CHO cells strikingly enhanced the sensitivity of these c

253 ty to coactivate integrin alphaIIbbeta3 in a CHO cell system when coexpressed with talin.

254 ay yield organics with lower genotoxicity to CHO cells than chlorine-based disinfection.

255 romoters for biopharmaceutical production in CHO cells that exhibited precisely designed activity dyn

256 PU-1-OBCAM and DIgLON:CEPU-1-LAMP but not on CHO cells that express single IgLONs CEPU-1 or OBCAM.

257 iminished in mitochondria isolated from glyA CHO cells that lack SHMT2 activity, as well as mitochond

258 AV2.5T binds but fails to be internalized by CHO cells that lack surface expression of sialic acid.

259 CHO cells that overexpress exogenous GULP (FL) demonstra

260 In mutant Chinese hamster ovary (CHO) cells that do not add galactose (Gal) to the GlcNAc

261 hibited the growth of Chinese hamster ovary (CHO) cells that expressed FRs but not the reduced folate

262 lex N-glycans of Lec8 Chinese hamster ovary (CHO) cells that lack UDP-Gal transporter activity and ex

263 In CHO cells, the AMPK activator 5-aminoimidazole-4-carboxa

264 In trigeminal neurons and CHO cells, the manipulation of cellular PIP(2) led to si

265 hat integrates the reported glycoproteome of CHO cells, the number of N-linked and O-GalNAc glycosyla

266 In CHO cells, this coincident signaling mechanism is involv

267 regulated kinases 1 and 2 (ERK1/2) in intact CHO cells to identify potential agonistic effects as wel

268 As part of an effort to bioengineer CHO cells to produce heparin, we previously showed that

269 by GnT1IP markedly increases the adhesion of CHO cells to TM4 Sertoli cells.

270 acute genotoxicity in Chinese hamster ovary (CHO) cells to compare the toxicity of analogous N-nitros

271 Similarly, CHO cells transfected with the full-length bovine beta(3

272 CHO cells transiently transfected with 24p3R or distal t

273 well as mitochondria isolated from wild-type CHO cells treated with methotrexate, a DHFR inhibitor.

274 When added to CHO cells, U-X crosslinked to NPC1.

275 We show that Chinese hamster ovary (CHO) cells used to express recombinant gp120 produced fi

276 of-function screen in Chinese hamster ovary (CHO) cells using insertional mutagenesis to identify gen

277 as uracil levels in mtDNA isolated from glyA CHO cells was 40% higher than observed in mtDNA isolated

278 rTNF-alpha3 produced in CHO cells was bioactive in different cell lines and prim

279 The oligomeric nature of Gi1 in live CHO cells was demonstrated by means of Forster resonance

280 fluorescent labeling of microtubules in live CHO cells was demonstrated with a long-wavelength photoa

281 we show that DLL1-induced NOTCH signaling in CHO cells was enhanced by LFNG, but this did not occur i

282 data for specific growth and productivity of CHO cells, we observe that the demand of NSs towards gly

283 nsfer with functional receptors expressed in CHO cells, we show that the cleft of the amino-terminal

284 d 9-13 toward FRalpha- and FRbeta-expressing CHO cells were only partly reflected in binding affiniti

285 R profiles might affect macrophage function, CHO cells were transfected with SR-AI/II, and phagocytos

286 blood cells (RBC), or Chinese hamster ovary (CHO) cells, were immobilized on the inside of the flow c

287 e reporter fusions in Chinese hamster ovary (CHO) cells, where the putative cis element required for

288 SV5 particles generated in CHO cells, which do not express CD46, did not have cofac

289 ssion, we transfected Chinese hamster ovary (CHO) cells, which lack EGFR expression, with EGFR expres

290 ocedure to vesiculate Chinese hamster ovary (CHO) cells, widely used for the expression of recombinan

291 Initially, we found that treating CHO cells with a potent inhibitor of GSL biosynthesis in

292 s, since it occurs even in erbB3-transfected CHO cells with disproportionally small amounts of erbB2.

293 In CHO cells with low levels of Slc35c2, both Delta1- and J

294 ates ENaC activity, we reconstituted ENaC in CHO cells with or without coexpressed beta1Pix and found

295 tivity when ENaC subunits are coexpressed in CHO cells with PPARgamma.

296 FULL LENGTH (FL)) co-expressed in L-cells or CHO cells with wild-type (WT) IL-4Ralpha.

297 ent of fibroblasts or Chinese hamster ovary (CHO) cells with 25OH caused a 50-70% reduction in Golgi-

298 G protein by 350% in Chinese hamster ovary (CHO) cells with genetically induced expression of AT1 an

299 rvey of bioengineered Chinese Hamster Ovary (CHO) cells with knock-in/out enzymes involved in protein

300 Treating Chinese hamster ovary (CHO) cells with monoHANs followed by the release from th