戻る
「早戻しボタン」を押すと検索画面に戻ります。

今後説明を表示しない

[OK]

コーパス検索結果 (left1)

通し番号をクリックするとPubMedの該当ページを表示します
1                                              CLA and CNA significantly reduced body weight and fat ma
2                                              CLA and CNA significantly reduced serum leptin and tumou
3                                              CLA and OCS act non-additively to activate atNHE1, indic
4                                              CLA attenuated the increase in BMI (0.5 +/- 0.8) compare
5                                              CLA exerts its anti-carcinogenic effect by reducing VEGF
6                                              CLA expressed on CD43 was found exclusively on the high-
7                                              CLA has a wide range of biological effects, including bo
8                                              CLA significantly increased bone markers without major c
9                                              CLA supplementation for 7 +/- 0.5 mo decreased body fatn
10                                              CLA supplementation significantly prevented ovariectomy-
11                                              CLA treatment increased serum parathyroid hormone (PTH)
12                                              CLA was microencapsulated by spray drying in ten varied
13                                              CLA(+) is expressed on the surface of circulating CD45RO
14                                              CLA(+) TH2 T cells were markedly expanded in both childr
15                                              CLA+ and alpha4beta7+ memory T cells were isolated and c
16                                              CLA+ CD43 purified from human T cells supported tetherin
17                                              CLA-rich eggs and soy control eggs were produced by addi
18                                              CLA-yolk mayonnaise was more viscous, had greater storag
19                                              CLA/ESL mediates adhesion of T cells to inflamed vascula
20                                              CLA:M:PPC (1:1:3) microparticles demonstrated better mor
21                                              CLA:M:PPC (1:1:3) showed the most promising results, thu
22 had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+) Th22 (P < .05) frequenc
23                    CD36 and LIMPII analog 1, CLA-1, and its splicing variant, CLA-2 (SR-BI and SR-BII
24 e members of the SR-B family, namely, CLA-1, CLA-2, and CD36, mediate recognition of bacteria not onl
25 t bacterial uptake that is enhanced by CLA-1/CLA-2 overexpression.
26  cytosolic accumulation of bacteria in CLA-1/CLA-2-overexpressing HeLa cells.
27 soy control eggs were produced by adding 10% CLA-rich soy oil or 10% of control unmodified soy oil to
28 from mountain areas showed average c-9, t-11 CLA content higher than those from prairie districts.
29 ilk yogurts showed lower values of c-9, t-11 CLA content on lipid basis compared to full-fat yogurts.
30 roducts naturally enriched in cis-9,trans-11 CLA (and trans-11 18:1) on the blood lipid profile, the
31  dairy products enriched with cis-9,trans-11 CLA and trans-11 18:1 did not significantly affect body
32 ducts naturally enriched with cis-9,trans-11 CLA and trans-11 18:1 do not appear to have a significan
33 erse association between the cis-9, trans-11 CLA in adipose tissue and diabetes risk is consistent wi
34                          The cis-9, trans-11 CLA isomer was associated with a lower risk of diabetes.
35 determine the association between the 9c,11t-CLA isomer in adipose tissue and risk of MI.
36                        Adipose tissue 9c,11t-CLA was associated with a lower risk of MI in basic and
37                                       9c,11t-CLA, which is present in meaningful amounts in the milk
38                         The trans-10, cis-12 CLA isomer was not detected in adipose tissue.
39     The cis-9, trans-11 and trans-10, cis-12 CLA isomers in adipose tissue and 48 other fatty acids w
40 nflammatory proteins were increased by 10,12-CLA compared with bovine serum albumin vehicle in the ad
41 ns-10,cis-12-conjugated linoleic acid (10,12-CLA) activate the inflammatory signaling that promotes i
42                                        10,12-CLA-induced IL-8, IL-6, IL-1beta, and COX-2 mRNA levels
43 ized that they played the same role in 10,12-CLA-mediated inflammation.
44            Consistent with these data, 10,12-CLA-mediated secretions of IL-8 and IL-6 from AD50 cultu
45 d the inflammatory capacity of CM from 10,12-CLA-treated cultures.
46               We conclude that the CHO-131(+)CLA(+) T cell subset is enriched in P-selectin binding c
47 mory cell markers, CHO-131(+) and CHO-131(-) CLA(+) T cells have an overlapping skin-tropic and memor
48 inding capacity of CHO-131(+) and CHO-131(-) CLA(+) T cells revealed a significantly greater P-select
49 te supplementation in rodents elevates NO(2)-CLA levels in plasma, urine, and tissues, which in turn
50 erexpression abrogated cell surface HECA-452/CLA expression, reduced the number of rolling leukocytes
51 specially formulated diet consisting of 0.5% CLA for 24 days.
52 re randomly assigned to receive 3 g/d of 80% CLA (50:50 cis-9,trans-11 and trans-10,cis-12 isomers) o
53  with the phosphatase inhibitor calyculin-A (CLA) increase Na(+) transport capacity without affecting
54                    Conjugated linoleic acid (CLA) and conjugated nonadecadienoic acid (CNA) have been
55 imental studies on conjugated linoleic acid (CLA) and insulin regulation suggested that CLA could be
56 of 2 dietary oils, conjugated linoleic acid (CLA) and safflower oil (SAF), on body weight and composi
57 ch in trans, trans conjugated linoleic acid (CLA) are significantly more viscous, have more phospholi
58 urally enriched in conjugated linoleic acid (CLA) exists.
59                    Conjugated linoleic acid (CLA) has been shown to be an effective supplement for re
60 n for the elderly; conjugated linoleic acid (CLA) has been shown to improve overall bone mass when ca
61                    Conjugated linoleic acid (CLA) has the unique property of inducing regression of p
62 tes the effects of conjugated linoleic acid (CLA) in preventing bone loss, using an ovariectomised mo
63  Many studies with conjugated linoleic acid (CLA) indicate that it has a protective effect against ma
64                    Conjugated linoleic acid (CLA) is a family of positional and geometric isomers wit
65                    Conjugated linoleic acid (CLA) is a supplemental dietary fatty acid that decreases
66                    Conjugated Linoleic Acid (CLA) is fatty acid found endogenously in food sources th
67            Herein, conjugated linoleic acid (CLA) is identified as the primary endogenous substrate f
68 GC-FID), including conjugated linoleic acid (CLA) isomeric profile (Ag(+)-HPLC), and nutritional valu
69  identification of conjugated linoleic acid (CLA) isomers has been developed in which silver ion liqu
70 e development of a conjugated linoleic acid (CLA) oil-in-water beverage emulsion containing acacia gu
71 n the synthesis of conjugated linoleic acid (CLA) partial glycerides, which presented nutraceutical p
72 sity is to consume conjugated linoleic acid (CLA) supplements containing isomers cis-9, trans-11 and
73 aturated FA (UFA), conjugated linoleic acid (CLA), n-3 FA, and C18:1cis9 to C16 ratio.
74 ty acids including conjugated linoleic acid (CLA), polyunsaturated fatty acids C18:2(n-6) and C18:3(n
75 ration products of conjugated linoleic acid (CLA).
76 the main source of conjugated linoleic acid (CLA; 18:2n-7t), which is produced by the ruminal biohydr
77 id, docosapentaenoic acid, arachidonic acid, CLA:9c11t and gamma linolenic acid.
78 g more n-3 FA and conjugated linoleic acids (CLA) than conventionally produced dairy products.
79  either a study of skin lesions or activated CLA(+) T-cell subsets in peripheral blood.
80 ho were receiving either 4 g/d of 78% active CLA isomers (3.2 g/d: 39.2% cis-9,trans-11 and 38.5% tra
81                     Chaperone-like activity (CLA) was evaluated by measuring the ability of alpha-cry
82 nd high levels of the skin-homing addressins CLA, CCR4, and CCR6.
83 ed to as cutaneous lymphocyte-associated Ag (CLA) T cells) correlates with E-selectin binding, yet wh
84 % of Treg expressed cutaneous lymphocyte Ag (CLA) and 73% expressed CCR6.
85 e not only the CLnA concentrations, but also CLA in egg-yolk lipids.
86  .001]), with less significant effects among CLA(+) T cells (IL-22: 11 vs 7.5, P = .04).
87 uencies, which were highly significant among CLA(-) cells (IL-22: 3.7 vs 1.7 [P < .001] and IL-17: 1.
88  group than in the PCV group (P = .014); and CLA was expressed more frequently in the pneumonia group
89 -) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+) Th22 (P < .05) frequencies than controls.
90 7pA) competed with E. coli K12 for CLA-1 and CLA-2 binding.
91 l adhesion and cytosolic invasion, CLA-1 and CLA-2 may play an important role in infection and sepsis
92  and replicate intracellularly in CLA-1- and CLA-2-overexpressing HeLa, but both L-37pA and D-37pA pr
93                    In this study, CLA-1- and CLA-2-stably transfected HeLa and HEK293 cells demonstra
94  gut (alpha4beta7, CCR6) and skin (CCR10 and CLA).
95 d the skin-homing receptors CCR4, CCR10, and CLA and migrated in response to CCL17/CCL27.
96 n of other skin addressins (CCR6, CCR10, and CLA).
97 tin and CD27 but strongly expressed CCR4 and CLA, a phenotype suggestive of skin resident effector me
98 d CD8(+) T cells were compared in CLA(-) and CLA(+) populations.
99 aneous lymphocyte antigen (CLA)-positive and CLA(-) T-cell subsets in patients with AD and control su
100     Cutaneous lymphocyte-associated antigen (CLA(+) ) T cells are specialized for skin homing and rep
101 hat cutaneous lymphocyte-associated antigen (CLA), a functional E-selectin ligand (ESL), is selective
102 med cutaneous lymphocyte-associated antigen (CLA), a skin-homing receptor, than do circulating HSV-sp
103 ope cutaneous lymphocyte-associated antigen (CLA).
104 CD)4+, CD45RO+, cutaneous leukocyte antigen (CLA)+ T cells that homes to the skin.
105 oming markers, cutaneous lymphocyte antigen (CLA) and alpha4beta7 integrin, are used to determine whe
106 high levels of cutaneous lymphocyte antigen (CLA) and chemokine receptor (CCR)4.
107  expression of cutaneous lymphocyte antigen (CLA) in ILCs.
108 -selectin, and cutaneous lymphocyte antigen (CLA) on S. pneumoniae-specific plasmablasts was examined
109 s positive for cutaneous lymphocyte antigen (CLA) were increased fourfold in all CD4+ and CD8+ T cell
110        T-cell, cutaneous lymphocyte antigen (CLA)(+) and CCR4(+) T-cell and cytokine responses were s
111 rities between cutaneous lymphocyte antigen (CLA)(+) polarized T-cell subsets in children versus adul
112  contained few cutaneous lymphocyte antigen (CLA)(+) T cells, the cell type thought to provide cutane
113 CXCR3, but not cutaneous lymphocyte antigen (CLA), on circulating T cell subsets was associated with
114 ubsets in both cutaneous lymphocyte antigen (CLA)-positive and CLA(-) T-cell subsets in patients with
115 mir1-mediated resistance to corn leaf aphid (CLA; Rhopalosiphum maidis), a phloem sap-sucking insect
116          These molecules were functional, as CLA+ Treg showed CD62E ligand activity and demonstrable
117 ng small interfering NFkappaB p65 attenuated CLA suppression of glucose transporter 4 and peroxisome
118           We further demonstrated that blood CLA+ Treg inhibited CD4+CD25- T cell proliferation induc
119 ain emphasized the divergent effects of both CLA isomers on different pathways, but also revealed a l
120 yvitamin D3 concentration was not changed by CLA.
121 pendent bacterial uptake that is enhanced by CLA-1/CLA-2 overexpression.
122               Furthermore, foliar feeding by CLA rapidly sends defensive signal(s) to the roots that
123                                   Feeding by CLA triggers the rapid accumulation of mir1 transcripts
124       We report circulating skin-homing CD22+CLA+B cells in healthy volunteers and melanoma patients
125                                  Circulating CLA(+) T cells may be a reliable surrogate marker of the
126  PTDM (N = 24), the frequency of circulating CLA(+) (skin-homing) Tregs was decreased (1.53% vs 3.99%
127 e latest advancements reached on circulating CLA(+) in AD and the great potential they harbor in unde
128     Isomerization of cis,trans and trans,cis CLA to trans,trans isomers was observed mainly for the m
129 e significantly higher in the trans10, cis12-CLA group, whereas plasma triglyceride, NEFA, glucose, a
130 rast, the treatment effect of trans10, cis12-CLA was mainly explained by up-regulation of key enzymes
131 of CLA, cis9, trans11-CLA and trans10, cis12-CLA, affected lipid and glucose metabolism, as well as h
132            AA patients had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and CLA(+)
133  the analysis of CLA isomers in a commercial CLA supplement, milk fat, and the lipid extract from a L
134 single-isomer studies, and results comparing CLA isomers were inconclusive.
135  due to inflammatory signaling and considers CLA's linkage with lipogenesis, lipolysis, thermogenesis
136                  The mobile phase containing CLA isomers eluting from the Ag(+)-LC column flows throu
137 djustment to the median dose of 3.2 g CLA/d, CLA was effective and produced a reduction in fat mass f
138 001) CONCLUSION: Given at a dose of 3.2 g/d, CLA produces a modest loss in body fat in humans.
139        The aim of our study was to determine CLA's effects on energy expenditure, macronutrient utili
140       The aim of this study was to determine CLA's efficacy with regard to change in fat and body mas
141                                      Dietary CLA and nitrite supplementation in rodents elevates NO(2
142  intravital microscopy, we show that, during CLA-induced regression of pre-established atherosclerosi
143 on of 113 liver cytosolic proteins by either CLA isomer.
144  was moderate, and the proportion expressing CLA was low.
145 nzymatic and cellular mechanisms account for CLA nitration, including reactions catalyzed by mitochon
146 pA and D-37pA) competed with E. coli K12 for CLA-1 and CLA-2 binding.
147 esented describe a novel functional role for CLA in the regulation of monocyte adhesion, polarization
148 itioned medium (CM) model, CM collected from CLA-treated AD50 but not AD0 cultures induced IL-8 and I
149 After adjustment to the median dose of 3.2 g CLA/d, CLA was effective and produced a reduction in fat
150 y 0.128-1.501, 0.405-1.250 and 0.433-0.976 g CLA/100 g fat.
151 loss compared with placebo was -0.024 kg x g CLA(-1) x wk(-1) (P=0.03).
152  P-selectin glycoprotein ligand-1 glycoform "CLA," and CD43.
153 n was reduced during sleep in the CLA group (CLA: -3.3 +/- 2.6%; placebo: 0.3 +/- 5.7%).
154 ine secretion in the order of CD36 > CLA-2 &gt; CLA-1 in HEK293 cells.
155 of cytokine secretion in the order of CD36 &gt; CLA-2 > CLA-1 in HEK293 cells.
156       After only 3h, OM-RML gave the highest CLA conversion (54% at 40 degrees C with 1:3M ratio of g
157 nt groups and Th2 skewing in the skin-homing CLA+ cells of peanut allergic patients.
158 roliferation predominates in the skin-homing CLA+ subset, whilst peanut-tolerant groups have a mixed
159 aimed to compare frequencies of skin homing (CLA(+) ) vs systemic (CLA(-) ) "polar" CD4(+) and CD8(+)
160 tion markers and frequencies of skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4 and CD8 T-c
161 vealed cytosolic accumulation of bacteria in CLA-1/CLA-2-overexpressing HeLa cells.
162 g CD4(+) and CD8(+) T cells were compared in CLA(-) and CLA(+) populations.
163  those in control subjects, but decreases in CLA(+) TH1 T-cell numbers were greater in children with
164 ke in adults, no imbalances were detected in CLA(-) T cells from pediatric patients with AD nor were
165 ol subjects, with significant differences in CLA(+) T-cell numbers (P < .01).
166  with AD, with no significant differences in CLA(-) T-cell numbers.
167  to survive and replicate intracellularly in CLA-1- and CLA-2-overexpressing HeLa, but both L-37pA an
168 ming growth factor beta are each involved in CLA expression by memory HSV type 2 (HSV-2)-specific CD4
169 sn-1 mono and sn-1,3 diacylglycerols rich in CLA, with a ratio of sn-1,3/sn-1,2 regioisomers of 21.8,
170                    AA patients had increased CLA(+) /CLA(-) Th2 (P < .007), CLA(+) Tc2 (P = .04), and
171 ly, use of bifidobacteria slightly increased CLA relative content in the conventional fermented milks
172 g bacterial adhesion and cytosolic invasion, CLA-1 and CLA-2 may play an important role in infection
173                                 Mixed isomer CLA supplementation, but not placebo, positively altered
174  oxidative stability of conjugated linoleic (CLA) and linoleic (LA) acids in different chemical forms
175 g cis-9 trans-11, C18:2 conjugated linoleic (CLA-1.4 times), and alpha-linolenic acids (ALA-1.6 times
176       We recruited 23 subjects from our main CLA efficacy study who were receiving either 4 g/d of 78
177 od T cells expressing tissue homing markers (CLA, beta7, CD49d).
178                                   Meanwhile, CLA significantly reduced femur tartrate resistant acid
179  the sympathetic nervous system in mediating CLA's antiobesity properties.
180 , whilst peanut-tolerant groups have a mixed CLA/alpha4beta7 response (P = 0.008).
181         In model 1, a differentiation model, CLA activation of MAPK and induction of interleukin-8 (I
182         Pasture-grazing dairy cows have more CLA in their milk than do grain-fed cows.
183 at three members of the SR-B family, namely, CLA-1, CLA-2, and CD36, mediate recognition of bacteria
184          Nitro-conjugated linoleic acid (NO2-CLA) is preferentially formed, constitutes the most abun
185 y, human serum albumin was found to bind NO2-CLA both non-covalently and to form covalent adducts at
186 presence of two electrophilic centers in NO2-CLA located on the beta- and delta-carbons with respect
187  new insights into the chemical basis of NO2-CLA signaling actions.
188  this work, we examined the reactions of NO2-CLA with low molecular weight thiols (glutathione, cyste
189 pwise mechanisms with thiolate attack on NO2-CLA as rate-controlling step.
190            Moreover, we estimate that 98% of CLA(+) effector memory T cells are resident in normal sk
191                      Although the ability of CLA to inhibit angiogenesis in the peripheral nervous sy
192 n (+/-SD) percentage of total fatty acids of CLA for the cis-9, trans-11 isomer in adipose tissue was
193  the study was to test whether the amount of CLA in adipose tissue is associated with risk of diabete
194 /O3-MS method was applied to the analysis of CLA isomers in a commercial CLA supplement, milk fat, an
195                        However, causality of CLA-mediated responses to body fat loss, particularly th
196 tives were (1) compare the FA composition of CLA-rich yolk granules and plasma, relative to standard
197                              The contents of CLA and n-3 FA in a serving of whole milk (3.25% fat) in
198                   Nitroalkene derivatives of CLA and their metabolites are detected in the plasma of
199 sed for the direct and fast determination of CLA isomers at low concentrations and in complex lipid m
200                                The effect of CLA in lowering BMI was detected during the last 8 wk of
201 to demonstrate the anti-angiogenic effect of CLA in the brain, and suggests that CLA be explored as a
202 This comparison indicated that the effect of CLA was linear for up to 6 mo and then slowly approached
203                However, potential effects of CLA and calcium on bone mass during a period of bone los
204 l models have reported beneficial effects of CLA on atherosclerosis.
205  investigation of the safety and efficacy of CLA supplementation in children is recommended.
206 ing in the skin, and thus, the evaluation of CLA(+) T cells in the blood may eliminate the need for s
207                                    Except of CLA(-) Tc1 cells (P = .03), IFN-gamma levels were mostly
208                             The expansion of CLA-expressing effector memory CD8+ T cells in response
209 s develop strong and selective expression of CLA and E-selectin ligand while responding to HSV antige
210 s was observed mainly for the methyl form of CLA.
211           A significantly lower frequency of CLA(+) IFN-gamma-producing cells was observed in patient
212       In this way, de novo identification of CLA positional isomers, i.e. without requiring compariso
213 ic cells are particularly potent inducers of CLA on HSV-reactive CD4 T cells.
214               We explored how two isomers of CLA, cis9, trans11-CLA and trans10, cis12-CLA, affected
215 in resident T cells expressed high levels of CLA, CCR4, and CCR6, and a subset expressed CCR8 and CXC
216           Proposed antiobesity mechanisms of CLA include regulation of (a) adipogenesis, (b) lipid me
217 ation were measured before and after 6 mo of CLA supplementation by using whole-room indirect calorim
218  labeled dietary fat oxidation after 6 mo of CLA supplementation given with a breakfast meal.
219 had higher Bet v 1-specific proliferation of CLA(+) and CCR4(+) T cells compared with patients with b
220 ility (CLA isomer profile, quantification of CLA and volatile compounds by SPME coupled with CG-MS) d
221  E-selectin on tumor vessels, recruitment of CLA(+) CD8(+) T cells, and histological evidence of tumo
222 ts are the most important dietary sources of CLA, we have investigated the CLA concentrations and add
223 entifying these cells as possible sources of CLA-promoting cytokines.
224 did not influence the oxidative stability of CLA, however its presence improved physical-chemical cha
225 gs and (3) compare the emulsion stability of CLA-yolk mayonnaise.
226    Interestingly, CHO-131 stains a subset of CLA(+) T cells.
227 d, double-blind, placebo-controlled trial of CLA in 62 prepubertal children aged 6-10 y who were over
228 CELL." E-Ig reactivity was most prominent on CLA in mouse cells and on HCELL in human cells.
229 uencies of TH22 T cells within the CLA(+) or CLA(-) compartments.
230 l women with type 2 diabetes received SAF or CLA (8 g oil/d) during two 16-wk diet periods separated
231 < .0001), and frequencies of IL-13-producing CLA(+) cells were also correlated with IgE levels and SC
232            Based on homing receptor profile, CLA+ Treg should enter normal skin.
233 ocytes and dendritic cells from PBMC reduces CLA expression by HSV-2-responsive CD4 lymphoblasts, whi
234 gC tagged alphaB-crystallin displayed robust CLA.
235 e change in energy expenditure during sleep (CLA: 0 +/- 38 kcal; placebo: -43 +/- 90 kcal).
236 here were no differences in Bet v 1-specific CLA(+) and CCR4(+) proliferation and cytokine secretion
237  state of PA correlates with peanut-specific CLA responses, with tolerance associated with predominan
238 cal characteristics and oxidative stability (CLA isomer profile, quantification of CLA and volatile c
239 aled that it is possible to produce a stable CLA oil-in-water emulsion for using in beverages.
240 cts of the addition of CPP on the structure, CLA, and cell transduction properties of alphaB-crystall
241                               In this study, CLA-1- and CLA-2-stably transfected HeLa and HEK293 cell
242 ies of skin-homing (CLA(+)) versus systemic (CLA(-)) "polar" CD4 and CD8 T-cell subsets in patients w
243 encies of skin homing (CLA(+) ) vs systemic (CLA(-) ) "polar" CD4(+) and CD8(+) and activated T-cell
244                                       c9,t11-CLA lowered triacylglycerol (P </= 0.01) and had no effe
245  and cell studies suggest that VA and c9,t11-CLA may be hypocholesterolemic and antiatherogenic, epid
246 -9,trans-11 conjugated linoleic acid (c9,t11-CLA), are less clear.
247      We determined the effects of VA, c9,t11-CLA, and iTFA, in the context of highly controlled diets
248 , with the requirement of VA, but not c9,t11-CLA, to be listed under TFA on the Nutrition Facts Panel
249 y 3% VA, approximately 3% iTFA, or 1% c9,t11-CLA.
250 g two months of storage at room temperature, CLA:PPC (1:4) was selected for comparisons.
251           In this study, we demonstrate that CLA inhibits CXCR4 expression, resulting in a failure of
252 tic-adhesion assay, we provide evidence that CLA prevents monocytes from binding to ICAM-1 and subseq
253  risk is consistent with the hypothesis that CLA may be involved in insulin regulation.
254                    Our results indicate that CLA administration significantly reduces angiogenesis in
255                                 We show that CLA inhibits human peripheral blood monocyte cell adhesi
256                      These data suggest that CLA instigates the release of inflammatory signals from
257                        The data suggest that CLA, along with dietary calcium, has great potential to
258  (CLA) and insulin regulation suggested that CLA could be associated with risk of diabetes, but epide
259 ffect of CLA in the brain, and suggests that CLA be explored as a therapeutic treatment for cancer an
260                                          The CLA(+) TH1/TH2 and TC1/TC2 ratio was highly imbalanced i
261                                          The CLA, vaccenic acid, C18:39c12c15c, total C18:1 trans and
262 in the change in fat utilization between the CLA (4 +/- 8 g) and placebo (-7 +/- 11 g) groups during
263   However, feeding regimens that enhance the CLA content of milk also increase concentrations of tran
264 and produced a reduction in fat mass for the CLA group alone (0.05 +/- 0.05 kg/wk; P<0.001) and for t
265 e (0.05 +/- 0.05 kg/wk; P<0.001) and for the CLA group compared with placebo (0.09 +/- 0.08 kg/wk; P<
266 al body weight was smaller (P = 0.02) in the CLA group (-0.09 +/- 0.9%) than in the placebo group (0.
267 bsorptiometry was smaller (P = 0.001) in the CLA group (-0.5 +/- 2.1%) than in the placebo group (1.3
268 creased significantly more (P = 0.05) in the CLA group (-5.1 +/- 7.3 mg/dL) than in the placebo group
269 ineral accretion was lower (P = 0.04) in the CLA group (0.05 +/- 0.03 kg) than in the placebo group (
270 from protein was reduced during sleep in the CLA group (CLA: -3.3 +/- 2.6%; placebo: 0.3 +/- 5.7%).
271  subjects completed the trial (n = 28 in the CLA group, n = 25 in the placebo group).
272 ary sources of CLA, we have investigated the CLA concentrations and additionally the fatty acid profi
273 skin-resident T cells, while maintaining the CLA(+)CCR4(+) skin-homing phenotype as well as a diverse
274      The physical-chemical properties of the CLA microparticles were characterised by core retention,
275                      The predominance of the CLA+ response to peanut in peanut allergic patients is c
276 y on T cells, can also be decorated with the CLA epitope and serve as an E-selectin ligand.
277 tered frequencies of TH22 T cells within the CLA(+) or CLA(-) compartments.
278 somers, i.e. without requiring comparison to CLA standards, was achieved.
279  40 degrees C with 1:3M ratio of glycerol to CLA).
280 eters aphid settling)-mediated resistance to CLA compared with B73 and Tx601 maize susceptible inbred
281  JA, contributed to heightened resistance to CLA in maize.
282 ir1-Cys Protease provides direct toxicity to CLA.
283 evated ratios of alpha(4)beta(7)(+) Tregs to CLA(+) Tregs (odds ratio, 18.1; P = .020).
284                         The content of total CLA and the percentage of its t11,c13 isomer were higher
285 plored how two isomers of CLA, cis9, trans11-CLA and trans10, cis12-CLA, affected lipid and glucose m
286 ere significantly lower in the cis9, trans11-CLA group, compared with control mice consuming linoleic
287 al properties or emulsion stability by using CLA-rich eggs.
288 I analog 1, CLA-1, and its splicing variant, CLA-2 (SR-BI and SR-BII in rodents), are human high dens
289 ace method was used and 10% w/w AG, 3.5% w/w CLA and 0.3% w/w XG was introduced as the optimum formul
290 l properties are needed to establish whether CLA(+) memory subsets can be used as biomarkers and a su
291                 This review examines whether CLA's antiobesity properties are due to inflammatory sig
292 ll characterized, it remains unknown whether CLA also affects vascular morphology in the central nerv
293 is a meta-analysis of human studies in which CLA was provided as a dietary supplement to test its eff
294 esulting search to identify studies in which CLA was provided to humans in randomized, double-blinded
295 e identified a novel mechanism through which CLA alters monocyte function.
296   Understanding the mechanisms through which CLA mediates its atheroprotective effect may help to ide
297 gical properties of mayonnaise prepared with CLA-rich eggs to control eggs and (3) compare the emulsi
298                         Supplementation with CLA and SAF exerted different effects on BMI, total and
299                         Supplementation with CLA reduced body mass index (BMI) (P = 0.0022) and total
300 aegypti mosquitoes infected with the wMelPop-CLA strain of Wolbachia and in Drosophila melanogaster a

WebLSDに未収録の専門用語(用法)は "新規対訳" から投稿できます。
 
Page Top