ライフサイエンスコーパス: CM

1 CM attachment to the ECM is mediated by integrin complex

2 CM exposure was reported by 35% of the women.

3 CM was significantly greater post-LASIK relative to pre-

4 CM-MSC may provide an effective cell-free therapy for in

5 CM-MSC treatment reduces cartilage damage and suppresses

6 CM-MSC treatment significantly reduced knee-joint swelli

7 admissions with the primary diagnosis (ICD-9-CM code) of cellulitis and abscess of finger and toe (68

8 In the outpatient setting, the ICD-9-CM codes 691.8 and 692.9 alone have poor PPV.

9 We sought to validate the use of ICD-9-CM codes for identifying AD.

10 Ninth Revision, Clinical Modification (ICD-9-CM) codes.

11 Ninth Revision, Clinical Modification [ICD-9-CM] code 007.1) from 2006 to 2010, and analyzed claims o

12 At age 4, we calculated a CM-risk score (n=386) as the sum of the individual age-,

13 3 post-transduction while still retaining a CM gene expression profile.

14 higher tryptase levels were found in active CM-FPIES versus resolved CM-FPIES.

15 and metastasis of CM, thus likely affecting CM prognosis.

16 taCNR) between liver and muscle tissue after CM-101 injection was used to quantify liver fibrosis.

17 nolayers, CF-conditioned media did not alter CM spreading or MEK-ERK activation.

18 ns than in SE and more in EAC than in BE and CM.

19 tabolism, and further imply that the DCM and CM degradation pathways are mechanistically distinct.

20 opulations responsible for anaerobic DCM and CM metabolism, and further imply that the DCM and CM deg

21 nterestingly, cultures that received DCM and CM together degraded both compounds concomitantly.

22 eonatal oral exposure to alpha-, gamma-, and CM-HBCD resulted in different serum metabolomic profiles

23 fied, including 13 miRNAs between the NC and CM, 17 between the NC and CS, and 14 between the CM and

24 rongly expressed in slowly expanding RPE and CM compartments, and the loss of mouse Nf2 causes hyperp

25 ells exhibiting features of both neurons and CMs.

26 In the fungal Ascomycota, CM is based on hyphal filaments and arose in the Pezizom

27 ctional changes in heart, presumably because CM Tln1 became up-regulated.

28 monstrates a reasonable relationship between CM determined by ELISA and HPLC-ESI-ITMS/MS and therefor

29 ony growth and gtfB activation in situ by BF-CM.

30 Intriguingly, BF-CM induced S. mutans gtfBC expression (responsible for G

31 r metabolites/chromatographic analyses of BF-CM revealed elevated amounts of formate and the presence

32 uced mice that had simultaneous loss of both CM Tln1 and Tln2 and found that cardiac dysfunction occu

33 Cell growth regulation by CM and TRAIL was associated with the modulation of p53/M

34 ricted the induction of oxalate transport by CM.

35 II GRs were treated with CAF (n = 17), CAF + CM (n = 17), CAF + EMD (n = 17), and CAF + CM + EMD (n =

36 signed to receive either CAF (n = 17); CAF + CM (n = 17); CAF + EMD (n = 17), or CAF + CM + EMD (n =

37 cantly superior to CAF alone (23.53%); CAF + CM (52.94%), and CAF + CM + EMD (51.47%) (P <0.05).

38 alone (23.53%); CAF + CM (52.94%), and CAF + CM + EMD (51.47%) (P <0.05).

39 + CM (n = 17), CAF + EMD (n = 17), and CAF + CM + EMD (n = 17).

40 gnificant for CAF + CM, CAF + EMD, and CAF + CM + EMD (P <0.05).

41 QoL after 6 months was significant for CAF + CM, CAF + EMD, and CAF + CM + EMD (P <0.05).

42 + CM (n = 17); CAF + EMD (n = 17), or CAF + CM + EMD (n = 17).

43 MI) and other components of cardiometabolic (CM) risk during childhood, but evidence is scarce and in

44 es such as zebrafish activate cardiomyocyte (CM) division after tissue damage to regenerate lost hear

45 eracts with its receptors on cardiomyocytes (CMs), resulting in redox imbalance and cardiac dysfuncti

46 ene expression as compared with control cell CM.

47 d adipocyte UCP1 mRNA induction by mast cell CM.

48 MFs were stimulated by TGF-beta, cancer cell-CM or cancer cells, with or without SB431542 and JSI-124

49 In contrast, cancer cell-CM or TGF-beta stimulated MFs to produce IL-6.

50 Our results show that certain CM formulas without allergenic and low proinflammatory p

51 Chloromethane (CM) was not an intermediate during DCM utilization consi

52 ted with individual outcomes or the combined CM-risk score.

53 Our aim was to compare CM formulas regarding the presence of immunoreactive CM

54 cardiometabolic risk, including a composite CM-risk score.

55 w that, for the inbreeding loads considered, CM leads to unacceptably high extinction risks and, as a

56 Considering CM-HBCD has been implicated in neurodevelopment and endo

57 In contrast, CM degradation in culture RM had a strict requirement fo

58 PELP1-cyto CM induced changes in THP-1 gene expression as compared

59 uman and mouse pluripotent stem cell-derived CMs (PSC-CMs) were transduced with the neurogenic transc

60 human induced pluripotent stem cell-derived CMs, decreasing expression of hypertrophic genes and reg

61 nderstanding of non-ultraviolet (UV)-derived CMs remains limited.

62 ried different sources of mitochondrial DNA, CMs derived from iPSC and nt-ESCs could both recapitulat

63 with the coiled-coil and marked-box domains (CMs) of E2Fs.

64 ckaged in the extracellular vesicles from EC-CM.

65 or endothelial cells conditional medium (EC-CM).

66 A blinded analysis of eight CM formulas, one nonhydrolyzed, two partially hydrolyzed

67 tings of injury- and/or Neuregulin1-elicited CM proliferation.

68 tal CF-conditioned media distinctly enhanced CM spreading and contractile activity, which was shown b

69 lute transfer of chylomicron retinyl esters (CM-REs), retinol bound to retinol-binding protein (RBP-R

70 ith single-cell qPCR, we temporally examined CM gene expression and observed increased expression of

71 ect the cognitive outcome after experimental CM.

72 After completing OIT for CM, children were able to reintroduce CM in their diet.

73 Patients treated with PBR for CM (with 100% of the dose given to the macula) present m

74 We propose a multistep process for CM pathology that is initiated by cytoadhesion of infect

75 yperoxaluric mice treated with O. formigenes CM reflects the in vivo retention of biologic activity a

76 pe 1, rectal administration of O. formigenes CM significantly reduced (>32.5%) urinary oxalate excret

77 l blood mononuclear cell (PBMC) culture from CM allergic patients and nonallergic individuals were as

78 However, recycling strategies for future CM quantities in end-of-life vehicles (ELVs) are poorly

79 hiPSC-CM treated with T3+Dex, but not with either T3 or Dex al

80 METHODS AND hiPSC-CM were generated using a standard chemical differentiat

81 tent stem cell-derived cardiomyocytes (hiPSC-CM) are increasingly being used for modeling heart disea

82 sisting of either fetal-like or mature hiPSC-CM monolayers.

83 led into question the applicability of hiPSC-CM findings to the adult heart.

84 ration method could advance the use of hiPSC-CM for disease modeling and cell-based therapy.

85 enabling technology for future use of hiPSC-CM tissues in human heart repair.

86 ure excitation-contraction coupling of hiPSC-CM when cultured on extracellular matrix with physiologi

87 tructural and functional maturation of hiPSC-CM, including lack of T-tubules, immature excitation-con

88 t of cardiomyocyte maturation state on hiPSC-CM drug responsiveness.

89 tent stem cell derived cardiomyocytes (hiPSC-CMs) offer a novel in vitro platform for pre-clinical ca

90 tent stem cell-derived cardiomyocytes (hiPSC-CMs) provide new possibilities for evaluating drug safet

91 tent stem cell-derived cardiomyocytes (hiPSC-CMs), but D-ala,RP produgs, including MK-3682, did not.

92 tent stem cell-derived cardiomyocytes (hiPSC-CMs), Tbx20 enhanced human KCNH2 gene expression and hER

93 human induced pluripotent stem cells (hiPSC-CMs), 41 were accurately quantified.

94 iPSCs) can be differentiated into CMs (hiPSC-CMs) that model cardiac contractile mechanical output mo

95 To date hiPSC-CMs used for cardiotoxicity testing display an immature,

96 0 did not increase KCNH2 expression in hiPSC-CMs, which led to decreased IhERG and increased APD.

97 diac function of single micropatterned hiPSC-CMs and determine contractile parameters that can be use

98 anical output of single micropatterned hiPSC-CMs from microscopy videos.

99 Here we show that maturation state of hiPSC-CMs determines the absolute pro-arrhythmia risk score ca

100 Thus, the maturation state of hiPSC-CMs should be considered prior to pro-arrhythmia and car

101 tform for 3D culture and maturation of hiPSC-CMs that after 5 weeks of differentiation show robust el

102 Under these conditions, hPSC-CMs were refractory to mitogenic stimuli, and we found t

103 tilization and functional maturation of hPSC-CMs facilitating the application of this promising cell

104 In this study, we report that shifting hPSC-CMs from glucose-containing to galactose- and fatty acid

105 radation is consumed by the hydrogenotrophic CM degrader, or may fuel other hydrogenotrophic processe

106 Total IgE and IgM, CM-specific IgG, and casein-specific IgE, IgG, IgG4, and

107 las regarding the presence of immunoreactive CM components, IgE reactivity, allergenic activity, abil

108 CM-OIT as well as the role of iNKT cells in CM allergy.

109 tural killer T cells (iNKTs) are involved in CM allergy sensitization and now examine their role in O

110 not studied at the ultrastructural level in CM.

111 herapy and supported the role of UV light in CM pathogenesis.

112 tentially other LV abnormalities observed in CM-AVM.

113 lead to the LV leakage defects that occur in CM-AVM is not understood.

114 issue thickness gain was significant only in CM-treated groups (P <0.05).

115 873997 G>A) that showed a predictive role in CM-specific survival, with an effect-allele-attributed h

116 idate mechanisms that underlie variations in CM function.

117 ave evaluated activation of MAPKs and Akt in CMs exposed to C5a in vitro and after cecal ligation and

118 vation (phosphorylation) of MAPKs and Akt in CMs, which required availability of both C5a receptors.

119 em cells (hiPSCs) can be differentiated into CMs (hiPSC-CMs) that model cardiac contractile mechanica

120 otent stem cells-derived cardiomyocytes (iPS-CMs).

121 The resulting iPS-CMs cells underwent spontaneous apoptosis which was furt

122 iPSC-CMs can integrate into host myocardium and create a biol

123 We now report pacemaker function of iPSC-CMs in a canine model.

124 Engineered heart muscles from TTS-iPSC-CMs showed an impaired force of contraction and a higher

125 hanced beta-adrenergic signaling in TTS-iPSC-CMs under catecholamine-induced stress increased express

126 e detected in catecholamine-treated TTS-iPSC-CMs, and were confirmed by differentially expressed lipi

127 ent stem cells derived cardiomyocytes (iPSCs-CM).

128 In iPSCs-CM, similar effects of M94I on reduction of peak INa wer

129 before and after injection of 10 micromol/kg CM-101.

130 a nevertheless possesses Pezizomycotina-like CM.

131 omotes their fast maturation into adult-like CMs with higher oxidative metabolism, transcriptional si

132 ed activation in vivo of MAPKs and Akt in LV CMs as well as the appearance of cytokines and histones

133 Ks and Akt occurred in left ventricular (LV) CMs, requiring both C5a receptors, C5aR1 and -2.

134 PlexA1 mutant mice maintain CM connections into adulthood and exhibit superior manua

135 sequential epitopes derived from five major CM proteins were measured with a peptide microarray-base

136 Cerebral malaria (CM) can be classified as retinopathy-positive or retinop

137 Cerebral malaria (CM) is a severe complication of Plasmodium falciparum in

138 is the salient feature of cerebral malaria (CM).

139 of mortality in pediatric cerebral malaria (CM).

140 ary malformation-arteriovenous malformation (CM-AVM) is a blood and lymphatic vessel (LV) disorder th

141 Childhood maltreatment (CM) confers deleterious long-term consequences, and grow

142 ompartments: the retina, the ciliary margin (CM), and the retinal pigment epithelium (RPE).

143 Here we describe the Cerutti Mastodon (CM) site, an archaeological site from the early late Ple

144 Maternal CM exposure was assessed using the Childhood Trauma Ques

145 Maternal CM exposure was associated with lower child intracranial

146 onal transmission of the effects of maternal CM exposure on child brain development and suggest this

147 ned the intergenerational effect of maternal CM exposure on child brain structure and also addressed

148 d the other forcing it (circular sib mating (CM)).

149 Considering xenogeneic collagen matrix (CM) and enamel matrix derivative (EMD) characteristics,

150 procedure with a xenogenous collagen matrix (CM) and/or enamel matrix derivative (EMD) in combination

151 FSP - 7.5:0.2:7.5%, w/w/w) and chia seed (MD:CM:CSP - 7.5:0.6:7.5%, w/w/w), respectively.

152 en with CM-FPIES, children with IgE-mediated CM allergy, and those tolerating CM.

153 with CM-FPIES versus those with IgE-mediated CM allergy.

154 Here, we have shown that conditioned medium (CM) containing gelsolin fragments or an N-terminal gelso

155 of O. formigenes culture conditioned medium (CM) on apical (14)C-oxalate uptake by human intestinal C

156 e UCP1 mRNA induction by conditioned medium (CM).

157 s were treated with HMEC-conditioned medium (CM).

158 of mesenchymal stem cell-conditioned medium (CM-MSC) as an alternative to cell therapy in an antigen-

159 Conjunctival melanoma (CM) is a highly aggressive ocular cancer for which treat

160 Genomic analyses of cutaneous melanoma (CM) have yielded biological and therapeutic insights, bu

161 Cutaneous melanoma (CM) is the most lethal skin cancer.

162 er increased steadily in the central memory (CM) and effector memory T cells over 48 weeks.

163 Coccidioidal meningitis (CM) is a devastating complication of coccidioidomycosis.

164 rus/AIDS-associated cryptococcal meningitis (CM) frequently experience clinical deterioration, known

165 Cryptococcus can cause meningoencephalitis (CM) among previously healthy non-HIV adults.

166 The functional improvements in mESC-CM + fetal CF patches were associated with differences i

167 In mESC-CM monolayers, CF-conditioned media did not alter CM spr

168 atches containing adult CFs, while pure mESC-CM patches did not form functional syncytium.

169 yonic stem cell-derived cardiomyocytes (mESC-CMs).

170 our studies show that 3D co-culture of mESC-CMs with embryonic CFs is superior to co-culture with ad

171 f paracrine signaling, we cultured pure mESC-CMs within miniature tissue "micro-patches" supplemented

172 f the major applications of critical metals (CMs) is in electrical and electronic equipment (EEE), wh

173 's esophagus (BE), columnar cell metaplasia (CM), squamous cell carcinoma (SCC), and squamous epithel

174 Recombinant IL-6 and MF-CM activated STAT3 and upregulated TGF-beta in cancer ce

175 timulated by IL-6, MF-conditioned medium (MF-CM) or MFs, with or without TGF-beta signaling inhibitor

176 vitamin D/100 mL) or nonfortified cow milk (CM) (0.02 mg Fe/100 mL; no vitamin D) for 20 wk.

177 Cow milk (CM) allergy (CMA) affects up to 3% of the paediatric pop

178 Although most of cow's milk (CM) allergic children will outgrow their allergy, the pa

179 e previously shown that maternal cow's milk (CM) elimination results in downregulation of CM-specific

180 in children with FPIES caused by cow's milk (CM).

181 timate six single and combined misalignment (CM) parameters, the root mean square (RMS) of the differ

182 chondrial activity-dependent dye Mitotracker CM-H2TMRosa, is higher in CTB than in SCT in culture and

183 HBCD is a commercial mixture (CM-HBCD) composed of three main stereoisomers: alpha-HBC

184 Moreover, CMs isolated from transverse aortic constriction mice tr

185 with the appearance of cortico-motoneuronal (CM) connections during the evolution of the mammalian co

186 The advent of complex multicellularity (CM) was a pivotal event in the evolution of animals, pla

187 and CagA and CagA with CagA multimerization (CM) sequence-positive H. pylori.

188 es of longitudinal (LM) and circular muscle (CM) layers.

189 TIONALE: During each beat, cardiac myocytes (CMs) generate the mechanical output necessary for heart

190 and stable connections of cardiac myocytes (CMs) with the extracellular matrix (ECM) to preserve sar

191 innocent bystanders in retinopathy-negative CM and the etiology of the coma is entirely non-malarial

192 , their contribution to retinopathy-negative CM is largely unknown.

193 und for the fraction of retinopathy-negative CM that would be prevented if malaria parasitemia were t

194 of malaria infection to retinopathy-negative CM.

195 to the pathogenesis of retinopathy-negative CM.

196 hest levels of CRC; however, the addition of CM increases gingival thickness.

197 sults suggested the promising application of CM-ALs (10%) scaffolds for drug delivery and bone tissue

198 om the period 2000-2014 to identify cases of CM.

199 To identify the molecular characteristics of CM using next-generation whole-exome sequencing (WES).

200 rachnoiditis is an important complication of CM in previously healthy individuals, requiring heighten

201 CM) elimination results in downregulation of CM-specific IgA antibody levels in BM, but not in serum,

202 re.VA supplementation redirected the flow of CM-REs from peripheral to central organs and accumulated

203 reflect inherited systemic immunogenicity of CM proteins in these families, although specific IgA in

204 of advanced HIV, with 2013-2014 incidence of CM comparable to pre-ART era rates in South Africa.

205 y known curative option in the management of CM.

206 on development, invasion, and metastasis of CM, thus likely affecting CM prognosis.

207 itic varicosities in the cortical neurons of CM-infected brain.

208 served group difference between offspring of CM-exposed mothers versus nonexposed mothers was 6%.

209 hat IL-22 is involved in the pathogenesis of CM.

210 In the presence of CM-MSC or MSCs, increases in IL-10 concentration were ob

211 the mechanical and degradation properties of CM-ALs (10%) scaffolds were comparable to that of PLLA/n

212 oal was to reduce the false positive rate of CM diagnosis, and so the algorithms were tuned at high s

213 study showed more sustained drug release of CM-AL-containing scaffolds than these of CM/nHA-AL and P

214 is study confirms the efficacy and safety of CM-OIT as well as the role of iNKT cells in CM allergy.

215 ribution, pharmacokinetics, and stability of CM-101 in rats were measured with mass spectrometry.

216 of CM-AL-containing scaffolds than these of CM/nHA-AL and PLLA/nHA/AL scaffolds, and the mechanical

217 the practice experience in the treatment of CM, preparation of IT AmBd, and the different dosing str

218 utilization of IT AmBd and the treatment of CM.

219 hredder residue may increase the recovery of CMs from ELVs.

220 Three-month-old CMs were subjected to catecholamine stimulation to simul

221 Following five consecutive transfers on CM and H2 , Acetobacterium 16S rRNA gene sequences domin

222 d a single gavage dose of alpha-, gamma-, or CM-HBCD at 3, 10, and 30 mg/kg.

223 Infantile mice exposed to alpha-, gamma-, or CM-HBCD demonstrated differences in endogenous metabolit

224 sidered the combined misalignment parameter (CM), which represents the total effect of all individual

225 d (NC), APEC-challenged with mild pathology (CM), and APEC-challenged with severe pathology (CS).

226 With the Pd/CM-phos catalyst system, the corresponding 3-arylated ox

227 mbinant CIDRalpha1.7 domain from a pediatric CM brain autopsy inhibited the barrier-protective proper

228 CM epitopes between patients with persistent CM allergy (CMA) and those that naturally became toleran

229 secreted by type II secretory system: PheA (CM-sec), LipA/LesA, VirK, and four families involved in

230 esis-ring-opening metathesis polymerization (CM-ROMP) strategy that affords functionalized Grubbs-II

231 to the pathogenesis of retinopathy-positive CM, their contribution to retinopathy-negative CM is lar

232 re Tln2 is the dominant isoform in postnatal CMs.

233 agen-targeted magnetic resonance (MR) probe, CM-101, and to assess its ability to help quantify liver

234 es, and drug response of cardiomyocytes (PSC-CMs) and endothelial cells (PSC-ECs) derived from geneti

235 mouse pluripotent stem cell-derived CMs (PSC-CMs) were transduced with the neurogenic transcription f

236 This study demonstrates that PSC-CMs are amenable to partial neuronal conversion, yieldin

237 achnoiditis among 26 consecutively recruited CM patients with normal CD4 counts who achieved microbio

238 IT for CM, children were able to reintroduce CM in their diet.

239 electrophysiological profile of reprogrammed CMs.

240 ost-transduction, we found that reprogrammed CMs expressed neuronal markers such as Tuj1, Map2, and N

241 ere found in active CM-FPIES versus resolved CM-FPIES.

242 patients with active and those with resolved CM-FPIES.

243 al Classification of Diseases-Ninth Revision-CM codes 35.05 and 35.06) between January and November 2

244 had undergone an OCTA after PBR for a small CM.

245 h proton beam radiotherapy (PBR) for a small CM.

246 Cardiac patches were created from spheroids (CM:FB:EC = 70:15:15, 70:0:30, 45:40:15) using a 3D biopr

247 a novel microspheres-scaffold hybrid system (CM-ALs) for drug delivery and bone tissue engineering ap

248 subtype associated with worse survival than CM, is needed to delineate non-UV oncogenic mechanisms.

249 ization consistent with the observation that CM could not replace DCM as a growth substrate.

250 Our results show that CM Tln2 is essential for proper beta1D-integrin expressi

251 We found that CMs adopted neuronal morphologies as early as day 3 post

252 The CM fascicles on the ventral surface of LES are arranged

253 (CI): 0.01, 0.21], and between PFNA and the CM-risk score (beta=0.60; 95% CI: 0.04, 1.16).

254 ce of an unidentified species of Homo at the CM site during the last interglacial period (MIS 5e; ear

255 proboscidean bone reduction, evident at the CM site, fits within a broader pattern of Palaeolithic b

256 17 between the NC and CS, and 14 between the CM and CS groups.

257 ilbenedisulfonic acid completely blocked the CM-induced oxalate transport.

258 the CS group compared to levels in both the CM and NC groups.

259 On the other hand, the CM fascicles from the two sides cross midline on dorsal

260 rge cobbles (hammerstones and anvils) in the CM bone bed display use-wear and impact marks, and are h

261 ntion was lower in the YCF group than in the CM group.

262 iptional regulation of Nf2 by Yap/Taz in the CM, activation of Nf2 expression by Mitf in the RPE and

263 Treatment with the CM from CYP4A10(high) M2 macrophages (M2) increased pre-

264 other costameric proteins were lost from the CMs, and membrane integrity was compromised.

265 ferences in IgE and IgG4 antibody binding to CM epitopes between patients with persistent CM allergy

266 sed binding of serum epitope-specific IgA to CM in mothers of infants with CMA may reflect inherited

267 increased binding of epitope-specific IgA to CM proteins compared to those with a non-CMA infant.

268 es between IgE and IgG4 binding intensity to CM peptides decreased when the patients became tolerant.

269 ing simplification and constraint leading to CM suggest a deterministic process where shared modes of

270 gE-mediated CM allergy, and those tolerating CM.

271 to higher primates, we identified transient CM connections in early postnatal mice, which are eventu

272 erves, and transthyretin cardiomyopathy (TTR-CM), which primarily affects the heart.

273 c therapies shown to improve survival in TTR-CM.

274 The natural history of TTR-CM is poorly characterized, which presents difficulties

275 safety of tafamidis for the treatment of TTR-CM.

276 Unlike CM, somatic alterations were dominated by structural var

277 outcomes obtained in clinical studies using CM formulas.

278 R = 2.08) were independently associated with CM in a sample of 115 Nigerian children with CM and 160

279 46) of IL22 and IL22RA2 were associated with CM in children from Nigeria and Mali.

280 ium with rs2227476, was also associated with CM in the combined cohort for these two populations, (P

281 ,855 SNPs were significantly associated with CM-specific survival at P < 0.05, of which 547 SNPs were

282 CM in a sample of 115 Nigerian children with CM and 160 controls.

283 In this study, 10 of the 11 children with CM induced anaphylaxis enrolled in a CMA OIT clinical tr

284 r cell counts were measured in children with CM-FPIES, children with IgE-mediated CM allergy, and tho

285 on after casein stimulation in children with CM-FPIES, results were similar to those in control subje

286 coronally advanced flap (CAF) combined with CM and/or EMD.

287 In contrast with CM, we observed PAK1 copy gains in 15% of patients, and

288 lls from healthy mice following culture with CM-MSC or co-culture with MSCs.

289 HBCD and was nonsignificantly decreased with CM-HBCD.

290 NPs) in 151 Wnt pathway autosomal genes with CM-specific survival and then validated significant SNPs

291 the brain sections of symptomatic mice with CM.

292 Patients with CM who experienced C-IRIS (N = 27) upon ART initiation w

293 om the archived specimens of 5 patients with CM who had been treated with surgical excision between 2

294 be biomarkers for outcomes of patients with CM, if validated by larger studies.

295 imulated T cells were found in patients with CM-FPIES versus those with IgE-mediated CM allergy.

296 paucity of humoral response in patients with CM-FPIES.

297 in acquisition of tolerance in patients with CM-FPIES.

298 Those with CM-associated cerebrovascular events (8%) may benefit fr

299 d lymph nodes of arthritic mice treated with CM-MSC or MSCs.

300 D4+ T cells from arthritic mice treated with CM-MSC showed increases in FOXP3 and IL-4 expression and

301 verexpression, but this could not restore WT CM integrin levels nor improve heart function.