ライフサイエンスコーパス: CNPase

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1 nestin and vimentin, and negative for A2B5, CNPase, neurofilament (NF), and neuron specific enolase

2 nestin and vimentin, and negative for A2B5, CNPase, NF, and NSE.

3 rkers neurofilament, GAP-43, synapsin-1, and CNPase--and on the perikarya of sensory neurons in the H

4 chemically GFAP positive, A2B5 negative, and CNPase negative [38].

5 press other oligodendroglial markers such as CNPase, RIP, and APC.

6 The central CNPase homology domain was sufficient for catalytic acti

7 s well as the oligodendroglial gene encoding CNPase (2',3' cyclic nucleotide 3'-phosphohydrolase).

8 ostaining for the myelin-synthesizing enzyme CNPase was observed in me/me mice, confirming the loss o

9 neurons, 3) abnormal expression patterns for CNPase and connexin 29 in the SGN clusters, 4) reduced S

10 nuated metabolism of 2',3'-cAMP to 2'-AMP in CNPase(-/-) mice.

11 ial function, number, area, and autophagy in CNPase-knockout (CNPase(-/-)) versus wild-type (WT) mice

12 (IMC-C225) for 2 weeks beginning at birth in CNPase-hEGFR mice normalized all pathologies at 3 months

13 histologic changes were minimal after IR in CNPase(-/-) mice.

14 IR did not affect these parameters in CNPase(-/-) mice.

15 hence the name goldfish regeneration-induced CNPase homolog (gRICH).

16 d accelerated autophagy/mitophagy in injured CNPase(-/-) mice.

17 ber, area, and autophagy in CNPase-knockout (CNPase(-/-)) versus wild-type (WT) mice using a unique t

18 , colocalizing with the Schwann cell marker, CNPase (2',3'-cyclic nucleotide 3'-phosphodiesterase), a

19 , both immature (A2B5+/O4+) and more mature (CNPase+) human oligodendrocytes in vitro expressed the s

20 rentiated into GFAP positive, A2B5 negative, CNPase negative astrocytes, we conclude that 1H91 cells

21 st AKI, it is conceivable that inhibition of CNPase protects against ischemia-reperfusion (IR) -induc

22 lin), astrocyte (GFAP), and oligodendrocyte (CNPase) markers revealed that differentiated neurons con

23 or mature astroglia (GFAP), oligodendroglia (CNPase), or neurons (MAP2).

24 rations in expression of Iba1 or in Olig2 or CNPase.

25 ',3'-cyclic nucleotide 3'-phosphodiesterase (CNPase) could be demonstrated for one representative clo

26 ',3'-cyclic nucleotide 3'-phosphodiesterase (CNPase) metabolizes 2',3'-cAMP to 2'-AMP; 2',3'-cAMP is

27 creased cyclic nucleotide phosphodiesterase (CNPase) expression, whereas FGF-2 deprivation alone atte

28 ,3'-cyclic-nucleotide 3'-phosphodiesterases (CNPases), hence the name goldfish regeneration-induced C

29 2;,3;-cyclic nucleotide 3;-phosphohydrolase (CNPase), and vimentin, suggesting that they were not dif

30 ns confirms that both gRICH proteins possess CNPase activity.

31 prolifera-tion in perinatal mutant nerve, so CNPase-hEGFR Schwann cell numbers correlate with the cet

32 y to mammalian myelin marker proteins termed CNPases.

33 We conclude that CNPase deletion attenuates IR-induced AKI, in part by ac

34 eins are responsible for the majority of the CNPase activity detected in regenerating goldfish retina

35 x17 in the oligodendrocyte lineage under the CNPase promoter.

36 In a mouse model in which the CNPase promoter drives expression of human EGFR in Schwa

37 Thus, CNPase enzyme activity is implicated for the first time

38 confirmed with NeuN) or glia (confirmed with CNPase).

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