ライフサイエンスコーパス: CP

1 CP 55 940 (0.001-3.0 mug/kg) functioned as a reinforcer

2 CP 55 940 was least potent to decrease food-maintained r

3 CP binding has an electrostatic contribution, but assemb

4 CP-B7 and >/=CP-B8 patients exhibited median overall sur

5 CP-dox treatment also repolarized intratumoral myeloid c

6 CP-enes are formed by dehydrochlorination of CPs and are

7 Allocations were preformed adding 1 CP at a time or all 11 CPs at once, and with and without

8 Furthermore, inclusion of 1 CP at a time increased matching in ICP by up to 33%, and

9 The 10% CP dietary treatment damaged ileal mucosal morphology, a

10 in ICP by up to 33%, and inclusion of all 11 CPs at once increased ICP matching by 50%.

11 re preformed adding 1 CP at a time or all 11 CPs at once, and with and without exclusion of unaccepta

12 dentulous sites was taken from GAgP (n =12), CP (n=12) and H (n=12) patients.

13 d, including 191 retrospective isolates (122 CP-CRE and 69 non-CP isolates) as well as 45 prospective

14 ction proteins (occludin and claudin) in 13% CP group were higher than the other two groups.

15 ae was higher in both ileum and colon of 13% CP group.

16 well as 45 prospective clinical isolates (15 CP-CRE and 30 non-CP-CRE) obtained over a 3-month period

17 MCCPs (C14-C17 CPs) and LCCPs (C>/=18 CPs) predominated in most sediments with average percent

18 ns demonstrated that HadA prefers 4-CP and 2-CP over CPs with multiple substituents.

19 The 20S proteasome core particle (20S CP) plays an integral role in cellular homeostasis by de

20 During the study period, 309 CPs were recorded during 59 644 PCI-CABG procedures with

21 A total of 376 CP were recorded from 26 807 CTO-PCI interventions (inci

22 r reactions demonstrated that HadA prefers 4-CP and 2-CP over CPs with multiple substituents.

23 rapid-quench flow experiments of HadA with 4-CP show the involvement of specific intermediates (C4a-h

24 bacteremia during the study period: 37 (45%) CP-CRE and 46 (55%) non-CP-CRE.

25 The differential reactivity of 6-CP under reducing and nonreducing conditions highlights

26 Structural studies with 6-CP and SAM also reveal electron density consistent with

27 sera from dosed subjects were analyzed in a CP activation assay evaluating C3d deposition on HLA-coa

28 h matched nonperforation survivors without a CP (P<0.0001).

29 h matched nonperforation survivors without a CP (P<0.0001).

30 bapenemase-producing Pseudomonas aeruginosa (CP-PA) and carbapenemase-producing Acinetobacter baumann

31 ith a legacy effect on later mortality after CP also observed.

32 C and the cannabinoid receptor (CBR) agonist CP 55 940 in Old World monkeys (rhesus and cynomolgus),

33 dation products such as chlorinated alkenes (CP-enes).

34 Here, it is shown that amorphous CP can be used as general synthesis precursors of highly

35 we demonstrate that Ca(2+)-permeable AMPARs (CP-AMPARs) mediate glutamatergic transmission at the adu

36 he first shared genetic risk loci of AgP and CP with genome-wide significance and highlights the role

37 combinations of isotope clusters of CPs and CP-enes.

38 In contrast, craving and CP-AMPAR levels remain high on WD60.

39 ficant on comparison between the healthy and CP groups and between the CP and MI groups.

40 asal forebrain, lobar holoprosencephaly, and CP.

41 spartate receptor antagonists ifenprodil and CP-101,606 blocked cocaine-induced habits; this was depe

42 revealing the importance of TFAP2A in NC and CP specification, and performing a small-molecule screen

43 Women with PCOS and CP (n = 60) were randomly divided into two groups.

44 t alone in management of women with PCOS and CP.

45 ever, the mice differed in CPP retention and CP-AMPAR incorporation.

46 me, the detailed interactions between SP and CP in a bacteriophage, providing unique insights into ma

47 was to evaluate the safety/tolerability and CP-blocking potential of 4 weekly doses (60 mg/kg) of th

48 en the number of complex strategies used and CP evident (P=0.008 for trend).

49 emoved using the CRF-R1 selective antagonist CP-154526, suggesting Ucn1 acts through CRF-R1 when prom

50 On average, CP-PCB_126 individually accounted for over 95% of the to

51 as6 controls both base-lid affinity and base-CP affinity through steric hindrance; Nas6 clashes with

52 Pase subunit of the base and antagonize base-CP interaction.

53 Nas6 interferes with base-lid, but not base-CP, association.

54 Among 265 eyes without any GA on baseline CP, 70 (26.4%) developed CP-visible GA.

55 apenemase-producing Acinetobacter baumannii (CP-AB) and perform a multicenter evaluation of the mCIM

56 k, we investigate Mn(II) competition between CP and two solute-binding proteins that Staphylococcus a

57 size cutoff that best discriminated between CP-CRE and members of the family Enterobacteriaceae that

58 Fs), and coplanar polychlorinated biphenyls (CP-PCBs) concentrations and age-corrected trends for lak

59 d swelling that can be prevented by blocking CP-AMPARs.

60 nt terminal responsiveness, whereas blocking CP-AMPARs protects terminals from excitotoxic swelling.

61 and extent of periodontal breakdown in both CP and AgP and could potentially be used for prevention

62 rystal structure of Ni(II)- and Ca(II)-bound CP, which reveals that CP binds Ni(II) ions at both its

63 rom bacterial Mn(II) acquisition proteins by CP, and molecular insight into the extracellular battle

64 ted from spectral deconvolution of the (13)C CP/MAS spectra and the results has shown that all the ex

65 MCCPs (C14-C17 CPs) and LCCPs (C>/=18 CPs) predominated in most sedimen

66 hat LCCPs from C18 to C36, as well as C8-C17 CPs, are persistent in sediments over the last 50-80 yea

67 C8-C36 CPs were found in sediment dating back to the 1930s.

68 Whereas in the absence of C4, CP can no longer activate C3, LP retains a residual but

69 tion of the corresponding mono-radicals CAAC-CP(.) .

70 Human calprotectin (CP, S100A8/S100A9 oligomer) is a metal-sequestering host

71 The pungent molecule capsaicin (CP) has a similar effect as AEA; however, CP acts by eng

72 We find that CCMV CP is also capable of packaging polyU RNAs, which-unlike

73 Due to their high degree of chlorination, CPs have highly complex isotopic mass patterns that ofte

74 nd catalytic efficiency toward their cognate CPs in comparison with noncognate substrates.

75 irst examples of main-chain boron-containing CPs without aromatic moieties within the polymer backbon

76 tive cysteine residues, the peroxidatic Cys (CP) and, if present, the resolving Cys (CR).

77 any GA on baseline CP, 70 (26.4%) developed CP-visible GA.

78 nd crystallization of 3a affords diamagnetic CP 3.

79 erein that Chimeric Polypeptide Doxorubicin (CP-Dox), a nanoparticle formulation of doxorubicin, enha

80 f inflammatory and fibrotic processes during CP.

81 ant to define allocation metrics that enable CP to receive a better-matched kidney, without disadvant

82 at identified compounds that further enhance CP differentiation.

83 ing carbapenem-resistant Enterobacteriaceae (CP-CRE) is an important element of the effort to prevent

84 t low risk of complications were as follows: CP <5; MELD <8; FIB-4 <3 for HD and HCC, and <2 for all-

85 ties were, respectively, 3%, 10%, and 0% for CP-A; 14%, 12%, and 6% for CP-B7; and 23%, 32%, and 3% f

86 , 10%, and 0% for CP-A; 14%, 12%, and 6% for CP-B7; and 23%, 32%, and 3% for >/=CP-B8.

87 rs was 0.81-0.82 for FIB-4 but 0.61-0.68 for CP and MELD scores.

88 n 14 days were more than 4 times greater for CP-CRE compared with non-CP-CRE bacteremic patients (adj

89 on of N; (2) demonstration that deltaN/N for CP-AFM junctions is remarkably small (</=2%) and that th

90 c states (641, 732, and 735 nm) observed for CP 3 and show that oligomer chains of length >/=14 repea

91 Median overall survival for CP-A patients was 13.3 mo (95% confidence interval [CI],

92 f the Development of ALLergy (MeDALL; EU FP7-CP-IP; project no: 261357; 2010-2015) studied the comple

93 fe (t1/2) and percent decreases for PCDD/Fs, CP-PCBs, and toxic equivalence (TEQ) (average -56 to 70%

94 r replicated in another sample of 399 German CP cases (disease onset <60 y of age) and 1,633 controls

95 CP-B7 and >/=CP-B8 patients exhibited median overall survival of 6.9

96 nd 6% for CP-B7; and 23%, 32%, and 3% for >/=CP-B8.

97 o prevent 1 baby from either dying or having CP.

98 cause KPD programs were not designed to help CP, it is important to define allocation metrics that en

99 n (CP) has a similar effect as AEA; however, CP acts by engagement of the vanilloid receptor TRPV1, c

100 l circulatory support (pMCS) device (Impella CP, Abiomed, Danvers, Massachusetts) decreases 30-day mo

101 that the SA signaling pathway is involved in CP-mediated plant defense .

102 anging 70% to 80% to improve HLA matching in CP recipients.

103 ctron bunch under resonant helical motion in CP laser fields.

104 an provide a better immunological profile in CP recipients who have a high EpMM to their own donor wh

105 two-, three- and multi-photon absorption in CPs and MOFs is further supplemented with application-or

106 tance Isolate Bank were evaluated, including CP isolates (Ambler class A, B, and D), non-carbapenemas

107 with TNT009 was safe and potently inhibited CP activity.

108 Ss is swapped onto the RNA otherwise lacking CP recognition motifs, assembly is partially restored, a

109 chronic phase chronic myelogenous leukemia (CP-CML), but blast crisis CML (BC-CML) and acute myeloid

110 with chronic phase chronic myeloid leukemia (CP-CML) are treated with tyrosine kinase inhibitors (TKI

111 ed to the ends of the heptameric barrel-like CP structure that occlude substrate entry pores, prevent

112 The maximum CP concentrations found in proximity to a metropolitan s

113 by conducting probe atomic force microscopy (CP-AFM) in which an Au-coated tip contacts a self-assemb

114 rm of stem-loops that each present a minimal CP-binding motif AXXA in the loops.

115 ns using 46 ICPs and 11 fully HLA-mismatched CPs were undertaken using the Australian KPD matching al

116 odel (NB-fit), compound Poisson mixed model (CP-fit), and the variance stabilizing transformed linear

117 tive clinical isolates (15 CP-CRE and 30 non-CP-CRE) obtained over a 3-month period.

118 udy period: 37 (45%) CP-CRE and 46 (55%) non-CP-CRE.

119 etrospective isolates (122 CP-CRE and 69 non-CP isolates) as well as 45 prospective clinical isolates

120 Ambler class A, B, and D carbapenemases, non-CP-CRE isolates, and carbapenem-susceptible isolates wer

121 s, likely due to restricted inclusion of non-CP-CRE to assess the specificity of the assays.

122 B, and D), non-carbapenemase-producing (non-CP) carbapenem-resistant isolates, and carbapenem-suscep

123 st that CP-CRE may be more virulent than non-CP-CRE and are associated with poorer outcomes.

124 ) in the CP-CRE group and 6 (13%) in the non-CP-CRE group.

125 enemase-producing (CP)-CRE compared with non-CP-CRE bacteremia.

126 4 times greater for CP-CRE compared with non-CP-CRE bacteremic patients (adjusted odds ratio, 4.92; 9

127 cholangiocarcinoma, n = 15) or nonmalignant (CP, n = 15) CBD stenosis.

128 t and walleye samples is due to the nonortho CP-PCBs (average = 79%) as compared with PCDD/Fs (averag

129 net (PNN) components well beyond the normal CP in V1 and mPFC.

130 a single intravenous (IV) administration of CP-Dox at the maximum tolerated dose increases the infil

131 ) ions, which enhance the Mn(II) affinity of CP.

132 09 was validated in an independent cohort of CP of European Americans (1,961 cases and 1,864 controls

133 is are thought to precede the development of CP.

134 Dissolution of CP 3 in DMSO favors Co-S bond heterolysis, yielding the

135 The effects of CP 55 940 (1.0-10 mug/kg, i.v.) and THC (3.0-300 mug/kg,

136 the withdrawal time when stable elevation of CP-AMPAR levels is detected, the cocaine group exhibited

137 The VLP-RNAi assembles upon co-expression of CP and the RNAi scaffold in E. coli.

138 ation) with late active ABMR and features of CP activation, such as capillary C4d or complement-fixin

139 d fetal outcomes, including survival free of CP, and whether effects differed by participant or treat

140 Here, the authors report the first GWAS of CP among a large community-based sample of Hispanics/Lat

141 ether, our results support the importance of CP-AMPARs in mediating transmission at the hair cell rib

142 Low levels of CP accumulated in vivo in the absence of detectable TCV

143 The majority of CP-CRE isolates were bla KPC (92%), followed by bla NDM

144 esent paper develops a mathematical model of CP based on a dynamic network that includes macrophages,

145 The well-characterized caerulein model of CP was used to assess the therapeutic efficacy of Jak1/2

146 In the caerulein-induced murine model of CP, administration of ruxolitinib for one week significa

147 ance, we studied GVL against mouse models of CP-CML, BC-CML, and AML generated by the transduction of

148 However, monomerization of CP 3 in dry 1,2-dichloroethane affords bright green diam

149 New onset of CP-visible GA and central GA.

150 , the incidence, predictors, and outcomes of CP during CTO-PCI were defined.

151 , the incidence, predictors, and outcomes of CP during PCI-CABG were defined.

152 A better understanding of the pathology of CP is needed to facilitate improved diagnosis and treatm

153 of MMP-2/TIMP-2 complex with the presence of CP (P = 0.008) and periodontal parameters PD, CAL, and B

154 alculator that computes the probabilities of CP-visible, new-onset GA and central GA after 1 to 5 yea

155 addition to their function as regulators of CP and actin.

156 factors associated with an increased risk of CP were age and female sex.

157 factors associated with an increased risk of CP were related to CTO complexity.

158 ion strongly related to an increased risk of CP with a close relationship between the number of compl

159 foundation for evaluating putative roles of CP in Ni(II) homeostasis at the host-microbe interface a

160 bsequent genomic and experimental studies of CP.

161 authors interrogated a quantitative trait of CP (mean interproximal clinical attachment level determi

162 ses, it is required for the translocation of CP-AMPARs to these synapses following peripheral inflamm

163 roscopic TPIAT (L-TPIAT) in the treatment of CP.

164 Implications for the use of CP 3 in electronic devices are discussed based on its de

165 designed to clarify the therapeutic value of CP blockade in transplantation.

166 degradation products in some applications of CPs, for example, as metal working fluids.

167 o linear combinations of isotope clusters of CPs and CP-enes.

168 CP-enes are formed by dehydrochlorination of CPs and are expected thermal degradation products in som

169 itions up to and including the generation of CPs.

170 on the nonlinear optical (NLO) properties of CPs and MOFs, with a closer look at the two-photon absor

171 It was shown that thermolysis of CPs follows first-order kinetics.

172 ic, treatment, and ocular characteristics on CP/FA and optical coherence tomography (OCT) as candidat

173 The temporal trends agree with statistics on CP importation in Sweden or local industrial activities.

174 Herein, we shine light on CPs and MOFs as optical media for state-of-the-art photo

175 primary infant composite outcome of death or CP (relative risk [RR] 0.94, 95% confidence interval (CI

176 The primary outcomes were infant death or CP and severe maternal outcome potentially related to tr

177 ignificant reduction in the risk of death or CP with magnesium sulphate treatment compared with no tr

178 s the combined risk of fetal/infant death or CP.

179 es from GAgP when compared with that of H or CP patients.

180 strated that HadA prefers 4-CP and 2-CP over CPs with multiple substituents.

181 hout palate (CLP) and isolated cleft palate (CP) are common human developmental malformations with a

182 tochemical analysis of chronic pancreatitis (CP) and peritumoral areas in PDAC tissues showed that AG

183 ement of patients with chronic pancreatitis (CP) can be challenging.

184 Chronic pancreatitis (CP) is a fibro-inflammatory disease leading to pain, mal

185 Chronic pancreatitis (CP) is a progressive inflammatory disease of the pancrea

186 = 5) or nonmalignant (chronic pancreatitis [CP], n = 15) origin.

187 Chlorinated paraffins (CPs) are high production volume chemicals and ubiquitous

188 26S holocomplex comprises the core particle (CP), where proteolysis takes place, and one or two regul

189 The sensor built on carbon paste (CP) modified with silver nanoparticles (AgNPs).

190 pathways of complement-the classic pathway (CP), the alternative pathway, and the lectin pathway (LP

191 erapeutic approach may be classical pathway (CP) inhibition at the level of early component C1.

192 The classical pathway (CP) of complement may contribute to the pathogenesis of

193 re moderate phenotype chronic periodontitis (CP) (993 cases, 1,419 controls).

194 h aggressive (AgP) or chronic periodontitis (CP) and healthy controls (HC), as well as elucidate its

195 periodontitis (GAgP), chronic periodontitis (CP) and in patients with no history of periodontitis (H)

196 Chronic periodontitis (CP) has a genetic component, particularly its severe for

197 ion studies (GWAS) of chronic periodontitis (CP) have been unsuccessful in discovering susceptibility

198 BDs) in patients with chronic periodontitis (CP).

199 uals with generalized chronic periodontitis (CP).

200 ) 24 individuals with chronic periodontitis (CP); and 2) 23 individuals without CP.

201 healthy (control); 2) chronic periodontitis (CP); and 3) myocardial infarction (MI).

202 y 10% of those who present in chronic phase (CP) can discontinue TKI treatment and maintain a therapy

203 and size of GA in digital color photographs (CPs) and fluorescein angiograms (FAs) taken at baseline

204 eographic atrophy (GA) on color photography (CP) is preceded by specific features on spectral-domain

205 Candidate phyla (CP) are broad phylogenetic clusters of organisms that la

206 ctoderm, neural crest (NC), cranial placode (CP), and non-neural ectoderm in multiple hPSC lines, on

207 ynamic (QED) regime of circularly-polarized (CP) laser-plasma interactions.

208 nanotubes (VACNT) and a conjugated polymer (CP) was fabricated.

209 nusual one-dimensional coordination polymer (CP), 3, prepared from S = 1/2 Co(TPP), 1 (TPP = 5,10,15,

210 -chain boron-containing conjugated polymers (CPs), boron "doped" polyacetylenes (BPAs), which represe

211 oach that lies behind coordination polymers (CPs) and metal-organic frameworks (MOFs) results not onl

212 h (ACD), lens thickness (LT), corneal power (CP), noncycloplegic subjective refraction, and lens nucl

213 ted that these are currently the predominant CPs in use.

214 erm birth for fetal neuroprotection prevents CP and reduces the combined risk of fetal/infant death o

215 It uses cumulative probability (CP) plots for spatially representative data sets, prefer

216 tween patients with carbapenemase-producing (CP)-CRE compared with non-CP-CRE bacteremia.

217 patoblasts (HBs), cholangiocyte progenitors (CPs) expressing early biliary markers and mature CLCs di

218 face recognition, congenital prosopagnosia (CP), and matched controls.

219 ivity and resistance toward capping protein (CP) differ dramatically between both conditions.

220 e actin-binding activity of capping protein (CP), a major capper of actin filament barbed ends in cel

221 P) for a binding site on the capsid protein (CP), and works by altering the angle between capsomers.

222 the Turnip crinkle virus (TCV) coat protein (CP) open reading frame (ORF) has been found to promote i

223 Its genome encodes only its coat protein (CP) subunit, relying on the polymerase of its helper vir

224 Coat protein (CP)- transgenic papaya lines resistant to PRSV isolates

225 gs, fed with 16%, 13% and 10% crude protein (CP) in diets, respectively, were investigated using Illu

226 pounds via functionalizing carrier proteins (CPs) of biosynthetic megaenzymes.

227 Cerato-platanin proteins (CPs), which are secreted by filamentous fungi, are phyto

228 d this study to compare Child-Turcotte-Pugh (CP), MELD, and FIB-4 scores for predicting clinical outc

229 particles (DIPs), into the caudate-putamen (CP) and scored for an innate immune response and inhibit

230 5-methyl-4-isoxazolepropionic acid receptor (CP-AMPAR) mechanism that requires GluA1 phosphorylation

231 urthermore, Ca(2+)-permeable AMPA receptors (CP-AMPARs) increase in the NAc core after 1 month of wi

232 ium-permeable AMPA-type glutamate receptors (CP-AMPARs) after drug withdrawal results in profound rem

233 l mutants further functionalized recombinant CP substrates.

234 varying specificities toward 11 recombinant CPs of different types of biosynthetic pathways from cya

235 Selective CP blockade may be a promising strategy to counteract re

236 Ex vivo serum CP activity was profoundly inhibited when TNT009 concent

237 The bonding in solid CP 3 involves noninteger redox states in a resonance hyb

238 ase membrane introduction mass spectrometry (CP-MIMS) is an online, in situ analysis technique for lo

239 was active in the gRNA invivo Since the TCV CP also serves as the viral silencing suppressor, early

240 sing complementary spectroscopic techniques (CP/MAS (13)C NMR, Raman, FT-IR, and XPS) and high-resolu

241 and SELE were highly expressed in GAgP than CP or H patients.

242 We also demonstrate that CP can sequester Ni(II) from two human pathogens, Staphy

243 EPR) spectroscopic analyses demonstrate that CP outcompetes staphylococcal MntC and streptococcal Psa

244 bullfrog auditory synapses demonstrate that CP-AMPARs mediate a major component of glutamatergic tra

245 We demonstrate that CP-AMPARs mediate transmission at this first-order senso

246 Collectively, our results indicate that CP-AMPAR-mediated maturation of silent synapses in the N

247 However, they raise the possibility that CP-AMPAR accumulation in the NAc core occurs in a popula

248 II)- and Ca(II)-bound CP, which reveals that CP binds Ni(II) ions at both its transition-metal-bindin

249 Here, we show that CP-CML, BC-CML, and AML stem cells upregulate MHCII in a

250 Our findings suggest that CP-CRE may be more virulent than non-CP-CRE and are asso

251 s over the last 50-80 years, indicating that CPs are persistent chemicals regardless of alkane-chain

252 The CP-binding domain of CARMIL consists of the CP-interacti

253 The CP-fit approach for assessing heritability was implement

254 en the healthy and CP groups and between the CP and MI groups.

255 complex, C4bC2a, which is formed by both the CP and the LP.

256 kines that are present at high levels in the CP microenvironment.

257 ed within 14 days, including 12 (32%) in the CP-CRE group and 6 (13%) in the non-CP-CRE group.

258 These include the CP-binding motif, the relative placement of PS stem-loop

259 unfolds and translocates substrates into the CP.

260 found to promote internal expression of the CP from the genomic RNA (gRNA) both in vitro and in vivo

261 lencing suppressor, early translation of the CP from the viral gRNA is likely important for counterin

262 Infusion of the CP-AMPAR-specific blocker Naspm into the NAc shell rever

263 CP-binding domain of CARMIL consists of the CP-interaction (CPI) and CARMIL-specific interaction (CS

264 f PRSV has allowed the virus to overcome the CP-mediated transgenic resistance.

265 ate (s4), the gate controlling access to the CP is open.

266 rd-order tensor, and decomposed it using the CP algorithm.

267 d with the CP and MI groups but not when the CP group was compared with the MI group.

268 when the healthy group was compared with the CP and MI groups but not when the CP group was compared

269 ked proteasome, but clashes instead with the CP in the ATP-hydrolysis-competent proteasome.

270 g in controls, this was not the case for the CPs.

271 qTOF-HRMS followed by quantification of the CPs by a mathematical algorithm.

272 CARMILs bind directly to CP and induce a conformational change that allostericall

273 cs, and retinal pathology-for progression to CP-visible GA over up to 5 years.

274 vidually accounted for over 95% of the total CP-PCB TEQ.

275 tion of cocaine craving and does not undergo CP-AMPAR plasticity.

276 catalyze oxidative dechlorination of various CPs, and as such it will serve as the basis for future i

277 Further, excess Ca(2+) entry via CP-AMPARs may underlie afferent terminal damage followin

278 iments in which viral RNA competes for viral CP with polyUs of equal length show that polyU, despite

279 Indication for surgery was CP attributable to genetic mutation (n = 9), idiopathic

280 The gain in EpMM was larger when CPs were listed with unacceptable antigens.

281 tion required IFN-gamma stimulation, whereas CP-CML MHC upregulation was independent of both the IFN-

282 aintained responding in the monkeys in which CP 55 940 functioned as a reinforcer.

283 dictors of body composition in children with CP between the ages of 18 and 60 mo.

284 The mean %BF of children with CP classified them as overfat or obese.

285 pulation-based cohort study of children with CP who were aged 18-60 mo (364 assessments in 161 childr

286 Saliva samples from individuals with CP (n = 75), AgP (n = 20), and HC (n = 69) were collecte

287 LRP3, ASC, and IL-1beta salivary levels with CP and AgP.

288 Treatment of mice with CP also leads to differentiation of a regulatory subset

289 Patients with CP (n = 14) with 42 contralateral 2- and 3-walled defect

290 and radiographic parameters of patients with CP at the 6-month follow-up.

291 elial function was impaired in patients with CP compared with healthy patients, and the dysfunction o

292 P plus locally delivered MF in patients with CP in both initial PD = 5 to 7 and >7 mm subgroups compa

293 TR in the treatment of IBDs in patients with CP in terms of both clinical and radiologic outcomes.

294 zed PSC obtained from mice and patients with CP or pancreatic cancer to examine the effect of Jak/STA

295 Thirty-two patients with CP presented for TPIAT at a tertiary hospital from Janua

296 s were significantly higher in patients with CP than in healthy individuals (P <0.001) and decreased

297 s and prognostic impact of 598 patients with CP-CML treated on clinical trials with various TKIs.

298 P<0.001) were more frequent in patients with CP.

299 Antagonism of CRF-R1 with CP-154526 caused an accumulation of intracellular calciu

300 dontitis (CP); and 2) 23 individuals without CP.