ライフサイエンスコーパス: CREB-binding protein

1 N-beta promoter and its coactivator protein (CREB-binding protein).

2 CBP (cAMP-response element-binding protein (CREB)-binding protein).

3 F-kappaB by physically interacting with p300/CREB binding protein.

4 omplexes on binding to the KIX domain of the CREB binding protein.

5 the transcriptional regulators E1A(p300) and CREB binding protein.

6 the levels of NF-kappaB p65 associated with CREB-binding protein.

7 gated the transcription-activating effect of CREB-binding protein.

8 n domains of the transcriptional coregulator CREB-binding protein.

9 ID folding upon binding to the KIX domain of CREB-binding protein.

10 -catenin to its transcriptional coactivator, CREB-binding protein.

11 tenin by PKA, promoting its interaction with CREB-binding protein.

12 osed of phosphorylated ATF-2, C/EBPbeta, and CREB-binding protein.

13 ver, these lysines become acetylated by p300/CREB-binding protein.

14 go structural changes when it interacts with CREB-binding protein.

15 3 bound to its transcriptional cofactor, the CREB-binding protein.

16 ses and the nuclear calcium signaling target CREB-binding protein.

17 d binding of the transcriptional coactivator CREB-binding protein 1 to transcriptional complexes cont

18 teraction of the Smad complex with p300/CBP (CREB-binding protein), a co-activator with intrinsic ace

19 nteract with the transcriptional coactivator CREB-binding protein, a function not shared with the clo

20 miR-BART16 directly targets CREB-binding protein, a key transcriptional coactivator

21 ate (cAMP) response element-binding protein (CREB)-binding protein-a histone acetyltransferase (HAT)

22 duction of this coactivator, but not that of CREB-binding protein, abolishes the synergistic effect i

23 of a p53 mutant with mutations of known p300/CREB-binding protein acetylation sites demonstrated that

24 The Snf-2-related CREB-binding protein activator protein (SRCAP) serves as

25 strated previously that SRCAP (SNF-2-related CREB-binding protein activator protein), the human ortho

26 e, EMSAs demonstrated that the attachment of CREB binding protein and activating transcription factor

27 pha, RNA polymerase II, and the coactivators CREB binding protein and p300 to the CD40 promoter, as w

28 ators cAMP-response element-binding protein (CREB)-binding protein and steroid receptor coactivators

29 as accompanied by reduced recruitment of the CREB-binding protein and altered post-translational modi

30 IKK-alpha interacts with CREB-binding protein and in conjunction with Rel A is re

31 Recent reports on two such factors, CREB-binding protein and methyl-CpG-binding protein 2, h

32 rs including the histone acetyl transferases CREB-binding protein and NCoA-1 and other proteins such

33 We also noted that the acetyltransferases CREB-binding protein and p300 both can acetylate ERK1/2.

34 t IRF3 and IRF7, which with the coactivators CREB-binding protein and P300 form the virus-activated f

35 s, c-Jun, and the histone acetyltransferases CREB-binding protein and p300 to the IL-6 promoter in vi

36 f the nuclear co-activator binding domain of CREB-binding protein and the activation domain from the

37 n the reaction between the KIX domain of the CREB-binding protein and the transactivation domain of c

38 ade of the transcriptional coactivators CBP (CREB binding protein) and p300 as an alternative approac

39 utoinhibition to allow interaction with CBP (CREB-binding protein) and facilitates phosphorylation at

40 he binding epitopes in the ZZ domain of CBP (CREB-binding protein) and SUMO1 using NMR spectroscopy.

41 tivity was augmented by the coactivator CBP (CREB-binding protein) and was dependent on the p38 pathw

42 romatin immunoprecipitations employing CREB, CREB-binding protein, and acetylated H4 antibodies ident

43 tional coactivators/acetyltransferases, p300/CREB-binding protein, and p300/CREB-binding protein-asso

44 anscriptional synergism with the coactivator CREB-binding protein, and rescue of susceptibility to vi

45 r of the thyroid and retinoic acid receptor, CREB-binding protein, and steroid receptor coactivator 1

46 prevents its interaction with a coactivator, CREB-binding protein, and subsequently reduces the BDNF

47 te transcription factors, the recruitment of CREB-binding protein, and the acetylation of histone H3

48 that include histone acetyltransferase p300/CREB-binding protein, as a critical mediator of acetylat

49 p300/CREB binding protein associated factor (PCAF/KAT2B) and

50 at acetylation of histone H3 by the p300 and CREB-binding protein associated factor, PCAF, suppressed

51 etylated by acetyltransferases p300 and p300/CREB-binding protein associated factor.

52 Thr312 phosphorylation, acetylation by p300/CREB binding protein-associated factor, and detachment f

53 the subsequent interaction of Fli1 with p300/CREB-binding protein-associated factor (PCAF) and an ace

54 The transcriptional coactivators, p300/CREB-binding protein-associated factor (PCAF) and hGCN5,

55 f WRN(K577M), diminishes recruitment of p300/CREB-binding protein-associated factor (PCAF) and positi

56 r of the tumor suppressor p53, inhibits p300/CREB-binding protein-associated factor (PCAF)-mediated p

57 ferases, p300/CREB-binding protein, and p300/CREB-binding protein-associated factor (PCAF); and induc

58 report that after differentiation, the p300/CREB-binding protein-associated factor is recruited by F

59 recruits the histone acetyltransferase p300/CREB-binding protein-associated factor to chromatin.

60 ely 1000-fold more efficient than PCAF (p300/CREB-binding protein-associated factor)-mediated acetyla

61 HDAC4, and a histone acetyltransferase, p300/CREB-binding protein-associated factor, associate with c

62 AMP response element-binding protein (CREB)/CREB-binding protein binding, accumulation of activating

63 inducible domain interacting (KIX) domain of CREB binding protein binds to multiple intrinsically dis

64 main of the transcriptional coactivator CBP (CREB binding protein) binds specifically to p53 at the C

65 om displacement of Smad binding to DNA or to CREB-binding protein but from the recruitment of Evi-1 b

66 caused by mutations in the gene encoding the CREB binding protein (CBP) and characterized by mental r

67 responsive element binding protein 1 (CREB)/CREB binding protein (CBP) and inhibits PAX7 transcripti

68 f the histone acetylase coactivator paralogs CREB binding protein (CBP) and p300 to the promoter.

69 IF1alpha protein and recruiting coactivators CREB binding protein (CBP) and p300, and RNA polymerase

70 preferential recruitment of the coactivators CREB binding protein (CBP) and p300.

71 CREB binding protein (CBP) and p300/CBP-associated facto

72 The transcriptional coactivators CREB binding protein (CBP) and the closely related p300,

73 structures of the lead compound bound to the CREB binding protein (CBP) and the first bromodomain of

74 esult of its avid binding to the coactivator CREB binding protein (CBP) and the mammalian mediator co

75 ctor 4alpha (HNF4alpha), and the coactivator CREB binding protein (CBP) are required for the glucose

76 hosphorylate the transcriptional coactivator CREB binding protein (CBP) at serine 436 via PKC iota/la

77 The histone acetyltransferase CREB binding protein (CBP) can bind to TCF and inhibit W

78 The KIX domain of CREB binding protein (CBP) forms a small three-helix bun

79 the Notch1 gene, where it serves to recruit CREB binding protein (CBP) histone acetyltransferase.

80 or the histone acetyltransferase activity of CREB binding protein (CBP) in long-term memory and plast

81 CREB binding protein (CBP) is a coactivator of transcrip

82 CREB binding protein (CBP) is a transcriptional coactiva

83 cible domain interacting (KIX) domain of the CREB binding protein (CBP) is capable of simultaneously

84 While the transcriptional coactivator CREB binding protein (CBP) is known to interact with Egr

85 binding protein (ChREBP) and the coactivator CREB binding protein (CBP) on the L-PK promoter.

86 We found that the monoallelic deletion of CREB binding protein (CBP) results in the induction of E

87 for recruiting the histone acetyltransferase CREB binding protein (CBP) to the FoxM1B transcriptional

88 the latter of which are known to recruit the CREB binding protein (CBP) transcriptional coactivator.

89 We identify the CREB binding protein (CBP) transcriptional cofactor as e

90 tivity (ir) detecting the Fos, pCREB, egr-1, CREB binding protein (CBP), and calbindin-D (28K) protei

91 AMP response element-binding protein (CREB), CREB binding protein (CBP), and histone deacetylases (HD

92 ATA-1 (FOG-1), the histone acetyltransferase CREB binding protein (CBP), and the key component of the

93 h as the general transcriptional coactivator CREB binding protein (CBP), its paralogue p300, and the

94 effects of TGFbeta+FSK on activated pSmad3, CREB binding protein (CBP), MAPKs, and RhoA were determi

95 Here we show that obesity promotes the CREB binding protein (CBP)-mediated acetylation of CREB

96 JIL-1 is also required for CREB binding protein (CBP)-mediated acetylation of Lys27

97 at polyglutamine-expanded AR interferes with CREB binding protein (CBP)-mediated transcription of vas

98 of the transcriptional coactivators p300 and CREB binding protein (CBP).

99 activators and transcriptional co-activator CREB binding protein (CBP).

100 n factor c-myb to the cotranscription factor CREB binding protein (CBP).

101 Phosphorylation of CREB at Ser133 recruits CREB binding protein (CBP)/p300 coactivators to activate

102 53 function requires an association with the CREB binding protein (CBP)/p300 coactivators, and a tern

103 Members of the CREB binding protein (CBP)/p300 family have been shown t

104 strongly recruits the cellular coactivators CREB binding protein (CBP)/p300 to the viral promoter co

105 l transcription factor binding domain of the CREB binding protein (CBP)/p300, KIX, we report the firs

106 ogous cAMP-response element-binding protein (CREB) binding protein (CBP) gene together are mutated in

107 AMP (cAMP) response element binding protein (CREB) binding protein (CBP).

108 include cyclic-AMP response element binding (CREB) binding protein (CBP)/p300 and the retinoblastoma

109 , the cAMP response element binding protein (CREB) binding protein (CBP)/p300 family and the recently

110 ts in cAMP response element-binding protein (CREB) binding protein (CBP; an essential cofactor for ac

111 sive element), activate CRE-binding protein (CREB)-binding protein (CBP) and gene activities causing

112 ate (cAMP) response element-binding protein (CREB)-binding protein (CBP) and is expressed in the deve

113 sine monophosphate response element binding (CREB)-binding protein (CBP) and p300 are multidomain tra

114 cyclic-AMP response element-binding protein (CREB)-binding protein (CBP) and p300, which activate tra

115 sferase cyclic-AMP response element binding (CREB)-binding protein (CBP) during this process.

116 y the cAMP-response element binding protein (CREB)-binding protein (CBP) mediates sensitivity to coca

117 cAMP response element-binding protein (CREB)-binding protein (CBP) that interacts preferentiall

118 ) and cAMP response element binding protein (CREB)-binding protein (CBP) to chromatin.

119 cyclicAMP response element binding protein (CREB)-binding protein (CBP), a histone acetyltransferase

120 p300/cAMP-response element-binding protein (CREB)-binding protein (CBP)-associated factor localize d

121 duced cAMP response element-binding protein (CREB)-binding protein (CBP)-mediated H3K9/14 hyperacetyl

122 tenin/cAMP-response element-binding protein (CREB)-binding protein (CBP)-mediated transcription, but

123 ty of cAMP-response element-binding protein (CREB)-binding protein (CBP)/p300.

124 he Drosophila melanogaster acetyltransferase CREB-binding protein (CBP) acetylates histone H3 lysine

125 PRLR-recruited CREB-binding protein (CBP) acetylates multiple lysine si

126 p300 and CREB-binding protein (CBP) act as multifunctional regula

127 to the nucleus where it forms a complex with CREB-binding protein (CBP) and acetylated RelA/p65 causi

128 REB-serine 133 phosphorylation as well as in CREB-binding protein (CBP) and Bcl-2 levels.

129 ershift EMSA identified FOXA2 to rs327T, and CREB-binding protein (CBP) and CCAAT displacement protei

130 inds to the MHC-II histone acetyltransferase CREB-binding protein (CBP) and is critical for the recru

131 CREB-binding protein (CBP) and its para-log p300 are tra

132 acts with transcriptional co-activators like CREB-binding protein (CBP) and its paralog p300 in addit

133 The histone acetyl transferases CREB-binding protein (CBP) and its paralog p300 play a c

134 association with transcription coactivators CREB-binding protein (CBP) and its paralog p300 to activ

135 resulting in recruitment of the coactivators CREB-binding protein (CBP) and p300 and subsequent activ

136 CREB-binding protein (CBP) and p300 are highly homologou

137 Similarly, the transcription cofactors CREB-binding protein (CBP) and p300 are reduced in the p

138 he related histone acetyltransferases (HATs) CREB-binding protein (CBP) and p300 are required for end

139 The co-activators CREB-binding protein (CBP) and p300 are transcriptional

140 Here, we identified CREB-binding protein (CBP) and p300 as major histone ace

141 s cells, N-terminal E1A mutants defective in CREB-binding protein (CBP) and p300 binding capacity exh

142 the homologous transcriptional coactivators CREB-binding protein (CBP) and p300 forms a complex with

143 s of specific acetyltransferases such as the CREB-binding protein (CBP) and p300 have been well docum

144 The transcriptional coactivators CREB-binding protein (CBP) and p300 undergo a particular

145 of the related transcriptional coactivators CREB-binding protein (CBP) and p300, an oxygen-regulated

146 are the histone acetyltransferase paralogues CREB-binding protein (CBP) and p300.

147 e T3-induced recruitment of the co-activator CREB-binding protein (CBP) and release of nuclear recept

148 o be composed of the enzymatic activities of CREB-binding protein (CBP) and SMARCAD1, which belongs t

149 The global transcriptional coactivators CREB-binding protein (CBP) and the closely related p300

150 Mutations in the coactivator CREB-binding protein (CBP) are a major cause of the huma

151 highly related acetyltransferases, p300 and CREB-binding protein (CBP) are coactivators of signal-re

152 e have determined that Ku70, Ku86, p300, and CREB-binding protein (CBP) are ESE-1 interacting protein

153 istone acetyltransferase paralogues p300 and CREB-binding protein (CBP) are key transcriptional co-ac

154 tional coactivator/histone acetyltransferase CREB-binding protein (CBP) as being highly expressed in

155 Mutations in the gene encoding CREB-binding protein (CBP) cause deficits in long-term p

156 l synergy was mediated by the recruitment of CREB-binding protein (CBP) coactivator through the HNF6

157 cruitment of the normally rate-limiting p300/CREB-binding Protein (CBP) coactivator to the transcript

158 teractions that preclude recruitment of p300/CREB-binding protein (CBP) coactivators.

159 Because p300 does not substitute for all CREB-binding protein (CBP) functions, we investigated wh

160 ouse MRTF-A(-/-) cells or upon inhibition of CREB-binding protein (CBP) histone acetyltransferase (HA

161 mmunoprecipitation assays revealed that p300/CREB-binding protein (CBP) histone acetyltransferases an

162 ls SATB1 family protein forms a complex with CREB-binding protein (CBP) important in transcriptional

163 REB function via brain viral delivery of the CREB-binding protein (CBP) improves learning and memory

164 e present study identifies the importance of CREB-binding protein (CBP) in facilitating TNF-alpha-med

165 eracts with the transcriptional co-activator CREB-binding protein (CBP) in nuclear speckle domains in

166 tivity of the histone acetyltransferase p300/CREB-binding protein (CBP) in regulating promoter-proxim

167 ecule specific inhibitor of the beta-catenin/Creb-binding protein (CBP) interaction.

168 CREB-binding protein (CBP) is a large, multi-domain prot

169 y reported that the lysine acetyltransferase CREB-binding protein (CBP) is required for HIF-2alpha ac

170 The histone acetyltransferase CREB-binding protein (CBP) mediates transcriptional acti

171 is associated with increased binding of p300/CREB-binding protein (CBP) molecules at the Fos promoter

172 followed by recruitment of the coactivators CREB-binding protein (CBP) or p300.

173 We recently showed that p300/CREB-binding protein (CBP) plays an important role in ma

174 The multifunctional Creb-binding protein (CBP) protein plays a pivotal role

175 that CIITA requires the ability to bind the CREB-binding protein (CBP) to effectively inhibit MMP-9

176 Apoptosis was mediated by recruitment of CREB-binding protein (CBP) to the gamma-activating seque

177 that the HoxA10 PQ-like domain recruited the CREB-binding protein (CBP) to the ITGB3 promoter.

178 gr-1, and Sp1 transcription factors and that CREB-binding protein (CBP) transcriptional complexes for

179 al coactivator and histone acetyltransferase CREB-binding protein (CBP) via the CREB-binding domain o

180 criptional coactivator and acetyltransferase CREB-binding protein (CBP) via the KIX domain of CBP.

181 grated by the acetyltransferase co-activator CREB-binding protein (CBP) was suggested by bioinformati

182 causes to the interactions of KIX domain of CREB-binding protein (CBP) with phosphorylated kinase in

183 nin with either the Kat3 coactivator CREBBP (CREB-binding protein (CBP)) or the highly homologous EP3

184 orepressors (e.g. Ski) or coactivators (e.g. CREB-binding protein (CBP)), respectively.

185 ntly reduced by siRNA-mediated knock down of CREB-binding protein (CBP), a transcriptional co-activat

186 m regulatory element, the recruitment of the CREB-binding protein (CBP), and chromatin remodeling inc

187 nteracts with a transcriptional coactivator, CREB-binding protein (CBP), in the nucleus.

188 ein tethered to the LacO locus recruits p300/CREB-binding protein (CBP), induces histone hyperacetyla

189 that is acetylated by its coactivator, p300/CREB-binding protein (CBP), resides in the C-terminal tr

190 kappaB and STAT1, as well as the coactivator CREB-binding protein (CBP), to facilitate transcriptiona

191 ingly, the nuclear transcription coactivator CREB-binding protein (CBP), which can bind to Nrf2 trans

192 rk, we showed that survivin is acetylated by CREB-binding protein (CBP), which restricts its localiza

193 o role of the histone acetyltransferase p300/CREB-binding protein (CBP), which was reported to be a c

194 omoting its association with the coactivator CREB-binding protein (CBP), which was required to regula

195 lear accumulation of survivin is promoted by CREB-binding protein (CBP)-dependent acetylation on lysi

196 ously reported that HOX proteins can inhibit CREB-binding protein (CBP)-histone acetyltransferase-med

197 otein and the KIX domain of its coactivator, CREB-binding protein (CBP).

198 tion domain of the histone acetyltransferase CREB-binding protein (CBP).

199 gulator in cooperation with FGF-2, RSK1, and CREB-binding protein (CBP).

200 well as its interaction with the coactivator CREB-binding protein (CBP).

201 pha), IFNalpha receptors recruit cytoplasmic CREB-binding protein (CBP).

202 he fourth intrinsically disordered linker of CREB-binding protein (CBP).

203 presence of its transcriptional coactivator, CREB-binding protein (CBP).

204 nase A allows recruitment of the coactivator CREB-binding protein (CBP).

205 ion of other transcription cofactors such as CREB-binding protein (CBP).

206 en CREB and its transcriptional coactivator, CREB-binding protein (CBP).

207 h the transcriptional co-activators p300 and CREB-binding protein (CBP).

208 onized by TRX-dependent H3K27 acetylation by CREB-binding protein (CBP).

209 g domain of the transcriptional coactivator, CREB-binding protein (CBP).

210 hypoxia-inducible factor (HIF)-1alpha or the CREB-binding protein (CBP).

211 usly demonstrated that human SREBPs bind the CREB-binding protein (CBP)/p300 acetyltransferase KIX do

212 The coactivator CREB-binding protein (CBP)/p300 binds to this promoter-b

213 CREB-binding protein (CBP)/p300 interacting transactivat

214 Although acetylation of p53 by CREB-binding protein (CBP)/p300 is known to be indispens

215 es and recruits transcriptional coactivators CREB-binding protein (CBP)/p300 to effect potent transcr

216 activation recruits CREB and the coactivator CREB-binding protein (CBP)/p300 to the endogenous CNS-1.

217 Smads and beta-catenin/TCF4 was dependent on CREB-binding protein (CBP)/P300, as demonstrated by over

218 iggers IRF-3 to react with the coactivators, CREB-binding protein (CBP)/p300, to form a complex that

219 tion of p65 at Thr-305 and Ser-319 increased CREB-binding protein (CBP)/p300-dependent activating ace

220 We have previously shown that CREB-binding protein (CBP/p300) act as an important SOX9

221 Here we show that PAF increases Acetyl-CREB-binding protein (CBP/p300) histone acetyltransferas

222 ociated with the transcriptional coactivator CREB-binding protein (CBP/p300).

223 of candidate enhancer regions using data on CREB-binding protein co-factor binding or ATAC-seq chrom

224 C/EBPbeta binding to the TAZ2 region of p300/CREB-binding protein coactivators.

225 oding the histone acetyl-transferases (HATs) CREB binding protein (CREBBP) and EP300 are recurrently

226 these are two compounds, ICG-001, targeting CREB binding protein (CREBBP), and PKF118-310, targeting

227 Selective inhibitors of the CREB binding protein (CREBBP)/EP300 bromodomains are req

228 CREB-binding protein (CREBBP) is important for the cell-

229 al coactivator and histone acetyltransferase CREB-binding protein (CREBBP, also known as CBP).

230 Importantly, we discovered that Drosophila CREB-binding protein (dCBP) is a co-activator for Caudal

231 Drosophila CREB-binding protein (dCBP) is a very large multidomain

232 beta-catenin C-terminal interactions inhibit Creb-binding protein-dependent p53 acetylation and p53 t

233 f the homologous Kat3 co-activators, p300 or CREB-binding protein, differentially regulates maintenan

234 usion of integration host factor beta or the CREB-binding protein domain upstream to GFP resulted in

235 activator in IFN signaling, thereby inducing CREB-binding protein downregulation in EBV-transformed B

236 CREB-binding protein dramatically decreased the half-lif

237 Significant alterations in CREB binding protein expression and localization were no

238 ly showed that in quiescent cells, p300/CBP (CREB-binding protein)family coactivators repress c-myc a

239 The nuclear coactivator binding domain of CREB binding protein folds into remarkably different str

240 ors depleted the transcriptional coactivator CREB-binding protein from the NF-kappaB complex in the n

241 Our data also show that a proposed CREB-binding protein histone acetyltransferase protein-r

242 , and cAMP-response element-binding protein (CREB)-binding protein in the common regulatory region of

243 estigated the role of p300 and its homologue CREB-binding protein in prostate cancer cells treated ch

244 5, and 4 and the transcriptional coactivator CREB-binding protein in the BMP-responsive element-prote

245 demonstrated that overexpression of p300 or CREB-binding protein increases the PKCdelta promoter act

246 Here we show that CREB-binding protein induced acetylation of Nrf2, increa

247 We found that p300 but not CREB-binding protein induced activation of PSA in these

248 Moreover, overexpression of TTP blocked CREB-binding protein-induced acetylation of p65/NF-kappa

249 and FRET microscopy for monitoring CREB and CREB-binding protein interaction in the nuclei of live c

250 Furthermore, CREB-binding protein markedly increased transcription of

251 a1 promoter, suggesting that competition for CREB-binding protein may contribute to STAT1-dependent d

252 nd the nuclear coactivator binding domain of CREB-binding protein (NCBD), along with their bimolecula

253 itment of HDAC1 and increased recruitment of CREB-binding protein on the Mcp-1 promoter in TTP(-/-) c

254 fnb1, along with IFN regulatory factor-3 and CREB binding protein only during concomitant UPR and LPS

255 nteracts with co-activators such as p300 and CREB-binding protein or co-repressors such as HDAC3.

256 Overexpression of the acetyltransferases CREB-binding protein or p300 resulted in the acetylation

257 as are the transcription coregulators p300, CREB-binding protein, p/CIP, and SRC-1.

258 ltered, GTP mutants no longer cooperate with CREB-binding protein, p300, and pCAF and are defective i

259 and the recruitment of STAT-3, c-Jun, c-Fos, CREB-binding protein, p300, and RNA polymerase II to the

260 ts of the transcriptional machinery, such as CREB-binding protein, p300, RNA polymerase II and histon

261 cting (KIX) domain of the master coactivator CREB binding protein/p300 is a conformationally dynamic

262 dition, the activation potential of SOX9 and CREB binding protein/p300 on the cd-rap promoter was enh

263 nted recruitment of IFN regulatory factor-3, CREB binding protein/p300, and transcriptional machinery

264 sequestering the transcriptional coactivator CREB binding protein/p300.

265 viral promoters through an interaction with CREB binding protein/p300.

266 onventional global transcription coactivator CREB-binding protein/p300 (CBP/p300) that binds to the H

267 MLL1 (mixed lineage leukemia 1) and MLL3 and CREB-binding protein/p300 bind to the promoter of HOTAIR

268 arly growth response factor 1, together with CREB-binding protein/p300 coactivators, bind to Ealpha a

269 he transcription factor TCF4 and coactivator CREB-binding protein/p300 in the Wnt pathway.

270 onist, TRPC4 channel blocker, and CaMKII and CREB-binding protein/p300 inhibitors.

271 iting HIF, and the oncogenic factor, CITED2 (CREB-binding protein/p300 interacting transactivator wit

272 However, overexpression of the coactivator CREB-binding protein/p300 reduced the inhibitory actions

273 mediates the recruitment of the coactivators CREB-binding protein/p300 to the HTLV-1 promoter, locate

274 rs by SoxE proteins, displacing coactivators CREB-binding protein/p300 while promoting the recruitmen

275 knocking down the histone acetyltransferase CREB-binding protein/p300, known to target H3K27, render

276 iated protein by interaction cloning was the CREB-binding protein/p300-interacting transactivator wit

277 /calmodulin-dependent protein kinase IV-CREB/CREB-binding protein pathway.

278 B/CBP (cAMP-response element-binding protein/CREB-binding protein) pathway that regulates transcripti

279 lear translocation, dimerization, binding to CREB-binding protein, recognition of DNA, and induction

280 We found that p300, a CREB-binding protein-related protein, interacts with KLF

281 association of activated C/EBPbeta with p300/CREB-binding protein requires the LX2 and AID auto-inhib

282 o the cAMP response element binding protein (CREB)-binding protein reveals that the pLxIS motif also

283 es against CREB, phospho-CREB, and CBP/p300 (CREB-binding protein) showed that these proteins associa

284 ivity, strengthened the affinity of Smad3 to CREB-binding protein, suggesting that linker phosphoryla

285 to the KIX-binding domain of the coactivator CREB-binding protein, supporting the binding and traffic

286 ions affecting the transcriptional regulator CREB binding protein, the small GTPase dynamin, the cyto

287 kappaB activation by blocking the binding of CREB binding protein to the NF-kappaB complex, thereby l

288 omplex with the transcriptional co-activator CREB-binding protein to activate transcription.

289 t from the recruitment of Evi-1 by Smad3 and CREB-binding protein to DNA.

290 ter of CNTF, and the recruitment of CREB and CREB-binding protein to the CNTF promoter by aspirin sug

291 preferential recruitment of the coactivator CREB-binding protein to those promoters.

292 biquitin ligase, and both interact with p300-CREB-binding protein transcriptional coactivator protein

293 the retinoblastoma protein family, the p300/CREB-binding protein transcriptional coactivators, and t

294 IF function requires the recruitment of p300/CREB-binding protein, two coactivators with histone acet

295 rdered nuclear coactivator binding domain of CREB binding protein (UniProt CBP_MOUSE P45481 ), residu

296 CREB, which interacts with the KIX domain of CREB-binding protein upon phosphorylation.

297 of Brd4 and BrdT, and the KIX domain of the CREB-binding protein) using commercially available fluor

298 1 when an antibody against CREB, Sp1, or the CREB-binding protein was used.

299 ubdomains interact with the coactivator CBP (CREB-binding protein), which is required for NFATp-depen

300 itment of a transcriptional coactivator CBP (CREB binding protein) without modulation of CREB binding