ライフサイエンスコーパス: CRP

1 CRP >/=100 mg/L substantially improved specificity over

2 CRP and SAP dose-dependently and substoichiometrically i

3 CRP concentrations were measured at study entry with a p

4 CRP level was positively correlated with diabetes preval

5 CRP may be useful for distinguishing bacterial from RSV-

6 CRP remained higher in HIV-exposed vs HIV-unexposed infa

7 CRP selectivity is obtained by means of the adhesion of

8 CRP was higher in HIV-exposed than HIV-unexposed infants

9 CRP, IL-6, and I-FABP were not associated with worse cog

10 CRP, in its native pentameric structural conformation, b

11 CRP, in its non-native pentameric structural conformatio

12 CRP, sCD14, and HA levels decreased during ART but remai

13 CRP, TNF, sIL-6R, I-FABP, sCD14, D-dimer, and HA levels

14 CRPs prevent attack on host cells by the complement syst

15 S [0-C-reactive protein (CRP) </= 10 mg/L, 1-CRP > 10 mg/L and albumin >/=35 g/L, 2-CRP > 10 mg/L and

16 FHR-1/CRP interactions increased complement activation via the

17 The pooled odds ratio estimate using 18 CRP genetic instruments was 0.90 (random effects 95% CI,

18 /L, 1-CRP > 10 mg/L and albumin >/=35 g/L, 2-CRP > 10 mg/L and albumin < 35 g/L], TNM stage, and canc

19 djusted for gender, recent infections, and a CRP genetic risk score, hs-CRP at 7 years was associated

20 -42, C4b-binding protein, and factor H, in a CRP concentration- and ligand concentration-dependent ma

21 ty is obtained by means of the adhesion of a CRP specific aptamer chain onto the ITO film using the L

22 erum concentrations of glucose, adiponectin, CRP, or IL-6 in the US compared with the Mexican diet.

23 Confounders included age, CRP, and lifestyle and sociodemographic factors.

24 and activated with the GPVI specific agonist CRP (collagen-related peptide).

25 0.273; Spearman rho = 0.581; P < 0.001) and CRP (R(2) = 0.132; Spearman rho = 0.455, P < 0.001).

26 : 1.70 (95% CI: 1.01, 2.88); P = 0.038], and CRP concentrations >/=10 mg/L [52% compared with 33%, re

27 Among biomarkers, we found GDF-15 and CRP to be strongly associated with all-cause mortality (

28 Inclusion of GDF-15 and CRP to the Society of Thoracic Surgeons score significan

29 Adipokines, IL-6 and CRP levels were measured at admission and on 3rd day of

30 nfected infants had highest sCD14, IL-6, and CRP concentrations (P < .001) and marginally higher I-FA

31 In addition, we studied H-NS, CpxR and CRP global regulators, on Longus expression.

32 behavioral, and cardiometabolic factors, and CRP and interleukin-6, each standard deviation increase

33 ctants proteins (fibrinogen, haptoglobin and CRP), cell adhesion molecules (VCAM-1), endothelial grow

34 tty acids, amino acids, small molecules, and CRP.

35 In contrast, H-NS and CRP function as negative regulators since expression of

36 H-NS and CRP were required for salt- and glucose-mediated regulat

37 or biomarkers shows that in 3rd POD, PCT and CRP have similar area under the curve (AUC) (0.775 vs 0.

38 Measuring together PCT and CRP significantly improves AL diagnosis in 5th POD (AUC:

39 ce supporting the interplay between PMAs and CRP in patients with VTE.

40 In women, the correlations between RBP and CRP and AGP were too weak to justify adjustments for inf

41 Decreases in sCD14 and CRP levels were correlated with increases in CD4 T-cell

42 TL and CRP levels may help predict the impact of BC exposure on

43 TL and CRP were associated neither with each other nor with MMS

44 GCKR function, increased triglycerides, and CRP.

45 n PSC.The relation between estimated VAD and CRP and AGP deciles followed a linear pattern in PSC.

46 fR concentrations incrementally decreased as CRP and AGP deciles decreased for PSC and WRA, but the e

47 n-store estimates incrementally increased as CRP and AGP deciles decreased (4% compared with 30%, and

48 Many studies associated CRP level with diabetes and glucose levels, but the asso

49 showed that patients with elevated baseline CRP levels receiving the higher dose of NP001 had signif

50 compared with patients with normal baseline CRP, regardless of whether patients with normal CRP leve

51 ns uncertain whether the association between CRP concentrations and AMD is causal.

52 do not support a causal association between CRP concentrations and AMD.

53 APPHIRe) to investigate associations between CRP polymorphisms, circulating CRP, diabetes, and glucos

54 l class of Arabidopsis thaliana pollen-borne CRPs, the PCP-Bs (for pollen coat protein B-class) that

55 eNOS, endothelial FcgammaRIIB activation by CRP blunts insulin delivery to skeletal muscle to cause

56 C-reactive protein (CRP) encoded by CRP gene is a reflection of systemic inflammation.

57 s with increased inflammation as measured by CRP.

58 6R (potentially mediated at least in part by CRP) on schizophrenia risk.

59 2.68; 95% CI: 1.06, 6.79; p = 0.04 for BC-by-CRP-interaction).

60 sensitivity and specificity of point-of-care CRP and WHO symptom-based screening in reference to cult

61 rence standard, sensitivity of point-of-care CRP and WHO symptom-based screening were similar (94% [7

62 Point-of-care CRP testing had 89% sensitivity (145 of 163, 95% CI 83-9

63 h WHO symptom-based screening, point-of-care CRP testing had lower sensitivity (difference -7%, 95% C

64 ts were enrolled and underwent point-of-care CRP testing.

65 IDS programmes should consider point-of-care CRP-based tuberculosis screening to improve the efficien

66 CDAI, CRP, fecal calprotectin and VCE Lewis inflammatory score

67 ide polymorphisms that influence circulating CRP concentrations are associated with late AMD.

68 sed on demonstrated influence on circulating CRP concentrations in the literature.

69 tions between CRP polymorphisms, circulating CRP, diabetes, and glucose levels.

70 These findings indicate that circulating CRP and SAA levels are highest when the concentration of

71 nd 30ng/mL (for inflammatory bowel diseases) CRP in 1000-fold diluted blood respectively.

72 tivator for xylose catabolic operons, either CRP or XylR, and these mutations are demonstrated to enh

73 Elevated CRP was positively associated with confirmed bacterial p

74 (iii) Are elevated CRP levels relevant?

75 nd obesity was associated with both elevated CRP and AGP in WRA.Recent morbidity and abnormal anthrop

76 rboring constitutively active eNOS, elevated CRP did not invoke insulin resistance.

77 ood cell count, but all of them had elevated CRP.

78 the sensitivity and specificity of elevated CRP for "confirmed" bacterial pneumonia (positive blood

79 ructed to assess the performance of elevated CRP in distinguishing these cases.

80 nd G at rs1205 were associated with elevated CRP level (each P < 1.2 x 10(-6)).

81 -40.2% in PSC and 7.9-29.5% in WRA (elevated CRP) and 21.2-64.3% in PSC and 7.1-26.7% in WRA (elevate

82 In WRA, elevated CRP was positively associated with obesity [body mass in

83 Class II promoter activity, whilst elevating CRP steady state levels, thus indirectly increasing Clas

84 nts, such as the rate-limiting C4, following CRP interaction and thereby inhibit classical pathway ac

85 es of never or almost never were as follows: CRP: 0.80 (0.69, 0.90), P-trend = 0.0003; and IL-6: 0.86

86 ria on the estimated VAD after adjusting for CRP and AGP.The use of regression correction (derived fr

87 come measures persisted after adjustment for CRP and other covariates.

88 95% CI 0.558-0.687, P < 0.01) as well as for CRP (0.731, 95% CI 0.673-0.789, P < 0.01) for the predic

89 were not measured; regression correction for CRP concentrations increased the estimated prevalence of

90 1 at high concentration competed with FH for CRP binding, indicating possible complement deregulation

91 d identify 8 new regulatory interactions for CRP, IHF or Fis responsible for the control of the promo

92 s, inflammation correction was done only for CRP concentrations because AGP concentrations were not m

93 effect was more pronounced for AGP than for CRP.

94 um CRP were combined into a weighted genetic CRP score (wGSCRP).

95 eactive protein (CRP) and a weighted genetic CRP score representing markers of inflammatory burden mo

96 virus (HIV)-negative tested controls, 3% had CRP >/=40 mg/L.

97 with confirmed bacterial pneumonia, 77% had CRP >/=40 mg/L compared with 17% of 556 RSV pneumonia ca

98 disappeared-because the stressors heightened CRP, SAA, sICAM-1 and sVCAM-1 responses to the sunflower

99 Furthermore, high CRP levels significantly correlated with reduced toleran

100 p, with stronger associations only at higher CRP (5th quintile) and reference TL level (1st quintile)

101 The CMV-positive HEU infants had higher CRP than the CMV-negative HEU infants; this association

102 However, CRP modified the BC-MMSE relationship, with stronger ass

103 gnificant difference in GCF TNF-alpha and hs-CRP levels between the groups (P >0.05).

104 Plasma magnesium and hs-CRP were measured by inductively coupled plasma mass spe

105 AC (>0), 31% had FH, and 58% had elevated hs-CRP (>/=2 mg/l).

106 ical therapy, STS further reduce elevated hs-CRP and other circulating inflammation markers in CAD pa

107 Elevated hs-CRP is associated with allergic sensitization in school-

108 GCF hs-CRP, IL-lbeta, and TNF-alpha levels were analyzed using

109 They also had higher hs-CRP (P=0.014), higher central augmentation index (P=0.01

110 me was an independent predictor of higher hs-CRP and augmentation index.

111 ardial infarction patients with increased hs-CRP level were randomly assigned to atorvastatin-based s

112 ur subsample analysis; the geometric mean hs-CRP concentration for ascending quartiles of plasma magn

113 450K BeadChip), and circulating levels of hs-CRP and IL-18 were assessed in the association between a

114 ) exhibited significantly lower levels of hs-CRP than the control group (n = 35) (1.72 vs 3.20 mg/L,

115 sium was inversely correlated with plasma hs-CRP in our subsample analysis; the geometric mean hs-CRP

116 y healthy individuals, we measured plasma hs-CRP levels to examine their relation to plasma magnesium

117 Demographic data, metabolic profiles, hs-CRP (high-sensitivity C-reactive protein) levels, oxidat

118 erum high sensitivity C-reactive protein (hs-CRP) and a visual analogue scale (VAS), respectively.

119 High-sensitivity C-reactive protein (hs-CRP) is independently associated with cardiovascular eve

120 High-sensitivity C-reactive protein (hs-CRP), interleukin-1beta (IL-1beta), IL-6, tumor necrosis

121 a, and high-sensitive C-reactive protein (hs-CRP); serum hs-CRP was also sampled.

122 infections, and a CRP genetic risk score, hs-CRP at 7 years was associated with concurrent elevated s

123 d higher levels of GCF IL-1beta and serum hs-CRP in patients with OSA.

124 Serum hs-CRP levels were significantly higher in patients with OS

125 sitive C-reactive protein (hs-CRP); serum hs-CRP was also sampled.

126 Serum hs-CRP was measured by latex-enhanced immunoturbidimetric a

127 y disease (CAD) patients and reducing the hs-CRP level may further benefit this population.

128 The primary outcome was hs-CRP level.

129 nd degree of inflammation correlated with hs-CRP (r = 0.58, p = 0.04).

130 SNA and reduced cBRS were associated with hs-CRP although confounded in multivariable analysis.

131 HR was independently associated with hs-CRP.

132 ptide, hs-TnI (high-sensitivity troponin I), CRP (C-reactive protein), GDF-15 (growth differentiation

133 Altogether, these results identify CRP as a ligand for FHR-1 and suggest that FHR-1 enhance

134 ptor 1-like protein y (CRRY) is an important CRP in mice.

135 Between DHA and EPA, changes in CRP (-7.9% +/- 5.0% compared with -1.8% +/- 6.5%, respec

136 halene was associated with a 35% increase in CRP (95% confidence interval = -0.13, 83.2), a 14% incre

137 -PAHs were associated with >20% increases in CRP.

138 0.84-0.97; P = .005) per 2-fold increment in CRP levels; consistent results were obtained using diffe

139 SEP inequalities in CRP emerged in 30s, increased up to mid-50s or early 60

140 imultaneously, the ceramide concentration in CRPs increases approximately twofold.

141 hsCRP, based on 16 SNPs from genes including CRP, was not associated with BMD.

142 Increased CRP similarly predicted decreased dorsal striatal to vmP

143 e previously revealed in mice that increased CRP causes insulin resistance and mice globally deficien

144 Whereas increased CRP caused insulin resistance in mice expressing endothe

145 the complement system by locally increasing CRP expression using targeted gene therapy represents a

146 r of glucose tolerance, this study indicated CRP gene is associated with glucose intolerance.

147 to assess the relation between inflammation (CRP concentration >5 mg/L or AGP concentration >1 g/L) a

148 he approach used to adjust for inflammation (CRP plus AGP), the estimated prevalence of depleted iron

149 he approach used to adjust for inflammation (CRP+AGP), the estimated prevalence of VAD decreased by a

150 s/wk was associated with significantly lower CRP (all P < 0.0001) and IL-6 (P ranges from 0.001 to 0.

151 ogress more rapidly than do those with lower CRP levels and that this elevation may reflect a neuroin

152 d with 29% from highest compared with lowest CRP deciles for pooled PSC and WRA, respectively, with s

153 pCRP* constitutes the major CRP species in human-inflamed tissue and allows binding

154 d 8-isoPF2alpha levels, and between maternal CRP levels and HNE-MA levels.

155 eric CRP, but it bound strongly to monomeric CRP via its C-terminal domains.

156 2 treatment of native CRP did not monomerize CRP and did not affect the PCh binding activity of CRP.

157 ed inhibition for collagen (IC50 = 6.7 muM), CRP-XL (IC50 = 53.5 muM), and convulxin (CVX) (IC50 = 5.

158 muM) and R (6d) (collagen, IC50 = 126.3 muM; CRP-XL, IC50 > 500 muM; CVX, IC50 = 86.8 muM).

159 antiomers S (6c) (collagen, IC50 = 25.3 muM; CRP-XL, IC50 = 181.4 muM; CVX, IC50 = 9 muM) and R (6d)

160 o losartan (LOS) (collagen, IC50 = 10.4 muM; CRP-XL, IC50 = 158 muM; CVX, IC50 = 11 muM) than any of

161 Controlled H2O2 treatment of native CRP did not monomerize CRP and did not affect the PCh bi

162 , in BPD, a potential role for variants near CRP gene is proposed.

163 , regardless of whether patients with normal CRP levels received NP001 or placebo (3.00 [3.62] vs -7.

164 ing was associated with elevated AGP but not CRP in PSC, and obesity was associated with both elevate

165 d did not affect the PCh binding activity of CRP.

166 The assessment of CRP levels may help to optimize the management of patien

167 s and glucose levels, but the association of CRP gene with these traits is unclear.

168 RETATION: The performance characteristics of CRP support its use as a tuberculosis screening test for

169 Effects of the concentrations of CRP and AGP on SF, sTfR, and TBI were generally linear,

170 Circulating concentrations of CRP and SAA in patients with a range of early to late ob

171 our findings suggest a protective effect of CRP and a risk-increasing effect of sIL-6R (potentially

172 We cannot verify any causal effect of CRP level on any of the other common somatic and neurops

173 gest that the ligand recognition function of CRP is dependent on the presence of an inflammatory micr

174 l connectivity was examined as a function of CRP using seeds for subdivisions of the ventral and dors

175 structure and ligand recognition function of CRP.

176 ation, on the ligand recognition function of CRP.

177 We hypothesize that one of the functions of CRP at sites of inflammation is to sense the inflammator

178 sleep, was associated with higher levels of CRP (ES .09; 95% CI = .01-.17) but not IL-6 (ES .03; 95%

179 uration was associated with higher levels of CRP (ES .17; 95% CI = .01-.34) and IL-6 (ES .11; 95% CI

180 Elevated levels of CRP are common and relevant in CSU patients.

181 etermine whether stratification by levels of CRP improves differentiation of responders and nonrespon

182 What is the prevalence of elevated levels of CRP in CSU?

183 t week of life the elevated plasma levels of CRP predicted the risk of BPD (OR 3.4, p = 2.9 x 10(-4))

184 Levels of CRP started to discriminate from day 3 onward with a spe

185 Higher levels of CRP were associated with ASST positivity (P = .009) and

186 Serum levels of CRP were measured by the nephelometric method.

187 Levels of CRP, but not SAA, were also found to be significantly in

188 ticipants had significantly higher levels of CRP, tumor necrosis factor, and interleukin 6 and shorte

189 third of CSU patients had elevated levels of CRP.

190 Why do CSU patients show elevated levels of CRP?

191 remain, however, regarding the regulation of CRP activity itself.

192 To examine the prognostic significance of CRP in ALS.

193 and did not involve the PCh-binding site of CRP.

194 evaluated the sensitivity and specificity of CRP for identifying bacterial vs respiratory syncytial v

195 trate that a lysine (K100) on the surface of CRP has a dual function: to promote CRP activity at Clas

196 tion in the plasma membrane, the quantity of CRPs, and their size.

197 mg/L substantially improved specificity over CRP >/=40 mg/L, though at a loss to sensitivity.

198 Circulating pentameric CRP (pCRP) localizes to damaged tissue where it leads to

199 FHR-1 did not bind to native, pentameric CRP, but it bound strongly to monomeric CRP via its C-te

200 Finally, plasma CRP levels from the first week of life and the risk of B

201 P rs3093059 associated nominally with plasma CRP levels.

202 ides are located in ceramide-rich platforms (CRPs) with a size of about 75 nm that are composed of at

203 rface of CRP has a dual function: to promote CRP activity at Class II promoters, and to ensure proper

204 at Class II promoters, and to ensure proper CRP steady state levels.

205 tionship between mGPS [0-C-reactive protein (CRP) </= 10 mg/L, 1-CRP > 10 mg/L and albumin >/=35 g/L,

206 ion molecule (ALCAM) and C-reactive protein (CRP) (p < 0.05), and IQR increases in OH-PAHs were assoc

207 d that prebiotics reduce C-reactive protein (CRP) [standardized mean difference (SMD): -0.60; 95% CI:

208 on biomarkers, including C-reactive protein (CRP) and a panel of cytokines (interleukin-6 (IL-6) and

209 culating levels of serum C-reactive protein (CRP) and a weighted genetic CRP score representing marke

210 Effects of C-reactive protein (CRP) and alpha1-acid glycoprotein (AGP) concentrations o

211 ammation -assessed using C-reactive protein (CRP) and fibrinogen- varied across the adult age span.

212 aluate the expression of C-reactive protein (CRP) and serum amyloid A (SAA), the prototype acute-phas

213 asma inflammation marker C-reactive protein (CRP) and the urinary oxidative stress markers 8-hydroxyd

214 ctive, fast and reusable C-reactive protein (CRP) aptasensors.

215 nterleukin 6 (IL-6), and C-reactive protein (CRP) at 6 weeks and 6 months of age in 272 HIV-infected

216 sion of individuals with C-reactive protein (CRP) concentrations >5 mg/L or alpha-1-acid glycoprotein

217 exclude individuals with C-reactive protein (CRP) concentrations >5 mg/L or alpha-1-acid glycoprotein

218 clusion of subjects with C-reactive protein (CRP) concentrations >5 mg/L or alpha-1-acid glycoprotein

219 .005) and >2-fold higher C-reactive protein (CRP) concentrations (P < .0001).

220 C-reactive protein (CRP) concentrations rise in response to tissue injury or

221 C-reactive protein (CRP) encoded by CRP gene is a reflection of systemic inf

222 as limited evaluation of C-reactive protein (CRP) for identifying bacterial pneumonia.

223 he comparison we use the C-reactive protein (CRP) induced agglutination of identical samples of 100nm

224 C-reactive protein (CRP) is a biomarker of the inflammatory response that sh

225 C-reactive protein (CRP) is a circulating inflammatory marker associated wit

226 C-reactive protein (CRP) is present at sites of inflammation including amylo

227 essed the performance of C-reactive protein (CRP) measured with a point-of-care assay as a screening

228 Baseline C-reactive protein (CRP) or cytokine levels did not predict treatment outcom

229 edimentation rate (ESR), C-reactive protein (CRP) values, haemoglobin, and leukocyte values.

230 Increased log plasma C-reactive protein (CRP) was significantly associated with increased log lef

231 r example, cytokines and C-reactive protein (CRP)) are reliably elevated in depressed patients.

232 ndependent of age and of C-reactive protein (CRP), a marker of inflammation.

233 Elevated levels of C-reactive protein (CRP), a sensitive marker of inflammation, have been cons

234 performance status (PS), C-reactive protein (CRP), albumin, the nutritional risk index, daily energy

235 rient biomarkers such as C-reactive protein (CRP), alpha-1-acid glycoprotein (AGP), ferritin, and ret

236 ds to another pentraxin, C-reactive protein (CRP), analyze the functional relevance of this interacti

237 (IGFBP-3), adiponectin, C-reactive protein (CRP), and interleukin 6 (IL-6), as well as the homeostas

238 e binding protein (LBP), C-reactive protein (CRP), ILT-4, C-C motif ligand 18 (PARC), and sialic acid

239 ed circulating levels of C-reactive protein (CRP), interleukin-1 receptor antagonist (IL-1Ra), and so

240 lyzed with assessment of C-reactive protein (CRP), interleukin-6 (IL-6), and tumor necrosis factor al

241 , TNFalpha), circulatory C-reactive protein (CRP), serum amyloid A (SAA) and haptoglobin (Hpt) were a

242 no prior day stressors, C-reactive protein (CRP), serum amyloid A (SAA), intercellular adhesion mole

243 as associated with serum C-reactive protein (CRP), tumor necrosis factor, interleukin 1beta, 6, and 1

244 nd procoagulant molecule C-reactive protein (CRP), which induces PMA formation in vitro, along with p

245 studies (EWAS) of serum C-reactive protein (CRP), which is a sensitive marker of low-grade inflammat

246 ce can be used to detect C-reactive protein (CRP)-a biomarker for neonatal sepsis, pelvic inflammator

247 , total cholesterol, and C-reactive protein (CRP).

248 transcription factor cAMP receptor protein (CRP) is responsible for much of this regulation.

249 ndex [BMI] and levels of C-reactive protein [CRP] and leptin).

250 LDH, bilirubin, D-dimer, C-reactive protein [CRP]) improved.

251 edimentation rate [ESR], C-reactive protein [CRP]), and DNI were measured.

252 acroglobulin [alpha-2M], C-reactive protein [CRP], haptoglobin, and serum amyloid protein A [SAA]), i

253 markers of inflammation (C-reactive protein [CRP], interleukin 6, soluble interleukin 6 receptor [sIL

254 S100 beta proteins and C-reactive proteins (CRP).

255 in complement negative regulatory proteins (CRPs), possibly owing to bisretinoid accumulation, may b

256 Highly diverse small cysteine-rich proteins (CRPs) have been found to play multiple roles in plant re

257 95% CI: -0.98, -0.23], and synbiotics reduce CRP (SMD: -0.40; 95% CI: -0.73, -0.06) and tumor necrosi

258 of the phase 2 trial of NP001 using the same CRP threshold showed that patients with elevated baselin

259 oluble CD163 (sCD163), soluble CD14 (sCD14), CRP, IL-6, and a gut microbial translocation marker (int

260 Higher sCD14, CRP, and D-dimer levels were associated with higher peri

261 Secreted CRPs found in the pollen coat of members of the Brassica

262 Serum CRP levels from the first examination were recorded to a

263 Serum CRP levels in the 394 patients with ALS correlated with

264 Serum CRP levels were assayed at a central laboratory, and sin

265 and this relationship was modified by serum CRP levels.

266 st that patients with ALS and elevated serum CRP levels progress more rapidly than do those with lowe

267 d nutrition group showed no changes in serum CRP concentration and significantly increased skeletal m

268 We measured serum CRP levels in cases with World Health Organization-defin

269 riodontitis and NAFLD within strata of serum CRP and separately within strata of the wGSCRP.

270 up showed significant up-regulation of serum CRP) levels and no significant difference in both skelet

271 Patients' serum CRP levels were evaluated from January 1, 2009, to June

272 on = 0.8) on the multiplicative scale, serum CRP modified the relationship between periodontitis and

273 % CI, 0.65-1.93) for participants with serum CRP >3 mg/L.

274 CI, 1.32-4.31) among participants with serum CRP <1 mg/L.

275 % CI, 0.57-1.66) for participants with serum CRP levels of 1 to 3 mg/L and 1.12 (95% CI, 0.65-1.93) f

276 on studies as robustly associated with serum CRP were combined into a weighted genetic CRP score (wGS

277 This includes novel developments in SI-CRP, as well as the emergence of novel applications such

278 tiated controlled radical polymerization (SI-CRP) techniques has become a powerful approach to tailor

279 dditionally, polymer brushes prepared via SI-CRP have been utilized to modify the surface of novel su

280 .601); in 5th POD, PCT has a better AUC than CRP and WBC (0.862 vs 0.806 vs 0.611).

281 We propose that CRP K100 acetylation could be a mechanism by which the c

282 istent trends were obtained, suggesting that CRP-based assays may be useful for estimating abiotic NA

283 SNP rs11265269, flanking the CRP gene, showed the strongest signal in GWAS (odds rati

284 esistance and mice globally deficient in the CRP receptor Fcgamma receptor IIB (FcgammaRIIB) were pro

285 A number of other SNPs in the CRP region, including rs3093059, had nominal association

286 In vitro, Cmr (a member of the CRP/FNR super-family of transcription regulators) bound

287 responsiveness was inversely related to the CRP level in elderly participants, but not seniors, and

288 Adjustment for malaria in addition to CRP and AGP did not substantially change the estimated p

289 was not generated following ES-62 binding to CRP, demonstrating that C2 cleavage was far less efficie

290 Fabricated devices show high selectivity to CRP when compared with other target molecules, such as u

291 ing assays, purified pentameric H2O2-treated CRP bound to a number of immobilized proteins including

292 presentative protein ligand for H2O2-treated CRP, we found that the binding occurred in a Ca(2+)-inde

293 mechanism by which the cell downwardly tunes CRP-dependent Class II promoter activity, whilst elevati

294 graphy (MRE), video capsule endoscopy (VCE), CRP, fecal calprotectin and CDAI.

295 WBC, CRP, ESR and DNI were higher in APN than in lower UTI (p

296 To assess whether CRP (OMIM 123260) single-nucleotide polymorphisms that i

297 e is strongly affected by IHF and Fis, while CRP seems to provide a fine-tuning mechanism.

298 As expected, IL-6 and IL-8 increased, while CRP decreased, in the tocilizumab group compared with th

299 PFASs were not associated with CRP in the subset of the population with available data

300 ntical samples of 100nm MNPs conjugated with CRP antibodies.