ライフサイエンスコーパス: CSF

1 CSF and serum pNfH concentrations are elevated in patien

2 CSF metabolite concentrations provide markers for neuroi

3 CSF tau levels were compared between groups and correlat

4 N-gamma) DNA or colony-stimulating factor 1 (CSF-1) DNA prior to ocular infection with HSV-1.

5 in concert with colony-stimulating factor 1 (CSF-1)-dependent donor macrophages, induce a transformin

6 from DNA-aptamer-based measurements of 1,128 CSF proteins.

7 1.16 per 0.10 decrease; 95% CI, 1.04-1.30), CSF culture positivity (HR, 1.37; 95% CI, 1.02-1.84), an

8 11) CJD and 104 negative control samples (54 CSF and 50 OM).

9 tination assay (CSF-TPPA) is sensitive and a CSF Treponema pallidum hemagglutination assay (CSF-TPHA)

10 who subsequently convert to having abnormal CSF Abeta42.

11 d to evaluate the relationships between age, CSF biomarkers of AD pathology, and quantitative magneti

12 has been a lack of consistent findings among CSF microRNAs studies.

13 NfL concentrations in blood and CSF were recently shown to have prognostic value for cli

14 ves as a biomarker of target engagement, and CSF protein biomarkers were evaluated.

15 age, non-motor assessments, DAT imaging, and CSF biomarkers.

16 Plasma and CSF collected pre-ART were assayed for cytokines and che

17 (CSF) viral rebound or sustained plasma and CSF viremia during treatment.

18 Pyrimethamine was measured in plasma and CSF.

19 Serum and CSF 24(S)-hydroxycholesterol (24[S]-HC), which serves as

20 s: (1) cross-sectional with paired serum and CSF samples (n = 142), and (2) longitudinal with repeate

21 ts (52.6%), and CrAg LFA titers in serum and CSF samples ranged from 1:5 to >/=1:2,560.

22 Diabetes occurred in the anti-CSF-1 receptor protected mice after treatment with a blo

23 onema pallidum particle agglutination assay (CSF-TPPA) is sensitive and a CSF Treponema pallidum hema

24 F Treponema pallidum hemagglutination assay (CSF-TPHA) titer of >/=1:640 is specific for neurosyphili

25 Extra-axial CSF volume at 6 months predicted which high-risk infants

26 in the subarachnoid space (i.e., extra-axial CSF) from 6 to 24 months of age.

27 The agreement between CSF Abeta42 measures from different immunoassays and vis

28 We found a strong association between CSF NfL and sNfL (beta = 0.589, p < 0.001).

29 To determine the concordance between CSF Abeta42 levels measured using 5 different immunoassa

30 The correlation between CSF Abeta levels and WM-LL suggests a direct link betwee

31 There was a positive correlation between CSF-1 and MMP-8, which both correlated negatively to IL-

32 We review post-mortem, serum-biomarker, CSF-biomarker, and neuroimaging studies that have examin

33 oroid plexus epithelium to disrupt the blood-CSF barrier.

34 Viral persistence in both CSF and LN correlated with upregulation of mechanistic t

35 iculon-1 in macrophage migration toward both CSF-1 and CCL2 was confirmed.

36 to govern antibody trafficking at the brain-CSF interface with relevance for immune surveillance in

37 strate that microenvironmental alteration by CSF-1R blockade renders tumor cells more susceptible to

38 We centrifuged CSF, resuspended the pellet in 2 mL of CSF, and tested 0

39 agnostic algorithm for sporadic CJD combines CSF and OM RT-QuIC testing to provide virtually 100% dia

40 hromatography (HPLC) approaches to determine CSF levels of monoamines and their cofactors.

41 Brain penetration (measured by drug CSF/serum level) of G6 dendrimers correlated with the se

42 es can be an option to design more effective CSF surveillance strategies in wild boar.

43 Some had paresthesias (17%), elevated CSF protein level (13%), and other features sometimes co

44 ng neuronal damage have resulted in elevated CSF concentrations.

45 ly in swine, 13d provided a 15-fold enhanced CSF-to-plasma ratio and a 9-fold enhanced brain-to-plasm

46 ve, 0.87, which is comparable to established CSF biomarkers).

47 Classical swine fever (CSF) is a notifiable, highly contagious viral disease of

48 Surveillance of Classical Swine Fever (CSF) should not only focus on livestock, but must also i

49 enrolled in a study of cerebrospinal fluid (CSF) abnormalities in syphilis underwent the mental alte

50 continuous lower normal cerebrospinal fluid (CSF) amyloid beta1-42 (>/=640pg/ml) levels were related

51 ase L1 (UCHL1) in mouse cerebrospinal fluid (CSF) and Abeta in the mouse brains.

52 in (APP) are present in cerebrospinal fluid (CSF) and their potential as biomarkers for Alzheimer's d

53 rformed and assayed for cerebrospinal fluid (CSF) biomarkers of AD pathology, including beta-amyloid

54 tomegalovirus (hCMV) in cerebrospinal fluid (CSF) by polymerase chain reaction (PCR) is a marker of c

55 Notably, cerebrospinal fluid (CSF) contains microRNAs that may serve as biomarkers for

56 etabolomics analysis of cerebrospinal fluid (CSF) from cognitively intact elderly patients (N = 28) w

57 ered SOD1 levels in the cerebrospinal fluid (CSF) in patients carrying SOD1 mutations linked to famil

58 der (ASD) had increased cerebrospinal fluid (CSF) in the subarachnoid space (i.e., extra-axial CSF) f

59 rphism, which predicted cerebrospinal fluid (CSF) leukocyte count and survival of Vietnamese patients

60 gated whether plasma or cerebrospinal fluid (CSF) levels of cytokines and chemokines predicted C-IRIS

61 em, was associated with cerebrospinal fluid (CSF) markers of neuronal injury in preclinical AD and ri

62 levels are elevated in cerebrospinal fluid (CSF) of patients with amyotrophic lateral sclerosis (ALS

63 Existing cerebrospinal fluid (CSF) or imaging (tau positron emission tomography) bioma

64 ologic prion protein in cerebrospinal fluid (CSF) or olfactory mucosa (OM) samples.

65 es have shown increased cerebrospinal fluid (CSF) OT levels following IN administration, this does no

66 condition caused by low cerebrospinal fluid (CSF) pressure, usually due to CSF leakage.

67 h additional and edited cerebrospinal fluid (CSF) regions to produce voxel-level absorbed dose per un

68 is period, serum and/or cerebrospinal fluid (CSF) samples from 3,969 patients were tested with the Cr

69 metabolite screening of cerebrospinal fluid (CSF) samples from a canine model of MPS I revealed a mar

70 the absence of definite cerebrospinal fluid (CSF) sterilization or related mortality.

71 ses that (1) antemortem cerebrospinal fluid (CSF) tau levels correlate with postmortem tau pathology

72 nimals with episodes of cerebrospinal fluid (CSF) viral rebound or sustained plasma and CSF viremia d

73 cells, are shed to the cerebrospinal fluid (CSF), and are detectable at low concentrations in periph

74 bility maps (nlTPMs) of cerebrospinal fluid (CSF), gray matter (GM), and white matter (WM) tissues; 3

75 detection of prions in cerebrospinal fluid (CSF), most recently, the measurements of prion seeding a

76 s brain tissue from the cerebrospinal fluid (CSF).

77 a-amyloid (Abeta) 42 in cerebrospinal fluid (CSF).

78 f an LD for drainage of cerebrospinal fluid (CSF; intervention group).

79 cord to circulate the cerebral spinal fluid (CSF).

80 We explored risk factors for CSF discordance and escape in patients presenting with d

81 otocol describes step-by-step procedures for CSF sample preparation for the analysis of different mol

82 The contrast sensitivity function (CSF) relates the visibility of a spatial pattern to both

83 g G-CSF treatment reduces excitability and G-CSF-induced visceral pain in vivo.

84 lowing administration of neutralizing anti-G-CSF antiserum.

85 A new G-CSF-driven (methylated BSA/G-CSF) arthritis model was established enabling us to demo

86 aftment efficiency than those mobilized by G-CSF.

87 This drug combination (AraC+CHK1i+G-CSF) will open the doors for a more efficient treatment

88 Five cytokines (G-CSF, GM-CSF, IL-1-ra, IL-2 and IL-16) were significantly

89 g CX3CR1 or nitric oxide production during G-CSF treatment reduces excitability and G-CSF-induced vis

90 ein granulocyte colony-stimulating factor (G-CSF) against storage at 4 degrees C and shipping tempera

91 es, granulocyte-colony stimulating factor (G-CSF) and stem cell factor (SCF) in a humanized murine mo

92 Granulocyte colony-stimulating factor (G-CSF) is used clinically to treat leukopenia and to enfor

93 and granulocyte colony-stimulating factor (G-CSF) levels in the amniotic fluid of ZIKV-positive pregn

94 ine granulocyte colony-stimulating factor (G-CSF) through complex mechanisms.

95 -8, granulocyte-colony stimulating factor (G-CSF), IL-33, IL-11, IL-1alpha, and IL-1beta.

96 of granulocyte colony-stimulating factor (G-CSF).

97 of granulocyte-colony stimulating factor (G-CSF); these effects are reversed following administratio

98 latency that can be reactivated following G-CSF treatment.

99 s adverse events were more frequent the in G-CSF and stem-cell infusion group (12 [43%] patients) tha

100 , dorsal root ganglion neurons cultured in G-CSF failed to respond to G-CSF in vitro, and Csf3r gene

101 INTERPRETATION: G-CSF with or without haemopoietic stem-cell infusion did

102 A new G-CSF-driven (methylated BSA/G-CSF) arthritis model was es

103 mice subjected to intrathecal injection of G-CSF exhibit pronounced visceral hypersensitivity, an eff

104 are preferentially mobilized to the PB on G-CSF treatment.

105 poiesis and EMH in response to bleeding or G-CSF treatment.

106 h untreated animals, animals that received G-CSF following radiation injury exhibited enhanced functi

107 dilution of dormant HSCs, suggesting that G-CSF does not stimulate dormant HSC proliferation.

108 sion group (12 [43%] patients) than in the G-CSF (three [11%] patients) and standard care (three [12%

109 uction of CEBPbeta, a key regulator of the G-CSF gene.

110 s of MELD change over time (p=0.55 for the G-CSF group vs standard care and p=0.75 for the G-CSF plus

111 y assigned to the standard care, 26 to the G-CSF group, and 28 to the G-CSF plus stem-cell infusion g

112 group vs standard care and p=0.75 for the G-CSF plus stem-cell infusion group vs standard care).

113 care, 26 to the G-CSF group, and 28 to the G-CSF plus stem-cell infusion group.

114 tations in the extracellular domain of the G-CSF receptor (CSF3R) have been reported only in severe c

115 ron-microglia interaction that responds to G-CSF by engaging Cathepsin S-CX3CR1-inducible NOS signali

116 ons cultured in G-CSF failed to respond to G-CSF in vitro, and Csf3r gene expression could not be det

117 thin the BM HSC compartment in response to G-CSF treatment.

118 GM-CSF alone did not improve 6-minute walk more than attent

119 GM-CSF has been portrayed as a critical cytokine in the pat

120 GM-CSF levels were increased in NPs compared with those in

121 GM-CSF neutralization in diet-induced obese mice significan

122 GM-CSF was identified as an upstream modulator.

123 hritis, and increased numbers of IL-17A(+)GM-CSF(+) double-producing CD4, CD8, gammadelta and NK cell

124 s, but rather promoted the expansion of a GM-CSF(+) Th17 cell subset, thereby enhancing its encephali

125 lease of thymic stromal lymphopoietin and GM-CSF from tracheal epithelial cells.

126 ranulocyte colony-stimulating factor, and GM-CSF levels.

127 ranulocyte colony-stimulating factor, and GM-CSF levels.

128 L-5 share a common ss-chain with IL-3 and GM-CSF receptors.

129 ation and reduced frequency of IL-17- and GM-CSF-producing CD4(+) T cells.

130 atherosclerotic plaques by both M-CSF and GM-CSF.

131 stance to lung infection through Nod2 and GM-CSF.

132 -IL5(OXA) EoE mice were treated with anti-GM-CSF neutralizing antibody or isotype control and assesse

133 rupture, a complication mitigated by anti-GM-CSF therapy.

134 tured CD103(neg)CD11c(+) cells induced by GM-CSF readily supported exponential growth of L. monocytog

135 or administration of exosomes produced by GM-CSF-expanded bone marrow cells.

136 TGF-beta and granulocyte-macrophage CSF (GM-CSF) enhanced the KDR/ID2 signaling axis in BMDCs.

137 mmation resolving) and granulocyte-M-CSF (GM-CSF; proinflammatory) may contribute to the inconsistenc

138 Five cytokines (G-CSF, GM-CSF, IL-1-ra, IL-2 and IL-16) were significantly increas

139 l expression of the inflammatory cytokine GM-CSF, concomitant with pancreatic infiltration of inflamm

140 he ES cell- and adult progenitor-derived, GM-CSF-instructed, nonconventional DC subsets.

141 ently, hearts of mice deficient in either GM-CSF or its receptor recruit fewer leukocytes and functio

142 virotherapy with an HSV vector expressing GM-CSF has been recently approved by the Food and Drug Admi

143 yte macrophage colony-stimulating factor (GM-CSF) and matrix metallopeptidase 9 (MMP9).

144 yte-macrophage colony-stimulating factor (GM-CSF) signaling, which stimulates pathogen killing and cl

145 yte-macrophage colony-stimulating factor (GM-CSF), mainly produced by MDSCs, was identified as a key

146 mice with transgenic expression of human GM-CSF, interleukin-3, and stem cell factor in a NOD/SCID-I

147 These results identify GM-CSF as both a key contributor to the pathogenesis of MI

148 C57BL/6 mice deficient in GM-CSF are resistant to EAE induced by immunization with my

149 1 diabetes was associated with increased GM-CSF, IL-4, and IL-13 cytokine secretion among Ag-stimula

150 Monocytes integrated GM-CSF and IL-4 stimulation combinatorically and temporally

151 BNSE or the GM-CSF-expressing RABV (LBNSE-GM-CSF).

152 r example, miR-466i functioned to mediate GM-CSF and IL-17 mRNA decay, which was confirmed by in vitr

153 Moreover, the co-expression of GM-CSF and its receptors as well as phosphorylated ERK1/2 a

154 ifies a progression-promoting function of GM-CSF in colon cancer by inducing EMT.

155 ein (MOG)35-55 The mechanism of action of GM-CSF in EAE is poorly understood.

156 lking exercise may augment the effects of GM-CSF in PAD, since exercise-induced ischemia enhances pro

157 ever, little is known about the effect of GM-CSF on cancer cells.

158 of CRC patients, high-level expression of GM-CSF positively correlates with local metastases in lymph

159 show that the dysregulated production of GM-CSF rather than IL-17 induces spontaneous immunopatholog

160 Here we show increased numbers of GM-CSF-producing CD4 and CD8 lymphocytes in the blood and j

161 NF-alpha (but not IL5, IL-3, eotaxin-1 or GM-CSF) was detected in supernatants of ex vivo eosinophil

162 In addition, unlike IL-5 or GM-CSF, IL-3 induced expression of CD32B/C (FCGRIIB/C) subt

163 n relatively well, whereas mice producing GM-CSF can succumb from left ventricular rupture, a complic

164 wed that FTY720 triggers MDSCs to release GM-CSF via S1P receptor 3 (S1pr3) through Rho kinase and ex

165 rapeutic target, bolstering the idea that GM-CSF is a major orchestrator of the leukocyte supply chai

166 Results demonstrate that GM-CSF is required for cholesterol clearance in macrophages

167 unized with the parent virus LBNSE or the GM-CSF-expressing RABV (LBNSE-GM-CSF).

168 d with GM-CSF alone; to determine whether GM-CSF alone improves 6-minute walk more than placebo and w

169 To determine whether GM-CSF combined with supervised treadmill exercise improves

170 red with exercise alone and compared with GM-CSF alone; to determine whether GM-CSF alone improves 6-

171 when CD4(+) T-cells are co-cultured with GM-CSF derived bone marrow dendritic cells (G-BMDCs).

172 atment mimicked the effects observed with GM-CSF neutralization and MMP9 inhibition, suggesting these

173 nistration with respect to achieving greater CSF concentrations of OT.

174 ne patient had MRI imaging but not CT; 5 had CSF pressure measurements.

175 g A*03:01 B*07:02 was associated with higher CSF amyloid levels (p = 0.03, beta +/- standard error =

176 tients, mortality was associated with higher CSF neutrophil counts (hazard ratio [HR], 1.10 per 10% i

177 by measurements of these compounds in human CSF and other biological samples.

178 strate that APP-CTFs are detectable in human CSF, being the most abundant a 25-kDa fragment, probably

179 served to characterize CTFs of APP in human CSF.

180 directions, velocities, and volumes of human CSF flow within the brain aqueduct as part of the intern

181 he results extend our understanding of human CSF flux and open important clinical implications, inclu

182 In humans, CSF spermine was elevated in neuropathic subtypes of MPS

183 etion of TLR4 or SPAK normalizes hyperactive CSF secretion rates and reduces PHH symptoms, as does tr

184 : The diagnostic performance of the improved CSF RT-QuIC is superior to surrogate marker tests for pr

185 n, brain growth, NAGLU enzymatic activity in CSF, and specific anti-NAGLU immune response for 30 mont

186 ay (0.5%) or cell-based assay (1.5%), and in CSF controls by cell-based assay (0.9%).

187 blasts, and of white and red blood cells, in CSF samples at diagnosis of acute lymphoblastic leukemia

188 Measles detection in CSF was performed by serology at the California Departme

189 No differences in CSF biomarker levels were observed between treatment gro

190 There were no significant differences in CSF cytokine or chemokine levels between cases and contr

191 es for the ability to infiltrate and grow in CSF, a remarkably acellular, mitogen-poor metastasis mic

192 -copy assay, we measured HIV-1 RNA levels in CSF and plasma specimens from 220 HIV-positive adults wh

193 Concentrations of NfL in CSF and plasma were correlated in mutation carriers (r=0

194 (CSF-VDRL), (ii) detection of T. pallidum in CSF by reverse transcriptase PCR, or (iii) new vision lo

195 These results suggest that hCMV-PCR in CSF may not be a useful prognostic marker in cCMV.

196 ects model showed a significant reduction in CSF SOD1 at visit 6 (p < 0.001) with a mean reduction of

197 se assays for the quantitation of HIV RNA in CSF and seminal plasma.

198 Low-level HIV-1 RNA in CSF is common during suppressive ART and is associated w

199 s detection of Treponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive

200 ption of criteria to screen HSV PCR tests in CSF represents a cost-effective approach.

201 ns of NfL in plasma correlated with those in CSF.

202 Increased CSF cytokine concentrations correlated with more-severe

203 ozygous for codon 129 generated intermediate CSF RT-QuIC patterns, whereas genetic prion diseases rev

204 INTERPRETATION: CSF phosphorylated-tau levels are positively associated

205 status, and lower ART drug distribution into CSF.

206 and their role in drainage of the brain ISF, CSF, CNS-derived molecules, and immune cells from the CN

207 5% confidence interval [CI], 1.04-1.16), low CSF to blood glucose ratio (HR, 1.16 per 0.10 decrease;

208 h more-severe disease, but patients with low CSF leukocytes and cytokine concentrations were more lik

209 ociated with the older age (0.008) and lower CSF Abeta1-42 (0.005) at baseline.

210 y accompanying sporadic FTLD, we found lower CSF phosphorylated tau levels in the TDP-43 group (media

211 NAGLU activity was detected in lumbar CSF and was 15-20% of that in unaffected children.

212 c activation and sterol biosynthesis after M-CSF stimulation.

213 OCs when they were cultured with RANKL and M-CSF.

214 in murine atherosclerotic plaques by both M-CSF and GM-CSF.

215 atherosclerosis (fractalkine/CX3CL1, CCL8, M-CSF, HGF), T-cell development/activation (CD40L, IL-7, C

216 e RNA: Emr1 (F4/80), Itgam (CD11b), Csf1r (M-CSF Receptor), Itgal (CD11a), Tnf, and Nos2 Additionally

217 The anti-inflammatory cytokine M-CSF, but not DC growth factors, sustains CD33/LAIR-1 exp

218 e, or macrophage colony-stimulated factor (M-CSF) significantly activated AMPK and promoted monocyte-

219 m by macrophage colony-stimulating factor (M-CSF; inflammation resolving) and granulocyte-M-CSF (GM-C

220 F; inflammation resolving) and granulocyte-M-CSF (GM-CSF; proinflammatory) may contribute to the inco

221 secreted TGF-beta and granulocyte-macrophage CSF (GM-CSF) enhanced the KDR/ID2 signaling axis in BMDC

222 are hypersensitive to granulocyte macrophage-CSF due to hyperactive RAS/ERK signaling.

223 rom ileum, rectum, lymph nodes, bone marrow, CSF, circulating CD4+ T cell subsets, and plasma.

224 nd an independent association of ante mortem CSF phosphorylated tau levels with postmortem cerebral t

225 ession tested the association of ante mortem CSF tau levels with postmortem tau pathology adjusting f

226 ed to similar levels in MPhiM-CSF and MPhiGM-CSF.

227 esis were induced to similar levels in MPhiM-CSF and MPhiGM-CSF.

228 . pallidum, CSF WBCs </=5/uL and nonreactive CSF-VDRL.

229 HR = 0.19, p < .01), indicating that normal CSF amyloid levels do not exclude incident Alzheimer dis

230 utcome, and more-precise characterization of CSF leukocytes could guide possible host-directed therap

231 Concentrations of CSF Abeta were assessed using an antibody-independent ma

232 zheimer disease should take the continuum of CSF amyloid beta1-42 levels within the normal range into

233 es were negatively associated with levels of CSF biomarkers across brain white matter and in areas pr

234 fuged CSF, resuspended the pellet in 2 mL of CSF, and tested 0.5 mL with mycobacteria growth indicato

235 Testing 6 mL or more of CSF was associated with more frequent detection of tuber

236 We found that large outbreaks of CSF would be rare and generated from a limited number of

237 erapy studies demonstrated that reduction of CSF spermine reflects correction of brain lesions in the

238 In this case-control study, samples of CSF and OM were collected from 86 patients with a clinic

239 iagnostic specificity and 92% sensitivity of CSF RT-QuIC in a blinded retrospective analysis matched

240 The similarities of CSF-c cells in chicken, Xenopus, and zebrafish suggest t

241 logical features may influence the spread of CSF in GB, highlighting the importance of between-farm b

242 visual PET readings may influence the use of CSF biomarkers and/or amyloid PET assessment in clinical

243 opens opportunities for broad utilization of CSF biomarkers in drug development and precision medicin

244 agnosis of CJD by using the RT-QuIC assay on CSF samples, OM samples, or both.

245 valuable stepping stone in guiding policy on CSF surveillance and control in GB.

246 tion of Treponema pallidum 16S RNA in CSF or CSF white blood cells (WBCs) >20/uL or a reactive CSF-Ve

247 were strongly associated with poor outcome (CSF culture positivity, CSF white blood cell count, hemo

248 was defined as undetectable CSF T. pallidum, CSF WBCs </=5/uL and nonreactive CSF-VDRL.

249 asymptomatic newborns (6.8%) had a positive CSF hCMV-PCR.

250 composite reference standard of any positive CSF tuberculous test.

251 d with poor outcome (CSF culture positivity, CSF white blood cell count, hemoglobin, Glasgow Coma Sca

252 esenting with diverse neurological problems, CSF discordance or escape was observed in 15%, with trea

253 ow the multi-dimensional output of the quick CSF can provide greater precision than scalar outcome me

254 showed that the CSFs derived from the quick CSF method well matched with those from the traditional

255 or can improve convergence rate of the quick CSF, and how the multi-dimensional output of the quick C

256 Neurosyphilis was defined as (i) a reactive CSF Venereal Disease Research Laboratory test (CSF-VDRL)

257 hite blood cells (WBCs) >20/uL or a reactive CSF-Venereal Disease Research Laboratory (VDRL) test; un

258 nse to colony-stimulating factor 1 receptor (CSF-1R) blockade and nanoparticle-based drug delivery in

259 hich 10 (6.1%) LPs in 9 patients represented CSF escape.

260 Unlike plasma/tissue/blood samples, CSF requires minimal sample preparation: in this protoco

261 ylvania Smell Inventory Test (UPSIT) scores, CSF amyloid - (Abeta42) to t-tau ratio, and APOE status

262 wenty-four (14.7%) LPs in 22 patients showed CSF discordance, of which 10 (6.1%) LPs in 9 patients re

263 common disease usually treated by suboptimal CSF shunting techniques.

264 Subsequently, CSF-TPPA reactivity at a 1:640 dilution was determined f

265 F Venereal Disease Research Laboratory test (CSF-VDRL), (ii) detection of T. pallidum in CSF by rever

266 vels of select restriction factor genes than CSF-1-Mphis.

267 We here provide evidence that CSF and blood NfL concentrations can be used as accessib

268 The CSF cell count, 14-3-3 protein detection and S100B were

269 The CSF level of this 25-kDa CTF is higher in subjects with

270 The CSF RT-QuIC differentiated 94% of cases of sporadic Creu

271 s, and associations between the QAlb and the CSF level of neurofilament light chain (NFL), the ratio

272 peripherally administered OT is entering the CSF.

273 E3 cells that may relay information from the CSF to underlying neural circuits along the ventral midl

274 cular access of the larger antibody from the CSF, with intrathecal 0.75 m mannitol increasing the num

275 nt in individuals with normal Abeta42 in the CSF and normal amyloid PET who subsequently convert to h

276 JC polyomavirus was detected in the CSF at the time of presentation.

277 miR-142-3p was upregulated in the CSF of MS patients and in EAE cerebellum.

278 observed that miR-142-3p is increased in the CSF of patients with active MS and in EAE brains.

279 CHIT1 concentrations in the CSF of patients with ALS may reflect the extent of micro

280 High levels of PPIA were found in the CSF of SOD1(G93A) mice and rats and sporadic ALS patient

281 is one of the most abundant proteins in the CSF, as well as provide new insights with respect to und

282 more efficient entry of the peptide into the CSF compared to the intravenous (IV) route, which requir

283 uses and 10% presented superinfection of the CSF temporary drainage/externalized peritoneal catheter.

284 pare two methods for rapid assessment of the CSF that were implemented on a tablet device.

285 r NOD mice with a monoclonal antibody to the CSF-1 receptor resulted in depletion of the resident mac

286 SFK) signaling is sufficient to transmit the CSF-1 lineage instructive signal.

287 Our results showed that the CSFs derived from the quick CSF method well matched with

288 ngs in a subset of HIV- subjects (n = 17) to CSF levels of neurofilament light chain (NFL), reflectiv

289 expansion of the cerebral ventricles due to CSF accumulation following intraventricular hemorrhage (

290 ospinal fluid (CSF) pressure, usually due to CSF leakage.

291 alization to the periphery after exposure to CSF-1 for 2.5 min was shown by immunofluorescence micros

292 AM heterogeneity, and successful response to CSF-1R blockade is characterized by enhanced TAM penetra

293 f producing a significant reduction in total CSF SOD1 protein content in patients with ALS caused by

294 ntly phagocytose via FcR and chemotax toward CSF and CX3CL1.

295 licated syphilis was defined as undetectable CSF T. pallidum, CSF WBCs </=5/uL and nonreactive CSF-VD

296 cute lymphoblastic leukemia (ALL), a uniform CSF and risk group classification schema was incorporate

297 Its effective disposal via various CSF routes has been demonstrated in animal models.

298 isposal of brain cellular waste products via CSF has been demonstrated repeatedly in animal models.

299 Here, we address which CSF-1-activated pathways are involved in transmitting th

300 AD with dementia, plasma NFL correlated with CSF NFL (Spearman rho = 0.59, P < .001).