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1                                              CSH also enhances NE inhibition of glutamate release fro
2                                              CSH enhanced the alpha2-adrenoreceptor agonist clonidine
3                                              CSH exposure increased evoked EPSC (eEPSC) amplitude via
4                                              CSH is associated with a significantly increased risk of
5                                              CSH was defined as a post-procedure hematoma requiring f
6                                              CSH was the only independent predictor and was associate
7 o retained heterozygosity (0.91; 0.56-1.48), CSH p=0.039.
8                                            A CSH approach should be used in conjunction with overall
9 drenoreceptor antagonist yohimbine abolished CSH-induced enhancement of NE inhibition of eEPSCs.
10                  H2O2 oxidizes capping agent CSH, modulating the growth of CSH-stabilized cadmium sul
11 y (CSH) influences virulence of C. albicans, CSH properties of C. dubliniensis were investigated and
12 oxic controls; i.e. acute hypoxia in CON and CSH, and normoxia in CSH.
13 increased ventilation 25% and 50% in CON and CSH, respectively, while NMDA doubled ventilation in bot
14              This column chemistry, known as CSH (charged-surface hybrid) C18, improves upon an alrea
15 ionary phase with a charged surface, such as CSH C18, holds significant promise for facilitating chal
16 eins, the lack of these virulence-associated CSH entities in C. dubliniensis could contribute to its
17 ospectively examined the association between CSH and subsequent device infection.
18 en non-accidental falls and cardioinhibitory CSH.
19 tly sensitized PN-, walnut- (WN) and cashew (CSH)-allergic mice received 1-day PN/WN/CSH rush OIT plu
20 -free and selective detection of cysteamine (CSH).
21 nsor for the electrooxidation of l-cysteine (CSH) in aqueous media.
22 ystem is based on the oxidation of cysteine (CSH) with hydrogen peroxide (H2O2) enzymatically generat
23 dentify prognostic factors for the following CSH rates: intestinal graft failure (IGF)/death due to r
24                      The detection limit for CSH is 150nM (S/N=3).
25 ther developed as a new detection scheme for CSH by chronoamperometry method and under optimized cond
26 he BSAGNCs allow sensitive detection of free CSH in the range of 500-10,000nM.
27          The use of a cause-specific hazard (CSH) approach may provide more precise identification an
28       A multivariable cause-specific hazard (CSH) rate analysis using Cox stepwise regression was per
29 e of clinically significant pocket hematoma (CSH).
30 tios with the chi(2) test for heterogeneity (CSH).
31 in and human chorionic somatotropin hormone (CSH) produced by the placental trophoblastic cells.
32  layer, concurrent calcium silicate hydrate (CSH) alteration to an amorphous zeolite and Ca-carbonate
33                    Calcium silicate hydrate (CSH) is the main binding phase of Portland cement, the s
34              As cell surface hydrophobicity (CSH) influences virulence of C. albicans, CSH properties
35 ioinhibitory carotid sinus hypersensitivity (CSH).
36 ter a chronic exposure to sustained hypoxia (CSH) (7-8 d at 10% FIO(2)).
37              With chronic sustained hypoxia (CSH), such as during acclimatization to high altitude, a
38 bably glutamate receptor modification, as in CSH rats the expression of phosphorylated NR1 and GluR1
39 oM) elicited greater inhibition of eEPSCs in CSH cells (63 +/- 2%; n = 16) than NORM cells (45 +/- 3%
40 ats, and completely blocked the acute HVR in CSH rats but had no effect on ventilation in normoxia.
41 (50) of NE inhibition of eEPSCs was lower in CSH cells (3.0 +/- 0.9 microM; n = 5) than in normoxic (
42 cute hypoxia in CON and CSH, and normoxia in CSH.
43 e and bonding of a large subset of the known CSH minerals.
44 ining heterozygosity than in those with LOH (CSH p=0.02).
45 1) the use of a new charge-surface-modified (CSH) C18 stationary phase to mitigate the challenges of
46 eated rats held in normoxia (CON) or 10% O2 (CSH) for 7 days and measured ventilation in conscious, u
47 Co/GCE) improves electrochemical behavior of CSH oxidation, as compared to the GO and PcCo.
48 ue to the complex structure and chemistry of CSH at various length scales, the focus has progressivel
49 a synergic effect in the electrooxidation of CSH.
50  capping agent CSH, modulating the growth of CSH-stabilized cadmium sulphide quantum dots (CdS QDs).
51            The sensing mechanism is based on CSH etching-induced fluorescence quenching of the bovine
52 d in 11% of patients (7 of 66) with previous CSH and in 1.5% (9 of 593) without CSH.
53 d on separations of a nine-peptide standard, CSH C18 was found to exhibit improved loadability, great
54 tural amino acids do not interfere with such CSH-induced etching process.
55               These results demonstrate that CSH increases evoked excitatory inputs to NTS neurons re
56                             It is found that CSH can etch the Au25 nanoclusters, exhibiting the poten
57             Previous studies have shown that CSH status of C. albicans involves multiple surface prot
58                                          The CSH approach should be more widely used in investigation
59  temperature is one factor which affects the CSH status of stationary-phase C. albicans.
60   Since protein glycosylation influences the CSH status of C. albicans, we compared the cell wall man
61 ed out towards a better understanding of the CSH-induced fluorescence quenching of the BSAGNCs.
62  associated with graft/patient survival, the CSH analysis more precisely identified their prognostic
63 ceTrap can be implemented online through the CSH Galaxy server http://cancan.cshl.edu/splicetrap and
64 nges represent central neural adaptations to CSH.
65 econdary increase persists after exposure to CSH and involves plasticity within the circuits in the c
66 krusei, C. parapsilosis, and C. tropicalis), CSH status correlated with coaggregation with the anaero
67 ition (40 microM; NORM, 36 +/- 2%, n = 11 vs CSH, 26 +/- 4%, n = 6; p < 0.05).
68 ibition (3 microM; NORM, 23 +/- 2%, n = 5 vs CSH, 44 +/- 5%, n = 4; p < 0.05) but attenuated alpha1-a
69 ough application to peptide mapping, wherein CSH C18 was found to aid the development of a high-resol
70 NTS contribute to plasticity in the HVR with CSH.
71  previous CSH and in 1.5% (9 of 593) without CSH.
72 hew (CSH)-allergic mice received 1-day PN/WN/CSH rush OIT plus 3 weeks of maintenance dosing, with or

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