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1 etter discriminate with respect to CYP17 and CYP19.
2 ffects of these two SNPs on transcription of CYP19.
3 transcribed from the same strand, closer to CYP19.
4 the crucial steroidogenic enzymes, CYP17 and CYP19.
5 cardiovascular diseases, dual inhibition of CYP19 and CYP11B2 (aldosterone synthase) is a promising
6 on of important structural features of known CYP19 and CYP11B2 inhibitors, we succeeded in obtaining
7 p as potent and selective dual inhibitors of CYP19 and CYP11B2, especially compound 3p, which exhibit
8 exhibited IC(50) values of 32 and 41 nM for CYP19 and CYP11B2, respectively, and a high selectivity
14 ncoding two steroidogenic enzymes, CYP17 and CYP19, and examining their expression patterns in the C.
16 a key steroidogenic enzyme [cytochrome P450(CYP19) aromatase] required for estrogen synthesis in ver
17 genetic polymorphisms in the aromatase gene, CYP19, as a step toward pharmacogenomic studies of aroma
20 , MAPK6 or TLN2), placing it upstream of the CYP19 coding region in the opposite strand, whereas a de
25 le arrest in granulosa cells of the KrasG12D;Cyp19-Cre mice but not in the KrasG12D;Pgr-Cre mice, doc
26 that did not occur in the Ptenfl/fl;KrasG12D;Cyp19-Cre or Ptenfl/fl;KrasG12D;Pgr-Cre mouse strains.
29 7 and rs7176005 in the 5'-flanking region of CYP19 exon 1.1) that were significantly associated with
32 rget of Dax-1 in Leydig cells, and increased Cyp19 expression may account, in part, for the infertili
34 n trophoblasts markedly inhibited endogenous CYP19 expression, differentiation of cultured human trop
35 mic sequences required for placenta-specific CYP19 expression, fusion genes containing 2,400 and 501
38 permia (NOA) revealed enhanced expression of CYP19, GAS6, and AXL, which suggests that the AROM+ mous
39 g DNA within 501 bp of exon I.1 of the human CYP19 gene contains cis-acting elements that bind placen
43 d within the 11.2 Mb region telomeric to the CYP19 gene in chromosome 15q21 cryptically upregulated a
44 ingle nucleotide polymorphisms (SNPs) in the CYP19 gene may alter the effectiveness of AI therapy in
46 P450 aromatase (aromatase), a product of the CYP19 gene, catalyzes the synthesis of estrogens from an
47 P450 aromatase (aromatase), a product of the CYP19 gene, catalyzes the synthesis of estrogens from an
54 intervening intron, representing duplicated CYP19 genes, were cloned and sequenced from collared pec
55 he hypothesis that women with the long-range CYP19 haplotype 2b-3c may be carriers of a predisposing
60 decisive structural features of CYP11B2 and CYP19 inhibitors into a common template, a series of pyr
62 e is known about whether common variation in CYP19 is associated with risk of hormone-related disease
65 B1 and good selectivity over human CYP17 and CYP19, it is a promising candidate for further developme
67 proximately 2.6 kb) spanning 189.4 kb of the CYP19 locus to characterize linkage disequilibrium (LD)
68 a polymorphic tetranucleotide marker at the CYP19 locus using fluorescence-based semiautomated genot
70 genes on chromosomes 2 (NIDDM1) and 15 (near CYP19) makes a contribution to susceptibility to type 2
72 ervations indicate that genetic variation in CYP19 might contribute to variation in the pathophysiolo
74 lizards compared to receptive PreOv animals; CYP19 mRNA levels in the VMN did not change across the o
76 In placenta, the 5' untranslated region of CYP19 mRNA transcripts is encoded by exon I.1, which lie
78 Previously, we found that Mash-2 inhibited CYP19 promoter activity through sequences within a 350-b
80 sed the SF-1-mediated transactivation of the Cyp19 promoter but did not inhibit the StAR or Cyp11a pr
83 AMP-->PKA-->CREB pathway leading to enhanced CYP19 transcription and increased aromatase activity.
84 E(2) (PGE(2)) that mediate the induction of CYP19 transcription in human adipocytes and breast cance
85 d a portion of the 3'-untranslated region of CYP19 using 240 DNA samples from four ethnic groups.
86 genetic polymorphisms in the aromatase gene CYP19 vary the response of breast cancer patients to aro
87 more, selectivities over CYP11B2, CYP17, and CYP19 were observed, as well as satisfying metabolic sta
89 sgenic mouse strain that overexpresses human CYP19, which encodes aromatase (AROM+ mice), and mice wi
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