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1 lphosphorylceramide, which renders the cells Ca2+-sensitive.
3 Adenylyl cyclase (AC) 1 and AC8, the major Ca2+-sensitive AC isoforms, are not crucial for the base
6 uanylyl cyclase activating protein (GCAP), a Ca2+-sensitive activator, to resynthesize light-depleted
7 of Ca2+ can lower cAMP through its action on Ca2+-sensitive adenylate cyclases or phosphodiesterases,
10 e findings not only extend the means whereby Ca2+-sensitive adenylyl cyclases may be regulated, they
11 -exist, cyclic nucleotide-gated channels and Ca2+-sensitive adenylyl cyclases may reciprocally modula
14 se [Ca2+] by laser photolysis of NP-EGTA was Ca2+ sensitive and biphasic: a rapid component approxima
15 4-aminopyridine (4-AP) which suppressed the Ca2+ sensitive and other K+ currents in rat carotid body
19 unctions as a Ca2+/K+ ion exchanger, and two Ca2+-sensitive channels, one to import K+ into the Ca2+-
20 with the recently described bovine tracheal, Ca2+-sensitive chloride channel protein (bCLCA1), bovine
22 investigating the possible contribution of a Ca2+-sensitive chloride conductance to the pathogenesis
23 rine role in regulating basolateral membrane Ca2+-sensitive Cl- conductance linked to Cl- and fluid t
24 th CLCA1 exhibited an increase in whole-cell Ca2+-sensitive Cl- currents that were outwardly rectifie
26 s of internal repeats, and the complex forms Ca2+-sensitive contractile fibers that function to reori
32 laser scanning confocal microscopy using the Ca2+-sensitive dye Fluo-4/AM, we determined that spontan
34 en they were monitored with the low-affinity Ca2+-sensitive dye fura-2FF, but not with the high-affin
35 oma cells and primary astrocytes loaded with Ca2+-sensitive dye reveals that XeC selectively blocks b
36 event of fusion (which was the diffusion of Ca2+-sensitive dyes from egg into sperm) and any change
37 we have adapted biolistic techniques to load Ca2+-sensitive dyes into guard cells of the flowering pl
39 the dynamics of [Ca2+]i that regulates this Ca2+-sensitive enzyme under a variety of physiological c
41 tricular cardiomyocytes were loaded with the Ca2+-sensitive fluorescent dye Fluo-3 and imaged by a di
43 Intracellular Ca2+ was analyzed with the Ca2+-sensitive fluorescent dye INDO-1 and confirmed that
44 stores using confocal microscopic imaging of Ca2+-sensitive fluorescent dye loaded into the cells.
46 2+ signals in strips of UBSM loaded with the Ca2+-sensitive fluorescent dye, fluo-4, using laser scan
47 an worm Cerebratulus lacteus were mixed with Ca2+-sensitive fluorescent dyes and injected into unfert
49 a2+ nanoscale resolution (SCCaNR), employing Ca2+-sensitive fluorescent dyes to localize stochastic o
50 n levator auris longus motor terminals using Ca2+-sensitive fluorescent indicator dyes (rhod-2, rhod-
54 s could not be determined precisely with the Ca2+-sensitive fluorophore, fura-2, because of its high
57 ession of putative Ca2+-insensitive, but not Ca2+-sensitive, forms of alpha-actinin reduced inactivat
58 inside-out membrane patches were 2-fold less Ca2+ sensitive in high-frequency than in low-frequency c
60 ase rate stimulated by the minifilaments was Ca2+-sensitive, indicating that single regulatory length
61 ped rat ventricular myocytes loaded with the Ca2+-sensitive indicator fluo-3, using confocal microsco
64 rocytic endfeet exhibited large-conductance, Ca2+-sensitive K+ (BK) channel currents that could be ac
65 om the SR (sparks), stimulating plasmalemmal Ca2+-sensitive K+ (BK) channels, determines the refracto
68 e inhibited by the intermediate conductance, Ca2+-sensitive K+ (IKCa) channel inhibitors, TRAM-34, an
69 evented by clotrimazole, an inhibitor of the Ca2+-sensitive K+ (KCa) channel, suggesting that it was
70 muscle, Ca2+ release through RyRs activates Ca2+-sensitive K+ (KCa) channels to oppose vasoconstrict
73 the human pore-forming alpha-subunit of the Ca2+-sensitive K+ channel, Hslo, and the alpha-isoform o
75 ve large conductance, voltage-dependent, and Ca2+-sensitive K+ channels are activated by cGMP-depende
76 that the alpha-subunit of large conductance Ca2+-sensitive K+ channels is substrate for G-Ialpha kin
78 Intracellular Ca2+ was assessed by measuring Ca2+-sensitive K+ currents or imaging the fluorescence o
79 ype of K+ channel that dominates the IK: the Ca2+-sensitive (K(Ca)) channel, delayed rectifier (K(Dr)
80 e plasma membrane (PM) Ca2+ influx or of the Ca2+-sensitive leak coefficient of the ryanodine recepto
87 ucocorticoid-treated thymocytes occurs via a Ca2+-sensitive mechanism and that exogenous Ca2+ promote
88 e surface membrane may explain how numerous (Ca2+)-sensitive membrane processes are activated at time
92 ere we demonstrate that myosin Vb (MyoVb), a Ca2+-sensitive motor, conducts spine trafficking during
93 bservations indicated the lack of Ang II and Ca2+-sensitive NO production in pericytes of the vasa re
96 to Ca2+, Sr2+, and Ba2+, the complex remains Ca2+- sensitive on fusion-incompetent CV, and disruption
98 ly demonstrated that farnesol did not affect Ca2+-sensitive pathways implicated in smooth muscle cont
101 TRP) proteins, can mediate activation of the Ca2+-sensitive phosphatase calcineurin in nonexcitable c
103 pool." Upon activation of PKC, this "highly Ca2+-sensitive pool" is enhanced in size to a greater ex
104 l organelle-specific fluorescent markers and Ca2+-sensitive probes were used to identify the source o
108 endent mechanism involving the activation of Ca2+-sensitive protein phosphatase 2B (PP-2B, calcineuri
109 fferential regulation of Ras function by two Ca2+-sensitive Ras inhibitors: Ca2+-promoted Ras activat
110 ggest that although GCAP1 is involved in the Ca2+-sensitive regulation of GC in rod and cone outer se
111 l cyclase activating protein-2 (GCAP-2) is a Ca2+-sensitive regulator of phototransduction in retinal
114 n is an adaptor that functions to localize a Ca2+ sensitive signal transduction machinery in sperm to
117 cts of luminal Ca2+ are mediated by distinct Ca2+-sensitive site(s) at the luminal face of the channe
118 esicles and by shifting vesicles to a highly Ca2+-sensitive state, enabling exocytosis at sites relat
119 pid deactivation of rhodopsin is therefore a Ca2+-sensitive step controlling the amplitude of the lig
124 des activation of PI3-kinase, IGF1R and Akt, Ca2(+)-sensitive transcription factors and also TGFbeta1
125 t PCE1 is a component of a cell-specific and Ca2+-sensitive transcriptional regulatory mechanism that
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