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1 lphosphorylceramide, which renders the cells Ca2+-sensitive.
2                                          The Ca2+-sensitive 85-kDa cytosolic phospholipase A2 (cPLA2)
3   Adenylyl cyclase (AC) 1 and AC8, the major Ca2+-sensitive AC isoforms, are not crucial for the base
4     We conclude that gelsolin is the primary Ca2+-sensitive actin filament recycling protein in the c
5                                          The Ca2+-sensitive actin-severing protein gelsolin concentra
6 uanylyl cyclase activating protein (GCAP), a Ca2+-sensitive activator, to resynthesize light-depleted
7 of Ca2+ can lower cAMP through its action on Ca2+-sensitive adenylate cyclases or phosphodiesterases,
8 biological properties of both aequorin and a Ca2+-sensitive adenylyl cyclase.
9                                              Ca2+-sensitive adenylyl cyclases are key integrators of
10 e findings not only extend the means whereby Ca2+-sensitive adenylyl cyclases may be regulated, they
11 -exist, cyclic nucleotide-gated channels and Ca2+-sensitive adenylyl cyclases may reciprocally modula
12       Previous studies have established that Ca2+-sensitive adenylyl cyclases, whether endogenously o
13                                              Ca2+-sensitive adenylyl cyclases, whether endogenously o
14 se [Ca2+] by laser photolysis of NP-EGTA was Ca2+ sensitive and biphasic: a rapid component approxima
15  4-aminopyridine (4-AP) which suppressed the Ca2+ sensitive and other K+ currents in rat carotid body
16                                              Ca2+-sensitive and H+-sensitive fluorescent probes were
17             This stimulation of cAMP was not Ca2+-sensitive and was unaffected by a range of protein
18 est degree of aggregation and being the most Ca2+ sensitive at a given protein concentration.
19 unctions as a Ca2+/K+ ion exchanger, and two Ca2+-sensitive channels, one to import K+ into the Ca2+-
20 with the recently described bovine tracheal, Ca2+-sensitive chloride channel protein (bCLCA1), bovine
21 cule-1 (Lu-ECAM-1), and the human intestinal Ca2+-sensitive chloride channel protein (hCLCA1).
22 investigating the possible contribution of a Ca2+-sensitive chloride conductance to the pathogenesis
23 rine role in regulating basolateral membrane Ca2+-sensitive Cl- conductance linked to Cl- and fluid t
24 th CLCA1 exhibited an increase in whole-cell Ca2+-sensitive Cl- currents that were outwardly rectifie
25 lations in action potential duration through Ca2+-sensitive conductances.
26 s of internal repeats, and the complex forms Ca2+-sensitive contractile fibers that function to reori
27 reviously identified by complementation of a Ca2+-sensitive (csg1) mutant.
28 n by whole-cell patch-clamp recording of the Ca2+-sensitive currents.
29 ular Ca2+ stores, but without increasing the Ca2+-sensitive currents.
30 ds using microscopic digital imaging and the Ca2+ sensitive dye fura-2.
31 e Ca2+ signal to AMPA was examined using the Ca2+ sensitive dye fura-2.
32 laser scanning confocal microscopy using the Ca2+-sensitive dye Fluo-4/AM, we determined that spontan
33 s was measured by microfluorimetry using the Ca2+-sensitive dye fura-2.
34 en they were monitored with the low-affinity Ca2+-sensitive dye fura-2FF, but not with the high-affin
35 oma cells and primary astrocytes loaded with Ca2+-sensitive dye reveals that XeC selectively blocks b
36  event of fusion (which was the diffusion of Ca2+-sensitive dyes from egg into sperm) and any change
37 we have adapted biolistic techniques to load Ca2+-sensitive dyes into guard cells of the flowering pl
38 urface because of the limited penetration of Ca2+-sensitive dyes.
39  the dynamics of [Ca2+]i that regulates this Ca2+-sensitive enzyme under a variety of physiological c
40 tic plasticity by differential activation of Ca2+ -sensitive enzymes such as calmodulin.
41 tricular cardiomyocytes were loaded with the Ca2+-sensitive fluorescent dye Fluo-3 and imaged by a di
42 entration ([Ca2+]i) were monitored using the Ca2+-sensitive fluorescent dye furaptra.
43     Intracellular Ca2+ was analyzed with the Ca2+-sensitive fluorescent dye INDO-1 and confirmed that
44 stores using confocal microscopic imaging of Ca2+-sensitive fluorescent dye loaded into the cells.
45                                    Using the Ca2+-sensitive fluorescent dye, Fluo-3, AM, and a trypan
46 2+ signals in strips of UBSM loaded with the Ca2+-sensitive fluorescent dye, fluo-4, using laser scan
47 an worm Cerebratulus lacteus were mixed with Ca2+-sensitive fluorescent dyes and injected into unfert
48                     Although a wide range of Ca2+-sensitive fluorescent dyes is available, they are o
49 a2+ nanoscale resolution (SCCaNR), employing Ca2+-sensitive fluorescent dyes to localize stochastic o
50 n levator auris longus motor terminals using Ca2+-sensitive fluorescent indicator dyes (rhod-2, rhod-
51                                          The Ca2+-sensitive fluorescent indicator rhod-2 was used to
52          We used two-photon imaging with the Ca2+-sensitive fluorescent protein G-CaMP to map the pri
53 etry using membrane vesicles loaded with the Ca2+-sensitive fluorophore fura-2.
54 s could not be determined precisely with the Ca2+-sensitive fluorophore, fura-2, because of its high
55 ling was monitored using cells loaded with a Ca2+-sensitive fluorophore.
56 e 1, 3, 7, 11, 14 and 21 days old, using the Ca2+-sensitive fluoroprobe fura-2.
57 ession of putative Ca2+-insensitive, but not Ca2+-sensitive, forms of alpha-actinin reduced inactivat
58 inside-out membrane patches were 2-fold less Ca2+ sensitive in high-frequency than in low-frequency c
59                                              Ca2+-sensitive inactivation only appeared when the membr
60 ase rate stimulated by the minifilaments was Ca2+-sensitive, indicating that single regulatory length
61 ped rat ventricular myocytes loaded with the Ca2+-sensitive indicator fluo-3, using confocal microsco
62 sured with fluorescent voltage-sensitive and Ca2+-sensitive indicators in rat brain slices.
63                                              Ca2+-sensitive involucrin AP-1 promotor activity was inc
64 rocytic endfeet exhibited large-conductance, Ca2+-sensitive K+ (BK) channel currents that could be ac
65 om the SR (sparks), stimulating plasmalemmal Ca2+-sensitive K+ (BK) channels, determines the refracto
66 um (Ca2+ sparks) activate large-conductance, Ca2+-sensitive K+ (BK) channels.
67           The activity of large conductance, Ca2+-sensitive K+ (BKCa) channels, known to control neur
68 e inhibited by the intermediate conductance, Ca2+-sensitive K+ (IKCa) channel inhibitors, TRAM-34, an
69 evented by clotrimazole, an inhibitor of the Ca2+-sensitive K+ (KCa) channel, suggesting that it was
70  muscle, Ca2+ release through RyRs activates Ca2+-sensitive K+ (KCa) channels to oppose vasoconstrict
71                                 Voltage- and Ca2+-sensitive K+ (MaxiK) channels play key roles in con
72                                          The Ca2+-sensitive K+ channel (K(Ca) channel) plays a key ro
73  the human pore-forming alpha-subunit of the Ca2+-sensitive K+ channel, Hslo, and the alpha-isoform o
74                 Increased [Ca2+]i stimulates Ca2+-sensitive K+ channels (IK-Ca), and this, in turn, h
75 ve large conductance, voltage-dependent, and Ca2+-sensitive K+ channels are activated by cGMP-depende
76  that the alpha-subunit of large conductance Ca2+-sensitive K+ channels is substrate for G-Ialpha kin
77 rnal stores and the subsequent activation of Ca2+-sensitive K+ channels.
78 Intracellular Ca2+ was assessed by measuring Ca2+-sensitive K+ currents or imaging the fluorescence o
79 ype of K+ channel that dominates the IK: the Ca2+-sensitive (K(Ca)) channel, delayed rectifier (K(Dr)
80 e plasma membrane (PM) Ca2+ influx or of the Ca2+-sensitive leak coefficient of the ryanodine recepto
81                  Synaptotagmin competes in a Ca2+-sensitive manner with binding of Gbetagamma to SNAP
82 -binding protein that activates RetGC-1 in a Ca2+-sensitive manner.
83 lyl cyclase-activating proteins (GCAPs) in a Ca2+-sensitive manner.
84 cetylases (HDAC4) regulate SRF activity in a Ca2+-sensitive manner.
85 ment membrane guanylate cyclase (RetGC) in a Ca2+-sensitive manner.
86 eptor membrane guanylyl cyclase, RetGC, in a Ca2+-sensitive manner.
87 ucocorticoid-treated thymocytes occurs via a Ca2+-sensitive mechanism and that exogenous Ca2+ promote
88 e surface membrane may explain how numerous (Ca2+)-sensitive membrane processes are activated at time
89                                          The Ca2+-sensitive microelectrodes were sensitive to intrace
90 rial matrix Ca2+ ([Ca2+]m) and activation of Ca2+-sensitive mitochondrial metabolism.
91 esis in order to understand the mechanism of Ca2+-sensitive modulation of GC1 activity.
92 ere we demonstrate that myosin Vb (MyoVb), a Ca2+-sensitive motor, conducts spine trafficking during
93 bservations indicated the lack of Ang II and Ca2+-sensitive NO production in pericytes of the vasa re
94 elium (47+/-8 U), indicating the presence of Ca2+-sensitive NO production.
95                We also find that MsNOS has a Ca2+-sensitive NO-producing activity similar to that of
96 to Ca2+, Sr2+, and Ba2+, the complex remains Ca2+- sensitive on fusion-incompetent CV, and disruption
97 2+]i due to Ca2+ release from IP3-sensitive, Ca2+-sensitive, or mitochondrial Ca2+ stores.
98 ly demonstrated that farnesol did not affect Ca2+-sensitive pathways implicated in smooth muscle cont
99       Deletion of either gene suppresses the Ca2+-sensitive phenotype of csg2Delta mutants, which ari
100  in a calcineurin mutant strain results in a Ca2+-sensitive phenotype.
101 TRP) proteins, can mediate activation of the Ca2+-sensitive phosphatase calcineurin in nonexcitable c
102                   This may signal a role for Ca2+-sensitive PKC isoforms in cardiac mechanisms involv
103  pool." Upon activation of PKC, this "highly Ca2+-sensitive pool" is enhanced in size to a greater ex
104 l organelle-specific fluorescent markers and Ca2+-sensitive probes were used to identify the source o
105                                             (Ca2+)-sensitive processes at cell membranes involved in
106                                We describe a Ca2+-sensitive protein complex involved in the regulatio
107                      Increased expression of Ca2+-sensitive protein kinase C (PKC) isoforms may be im
108 endent mechanism involving the activation of Ca2+-sensitive protein phosphatase 2B (PP-2B, calcineuri
109 fferential regulation of Ras function by two Ca2+-sensitive Ras inhibitors: Ca2+-promoted Ras activat
110 ggest that although GCAP1 is involved in the Ca2+-sensitive regulation of GC in rod and cone outer se
111 l cyclase activating protein-2 (GCAP-2) is a Ca2+-sensitive regulator of phototransduction in retinal
112 t is important for the regulation of several Ca2+-sensitive responses.
113       The homogenate-activated channels were Ca2+ sensitive, selective for Ca2+ over Cs+, and driven
114 n is an adaptor that functions to localize a Ca2+ sensitive signal transduction machinery in sperm to
115 trations that should maximally activate most Ca2+-sensitive signaling kinases and phosphatases.
116                          The manner in which Ca2+-sensitive signaling proteins are activated in contr
117 cts of luminal Ca2+ are mediated by distinct Ca2+-sensitive site(s) at the luminal face of the channe
118 esicles and by shifting vesicles to a highly Ca2+-sensitive state, enabling exocytosis at sites relat
119 pid deactivation of rhodopsin is therefore a Ca2+-sensitive step controlling the amplitude of the lig
120            We investigated the properties of Ca2+-sensitive steps in the cycling of synaptic vesicles
121 lved in PC2 oligomerization, and PC2-EF is a Ca2+-sensitive switch.
122 ular cues to the regulation of intracellular Ca2+-sensitive targets.
123 a2+]m) increase regulates intramitochondrial Ca2+-sensitive targets.
124 des activation of PI3-kinase, IGF1R and Akt, Ca2(+)-sensitive transcription factors and also TGFbeta1
125 t PCE1 is a component of a cell-specific and Ca2+-sensitive transcriptional regulatory mechanism that
126           Exon 6b encodes part of a putative Ca2+-sensitive troponin binding site in striated muscle

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