1 logically modulated by dihydropyridine (DHP) Ca2+ antagonists and agonists.

2                 These two clones, a panel of Ca2+ antagonists, and agonists were used to determine th

3 -[diethylamino]octyl ester, an intracellular Ca2+ antagonist, did not change the potentiation by insu

4                          The dihydropyridine Ca2+ antagonist drugs used in the therapy of cardiovacul

5 ntial of -60 mV and was not inhibited by the Ca2+ antagonists nicardipine (1 microM) or D600 (10 micr

6                               The effects of Ca2+ antagonists on ABA-induced stomatal closure and the

7 nversely, pretreatment with an intracellular Ca2+ antagonist or an antioxidant blocked AngII-induced

8  whose mutation reduced the affinity for the Ca2+ antagonist PN200-110 by 2-25-fold.

9 creased the affinity for the dihydropyridine Ca2+ antagonist PN200-110 by 416-fold with no effect on