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1 CaCl2 also improved the retention of Zn and P in the fle
2 CaCl2 destabilized GO more aggressively than MgCl2 and N
3 CaCl2 had no effect on troponin binding to the thin fila
4 CaCl2 improved both systolic and diastolic function and
5 CaCl2, but not neomycin or KC1, could overcome the inhib
6 mposition of 0.5% alginate, 400ml oil, 0.05M CaCl2 and 100ml surfactant was recognized as the optimiz
9 the selective precipitation of RNA with 1.4M CaCl2, followed by a final selective precipitation of th
14 heat stability in autumn, whereas with added CaCl2, the best heat stability was observed in spring.
15 ion in cells shocked in buffer without added CaCl2 than in cells shocked in 200 or 500 microM CaCl2 o
16 ose in cells shocked in buffer without added CaCl2 were measured in cells following acid shock in buf
19 dependent increase in [Ca2+](m) after adding CaCl2 equivalent to 20, 114, and 485 nM extramatrix free
20 work, the impact was evaluated of alginate, CaCl2, oil and surfactant content on the size and encaps
21 study the influence of pH (3.5 and 7.0) and CaCl2 and MgCl2 addition on heat-set gelation of a quino
23 ch as calcium gluconate, calcium acetate and CaCl2, had greater effects on the rate and extent of fre
29 cts of the substitution of NaCl with KCl and CaCl2 on the physicochemical, mineral and sensory profil
30 in aqueous solutions by HCl, NaCl, KCl, and CaCl2 and in acetonitrile by CF3COOH and the supporting
34 with individual salts (NaCl, KCl, MgCl2, and CaCl2) using deionized water with a known stable isotope
38 ininogen (HK, 45 nM), ZnCl2 (25 microM), and CaCl2 (2 mM), conditions optimal for factor XI binding t
42 es the effect of including hydrated NaCl and CaCl2 (up to 6% w/w) on the physicochemical properties o
43 logenin as a function of increasing NaCl and CaCl2 concentration beginning with solution conditions o
44 ited on silica surfaces at elevated NaCl and CaCl2 concentrations before being rinsed with eluents of
49 n a variety of salt solutions (KCl, NaCl and CaCl2) at low salt concentrations (<10(-4) M) showed sev
51 in the presence of different salts (NaCl and CaCl2), ionic strengths, pHs, and temperatures were cons
52 da tympani responses to NaCl, KCl, NH4Cl and CaCl2 were recorded in Sprague-Dawley rats, and in wild-
54 CaM Mr in presence of synthetic peptide and CaCl2 in 4 M urea polyacrylamide gel electrophoresis.
56 However, the presence of both sucrose and CaCl2 restored the ABA response to 20-40% of the maximum
58 er incubation in media amended with urea and CaCl2 and/or SrCl2 , calcite (CaCO3 ), strontianite (SrC
59 l filtration with a combination of anhydrous CaCl2 (100mg) and primary-secondary amine (PSA, 25mg) as
60 re, hydrocooling cherry fruit in appropriate CaCl2 solutions (i.e., 0.2-0.5%) for 5 min and then pass
61 theart' and 39-188% linearly for 'Lapins' as CaCl2 rate increased from 0.2% to 2.0% at 0 degrees C fo
62 ersely, favorable deposition was observed at CaCl2 concentrations above 0.5 mM when the charge of SLB
67 ing reaction in EDTA followed immediately by CaCl2, the looped DNA was blocked from recombination whi
70 capsules cross-linked with divalent cationic CaCl2 salt (MCS), and the third group consisted of contr
71 s avium L.), the effect of calcium chloride (CaCl2) added to hydro-cooling water on physiological and
72 enzylaminopurine (BAP) and calcium chloride (CaCl2) could induce tolerance to chilling and could cons
73 with low concentrations of calcium chloride (CaCl2) induced chronic degeneration and calcification of
74 agnesium chloride (MgCl2), calcium chloride (CaCl2)], and electrolyte mixtures (KCl + CaCl2) through
77 r a wide range of mono- (NaCl) and divalent (CaCl2) electrolytes and using time-resolved dynamic ligh
84 he role of calcium during APC, extracellular CaCl2 was decreased to 0.5 mM but only during the APC ep
93 d C(salt), in concentrated aqueous LiCl-HCl, CaCl2-HCl, and AlCl3-HCl solutions (C(acid) at 5-6 M; C(
94 ually no response to stimuli (including high CaCl2 concentration) and inhibitors (CaCl2 omission bein
97 ted and measured dissolved concentrations in CaCl2 soil extracts, using the different extractions as
98 h MWNTs and silica surfaces with the drop in CaCl2 concentration likely resulted in the decrease in t
99 nced colloidal stability of nanoparticles in CaCl2 solution but rapid nanoparticle-nanoparticle aggre
101 gible amount of deposited GO was released in CaCl2 under favorable conditions due to the binding of G
102 CFM reveals stronger adhesion on silica in CaCl2 compared with the other electrolytes, and shows a
103 2 Hz) was deposited on the silica surface in CaCl2 due to the bridging ability of Ca(2+) ions with GO
104 quadratic polynomial manner with increasing CaCl2 concentration from 0.2% to 2.0% in cold water (0 d
107 In this work, the effect of an inorganic (CaCl2) and an organic (tetraalkylphosphonium chloride) m
109 de (CaCl2)], and electrolyte mixtures (KCl + CaCl2) through a gated nanofluidic device was performed.
110 s in aqueous solutions containing NaCl, KCl, CaCl2, and Tris buffer show that the magnitude of the ef
111 5-minute intracoronary infusion of 20 mmol/L CaCl2 or saline before undergoing 1 hour of coronary occ
112 were diluted in a buffer containing 5 mmol/L CaCl2, phospholipid vesicles (10 micromol/L), and APC.
113 t (TIMP-1-/-) mice were exposed to 0.5 mol/L CaCl2 for 15 minutes, with terminal studies performed at
114 d Cu, and then extracted using either 0.01 M CaCl2 or 0.005 M diethylenetriaminepentaacetic acid (DTP
115 and soil in synthetic soil solution (0.01 M CaCl2), but in synthetic earthworm guts desorption was h
116 mmon leaching solutions (0.5 M K2SO4, 0.01 M CaCl2, 2 M KCl, and H2O) were used to extract WSOM from
118 ic chemistries (pH, salt types (NaCl, MgCl2, CaCl2), ionic strength) relevant to natural and engineer
119 on of three commonly prevalent salts, MgCl2, CaCl2, and NaCl, next to a sapphire substrate using surf
121 ric monitoring of NADH removal in 100 microM CaCl2 at ionic strength 0.15 M, pH 7.0 and 21 degreesC.
122 g acid shock in buffer containing 200 microM CaCl2 and supplemented with neomycin, ruthenium red, or
123 ocked in 500 microM CaCl2 than in 200 microM CaCl2, although the accumulation durations were similar.
125 greater in cells acid shocked in 500 microM CaCl2 than in 200 microM CaCl2, although the accumulatio
127 um; HC = imbibed and raised in 10 millimolar CaCl2), in the absence of additional Ca2+ (intermediate
130 ckground electrolyte of 100 mM NaCl and 1 mM CaCl2 in equilibration with 400 ppmv CO2(g) ([U(VI)] = 4
135 ations ranging from 0% to 65% (w/w) in 10 mM CaCl2 solution, thus producing formulations with a total
136 Cl2 and a binding isotherm measured in 10 mM CaCl2 using gel filtration to separate enzyme-bound and
142 00, and 70 mM NaCl and 10, 12, 6, and 7.5 mM CaCl2 for nC60, nC70, nC76, and nC84, respectively.
144 ffer (pH 7.2) containing 0.14 M NaCl, 0.5 mM CaCl2, and 0.15 mM MgCl2 or with this buffer and either
145 M reduced and oxidized glutathione, and 5 mM CaCl2, followed by buffer exchange to remove denaturant
149 der favorable deposition conditions (1.50 mM CaCl2 and pH 7.1) and the deposited MWNTs were then rins
152 IC50 of 1.79 and 0.47 mM when 10 and 1.8 mM CaCl2, respectively, was used as the charge carrier.
154 istribution, and protein solubility in model CaCl2 solutions (50 mmol L(-1)) or rennet casein dispers
155 bons through cathodic polarization in molten CaCl2 at temperatures of about 1100 K, which generates p
156 omparing sorption from 0.01 N KCl and 0.01 N CaCl2; however, no significant differences were observed
158 ynthetic formation brine, and synthetic NaCl+CaCl2 brine were collected at the pressures from 100 to
161 a function of GO concentration and in NaCl, CaCl2, and MgCl2 as a function of ionic strength (IS).
163 of aggregates in the presence or absence of CaCl2, and CaCl2 could not reduce pre-formed aggregates.
164 f gyration of bacterial EPS with addition of CaCl2 was 20 times larger than that with the addition of
165 se parameters were enhanced with addition of CaCl2 while NaCl exhibited an opposite trend for all of
166 radius of bacterial EPS with the addition of CaCl2, but no change was observed after addition of MgCl
171 rysm formation induced by the application of CaCl2 to the aortic surface was almost completely ablate
172 erve as an alternative to the application of CaCl2 used to extend the shelf life of numerous agricult
176 n with urea and equivalent concentrations of CaCl2 and SrCl2 was identified as hydrated Ca and Sr car
177 the presence of increasing concentrations of CaCl2, but the opposite trend was observed in the presen
179 y the re-addition of calcium (in the form of CaCl2), but not by the addition of magnesium (MgCl2).
183 KO, n =11) mice were exposed to 0.5 mol/L of CaCl2 for 15 minutes with terminal studies performed at
186 ) and PF1.2 (but not PF1) in the presence of CaCl2 (2 mM) were able to replace HK (45 nM) in the pres
188 egation by alpha-thrombin in the presence of CaCl2 but not with EDTA, suggesting that fibrinogen cros
191 e-derived ligand provided in the presence of CaCl2 Michael adducts in moderate to high enantioselecti
192 r proteolytic degradation in the presence of CaCl2 than in the presence of EDTA, demonstrating that c
201 t crosslinking of proteins by citric acid or CaCl2 resulted in the formation of cold-set hydrogels.
202 se, in buffers that contained either EDTA or CaCl2 so as to preclude DNA cleavage by the endonuclease
211 icated that soil characteristics, namely, pH(CaCl2) for Fe and Cr, silt and fine-sand for Al and V, O
212 of FKS2 expression in response to pheromone, CaCl2, or loss of FKS1 function requires the Ca2+/calmod
213 groups were also prepared-calcium phosphate (CaCl2.2H2O + K2HPO4 in buffer solution) and SDF (Ag[NH3]
214 using low-melting-point ternary molten salts CaCl2 -MgCl2 -NaCl, which still retains high CaSiO3 solu
216 alues obtained suggests the presence of some CaCl2/lactose aggregates in the media, which are influen
219 nhibitors of elastin degradation) before the CaCl2-mediated injury significantly reduced elastin calc
224 reatment in Ca2+-free medium) and exposed to CaCl2 or MnCl2 had a greater rate of Ca2+ entry (18.6 +/
227 und that MMP-knockout mice were resistant to CaCl2-mediated aortic injury and did not develop elastin
229 ned the surface of PG starch particles while CaCl2 did not bring about obvious changes on their morph
230 were embedded in alginate cross-linked with CaCl2 and cultured in modified Ham's F10 medium with 10%
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