ライフサイエンスコーパス: Caenorhabditis

1 mediated killing is unique to androdioecious Caenorhabditis, and may reduce the number of males in he

2 g an independent introgression fragment from Caenorhabditis briggsae X Chromosome in an otherwise Cae

3 ytogenes in vitro, in cell culture, and in a Caenorhabditis elegans (C. elegans) infection model.

4 The nematode worm Caenorhabditis elegans (C. elegans) is a versatile and w

5 Using the Caenorhabditis elegans (C. elegans) model, various studi

6 CblC from Caenorhabditis elegans (ceCblC) also exhibits a robust t

7 ing in two pivotal model animals - mouse and Caenorhabditis elegans - and compare them to consider wh

8 forward genetic screens, we isolated a novel Caenorhabditis elegans active zone gene, clarinet (cla-1

9 smitter release, we identified a mutation in Caenorhabditis elegans AIPR-1 (AIP-related-1), which cau

10 We expressed human and Caenorhabditis elegans AMPylation enzymes-huntingtin yea

11 In Caenorhabditis elegans an increase in the refractility o

12 Using RNAi screening for defects in Caenorhabditis elegans anchor cell (AC) invasion, we fou

13 activate a canonical cannabinoid receptor in Caenorhabditis elegans and also modulate monoaminergic s

14 , where biochemical and molecular studies in Caenorhabditis elegans and Ascaris suum have identified

15 Recent research in the invertebrates Caenorhabditis elegans and Drosophila melanogaster has l

16 ntly targets the degradation of cyclin B1 in Caenorhabditis elegans and human cells.

17 matin-associated protein degradation in both Caenorhabditis elegans and humans, which is relevant to

18 se analyses and cross-species experiments in Caenorhabditis elegans and in chondrogenic cell lines im

19 tional carriers of epigenetic information in Caenorhabditis elegans and in other organisms.

20 Caenorhabditis elegans and its bacterial diet provide a

21 flux assays in multiple tissues of wild-type Caenorhabditis elegans and long-lived daf-2/insulin/IGF-

22 a during gut infection in two animal models (Caenorhabditis elegans and mice).

23 C increase lifespan and stress resistance in Caenorhabditis elegans and reduce accumulation of reacti

24 splatin mutation signatures in XPF-deficient Caenorhabditis elegans and supports a model in which tra

25 re, we study this process using the predator Caenorhabditis elegans and the bacterial prey Streptomyc

26 e been shown to modulate organismal aging in Caenorhabditis elegans and to impact on age-related dise

27 on-apoptotic cell death process operating in Caenorhabditis elegans and vertebrate development, and i

28 rase (QPRTase) is not encoded in its genome, Caenorhabditis elegans are reported to lack a de novo NA

29 Here we use Caenorhabditis elegans as a model in which to study RIBE

30 Using Caenorhabditis elegans as a model, we examine how both a

31 We have used Caenorhabditis elegans as an invertebrate model to ident

32 de reporter to measure lysosomal chloride in Caenorhabditis elegans as well as murine and human cell

33 rofile nearly 50,000 cells from the nematode Caenorhabditis elegans at the L2 larval stage, which pro

34 In Caenorhabditis elegans AWA neurons, which are crucial fo

35 ance, we pursued a forward genetic screen in Caenorhabditis elegans based on the phenotype swimming-i

36 e used as experimental models decades before Caenorhabditis elegans became known as 'the worm'.

37 We investigated low zinc homeostasis in Caenorhabditis elegans because the genome encodes 14 evo

38 tes of anthelmintics have been restricted to Caenorhabditis elegans because they have failed when app

39 e that IL-17 plays a neuromodulatory role in Caenorhabditis elegans by acting directly on neurons to

40 arget sites, we isolated AGO-bound RNAs from Caenorhabditis elegans by individual-nucleotide resoluti

41 athway that controls mitochondrial fusion in Caenorhabditis elegans by repressing the expression of t

42 The free-living nematode Caenorhabditis elegans can adapt to harsh environments b

43 Here, we show that Caenorhabditis elegans can alternate between attractive

44 Here we show that the nematode Caenorhabditis elegans can discriminate spatial patterns

45 We show that Caenorhabditis elegans changes how it processes sensory

46 of gene mutations on the trafficking of the Caenorhabditis elegans choline transporter orthologue re

47 We utilized the Caenorhabditis elegans CLC-1/2/Ka/Kb anion channel homol

48 Using a computational model of the Caenorhabditis elegans connectome dynamics, we show that

49 Caenorhabditis elegans contains 25 Argonautes, of which,

50 We show that PKC-2, a Caenorhabditis elegans cPKC, is essential for a complex

51 ontig in a few minutes, and assemble 45-fold Caenorhabditis elegans data in 9 min, orders of magnitud

52 J and L3MBTL3 in Drosophila melanogaster and Caenorhabditis elegans demonstrate that the functional l

53 A new study in Caenorhabditis elegans demonstrates that changes in neur

54 small RNAs comprising six miRNAs involved in Caenorhabditis elegans development and two controls were

55 ccurs in a highly reproducible manner during Caenorhabditis elegans development.

56 CDC-42 in AJ formation and regulation during Caenorhabditis elegans embryo elongation, a process driv

57 ioning of mitotic spindle in the single-cell Caenorhabditis elegans embryo is achieved initially by t

58 n kinetochore-microtubule attachments in the Caenorhabditis elegans embryo.

59 ercellular surface during cell division in a Caenorhabditis elegans embryo.

60 The Caenorhabditis elegans embryonic nervous system, compris

61 Spindly to recruit dynein to kinetochores in Caenorhabditis elegans embryos and human cells.

62 The lack of physiological recordings from Caenorhabditis elegans embryos stands in stark contrast

63 Here we use computational simulations of Caenorhabditis elegans embryos to address this fundament

64 luorescently labeling endogenous proteins in Caenorhabditis elegans embryos, we show that dynein exis

65 erties on gene expression and development in Caenorhabditis elegans embryos.

66 components on AIN-1/GW182 and NTL-1/CNOT1 in Caenorhabditis elegans embryos.

67 physical properties of constricting rings in Caenorhabditis elegans embryos.

68 chromosomal loci in the interphase nuclei of Caenorhabditis elegans embryos.

69 The nceh-1 gene of Caenorhabditis elegans encodes an ortholog of neutral ch

70 g these questions using the robust number of Caenorhabditis elegans epidermal stem cells, known as se

71 We demonstrate that the Caenorhabditis elegans Epithelial Fusion Failure 1 (EFF-

72 and downstream motor neurons (A-MNs) in the Caenorhabditis elegans escape circuit, we found that dis

73 Dauer larvae of the nematode Caenorhabditis elegans exhibit a phoretic behavior calle

74 ex, time- and resource-consuming, transgenic Caenorhabditis elegans expressing hTTR provide an optima

75 Here we show that adult neurons from Caenorhabditis elegans extrude large (approximately 4 mu

76 autophagy is induced in multiple tissues of Caenorhabditis elegans following hormetic heat stress or

77 o studies, using small animal models such as Caenorhabditis elegans for hit identification and lead o

78 Finally, D-RR4 protected Caenorhabditis elegans from lethal infections of P. aeru

79 ere, we investigated the temporal aspects of Caenorhabditis elegans gene expression changes using ave

80 time-lapse in vivo single-cell analysis and Caenorhabditis elegans genetics, our evidence does not s

81 The Caenorhabditis elegans germline provides a tractable sys

82 hich we call SIP-HAVA-seq, by characterizing Caenorhabditis elegans germline stem cell mutation accru

83 the CED-3 caspase in distinct regions of the Caenorhabditis elegans germline.

84 ultiple small RNA-seq datasets from the worm Caenorhabditis elegans had shorter forms of miRNAs that

85 Since 1999, Caenorhabditis elegans has been extensively used to stud

86 The nematode Caenorhabditis elegans has been particularly instrumenta

87 ting gene expression, but its application in Caenorhabditis elegans has not been described.

88 Work in Caenorhabditis elegans has shown that the UPR(mt) is reg

89 s (OV) as a natural pathogen of the nematode Caenorhabditis elegans has stimulated interest in explor

90 ies such as larval zebrafish, Drosophila, or Caenorhabditis elegans have become key model organisms i

91 to unravel the sex determination pathway in Caenorhabditis elegans He inferred the order of genes in

92 ammals, satiety signals induce quiescence in Caenorhabditis elegans Here we report that the C. elegan

93 A comparison of the connectomes of the Caenorhabditis elegans hermaphrodite and male nervous sy

94 ctory long-term associative memory (LTAM) in Caenorhabditis elegans hermaphrodites.

95 We have identified atx-2, the Caenorhabditis elegans homolog of the human ATXN2L and A

96 t comprise the whole body of the small worm, Caenorhabditis elegans However, to fully elucidate the n

97 n of the first miRNA, lin-4, in the nematode Caenorhabditis elegans in 1993, thousands of miRNAs have

98 ebrate representative twitchin (UNC-22) from Caenorhabditis elegans In in vitro experiments, change o

99 ed in determining a stimulatory phenotype of Caenorhabditis elegans in response to physiologically re

100 re of a cyclic-nucleotide-gated channel from Caenorhabditis elegans in the cyclic guanosine monophosp

101 ng followed by mass spectrometry analysis of Caenorhabditis elegans infected with two species of Nema

102 ochastic bacterial community assembly in the Caenorhabditis elegans intestine is sufficient to produc

103 Caenorhabditis elegans is among the most powerful model

104 research, the glycome of the model nematode Caenorhabditis elegans is still not fully understood.

105 endocrine regulation of diverse behaviors of Caenorhabditis elegans is under the control of the DAF-7

106 Caenorhabditis elegans lacking ADARs exhibit reduced che

107 In Caenorhabditis elegans larvae, sleep is associated with

108 e that during periods of acute starvation in Caenorhabditis elegans larvae, the master metabolic regu

109 Long-term studies of Caenorhabditis elegans larval development traditionally

110 n exposure early in the life of the nematode Caenorhabditis elegans leads to a long-lasting aversion

111 pletion of casein kinase 1 gamma (CSNK-1) in Caenorhabditis elegans led to the formation of large pol

112 on of SLO-2 (a homolog of mammalian Slo2) in Caenorhabditis elegans Loss-of-function (lf) mutants of

113 Mutations in Caenorhabditis elegans mafr-1 that truncate the C-box re

114 Here, we describe two distinct ways in which Caenorhabditis elegans males cause faster somatic aging

115 ic reticulum-resident molecular chaperone in Caenorhabditis elegans MEC-6 modulates the expression of

116 Here, using Caenorhabditis elegans mechanosensory neurons, we addres

117 s full recovery of function after axotomy of Caenorhabditis elegans mechanosensory neurons.

118 tant is hypervirulent both in vitro and in a Caenorhabditis elegans model in vivo.

119 ecules in human cerebrospinal fluid and in a Caenorhabditis elegans model of AD.

120 for cells in biofilms, and to virulence in a Caenorhabditis elegans model of infection.

121 ted tauopathy in multiple models including a Caenorhabditis elegans model of tauopathy.

122 tand cannabinoid signaling, we have used the Caenorhabditis elegans model to examine the effects of c

123 uted to the in vivo virulence of PA14 in the Caenorhabditis elegans model.

124 Moreover, in genetically tractable Caenorhabditis elegans models, expression of alpha-synuc

125 , we made a series of observations utilizing Caenorhabditis elegans models, mammalian cell lines, pri

126 asynaptic acetylcholine receptors (AChRs) in Caenorhabditis elegans muscle cells.

127 S) microscopy and systematically identify 57 Caenorhabditis elegans mutants with altered lipid distri

128 Using Caenorhabditis elegans mutants, we identify DNA repair f

129 Here we describe a novel role for the Caenorhabditis elegans NCLX-type protein, NCX-9, in neur

130 l variation to behavior by monitoring single Caenorhabditis elegans nematodes over their complete dev

131 e low-dimensional functional response of the Caenorhabditis elegans network of neurons to propriocept

132 Using Caenorhabditis elegans neuromuscular junction as a model

133 rgy demands and support synaptic function in Caenorhabditis elegans neurons.

134 Here we describe a novel Caenorhabditis elegans nuclear receptor, HIZR-1, that is

135 conducted an RNA interference screen of the Caenorhabditis elegans nucleome in a strain carrying an

136 In the Caenorhabditis elegans one-cell embryo, the astral micro

137 During Caenorhabditis elegans oocyte meiosis, a multi-protein r

138 d ablation, and genetic perturbations in the Caenorhabditis elegans oocyte, we studied the mechanism

139 ochore attachments have not been observed in Caenorhabditis elegans oocytes and chromosomes instead a

140 We find that Caenorhabditis elegans oocytes whose maturation is arres

141 We adapted a Caenorhabditis elegans organogenesis model to enable a g

142 r investigations of bpl-1, which encodes the Caenorhabditis elegans ortholog of HCS.

143 factor (erythroid-derived 2)-like 2 and its Caenorhabditis elegans ortholog, SKN-1, are transcriptio

144 ven decision-theoretical model of feeding in Caenorhabditis elegans Our central assumption is that fo

145 quired for the first embryonic abscission in Caenorhabditis elegans Our findings indicate that membra

146 aviors, we utilized the genetic model system Caenorhabditis elegans Our studies demonstrate that grk-

147 Here, we implicate the two Caenorhabditis elegans PABPs (PAB-1 and PAB-2) in miRNA-

148 that mammalian PKDs 1-3 and the prototypical Caenorhabditis elegans PKD, DKF-2A, are exclusively (hom

149 Caenorhabditis elegans possesses 19 GABAergic motor neur

150 Nematodes such as the model organism Caenorhabditis elegans produce various homologous series

151 5 orthologues in Drosophila melanogaster and Caenorhabditis elegans promotes longevity.

152 These include the Caenorhabditis elegans protein LAF-1, which forms P gran

153 he well-studied sex determination pathway of Caenorhabditis elegans Repression of nhl-2 by the mir-35

154 edator-prey coevolution, we investigated how Caenorhabditis elegans responds to the predatory fungus

155 neuronal cultures and functional analyses in Caenorhabditis elegans revealed that the UNC13A variant

156 We show that the Caenorhabditis elegans RMI1 homolog-1 (RMH-1) functions

157 of synapse formation and axon termination in Caenorhabditis elegans RPM-1 functions in a ubiquitin li

158 New work in Caenorhabditis elegans shows that during embryogenesis e

159 is of a previous genetic screening result in Caenorhabditis elegans shows that homo-trimerization is

160 3 domain-containing protein family member in Caenorhabditis elegans SORB-1 is strongly localized to i

161 inated with extraintestinal copper levels in Caenorhabditis elegans Specifically, we show that CUA-1,

162 Under laboratory conditions, Caenorhabditis elegans sperm are very efficient at navig

163 In this study, we find that Caenorhabditis elegans sperm DNA stays in a fixed positi

164 The Caenorhabditis elegans spermatheca is a bag-like organ o

165 rther examine the effects of squalamine in a Caenorhabditis elegans strain overexpressing alpha-synuc

166 on of a deleterious mtDNA in a heteroplasmic Caenorhabditis elegans strain that stably expresses wild

167 The genomes of two Caenorhabditis elegans strains, a wild-type strain and a

168 Forward chemical screening conducted in Caenorhabditis elegans suggested that pOPCs reduced the

169 family homologs in chicken, Drosophila, and Caenorhabditis elegans suggests this antagonism is conse

170 The Caenorhabditis elegans SUN domain protein, UNC-84, funct

171 dentify a TRH-like neuropeptide precursor in Caenorhabditis elegans that belongs to a bilaterian fami

172 Here, we show in the model system Caenorhabditis elegans that expression of the arginine-c

173 loid-beta proteotoxicity in human, mouse and Caenorhabditis elegans that involves the mitochondrial u

174 al. identify a genetic locus in the nematode Caenorhabditis elegans that underlies nictation and cont

175 crosses between wild strains of the nematode Caenorhabditis elegans The element is made up of sup-35,

176 responses to oxygen, pheromones, and food in Caenorhabditis elegans The molecular composition of the

177 ilia in chemosensory neurons of the nematode Caenorhabditis elegans The trafficking defect caused by

178 bilization at the transition to adulthood in Caenorhabditis elegans This novel connection involves cr

179 r organization of meiotic chromosome axes in Caenorhabditis elegans through STORM (stochastic optical

180 Studies in metazoan models ranging from Caenorhabditis elegans to mammals have revealed cell-aut

181 Using the Caenorhabditis elegans touch receptor neurons, we analyz

182 (NAM) form of vitamin B3 is an agonist of a Caenorhabditis elegans TRPV channel.

183 The Caenorhabditis elegans UBCH7 homolog, UBC-18, plays a cr

184 a gain-of-function in rescue experiments in Caenorhabditis elegans unc-18 nulls.

185 her cell types, Munc13 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 13) proteins p

186 The nematode Caenorhabditis elegans uses simple building blocks from

187 Caenorhabditis elegans vulva development provides an in

188 ensitizing miRNAs, we initially utilized the Caenorhabditis elegans vulval cell model, an in vivo sys

189 The pha-1 gene of Caenorhabditis elegans was originally heralded as a mast

190 ed with feeding and fasting in the roundworm Caenorhabditis elegans We identified neural circuits thr

191 ius tipulae, a distant relative of the model Caenorhabditis elegans We used this draft to identify th

192 ere we identify life-limiting pathologies in Caenorhabditis elegans with a necropsy analysis of worms

193 ly affects behavior in mice, Drosophila, and Caenorhabditis elegans Yet, the mechanisms that modulate

194 Using the Caenorhabditis elegans zygote as a model, we find that t

195 odel to simulate contractile dynamics in the Caenorhabditis elegans zygote cytokinetic ring.

196 complex composition and stoichiometry during Caenorhabditis elegans zygote polarization, which takes

197 shment of anterior-posterior polarity in the Caenorhabditis elegans zygote requires two different pro

198 Here, we report that in the Caenorhabditis elegans zygote, feedback between active R

199 ent cortical actomyosin contractility in the Caenorhabditis elegans zygote.

200 s of NALCN-deficient animals (Drosophila and Caenorhabditis elegans) and the major symptoms of Parkin

201 itic (Haemonchus contortus) and free-living (Caenorhabditis elegans) nematodes.

202 tebrate pattern (Drosophila melanogaster and Caenorhabditis elegans) profoundly diverged.

203 In Caenorhabditis elegans, a specialized condensin forms th

204 framework to the connectome of the nematode Caenorhabditis elegans, allowing us to predict the invol

205 er context of similar phenomena described in Caenorhabditis elegans, and an analogy with quorum sensi

206 l response to vitamin B2 (VB2) deficiency in Caenorhabditis elegans, and demonstrated that VB2 level

207 model organisms, such as moths, Drosophila, Caenorhabditis elegans, and Mus musculus, a complete sig

208 on density, suppress exploratory foraging in Caenorhabditis elegans, and that heritable variation in

209 ncode food abundance to modulate lifespan in Caenorhabditis elegans, and uncovered cross- and self-re

210 difying the first two tryptophans, occurs in Caenorhabditis elegans, but four putative enzymes (DPY-1

211 16/FoxO is required to survive starvation in Caenorhabditis elegans, but how daf-16IFoxO promotes sta

212 paternal mitochondrial elimination (PME) in Caenorhabditis elegans, but how paternal mitochondria, b

213 the structure of the pore domain of MCU from Caenorhabditis elegans, determined using nuclear magneti

214 In species ranging from humans to Caenorhabditis elegans, dietary restriction (DR) grants

215 sensation across different species including Caenorhabditis elegans, Drosophila and zebrafish.

216 d healthspan of diverse organisms, including Caenorhabditis elegans, Drosophila melanogaster, and mic

217 ing factors known to mediate regeneration in Caenorhabditis elegans, Drosophila, and mammals.

218 is expressed in all developmental stages of Caenorhabditis elegans, enabling the analysis of hTTR me

219 hanges in a whole animal, the model organism Caenorhabditis elegans, from embryogenesis to adulthood.

220 geometric model fit to vulval development in Caenorhabditis elegans, implies a phase diagram where ce

221 drodioecy (males/hermaphrodites) as found in Caenorhabditis elegans, is thought to have evolved from

222 Although Gene Ontology (GO) is available for Caenorhabditis elegans, it does not include anatomical i

223 In Caenorhabditis elegans, mild developmental mitochondrial

224 In Caenorhabditis elegans, mitochondrial damage leads to nu

225 In Caenorhabditis elegans, nuclear RNAi ensures robust inhe

226 In Caenorhabditis elegans, removing germ cells slows aging

227 re the intermediate steps of RB formation in Caenorhabditis elegans, Rhabditis sp. SB347 (recently na

228 In Caenorhabditis elegans, stress-induced sleep(SIS) is reg

229 izes to a small subset of nonmotile cilia in Caenorhabditis elegans, suggesting an evolutionary adapt

230 Here, we demonstrate, using Caenorhabditis elegans, that linear DNAs with short homo

231 In Caenorhabditis elegans, the AWC neurons are thought to d

232 In the nematode Caenorhabditis elegans, the biogenic amines serotonin (5

233 e protein, such as epithelial cell fusion in Caenorhabditis elegans, the cell fusion step in osteocla

234 Here, we show that both in human cells and Caenorhabditis elegans, the Polo-like kinase 1 (PLK-1) i

235 ctor in seam cells, a stem-like cell type in Caenorhabditis elegans, thereby ensuring proper temporal

236 In Caenorhabditis elegans, this linkage is achieved by the

237 In the nematode Caenorhabditis elegans, this process is regulated by a t

238 In Caenorhabditis elegans, this response requires SKN-1, a

239 Here we use the nematode, Caenorhabditis elegans, to explore these issues using th

240 nient, low cost assay utilising the nematode Caenorhabditis elegans, to rapidly assess both acute tox

241 pattern of protein N-terminal acetylation in Caenorhabditis elegans, uncovering a conserved set of ru

242 In the nematode Caenorhabditis elegans, unfavorable environmental condit

243 In the nematode Caenorhabditis elegans, we applied RNA interference on m

244 Applying this technique in Caenorhabditis elegans, we comprehensively screened inte

245 Using Caenorhabditis elegans, we demonstrate that two compound

246 Using the hermaphroditic nematode Caenorhabditis elegans, we here show that the experiment

247 ing a forward genetic screen in the nematode Caenorhabditis elegans, we implicate the atypical mitoge

248 ver 800,000 DNA variants in wild isolates of Caenorhabditis elegans, we made a discovery that the pro

249 erence (RNAi) is best understood in the worm Caenorhabditis elegans, where the dsRNA-binding protein

250 origin of gene expression differentiation in Caenorhabditis elegans, which could not be detected by a

251 CPVT inducing mutations into the pharynx of Caenorhabditis elegans, which we previously established

252 Here, we report on a Caenorhabditis elegans-Escherichia coli (worm-bacteria)

253 RpoN* in vitro, we explored its effects in a Caenorhabditis elegans-P. aeruginosa infection model.

254 polysis in somatic tissues on oocyte fate in Caenorhabditis elegans.

255 etosensitive neuron pair AFD in the nematode Caenorhabditis elegans.

256 iomarker and predictor of life expectancy in Caenorhabditis elegans.

257 lectrophysiology inside an intact roundworm, Caenorhabditis elegans.

258 l non-pathogenic biofilm-forming bacteria on Caenorhabditis elegans.

259 neural function in diverse cell contexts in Caenorhabditis elegans.

260 d genome editing (CRISPR/Cas9) approaches in Caenorhabditis elegans.

261 issue-specific visualization of ribosomes in Caenorhabditis elegans.

262 sis in murine models and extends lifespan of Caenorhabditis elegans.

263 ed functions is relatively limited, even for Caenorhabditis elegans.

264 NPs) in exposed individuals of the nematode Caenorhabditis elegans.

265 when deleted, increase longevity in the host Caenorhabditis elegans.

266 ation of cell-specific ciliary structures in Caenorhabditis elegans.

267 id deposition and fatty acid desaturation in Caenorhabditis elegans.

268 to oxidative stress and organismal aging in Caenorhabditis elegans.

269 MAP2-like and MEC-7 beta-tubulin proteins in Caenorhabditis elegans.

270 ire subclass-specific traits in the nematode Caenorhabditis elegans.

271 ocal imaging of anchor-cell invasion in live Caenorhabditis elegans.

272 cted with several nematode species including Caenorhabditis elegans.

273 losely related to the genetic model organism Caenorhabditis elegans.

274 lved in ventral nerve cord (VNC) assembly in Caenorhabditis elegans.

275 IL-17 has neuromodulator-like properties in Caenorhabditis elegans.

276 histone H3 (H3K4me3), regulates lifespan in Caenorhabditis elegans.

277 chanism in wild-type or BK channel-humanized Caenorhabditis elegans.

278 ceptors have been identified in the nematode Caenorhabditis elegans.

279 of the mechanistic insight has emerged from Caenorhabditis elegans.

280 al tissues against Pseudomonas aeruginosa in Caenorhabditis elegans.

281 B16 uses a "Trojan horse" mechanism to kill Caenorhabditis elegans.

282 identify a new heterochronic gene, lep-2, in Caenorhabditis elegans.

283 growth arrest state called dauer arrest, in Caenorhabditis elegans.

284 ion of single endosomes in specific cells in Caenorhabditis elegans.

285 types in the nervous system of the nematode Caenorhabditis elegans.

286 s in locomotion, egg-laying, and survival in Caenorhabditis elegans.

287 pendent developmental delay and lethality in Caenorhabditis elegans.

288 athways and the transcriptional machinery in Caenorhabditis elegans.

289 ocrine axis of serotonergic body fat loss in Caenorhabditis elegans.

290 ateral line of Danio rerio and the embryo of Caenorhabditis elegans.

291 y hlh-8, the single Twist homolog present in Caenorhabditis elegans.

292 ulated tyrosine kinase substrate (HGRS-1) in Caenorhabditis elegans] mediate cargo degradation, conce

293 Caenorhabditis eleganssenses gentle touch via a mechanot

294 Androdioecious Caenorhabditis have a high frequency of self-compatible

295 Here we report results from the Caenorhabditis Intervention Testing Program in assessing

296 haviors in longevity regulation, we examined Caenorhabditis male lifespan under solitary, grouped, an

297 bditis briggsae X Chromosome in an otherwise Caenorhabditis nigoni background, which demonstrate simi

298 esting that MMB might mimic sex pheromone in Caenorhabditis species.

299 ex- and stage-specific attraction in several Caenorhabditis species.

300 m in assessing longevity variation across 22 Caenorhabditis strains spanning 3 species, using multipl