ライフサイエンスコーパス: Caenorhabditis elegans

1 polysis in somatic tissues on oocyte fate in Caenorhabditis elegans.

2 etosensitive neuron pair AFD in the nematode Caenorhabditis elegans.

3 iomarker and predictor of life expectancy in Caenorhabditis elegans.

4 l non-pathogenic biofilm-forming bacteria on Caenorhabditis elegans.

5 lectrophysiology inside an intact roundworm, Caenorhabditis elegans.

6 neural function in diverse cell contexts in Caenorhabditis elegans.

7 d genome editing (CRISPR/Cas9) approaches in Caenorhabditis elegans.

8 issue-specific visualization of ribosomes in Caenorhabditis elegans.

9 sis in murine models and extends lifespan of Caenorhabditis elegans.

10 ed functions is relatively limited, even for Caenorhabditis elegans.

11 NPs) in exposed individuals of the nematode Caenorhabditis elegans.

12 when deleted, increase longevity in the host Caenorhabditis elegans.

13 ation of cell-specific ciliary structures in Caenorhabditis elegans.

14 to oxidative stress and organismal aging in Caenorhabditis elegans.

15 MAP2-like and MEC-7 beta-tubulin proteins in Caenorhabditis elegans.

16 id deposition and fatty acid desaturation in Caenorhabditis elegans.

17 ire subclass-specific traits in the nematode Caenorhabditis elegans.

18 cted with several nematode species including Caenorhabditis elegans.

19 ocal imaging of anchor-cell invasion in live Caenorhabditis elegans.

20 losely related to the genetic model organism Caenorhabditis elegans.

21 lved in ventral nerve cord (VNC) assembly in Caenorhabditis elegans.

22 IL-17 has neuromodulator-like properties in Caenorhabditis elegans.

23 chanism in wild-type or BK channel-humanized Caenorhabditis elegans.

24 histone H3 (H3K4me3), regulates lifespan in Caenorhabditis elegans.

25 ceptors have been identified in the nematode Caenorhabditis elegans.

26 of the mechanistic insight has emerged from Caenorhabditis elegans.

27 al tissues against Pseudomonas aeruginosa in Caenorhabditis elegans.

28 B16 uses a "Trojan horse" mechanism to kill Caenorhabditis elegans.

29 identify a new heterochronic gene, lep-2, in Caenorhabditis elegans.

30 growth arrest state called dauer arrest, in Caenorhabditis elegans.

31 types in the nervous system of the nematode Caenorhabditis elegans.

32 ion of single endosomes in specific cells in Caenorhabditis elegans.

33 -induced germ cell apoptosis in the nematode Caenorhabditis elegans.

34 s in locomotion, egg-laying, and survival in Caenorhabditis elegans.

35 pendent developmental delay and lethality in Caenorhabditis elegans.

36 athways and the transcriptional machinery in Caenorhabditis elegans.

37 ocrine axis of serotonergic body fat loss in Caenorhabditis elegans.

38 ateral line of Danio rerio and the embryo of Caenorhabditis elegans.

39 y hlh-8, the single Twist homolog present in Caenorhabditis elegans.

40 In Caenorhabditis elegans, a specialized condensin forms th

41 forward genetic screens, we isolated a novel Caenorhabditis elegans active zone gene, clarinet (cla-1

42 smitter release, we identified a mutation in Caenorhabditis elegans AIPR-1 (AIP-related-1), which cau

43 framework to the connectome of the nematode Caenorhabditis elegans, allowing us to predict the invol

44 We expressed human and Caenorhabditis elegans AMPylation enzymes-huntingtin yea

45 In Caenorhabditis elegans an increase in the refractility o

46 Using RNAi screening for defects in Caenorhabditis elegans anchor cell (AC) invasion, we fou

47 activate a canonical cannabinoid receptor in Caenorhabditis elegans and also modulate monoaminergic s

48 , where biochemical and molecular studies in Caenorhabditis elegans and Ascaris suum have identified

49 Recent research in the invertebrates Caenorhabditis elegans and Drosophila melanogaster has l

50 ntly targets the degradation of cyclin B1 in Caenorhabditis elegans and human cells.

51 matin-associated protein degradation in both Caenorhabditis elegans and humans, which is relevant to

52 se analyses and cross-species experiments in Caenorhabditis elegans and in chondrogenic cell lines im

53 tional carriers of epigenetic information in Caenorhabditis elegans and in other organisms.

54 Caenorhabditis elegans and its bacterial diet provide a

55 flux assays in multiple tissues of wild-type Caenorhabditis elegans and long-lived daf-2/insulin/IGF-

56 a during gut infection in two animal models (Caenorhabditis elegans and mice).

57 C increase lifespan and stress resistance in Caenorhabditis elegans and reduce accumulation of reacti

58 splatin mutation signatures in XPF-deficient Caenorhabditis elegans and supports a model in which tra

59 re, we study this process using the predator Caenorhabditis elegans and the bacterial prey Streptomyc

60 e been shown to modulate organismal aging in Caenorhabditis elegans and to impact on age-related dise

61 on-apoptotic cell death process operating in Caenorhabditis elegans and vertebrate development, and i

62 ing in two pivotal model animals - mouse and Caenorhabditis elegans - and compare them to consider wh

63 s of NALCN-deficient animals (Drosophila and Caenorhabditis elegans) and the major symptoms of Parkin

64 ellow fluorescent protein producing nematode Caenorhabditis elegans, and a fluorescent signal was col

65 er context of similar phenomena described in Caenorhabditis elegans, and an analogy with quorum sensi

66 l response to vitamin B2 (VB2) deficiency in Caenorhabditis elegans, and demonstrated that VB2 level

67 model organisms, such as moths, Drosophila, Caenorhabditis elegans, and Mus musculus, a complete sig

68 on density, suppress exploratory foraging in Caenorhabditis elegans, and that heritable variation in

69 ncode food abundance to modulate lifespan in Caenorhabditis elegans, and uncovered cross- and self-re

70 rase (QPRTase) is not encoded in its genome, Caenorhabditis elegans are reported to lack a de novo NA

71 Here we use Caenorhabditis elegans as a model in which to study RIBE

72 Using Caenorhabditis elegans as a model, we examine how both a

73 We have used Caenorhabditis elegans as an invertebrate model to ident

74 de reporter to measure lysosomal chloride in Caenorhabditis elegans as well as murine and human cell

75 rofile nearly 50,000 cells from the nematode Caenorhabditis elegans at the L2 larval stage, which pro

76 In Caenorhabditis elegans AWA neurons, which are crucial fo

77 ance, we pursued a forward genetic screen in Caenorhabditis elegans based on the phenotype swimming-i

78 e used as experimental models decades before Caenorhabditis elegans became known as 'the worm'.

79 We investigated low zinc homeostasis in Caenorhabditis elegans because the genome encodes 14 evo

80 tes of anthelmintics have been restricted to Caenorhabditis elegans because they have failed when app

81 difying the first two tryptophans, occurs in Caenorhabditis elegans, but four putative enzymes (DPY-1

82 16/FoxO is required to survive starvation in Caenorhabditis elegans, but how daf-16IFoxO promotes sta

83 paternal mitochondrial elimination (PME) in Caenorhabditis elegans, but how paternal mitochondria, b

84 e that IL-17 plays a neuromodulatory role in Caenorhabditis elegans by acting directly on neurons to

85 arget sites, we isolated AGO-bound RNAs from Caenorhabditis elegans by individual-nucleotide resoluti

86 athway that controls mitochondrial fusion in Caenorhabditis elegans by repressing the expression of t

87 ytogenes in vitro, in cell culture, and in a Caenorhabditis elegans (C. elegans) infection model.

88 The nematode worm Caenorhabditis elegans (C. elegans) is a versatile and w

89 Using the Caenorhabditis elegans (C. elegans) model, various studi

90 The free-living nematode Caenorhabditis elegans can adapt to harsh environments b

91 Here, we show that Caenorhabditis elegans can alternate between attractive

92 Here we show that the nematode Caenorhabditis elegans can discriminate spatial patterns

93 CblC from Caenorhabditis elegans (ceCblC) also exhibits a robust t

94 Here, we reveal Caenorhabditis elegans CEP-290 (mammalian Cep290/Mks4/Np

95 We show that Caenorhabditis elegans changes how it processes sensory

96 of gene mutations on the trafficking of the Caenorhabditis elegans choline transporter orthologue re

97 We utilized the Caenorhabditis elegans CLC-1/2/Ka/Kb anion channel homol

98 Using a computational model of the Caenorhabditis elegans connectome dynamics, we show that

99 Caenorhabditis elegans contains 25 Argonautes, of which,

100 We show that PKC-2, a Caenorhabditis elegans cPKC, is essential for a complex

101 ontig in a few minutes, and assemble 45-fold Caenorhabditis elegans data in 9 min, orders of magnitud

102 J and L3MBTL3 in Drosophila melanogaster and Caenorhabditis elegans demonstrate that the functional l

103 A new study in Caenorhabditis elegans demonstrates that changes in neur

104 the structure of the pore domain of MCU from Caenorhabditis elegans, determined using nuclear magneti

105 small RNAs comprising six miRNAs involved in Caenorhabditis elegans development and two controls were

106 ccurs in a highly reproducible manner during Caenorhabditis elegans development.

107 In species ranging from humans to Caenorhabditis elegans, dietary restriction (DR) grants

108 sensation across different species including Caenorhabditis elegans, Drosophila and zebrafish.

109 d healthspan of diverse organisms, including Caenorhabditis elegans, Drosophila melanogaster, and mic

110 ing factors known to mediate regeneration in Caenorhabditis elegans, Drosophila, and mammals.

111 CDC-42 in AJ formation and regulation during Caenorhabditis elegans embryo elongation, a process driv

112 ioning of mitotic spindle in the single-cell Caenorhabditis elegans embryo is achieved initially by t

113 n kinetochore-microtubule attachments in the Caenorhabditis elegans embryo.

114 ercellular surface during cell division in a Caenorhabditis elegans embryo.

115 The Caenorhabditis elegans embryonic nervous system, compris

116 Spindly to recruit dynein to kinetochores in Caenorhabditis elegans embryos and human cells.

117 The lack of physiological recordings from Caenorhabditis elegans embryos stands in stark contrast

118 Here we use computational simulations of Caenorhabditis elegans embryos to address this fundament

119 ere, we show that in asymmetrically dividing Caenorhabditis elegans embryos, the cortical PAR protein

120 luorescently labeling endogenous proteins in Caenorhabditis elegans embryos, we show that dynein exis

121 erties on gene expression and development in Caenorhabditis elegans embryos.

122 components on AIN-1/GW182 and NTL-1/CNOT1 in Caenorhabditis elegans embryos.

123 physical properties of constricting rings in Caenorhabditis elegans embryos.

124 chromosomal loci in the interphase nuclei of Caenorhabditis elegans embryos.

125 is expressed in all developmental stages of Caenorhabditis elegans, enabling the analysis of hTTR me

126 The nceh-1 gene of Caenorhabditis elegans encodes an ortholog of neutral ch

127 g these questions using the robust number of Caenorhabditis elegans epidermal stem cells, known as se

128 We demonstrate that the Caenorhabditis elegans Epithelial Fusion Failure 1 (EFF-

129 and downstream motor neurons (A-MNs) in the Caenorhabditis elegans escape circuit, we found that dis

130 Here, we report on a Caenorhabditis elegans-Escherichia coli (worm-bacteria)

131 Dauer larvae of the nematode Caenorhabditis elegans exhibit a phoretic behavior calle

132 ex, time- and resource-consuming, transgenic Caenorhabditis elegans expressing hTTR provide an optima

133 Here we show that adult neurons from Caenorhabditis elegans extrude large (approximately 4 mu

134 autophagy is induced in multiple tissues of Caenorhabditis elegans following hormetic heat stress or

135 o studies, using small animal models such as Caenorhabditis elegans for hit identification and lead o

136 Finally, D-RR4 protected Caenorhabditis elegans from lethal infections of P. aeru

137 hanges in a whole animal, the model organism Caenorhabditis elegans, from embryogenesis to adulthood.

138 ere, we investigated the temporal aspects of Caenorhabditis elegans gene expression changes using ave

139 time-lapse in vivo single-cell analysis and Caenorhabditis elegans genetics, our evidence does not s

140 The Caenorhabditis elegans germline provides a tractable sys

141 hich we call SIP-HAVA-seq, by characterizing Caenorhabditis elegans germline stem cell mutation accru

142 the CED-3 caspase in distinct regions of the Caenorhabditis elegans germline.

143 ultiple small RNA-seq datasets from the worm Caenorhabditis elegans had shorter forms of miRNAs that

144 Since 1999, Caenorhabditis elegans has been extensively used to stud

145 The nematode Caenorhabditis elegans has been particularly instrumenta

146 ting gene expression, but its application in Caenorhabditis elegans has not been described.

147 Work in Caenorhabditis elegans has shown that the UPR(mt) is reg

148 s (OV) as a natural pathogen of the nematode Caenorhabditis elegans has stimulated interest in explor

149 ies such as larval zebrafish, Drosophila, or Caenorhabditis elegans have become key model organisms i

150 to unravel the sex determination pathway in Caenorhabditis elegans He inferred the order of genes in

151 ammals, satiety signals induce quiescence in Caenorhabditis elegans Here we report that the C. elegan

152 A comparison of the connectomes of the Caenorhabditis elegans hermaphrodite and male nervous sy

153 ctory long-term associative memory (LTAM) in Caenorhabditis elegans hermaphrodites.

154 We have identified atx-2, the Caenorhabditis elegans homolog of the human ATXN2L and A

155 t comprise the whole body of the small worm, Caenorhabditis elegans However, to fully elucidate the n

156 geometric model fit to vulval development in Caenorhabditis elegans, implies a phase diagram where ce

157 n of the first miRNA, lin-4, in the nematode Caenorhabditis elegans in 1993, thousands of miRNAs have

158 ates PARP1 and leads to accelerated aging in Caenorhabditis elegans In conclusion, this work supports

159 ebrate representative twitchin (UNC-22) from Caenorhabditis elegans In in vitro experiments, change o

160 ed in determining a stimulatory phenotype of Caenorhabditis elegans in response to physiologically re

161 re of a cyclic-nucleotide-gated channel from Caenorhabditis elegans in the cyclic guanosine monophosp

162 ng followed by mass spectrometry analysis of Caenorhabditis elegans infected with two species of Nema

163 ochastic bacterial community assembly in the Caenorhabditis elegans intestine is sufficient to produc

164 Caenorhabditis elegans is among the most powerful model

165 research, the glycome of the model nematode Caenorhabditis elegans is still not fully understood.

166 endocrine regulation of diverse behaviors of Caenorhabditis elegans is under the control of the DAF-7

167 drodioecy (males/hermaphrodites) as found in Caenorhabditis elegans, is thought to have evolved from

168 Although Gene Ontology (GO) is available for Caenorhabditis elegans, it does not include anatomical i

169 Caenorhabditis elegans lacking ADARs exhibit reduced che

170 In Caenorhabditis elegans larvae, sleep is associated with

171 e that during periods of acute starvation in Caenorhabditis elegans larvae, the master metabolic regu

172 Long-term studies of Caenorhabditis elegans larval development traditionally

173 n exposure early in the life of the nematode Caenorhabditis elegans leads to a long-lasting aversion

174 pletion of casein kinase 1 gamma (CSNK-1) in Caenorhabditis elegans led to the formation of large pol

175 nd that a subset of sensory neurons shortens Caenorhabditis elegans' life span by differentially regu

176 on of SLO-2 (a homolog of mammalian Slo2) in Caenorhabditis elegans Loss-of-function (lf) mutants of

177 Mutations in Caenorhabditis elegans mafr-1 that truncate the C-box re

178 Here, we describe two distinct ways in which Caenorhabditis elegans males cause faster somatic aging

179 ic reticulum-resident molecular chaperone in Caenorhabditis elegans MEC-6 modulates the expression of

180 Here, using Caenorhabditis elegans mechanosensory neurons, we addres

181 s full recovery of function after axotomy of Caenorhabditis elegans mechanosensory neurons.

182 ulated tyrosine kinase substrate (HGRS-1) in Caenorhabditis elegans] mediate cargo degradation, conce

183 In Caenorhabditis elegans, mild developmental mitochondrial

184 In Caenorhabditis elegans, mitochondrial damage leads to nu

185 tant is hypervirulent both in vitro and in a Caenorhabditis elegans model in vivo.

186 ecules in human cerebrospinal fluid and in a Caenorhabditis elegans model of AD.

187 for cells in biofilms, and to virulence in a Caenorhabditis elegans model of infection.

188 ted tauopathy in multiple models including a Caenorhabditis elegans model of tauopathy.

189 tand cannabinoid signaling, we have used the Caenorhabditis elegans model to examine the effects of c

190 uted to the in vivo virulence of PA14 in the Caenorhabditis elegans model.

191 Moreover, in genetically tractable Caenorhabditis elegans models, expression of alpha-synuc

192 , we made a series of observations utilizing Caenorhabditis elegans models, mammalian cell lines, pri

193 asynaptic acetylcholine receptors (AChRs) in Caenorhabditis elegans muscle cells.

194 S) microscopy and systematically identify 57 Caenorhabditis elegans mutants with altered lipid distri

195 Using Caenorhabditis elegans mutants, we identify DNA repair f

196 Here we describe a novel role for the Caenorhabditis elegans NCLX-type protein, NCX-9, in neur

197 l variation to behavior by monitoring single Caenorhabditis elegans nematodes over their complete dev

198 itic (Haemonchus contortus) and free-living (Caenorhabditis elegans) nematodes.

199 e low-dimensional functional response of the Caenorhabditis elegans network of neurons to propriocept

200 Using Caenorhabditis elegans neuromuscular junction as a model

201 rgy demands and support synaptic function in Caenorhabditis elegans neurons.

202 Here we describe a novel Caenorhabditis elegans nuclear receptor, HIZR-1, that is

203 In Caenorhabditis elegans, nuclear RNAi ensures robust inhe

204 conducted an RNA interference screen of the Caenorhabditis elegans nucleome in a strain carrying an

205 In the Caenorhabditis elegans one-cell embryo, the astral micro

206 During Caenorhabditis elegans oocyte meiosis, a multi-protein r

207 d ablation, and genetic perturbations in the Caenorhabditis elegans oocyte, we studied the mechanism

208 ochore attachments have not been observed in Caenorhabditis elegans oocytes and chromosomes instead a

209 We find that Caenorhabditis elegans oocytes whose maturation is arres

210 In Caenorhabditis elegans, opioid receptor agonists, such a

211 We adapted a Caenorhabditis elegans organogenesis model to enable a g

212 r investigations of bpl-1, which encodes the Caenorhabditis elegans ortholog of HCS.

213 factor (erythroid-derived 2)-like 2 and its Caenorhabditis elegans ortholog, SKN-1, are transcriptio

214 ven decision-theoretical model of feeding in Caenorhabditis elegans Our central assumption is that fo

215 quired for the first embryonic abscission in Caenorhabditis elegans Our findings indicate that membra

216 aviors, we utilized the genetic model system Caenorhabditis elegans Our studies demonstrate that grk-

217 RpoN* in vitro, we explored its effects in a Caenorhabditis elegans-P. aeruginosa infection model.

218 Here, we implicate the two Caenorhabditis elegans PABPs (PAB-1 and PAB-2) in miRNA-

219 that mammalian PKDs 1-3 and the prototypical Caenorhabditis elegans PKD, DKF-2A, are exclusively (hom

220 Caenorhabditis elegans possesses 19 GABAergic motor neur

221 Nematodes such as the model organism Caenorhabditis elegans produce various homologous series

222 tebrate pattern (Drosophila melanogaster and Caenorhabditis elegans) profoundly diverged.

223 5 orthologues in Drosophila melanogaster and Caenorhabditis elegans promotes longevity.

224 These include the Caenorhabditis elegans protein LAF-1, which forms P gran

225 In Caenorhabditis elegans, removing germ cells slows aging

226 he well-studied sex determination pathway of Caenorhabditis elegans Repression of nhl-2 by the mir-35

227 edator-prey coevolution, we investigated how Caenorhabditis elegans responds to the predatory fungus

228 neuronal cultures and functional analyses in Caenorhabditis elegans revealed that the UNC13A variant

229 re the intermediate steps of RB formation in Caenorhabditis elegans, Rhabditis sp. SB347 (recently na

230 We show that the Caenorhabditis elegans RMI1 homolog-1 (RMH-1) functions

231 of synapse formation and axon termination in Caenorhabditis elegans RPM-1 functions in a ubiquitin li

232 New work in Caenorhabditis elegans shows that during embryogenesis e

233 is of a previous genetic screening result in Caenorhabditis elegans shows that homo-trimerization is

234 3 domain-containing protein family member in Caenorhabditis elegans SORB-1 is strongly localized to i

235 inated with extraintestinal copper levels in Caenorhabditis elegans Specifically, we show that CUA-1,

236 Under laboratory conditions, Caenorhabditis elegans sperm are very efficient at navig

237 In this study, we find that Caenorhabditis elegans sperm DNA stays in a fixed positi

238 The Caenorhabditis elegans spermatheca is a bag-like organ o

239 rther examine the effects of squalamine in a Caenorhabditis elegans strain overexpressing alpha-synuc

240 on of a deleterious mtDNA in a heteroplasmic Caenorhabditis elegans strain that stably expresses wild

241 The genomes of two Caenorhabditis elegans strains, a wild-type strain and a

242 In Caenorhabditis elegans, stress-induced sleep(SIS) is reg

243 Forward chemical screening conducted in Caenorhabditis elegans suggested that pOPCs reduced the

244 izes to a small subset of nonmotile cilia in Caenorhabditis elegans, suggesting an evolutionary adapt

245 family homologs in chicken, Drosophila, and Caenorhabditis elegans suggests this antagonism is conse

246 The Caenorhabditis elegans SUN domain protein, UNC-84, funct

247 dentify a TRH-like neuropeptide precursor in Caenorhabditis elegans that belongs to a bilaterian fami

248 Here, we show in the model system Caenorhabditis elegans that expression of the arginine-c

249 loid-beta proteotoxicity in human, mouse and Caenorhabditis elegans that involves the mitochondrial u

250 al. identify a genetic locus in the nematode Caenorhabditis elegans that underlies nictation and cont

251 Here, we demonstrate, using Caenorhabditis elegans, that linear DNAs with short homo

252 crosses between wild strains of the nematode Caenorhabditis elegans The element is made up of sup-35,

253 responses to oxygen, pheromones, and food in Caenorhabditis elegans The molecular composition of the

254 ilia in chemosensory neurons of the nematode Caenorhabditis elegans The trafficking defect caused by

255 In Caenorhabditis elegans, the AWC neurons are thought to d

256 In the nematode Caenorhabditis elegans, the biogenic amines serotonin (5

257 e protein, such as epithelial cell fusion in Caenorhabditis elegans, the cell fusion step in osteocla

258 Here, we show that both in human cells and Caenorhabditis elegans, the Polo-like kinase 1 (PLK-1) i

259 ctor in seam cells, a stem-like cell type in Caenorhabditis elegans, thereby ensuring proper temporal

260 Organismic ageing in Caenorhabditis elegans therefore appears to involve aspe

261 nsgene can occur between intestinal cells in Caenorhabditis elegans These differences are caused by g

262 bilization at the transition to adulthood in Caenorhabditis elegans This novel connection involves cr

263 In Caenorhabditis elegans, this linkage is achieved by the

264 In the nematode Caenorhabditis elegans, this process is regulated by a t

265 In Caenorhabditis elegans, this response requires SKN-1, a

266 r organization of meiotic chromosome axes in Caenorhabditis elegans through STORM (stochastic optical

267 bors of PVD and FLP somatosensory neurons in Caenorhabditis elegans through the leucine-rich transmem

268 Studies in metazoan models ranging from Caenorhabditis elegans to mammals have revealed cell-aut

269 Here we use the nematode, Caenorhabditis elegans, to explore these issues using th

270 nient, low cost assay utilising the nematode Caenorhabditis elegans, to rapidly assess both acute tox

271 Using the Caenorhabditis elegans touch receptor neurons, we analyz

272 (NAM) form of vitamin B3 is an agonist of a Caenorhabditis elegans TRPV channel.

273 The Caenorhabditis elegans UBCH7 homolog, UBC-18, plays a cr

274 a gain-of-function in rescue experiments in Caenorhabditis elegans unc-18 nulls.

275 her cell types, Munc13 (mammalian homolog of Caenorhabditis elegans uncoordinated gene 13) proteins p

276 pattern of protein N-terminal acetylation in Caenorhabditis elegans, uncovering a conserved set of ru

277 In the nematode Caenorhabditis elegans, unfavorable environmental condit

278 The nematode Caenorhabditis elegans uses simple building blocks from

279 Caenorhabditis elegans vulva development provides an in

280 ensitizing miRNAs, we initially utilized the Caenorhabditis elegans vulval cell model, an in vivo sys

281 The pha-1 gene of Caenorhabditis elegans was originally heralded as a mast

282 ed with feeding and fasting in the roundworm Caenorhabditis elegans We identified neural circuits thr

283 ius tipulae, a distant relative of the model Caenorhabditis elegans We used this draft to identify th

284 In the nematode Caenorhabditis elegans, we applied RNA interference on m

285 Applying this technique in Caenorhabditis elegans, we comprehensively screened inte

286 Using Caenorhabditis elegans, we demonstrate that two compound

287 Using the hermaphroditic nematode Caenorhabditis elegans, we here show that the experiment

288 ing a forward genetic screen in the nematode Caenorhabditis elegans, we implicate the atypical mitoge

289 ver 800,000 DNA variants in wild isolates of Caenorhabditis elegans, we made a discovery that the pro

290 erence (RNAi) is best understood in the worm Caenorhabditis elegans, where the dsRNA-binding protein

291 origin of gene expression differentiation in Caenorhabditis elegans, which could not be detected by a

292 CPVT inducing mutations into the pharynx of Caenorhabditis elegans, which we previously established

293 ere we identify life-limiting pathologies in Caenorhabditis elegans with a necropsy analysis of worms

294 ly affects behavior in mice, Drosophila, and Caenorhabditis elegans Yet, the mechanisms that modulate

295 Using the Caenorhabditis elegans zygote as a model, we find that t

296 odel to simulate contractile dynamics in the Caenorhabditis elegans zygote cytokinetic ring.

297 complex composition and stoichiometry during Caenorhabditis elegans zygote polarization, which takes

298 shment of anterior-posterior polarity in the Caenorhabditis elegans zygote requires two different pro

299 Here, we report that in the Caenorhabditis elegans zygote, feedback between active R

300 ent cortical actomyosin contractility in the Caenorhabditis elegans zygote.