ライフサイエンスコーパス: Cajal

1 f gut pacemaker cells (interstitial cells of Cajal).

2 tor nucleus, and the interstitial nucleus of Cajal.

3 s of muscle fibers and interstitial cells of Cajal.

4 ons, smooth muscle, or interstitial cells of Cajal.

5 stricted to muscle and interstitial cells of Cajal.

6 d gametogenesis and in interstitial cells of Cajal.

7 system, including the interstitial cells of Cajal.

8 described more than 100 years ago by Ramon y Cajal.

9 ed by mechanosensitive interstitial cells of Cajal.

10 of enteric neurons and interstitial cells of Cajal.

11 in enteric neurons or interstitial cells of Cajal.

12 ocated on non-neuronal interstitial cells of Cajal.

13 ct and arises from the interstitial cells of Cajal.

14 ated near Kit-positive interstitial cells of Cajal.

15 also projects to the commissural nucleus of Cajal, a general visceral sensory center.

16 ellular origin from the interstitial cell of Cajal and distinctness from smooth muscles tumors were o

17 plexus, had functional interstitial cells of Cajal and had an electromechanical coupling that regulat

18 on points to a role of interstitial cells of Cajal and mast cells.

19 volutionized at the meso- and microlevels by Cajal and others in the late nineteenth century, and rea

20 Intramuscular interstitial cells of Cajal and platelet-derived growth factor receptor alpha-

21 membraneless organelles, including nucleoli, Cajal and PML bodies, and stress granules.

22 ns, were first described by Santiago Ramon y Cajal as "protoplasmic kisses that appear to constitute

23 vel, the importance of interstitial cells of Cajal as pacemakers, neuromodulators and stretch recepto

24 Interstitial cells of Cajal at the level of the deep muscular plexus (ICC-DMP)

25 ole of mouse Drp2 in apposition assembly and Cajal band function and compared it with periaxin.

26 Cajal bands are cytoplasmic channels flanked by appositi

27 Loss of periaxin disrupts appositions and Cajal bands in Schwann cells and causes a severe demyeli

28 e 1), drives phosphorylation of NDRG1 in the Cajal bands of the abaxonal compartment.

29 Schwann cells have disrupted appositions and Cajal bands, and they undergo focal hypermyelination and

30 of myelin decompaction, a reduced number of Cajal bands.

31 aparanodes, Schmidt-Lanterman incisures, and Cajal bands.

32 ), nuclear speckles (27), paraspeckles (24), Cajal bodies (17), Sam68 nuclear bodies (5), Polycomb bo

33 (ugAG) required for targeting of scaRNAs to Cajal bodies (CB) and an uncharacterized sequence requir

34 lear phosphoprotein that concentrates within Cajal bodies (CBs) and impacts small nuclear ribonucleop

35 Cajal bodies (CBs) are nuclear organelles that are usual

36 Cajal bodies (CBs) are nuclear organelles that occur in

37 Cajal bodies (CBs) are nuclear suborganelles that nonran

38 The nucleolus and Cajal bodies (CBs) are prominent interacting subnuclear

39 Cajal bodies (CBs) are subnuclear domains implicated in

40 nd the density and permeability of nucleoli, Cajal bodies (CBs), and speckles in the Xenopus oocyte n

41 role of various nuclear organelles, such as Cajal bodies (CBs), in their nuclear maturation phase ha

42 re we examined the non-random positioning of Cajal bodies (CBs), major NBs involved in spliceosomal s

43 omposition of subnuclear organelles, such as Cajal bodies (CBs), nucleoli, and other nuclear bodies,

44 Coilin is known as the marker protein for Cajal bodies (CBs), subnuclear domains important for the

45 Coilin protein scaffolds Cajal bodies (CBs)-subnuclear compartments enriched in s

46 displacements of coilin and SMN proteins in Cajal bodies and direct dissociation of coilin-SMN assoc

47 Specifically, SMN localization to Cajal bodies and gems was not observed in cells derived

48 ompartments including chromosomes, nucleoli, Cajal bodies and histone locus bodies.

49 AIDA-1d to the nucleus, where it couples to Cajal bodies and induces Cajal body-nucleolar associatio

50 replaces NAF1 to yield mature H/ACA RNPs in Cajal bodies and nucleoli.

51 eins potentially involved in the assembly of Cajal bodies and paraspeckles.

52 on of SMN function results in disassembly of Cajal bodies and relocalization of the marker protein, c

53 (i) hCaf1z and hCcr4d concentrate in nuclear Cajal bodies and shuttle between the nucleus and cytopla

54 uggest that the trafficking of telomerase to Cajal bodies and telomeres in cancer cells correlates wi

55 The localization of hTR to Cajal bodies and telomeres is specific to cancer cells w

56 cancer cells leads to loss of hTR from both Cajal bodies and telomeres without affecting hTR levels.

57 l cycle, accompanied by its concentration in Cajal bodies and transient colocalization with telomeres

58 Cajal bodies are believed to be the storage and processi

59 Cajal bodies are conserved sites for the maturation of r

60 Cajal bodies are nuclear sites of small ribonucleoprotei

61 Cajal bodies are nuclear structures that host RNA modifi

62 Our results suggest a function for Cajal bodies as a center for the assembly of an AGO4/NRP

63 olocalize to small nuclear bodies similar to Cajal bodies but lacking the Cajal body marker Atcoilin.

64 the trafficking of hTR to both telomeres and Cajal bodies depends on hTERT.

65 ions of both hTR and hTERT with nucleoli and Cajal bodies during S phase, implicating both structures

66 Other protein pairs in Cajal bodies exhibit different magnitudes of fluorescenc

67 inhibitor Imetelstat (GRN163L), a time when Cajal bodies fail to deliver telomerase RNA to telomeres

68 e imported into the nucleus, accumulating in Cajal bodies for additional snRNA maturation steps befor

69 ult implies that processing of telomerase by Cajal bodies may affect its processivity.

70 t structural changes of protein complexes in Cajal bodies may represent a unique mechanism of mechano

71 We used the accumulation of SMN in Cajal bodies of intact proliferating cells, which active

72 localized to the nucleoplasm, nucleoli, and Cajal bodies of transfected cells.

73 on factor NPAT in subnuclear foci, including Cajal bodies that associate with histone gene clusters.

74 protein localized primarily in nucleoli and Cajal bodies that was identified as a downstream target

75 jal bodies, while others must concentrate in Cajal bodies to function.

76 ogated, because telomerase mislocalizes from Cajal bodies to nucleoli within the iPSCs.

77 SPN also relocalizes to Cajal bodies upon treatment with leptomycin B.

78 Interestingly, SPN targets to Cajal bodies when U2 but not U1 snRNPs are imported as c

79 ce analyses, all 3 sera showed nuclear dots (Cajal bodies) and cytoplasmic staining.

80 eted to, and colocalize at, subnuclear foci (Cajal bodies) in a cell cycle-dependent manner.

81 he endogenous FRG1 is localized in nucleoli, Cajal bodies, and actively transcribed chromatin; howeve

82 pathway for enzyme assembly, localization in Cajal bodies, and association with telomeres.

83 n discrete organelles that include speckles, Cajal bodies, and histone locus bodies.

84 plicing factors and the telomerase enzyme to Cajal bodies, and its functional loss has been linked to

85 lex, its intracellular trafficking involving Cajal bodies, and its recruitment to telomeres.

86 cells involves trafficking of telomerase to Cajal bodies, and telomerase is thought to be recruited

87 rference prevents TERC from associating with Cajal bodies, disrupts telomerase-telomere association,

88 body protein 1), that is notably enriched in Cajal bodies, nuclear sites of RNP processing that are i

89 Nuclear bodies including nucleoli, Cajal bodies, nuclear speckles, Polycomb bodies, and par

90 as nucleolar import of the ORF3 protein via Cajal bodies, relocalization of some fibrillarin from th

91 in TCAB1 disrupt telomerase localization to Cajal bodies, resulting in misdirection of telomerase RN

92 phatases interfered with SMN accumulation in Cajal bodies, suggesting impaired SMN complex function,

93 phase lags between various protein pairs in Cajal bodies, suggesting viscoelastic interactions betwe

94 NA) binding protein SmD3; and two markers of Cajal bodies, trimethylguanosine-capped snRNAs and the U

95 Some RNAs transit Cajal bodies, while others must concentrate in Cajal bod

96 disrupting the recruitment of the protein to Cajal bodies.

97 hat facilitates trafficking of telomerase to Cajal bodies.

98 histone locus bodies) that are distinct from Cajal bodies.

99 of the cell cycle hTR is found primarily in Cajal bodies.

100 sm and in distinct nuclear structures called Cajal bodies.

101 uclear dicing bodies (D-bodies), differ from Cajal bodies.

102 supporting the idea that NF7 associates with Cajal bodies.

103 to associate with U2 snRNA or accumulate in Cajal bodies.

104 ls coincident with TCAB1 delocalization from Cajal bodies.

105 so recruits key spliceosomal components from Cajal bodies.

106 spliceosomal snRNPs and localizes to nuclear Cajal bodies.

107 entrates in two subnuclear structures termed Cajal body (CB) and gems.

108 We show here that one of them, the Cajal body (CB), can be formed de novo.

109 Because of their localization in the nuclear Cajal body (CB), these guide RNAs are known as small CB-

110 Small Cajal body (CB)-specific RNPs (scaRNPs) function in post

111 nthesis, snoRNAs transiently localize to the Cajal body (in plant and animal cells) or the homologous

112 increase more than those of SMN in the same Cajal body after dynamic force application.

113 STA1 signals are exclusively present in the Cajal body and overlap with AGO4 in most nuclei.

114 SMN complex was identified as a new Cajal body autoantigen recognized by sera from white pat

115 the elimination of the telomerase holoenzyme Cajal body chaperone TCAB1 or the Cajal body scaffold pr

116 Our results also demonstrate that Cajal body homeostasis requires SMN and ongoing snRNP bi

117 through direct binding of TERC and regulates Cajal body localization of the telomerase.

118 reen in human cells for factors required for Cajal body localization.

119 dies similar to Cajal bodies but lacking the Cajal body marker Atcoilin.

120 embly factor, survival motor neuron, and the Cajal body marker, coilin.

121 increased SMN protein levels and nuclear gem/Cajal body numbers in patient-derived cells.

122 riments include increased hTR RNA levels and Cajal body numbers, and expression of SV40 large T antig

123 In this issue, Jady et al. place the Cajal body on the intranuclear traffic route of telomera

124 tify a holoenzyme subunit, TCAB1 (telomerase Cajal body protein 1), that is notably enriched in Cajal

125 Box C/D small nucleolar and Cajal body ribonucleoprotein particles (sno/scaRNPs) dir

126 H/ACA small nucleolar and Cajal body ribonucleoproteins (RNPs) function in site-sp

127 ing, only a subset copurifies telomerase and Cajal body ribonucleoproteins.

128 efficient approach to deplete snoRNA, small Cajal body RNA (scaRNA) and small nuclear RNA in human a

129 We show that a Cajal body RNA localization motif determines this specif

130 controls trafficking of telomerase and small Cajal body RNAs (scaRNAs).

131 human telomerase RNA and individual resident Cajal body RNAs is associated with Sm proteins.

132 established telomerase components, and small Cajal body RNAs that are involved in modifying splicing

133 holoenzyme Cajal body chaperone TCAB1 or the Cajal body scaffold protein Coilin.

134 e core, conserved regions 4 and 5, and small Cajal body specific RNA domains of human TR have emerged

135 of the exon 2b encoded domain that mediates Cajal body targeting and self-association.

136 is widely known as the protein marker of the Cajal body, a subnuclear domain important to the biogene

137 wo nuclear organelles, the nucleolus and the Cajal body, respond to stress.

138 dy describes the discovery of the Drosophila Cajal body, revealing some interesting insights into the

139 In contrast, the telomerase holoenzyme Cajal body-associated protein, TCAB1, was released from

140 ncident with the histone locus body (HLB), a Cajal body-like nuclear structure associated with the hi

141 where it couples to Cajal bodies and induces Cajal body-nucleolar association.

142 t of a Box H/ACA sno/scaRNP (small nucleolar/Cajal body-specific ribonucleoprotein).

143 Small nucleolar RNAs (snoRNAs) and small Cajal body-specific RNAs (scaRNAs) are non-coding RNAs w

144 eukaryotic box C/D small nucleolar (sno) or Cajal body-specific RNAs guide base pairing with target

145 eins (RNPs), small nucleolar RNPs, and small Cajal body-specific RNPs, is sufficient for the formatio

146 levels and a loss of U3 snoRNA signal in the Cajal body.

147 neurons were originally described by Ramon y Cajal, but have not been thoroughly studied.

148 e peptide (GLP-1), in deep short axon cells (Cajal cells) of the olfactory bulb and its neuromodulato

149 lfactory bulb neuron (deep short axon cells, Cajal cells) that could be capable of modifying mitral c

150 Ramon y Cajal described longitudinal and transverse bands of cyt

151 A century ago, Ramon y Cajal described the generalized response of regenerating

152 ich lack intramuscular interstitial cells of Cajal, did not affect membrane depolarization or slow-wa

153 muscle cells (CSMCs), interstitial cells of Cajal distributed in the myenteric region (ICC-MY) and f

154 n his theory of functional polarity, Ramon y Cajal first identified the soma and dendrites as the pri

155 Santiago Ramon y Cajal had referred to neurons as the 'mysterious butterf

156 ored in 151 MS patients recruited at Ramon y Cajal Hospital in Madrid, Spain.

157 ingle-mutant mice, both interstitial cell of Cajal hyperplasia and mast cell hyperplasia were exacerb

158 ped gastric and colonic interstitial cell of Cajal hyperplasia as well as cecal GIST.

159 (GIST) are related to interstitial cells of Cajal (ICC) and often contain activating stem cell facto

160 lectrically coupled to interstitial cells of Cajal (ICC) and PDGFRalpha(+) cells.

161 h muscle cells (SMCs), interstitial cells of Cajal (ICC) and platelet-derived growth factor receptor

162 ialized cells known as interstitial cells of Cajal (ICC) are distributed in specific locations within

163 Interstitial cells of Cajal (ICC) are pacemaker cells that generate electrical

164 s to determine whether interstitial cells of Cajal (ICC) are present in the guinea pig extrahepatic b

165 Interstitial cells of Cajal (ICC) are putative pacemaker cells in the rabbit u

166 Interstitial cells of Cajal (ICC) are required for normal gastrointestinal mot

167 Interstitial cells of Cajal (ICC) are unique cells that generate electrical pa

168 Interstitial cells of Cajal (ICC) control intestinal smooth muscle contraction

169 istinct populations of interstitial cells of Cajal (ICC) exist within the tunica muscularis of the ga

170 Interstitial cells of Cajal (ICC) express c-kit; however, it is unknown whethe

171 Interstitial cells of Cajal (ICC) express the receptor tyrosine kinase, KIT, t

172 KEY POINTS: Interstitial cells of Cajal (ICC) from murine colonic muscles express genes en

173 Interstitial cells of Cajal (ICC) generate pacemaker activity (slow waves) in

174 Interstitial cells of Cajal (ICC) generate slow waves.

175 Interstitial cells of Cajal (ICC) have been identified in urinary bladder of s

176 Nitrergic nerves and interstitial cells of Cajal (ICC) have been implicated in the regulation of py

177 ns and distribution of interstitial cells of Cajal (ICC) in human gastric muscles.

178 phological features of interstitial cells of Cajal (ICC) in the gastrointestinal (GI) tract are descr

179 generally assumed that interstitial cells of Cajal (ICC) in the human gastrointestinal tract have sim

180 tegrity of networks of interstitial cells of Cajal (ICC) is essential to preserve orderly contractile

181 uction by hyperplastic interstitial cells of Cajal (ICC) or GISTs.

182 scle, enteric neurons, interstitial cells of Cajal (ICC) or other cellular components.

183 Interstitial cells of Cajal (ICC) provide important regulatory functions in th

184 Interstitial cells of Cajal (ICC) provide pacemaker activity and functional br

185 es has determined that interstitial cells of Cajal (ICC) serve as pacemaker cells, conduits for activ

186 about the function of interstitial cells of Cajal (ICC) since their discovery more than 100 years ag

187 Interstitial cells of Cajal (ICC) were described more than 100 years ago by Ra

188 Interstitial cells of Cajal (ICC) were proposed as potential mediators in moto

189 scles are generated by interstitial cells of Cajal (ICC), and these events actively propagate through

190 beta for glia, Kit for interstitial cells of Cajal (ICC), CD45 and CD68 for immune cells, and smoothe

191 sion, enteric neurons, interstitial cells of Cajal (ICC), smooth muscle cells and electrical activity

192 elopment, although the interstitial cells of Cajal (ICC), the cells of origin of GIST, were normal.

193 number and function of interstitial cells of Cajal (ICC), the gastrointestinal pacemaker cells, under

194 ls, but present in the interstitial cells of Cajal (ICC), the pacemaker cells that control smooth mus

195 master regulator in the intestinal cells of Cajal (ICC), thought to be the cells of origin of GIST.

196 ointestinal tract, the interstitial cells of Cajal (ICC), where activation triggers ICC proliferation

197 ses leading to loss of interstitial cells of Cajal (ICC), which generate intestinal pacemaker activit

198 athy, and depletion of interstitial cells of Cajal (ICC), which may cause dysrhythmias and impaired n

199 actively propagated by interstitial cells of Cajal (ICC).

200 pacemaker cells termed interstitial cells of Cajal (ICC).

201 Intramuscular interstitial cells of Cajal (ICC-IM) are closely associated with enteric motor

202 iated in intramuscular interstitial cells of Cajal (ICC-IM) by activation of Ca(2+) -activated Cl(-)

203 iated in intramuscular interstitial cells of Cajal (ICC-IM) by activation of Ca(2+) -activated Cl(-)

204 rves and intramuscular interstitial cells of Cajal (ICC-IM) in the stomach and colon and ICC in the d

205 Intramuscular interstitial cells of Cajal (ICC-IM) play a critical role in enteric neural re

206 Intramuscular interstitial cells of Cajal (ICC-IMs) and cholinesterases restrict ACh accessi

207 enteric plexus, called interstitial cells of Cajal (ICC-MY).

208 aker cells such as the interstitial cells of Cajal (ICCs) and atypical SMCs that control other tonic

209 antifying densities of interstitial cells of Cajal (ICCs) and mapping slow-wave abnormalities in pati

210 to investigate whether interstitial cells of Cajal (ICCs) at these borders generated distinct rhythms

211 D & AIMS: Depletion of interstitial cells of Cajal (ICCs) is common in diabetic gastroparesis.

212 e stem cells (SCs) for interstitial cells of Cajal (ICCs), electrical pacemaker, and neuromodulator c

213 ate cells, such as the interstitial cells of Cajal (ICCs), might detect nitrergic signals to indirect

214 is highly expressed in interstitial cells of Cajal (ICCs)-the presumed cell of origin for GIST-as wel

215 bon monoxide (CO) from interstitial cells of Cajal (ICCs).

216 hanosensitive role for interstitial cells of Cajal in smooth muscle tissues.

217 Interstitial cells of Cajal in the deep muscular plexus of the small intestine

218 W-v) mice, they lacked interstitial cells of Cajal in the gut and exhibited bile reflux into the stom

219 Since the work of Ramon y Cajal in the late 19th and early 20th centuries, neurosc

220 een myenteric neurons, interstitial cells of Cajal in the myenteric region (ICC-MY) and smooth muscle

221 The interstitial nucleus of Cajal (InC) controls eye position for vertical eye movem

222 nduction pattern in the interstitial cell of Cajal is responsible for the generation of the full spat

223 Since the work of Golgi and Cajal, light microscopy has remained a key tool for neur

224 y of research suggests that impaired bladder Cajal-like interstitial cells (ICCs) are a important com

225 m of KIT, expression of Interstitial Cell of Cajal-like markers, and release of various matrix metall

226 Starting with the work of Cajal more than 100 years ago, neuroscience has sought t

227 A century ago, Cajal noted striking similarities between the neural cir

228 Since the groundbreaking work of Ramon y Cajal over a century ago, defining the neural circuits u

229 raised by neuroanatomical studies of Ramon y Cajal over a century ago.

230 s, and specifically in interstitial cells of Cajal, provides a means of transmitter disposal after st

231 ontrols spatial ordering of cortical layers, Cajal-Retzius (C-R) cells play a crucial role in cortica

232 Cajal-Retzius (CR) cells are a transient cell population

233 Cajal-Retzius (CR) cells are among the earliest born cor

234 est populations of neurons in the brain, the Cajal-Retzius (CR) cells in the neocortex, which are kno

235 Cajal-Retzius (CR) cells play a crucial role in the form

236 Cajal-Retzius (CR) cells play a key role in the formatio

237 ring the development of the cerebral cortex, Cajal-Retzius (CR) cells settle in the preplate and coor

238 Cajal-Retzius (CR) cells, the earliest-born neurons in t

239 Cajal-Retzius (CR) cells, the predominant source of reel

240 , a progenitor pool that first gives rise to Cajal-Retzius (CR) cells, which populate layer I of all

241 In contrast to the premature loss of Cajal-Retzius (CR) neurons in PS1-null mice, generation

242 The generation of Cajal-Retzius (CR) neurons is restricted to discrete sit

243 Here, we investigated how early born Cajal-Retzius (CR) neurons, which regulate the assembly

244 Foxg1 is constitutively required to suppress Cajal-Retzius cell fate.

245 signaling is required for the maintenance of Cajal-Retzius cell position in the marginal zone during

246 We propose that this newly defined Cajal-Retzius cell-dependent microcircuit may regulate s

247 CXCL12, indicating the existence of a direct Cajal-Retzius cell-interneuron monosynaptic connection.

248 l primordium overexpand, while production of Cajal-Retzius cells and hippocampal neurons decreases, r

249 se depends on both the membrane potential of Cajal-Retzius cells and the kinetics of the received GAB

250 Cajal-Retzius cells are a class of neurons believed to p

251 xpression of functional glutamate receptors, Cajal-Retzius cells are integrated in the synaptic netwo

252 The marginal zone and Cajal-Retzius cells are key players in cortical developm

253 opment of earliest-born preplate neurons and Cajal-Retzius cells at the expense of progenitors.

254 n of the CXC chemokine receptor 4 (CXCR4) in Cajal-Retzius cells by CXC chemokine ligand 12 (CXCL12)

255 anscription factor Tbr2 is expressed in both Cajal-Retzius cells derived from the cortical hem that g

256 s in the main receptor for SDF1 (CXCR4) have Cajal-Retzius cells displaced to deeper cortical layers.

257 XCL12 and gp120 modulate the excitability of Cajal-Retzius cells in opposite directions.

258 resynaptic GABAergic interneurons contacting Cajal-Retzius cells is important to understand the micro

259 We analyzed the connections that Cajal-Retzius cells make and receive, and found that the

260 ow that GABAergic evoked synaptic input onto Cajal-Retzius cells may either increase their excitabili

261 rom hippocampal stratum lacunosum-moleculare Cajal-Retzius cells of the CXCR4-EGFP mouse.

262 have addressed these questions by activating Cajal-Retzius cells optogenetically in mouse hippocampal

263 n cortical GABAergic interneurons, including Cajal-Retzius cells or in the basal ganglia.

264 Cajal-Retzius cells orchestrate the development of corti

265 entorhinal cortex-CA1 synapse, suggest that Cajal-Retzius cells produce a diffuse output that may im

266 , and pharmacological experiments shows that Cajal-Retzius cells receive GABAergic input from oriens

267 the neocortex and hippocampus has shown that Cajal-Retzius cells receive predominantly, if not exclus

268 r paired recording experiments indicate that Cajal-Retzius cells receive small-amplitude, kinetically

269 Furthermore, we show that the axons of Cajal-Retzius cells target specifically the stratum lacu

270 ee different types of forebrain neurons: the Cajal-Retzius cells that populate the surface of the tel

271 marginal zone dysplasia with displacement of Cajal-Retzius cells to deeper cortical layers.

272 The connectivity from presynaptic Cajal-Retzius cells to interneurons was strong enough to

273 ice Tbr2 is required for proper migration of Cajal-Retzius cells to the DG; and, in the absence of Tb

274 a dramatically reduces spontaneous firing in Cajal-Retzius cells via hyerpolarization, and that cessa

275 comparing the properties of CXCR4-expressing Cajal-Retzius cells vs. CXCR4-non-expressing interneuron

276 TX, and decreased when presynaptic firing in Cajal-Retzius cells was reduced by the chemokine CXCL12,

277 so expressed in neurons in layer I (presumed Cajal-Retzius cells), and white matter (interstitial) ne

278 horage of radial glial endfeet, mislocalized Cajal-Retzius cells, and neuronal overmigration.

279 ties in the brain surface basement membrane, Cajal-Retzius cells, and radial glia.

280 mix of future marginal zone cells, including Cajal-Retzius cells, and subplate cells.

281 n the migration of forebrain neurons such as Cajal-Retzius cells, interneurons moving to the ventral

282 kinergic modulation of spontaneous firing of Cajal-Retzius cells, mediated by the chemokine (C-X-C mo

283 f radial glial processes and displacement of Cajal-Retzius cells.

284 Foxg1 null mutants, we observed an excess of Cajal-Retzius neuron production in the cortex.

285 ber, GDF5, which is produced by the adjacent Cajal-Retzius neurons and turns on before outgrowth of t

286 ne (HAR1F) that is expressed specifically in Cajal-Retzius neurons in the developing human neocortex

287 1F is co-expressed with reelin, a product of Cajal-Retzius neurons that is of fundamental importance

288 of the marginal zone with redistribution of Cajal-Retzius neurons to deeper layers.

289 neurons (i.e., interneurons, neurogliaform, Cajal-Retzius neurons) resembled those observed in other

290 produces morphogens such as Wnt3a, generates Cajal-Retzius neurons, and is regulated by Lhx2.

291 vivo order: first preplate cells, including Cajal-Retzius neurons, then deep and finally superficial

292 he adjacent hippocampus instead of producing Cajal-Retzius neurons.

293 onal cell bodies (cortical, subcortical, and Cajal-Retzius).

294 Since Cajal's first drawings of Golgi stained neurons, generat

295 nal studies have provided strong support for Cajal's view.

296 restore the numbers of interstitial cells of Cajal that are reduced in the nNOS(-/-) colon.

297 Since the days of Cajal, the CA1 pyramidal cell has arguably received more

298 ed to the Desensitization Program at Ramon y Cajal University Hospital (RCUH).

299 ed to the Desensitization Program at Ramon y Cajal University Hospital.

300 Interstitial cells of Cajal, which express the receptor tyrosine kinase Kit, a