ライフサイエンスコーパス: Campylobacter

1 involved in generating the helical shape of Campylobacter.

2 erotoxigenic Escherichia coli, Shigella, and Campylobacter.

3 ema, Selenomonas, Parvimonas, Dialister, and Campylobacter.

4 mer products rather than complete absence of Campylobacter.

5 uggested a new model of FeEnt acquisition in Campylobacter.

6 eneralist lineages of the zoonotic bacterium Campylobacter.

7 ng that two resistance mechanisms existed in Campylobacter.

8 adequate for protecting against exposure to Campylobacter.

9 ylobacter, indicating their functionality in Campylobacter.

10 of infection is an estimated 1 in 23500 for Campylobacter, 1 in 5050000 for Cryptosporidium, and 1 i

11 pathogens Shigella (36%), Giardia (33%), and Campylobacter (30%) predominated, but their presence was

12 753 pathogen-specific gastroenteritis cases (Campylobacter: 738, Salmonella: 624, Shigella: 376, Yers

13 Presently, the anti-Campylobacter activity of thymol was compared against th

14 e results identify a key virulence factor in Campylobacter and a potential target for the control of

15 erythromycin, a total of eight strains (four Campylobacter and four Enterococcus) obtained macrolide-

16 motors that produce different torques, from Campylobacter and Vibrio species.

17 TM7, and 11 genera, including Anaeroplasma, Campylobacter, and Clostridium, were correlated with app

18 e for the detection of Salmonella, Shigella, Campylobacter, and Shiga toxin-producing enterohemorrhag

19 er in Actinomyces, Selenomonas, Veillonella, Campylobacter, and TM7 [G-1] than the Swedish groups.

20 Campylobacter antigen detection by enzyme immunoassay (E

21 STAT! Campy immunochromatographic assay for Campylobacter antigen was compared to culture for 500 cl

22 Pathogenic species within the genus Campylobacter are responsible for a considerable burden

23 ed to estimate aerobic plate count (APC) and Campylobacter as well as Salmonella prevalence.

24 etected Salmonella, EHEC O157, Shigella, and Campylobacter at concentrations 1- to 2-log10 lower than

25 In microaerophilic organisms such as Campylobacter, biofilms play a key role in transmission

26 A high Campylobacter burden was associated with a lower length-

27 omach, including Fusobacterium, Megasphaera, Campylobacter, Capnocytophaga, and Dialister.

28 We propose to rename Tlp11 as CcrG, Campylobacter ChemoReceptor for Galactose.

29 Short-read data from 384 Campylobacter clinical isolates collected over 4 months

30 s, and provide improved resolution (i) among Campylobacter clonal complexes and (ii) between very clo

31 We purified R.PabI homologs from Campylobacter coli (R.CcoLI) and Helicobacter pylori (R.

32 p., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA for Shiga toxins 1 and 2.

33 When treated with 1 mM thymol, Campylobacter coli and jejuni were reduced during pure o

34 rolide-resistant mutants were induced in one Campylobacter coli and one Enterococcus faecium strain,

35 Campylobacter jejuni and Campylobacter coli are zoonotic pathogens once considere

36 ry, clinical and environmental C. jejuni and Campylobacter coli contained genetic changes within the

37 Interestingly, the OSTs from Campylobacter coli, Campylobacter upsaliensis, Desulfovi

38 diarrhoea-including Campylobacter jejuni and Campylobacter coli, Cryptosporidium spp, enteropathogeni

39 or the detection of Campylobacter jejuni and Campylobacter coli, leading global causes of bacterial g

40 /89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.

41 n order to determine the natural patterns of Campylobacter colonization over a period of 63 weeks.

42 Campylobacter concisus is an emergent pathogen that play

43 s of PglC, a prototypic dual domain PGT from Campylobacter concisus Using a luminescence-based assay,

44 Clostridium difficile, Campylobacter jejuni, Campylobacter concisus, and Salmonella enterica were rec

45 nocytophaga gingivalis, Eikenella corrodens, Campylobacter concisus, Porphyromonas gingivalis, Tanner

46 We used reference pathogens Campylobacter, Cryptosporidium, and rotavirus as conserv

47 enterotoxigenic Escherichia coli, Shigella, Campylobacter, Cryptosporidium, norovirus GII, and adeno

48 Campylobacter data collected from several urban stormwat

49 Campylobacter data were used to estimate gastrointestina

50 s positively correlated with the presence of Campylobacter, Deinococcus, and Sulfurospirillum Finally

51 rch is required to better define whether the Campylobacter detectable in stormwater are pathogenic to

52 Factors associated with a reduced risk of Campylobacter detection included exclusive breastfeeding

53 ed excellent sensitivity and specificity for Campylobacter detection with low cross reactivity for ot

54 ntigen CIDTs compared to culture and PCR for Campylobacter detection.

55 Given the relatively low incidence of Campylobacter disease and the generally poor diagnostic

56 lts suggest that there is natural control of Campylobacter dynamics within a flock which could potent

57 Enteroaggregative Escherichia coli, Campylobacter, enteropathogenic E. coli, rotavirus, and

58 he potentially deleterious implications of a Campylobacter enzyme immunoassay (EIA) result and the in

59 osis cases are acute and self-limiting, with Campylobacter excretion ceasing a few weeks after sympto

60 Historically, genetically diverse Campylobacter fetus and Campylobacter jejuni strains hav

61 Campylobacter fetus can cause intestinal illness and, oc

62 Campylobacter fetus infection should be suspected partic

63 4 to December 2016, a cluster of 13 cases of Campylobacter fetus intestinal and extraintestinal infec

64 Campylobacter fetus is a venereal pathogen of cattle and

65 Campylobacter fetus is rarely isolated from food, albeit

66 Classifications of the Campylobacter fetus subspecies fetus and venerealis were

67 Pathogens, including norovirus, Campylobacter fetus, Helicobacter pylori, Salmonella ent

68 he substantial burden of apparently sporadic Campylobacter from cattle where transmission routes are

69 A rise in Campylobacter gastroenteritis in an isolated population

70 , Parvimonas micra, Eubacterium nodatum, and Campylobacter gracilis, a significant positive correlati

71 ture-independent detection tests (CIDTs) for Campylobacter have become an area of intense controversy

72 the FlgK junction onto the structure of the Campylobacter hook provides some clues about its diverge

73 uctural constraints, due to the structure of Campylobacter hook, causes divergence of one element of

74 new insights into the adaptive mechanisms of Campylobacter in animal food production environments.

75 ts, as standalone tests for the detection of Campylobacter in stool is increasing.

76 as standalone tests for direct detection of Campylobacter in stool.

77 t diminished the abundance of non-pathogenic Campylobacter in the juvenile gut, suggesting a potentia

78 on the epidemiology of antibiotic-resistant Campylobacter in the ruminant reservoir.

79 ndings reveal the rising prevalence of FQ(R) Campylobacter in the U.S. and provide novel information

80 the TA systems, pVir was readily cured from Campylobacter, indicating their functionality in Campylo

81 rade 3]; diarrhoea and fever associated with Campylobacter infection [grade 3]; recurrence of abdomin

82 ntibiotic treatment may reduce the burden of Campylobacter infection and improve growth in children i

83 , we describe the epidemiology and impact of Campylobacter infection in the first 2 years of life.

84 Campylobacter infection is a leading cause of ovine abor

85 Campylobacter infection is the most commonly notified ba

86 Campylobacter infection was also associated with increas

87 oxacin, an antibiotic of choice for treating Campylobacter infection, through the pore of MOMP reveal

88 mmunological profile of the host response to Campylobacter infection.

89 of the gastrointestinal tract as a result of Campylobacter infection.

90 dentify risk factors for sporadic intestinal Campylobacter infections and to determine the relative i

91 In 2012, a multistate outbreak of Campylobacter infections associated with unpasteurized m

92 scertain burden, and can distinguish between Campylobacter infections at the species level.

93 We describe the epidemiology of Campylobacter infections in the United States during 200

94 ential for understanding the epidemiology of Campylobacter infections, a major worldwide cause of bac

95 rvoir responsible for up to 80% of the human Campylobacter infections.

96 out were the most important risk factors for Campylobacter infections.

97 Campylobacter is a common bacterial enteropathogen that

98 Campylobacter is a common cause of human gastroenteritis

99 Campylobacter is a pathogen frequently detected in urban

100 macrolides, in the major foodborne pathogen Campylobacter is considered a serious threat to public h

101 Campylobacter is the most common cause of foodborne bact

102 , this 4-gene operon is only present in some Campylobacter isolates and other arsenic resistance mech

103 Sixteen Campylobacter isolates were collected from the patient d

104 O157), shiga-toxin producing E. coli (stx2), Campylobacter jejuni (mapA), Shigella spp. (ipaH), and a

105 a patient with fecal specimens positive for Campylobacter jejuni (ST45) intermittently during a 10-y

106 Human campylobacteriosis, caused by Campylobacter jejuni and C. coli, remains a leading caus

107 rized two FeEnt receptors (CfrA and CfrB) in Campylobacter jejuni and C. coli, the enteric human path

108 Campylobacter jejuni and Campylobacter coli are zoonotic

109 athogens associated with diarrhoea-including Campylobacter jejuni and Campylobacter coli, Cryptospori

110 t targets mapA and ceuE for the detection of Campylobacter jejuni and Campylobacter coli, leading glo

111 Monomeric OTases, such as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze tr

112 genes with higher sequence diversity in the Campylobacter jejuni and Neisseria meningitidis genomes

113 says demonstrate that LpxJ and homologues in Campylobacter jejuni and Wolinella succinogenes can act

114 inked protein glycosylation (Pgl) pathway of Campylobacter jejuni are evaluated for their tolerance f

115 Despite the importance of Campylobacter jejuni as a pathogen, little is known abou

116 The central enzyme in the Campylobacter jejuni asparagine-linked glycosylation pat

117 icated a moderate probability of illness for Campylobacter jejuni at the study beaches, especially wh

118 The Campylobacter jejuni capsular polysaccharide is importan

119 Here, we report the crystal structures of Campylobacter jejuni Cas9 (CjCas9), one of the smallest

120 To investigate how Campylobacter jejuni causes the clinical symptoms of dia

121 he structures and functional dynamics of the Campylobacter jejuni CmeB multidrug efflux pump.

122 Here the authors present the structure of Campylobacter jejuni CmeB pump combined with functional

123 One phase-variable gene of Campylobacter jejuni encodes a homologue of an unusual T

124 acterial oligosaccharyltransferase, PglB, of Campylobacter jejuni favors acceptor proteins with conse

125 The Campylobacter jejuni flagellum exports both proteins tha

126 We discovered that Campylobacter jejuni FlhG is at the center of a multipar

127 Campylobacter jejuni harbors a branched electron transpo

128 A lack of relevant disease models for Campylobacter jejuni has long been an obstacle to resear

129 Campylobacter jejuni helical shape is important for colo

130 treptococcus pneumoniae in mice, and against Campylobacter jejuni in chicken.

131 osphorylase (PNPase) facilitates survival of Campylobacter jejuni in low temperatures and favors swim

132 Campylobacter jejuni is a commensal bacterium in the int

133 Campylobacter jejuni is a common cause of diarrhea and i

134 Campylobacter jejuni is a helix-shaped enteric bacterial

135 Campylobacter jejuni is a leading cause of acute gastroe

136 Campylobacter jejuni is a leading cause of bacterial gas

137 Campylobacter jejuni is a leading cause of bacterial gas

138 Campylobacter jejuni is a leading cause of gastrointesti

139 Campylobacter jejuni is a major cause of bacterial diarr

140 Campylobacter jejuni is a major cause of bacterial gastr

141 Campylobacter jejuni is a major human pathogen and a lea

142 Campylobacter jejuni is a major zoonotic pathogen, and i

143 Campylobacter jejuni is a natural commensal of the avian

144 Campylobacter jejuni is a zoonotic pathogen, and a hyper

145 Campylobacter jejuni is an important zoonotic pathogen t

146 The human pathogen Campylobacter jejuni is naturally competent for transfor

147 Campylobacter jejuni is one of the leading infectious ca

148 c acid cycle in the microaerophilic pathogen Campylobacter jejuni is potentially vulnerable, as it em

149 Campylobacter jejuni is the leading cause of foodborne b

150 Campylobacter jejuni is the leading cause of human bacte

151 The Gram-negative organism Campylobacter jejuni is the major cause of food poisonin

152 Campylobacter jejuni is the most common bacterial cause

153 The Gram-negative pathogen Campylobacter jejuni is the most common cause of bacteri

154 sourceR is demonstrated using Campylobacter jejuni isolate data collected in New Zeala

155 Campylobacter jejuni isolates from human (n = 65), bovin

156 he genetic basis of biofilm formation in 102 Campylobacter jejuni isolates.

157 The Campylobacter jejuni N-linked glycosylation pathway has

158 o-L-gluco-heptopyranose residue found in the Campylobacter jejuni NCTC11168 (HS:2) capsular polysacch

159 75-year-old man was diagnosed with probable Campylobacter jejuni prosthetic knee infection after a d

160 complex L-gluco-heptose synthesis pathway of Campylobacter jejuni strain NCTC 11168.

161 genetically diverse Campylobacter fetus and Campylobacter jejuni strains have been implicated in suc

162 n (strain VPI-5482) [PDB:3KZT], Cj0202c from Campylobacter jejuni subsp. jejuni serotype O:2 (strain

163 ber 2013, sexual transmission of 2 clades of Campylobacter jejuni subspecies jejuni isolates resulted

164 In Campylobacter jejuni the periplasmic binding protein Ceu

165 transfer of an N-glycosylation pathway from Campylobacter jejuni to Escherichia coli in 2002 can be

166 owth of the microaerophilic mucosal pathogen Campylobacter jejuni under oxygen-limited conditions was

167 The microaerophilic food-borne pathogen Campylobacter jejuni uses complex cytochrome-rich respir

168 ed sites (eg, Aeromonas, Vibrio cholerae O1, Campylobacter jejuni).

169 Campylobacter jejuni, a leading bacterial cause of foodb

170 Here we show that Campylobacter jejuni, a leading bacterial cause of human

171 Campylobacter jejuni, a leading cause of bacterial gastr

172 detection of Salmonella spp., Shigella spp., Campylobacter jejuni, and Campylobacter coli and an EIA

173 classes of inhibitors of Bacillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs

174 Enterovirus, adenovirus A, Salmonella spp., Campylobacter jejuni, bovine polyomavirus, and bovine ro

175 ase 3, sequences from Clostridium difficile, Campylobacter jejuni, Campylobacter concisus, and Salmon

176 The leading foodborne pathogen, Campylobacter jejuni, can carry multiple plasmids associ

177 erial species (Bacteroides thetaiotaomicron, Campylobacter jejuni, Enterococcus faecalis, Escherichia

178 d to spray-irrigated dairy manure containing Campylobacter jejuni, enterohemorrhagic Escherichia coli

179 ty against Gram-negative bacteria, including Campylobacter jejuni, Escherichia coli O157:H7, and mult

180 The dispersion of pathogens (Campylobacter jejuni, Escherichia coli O157:H7, non-O157

181 Campylobacter jejuni, known for being a major cause of b

182 d the highest antimicrobial activity against Campylobacter jejuni, L. monocytogenes, and Pseudomonas

183 The foodborne microaerophilic pathogen, Campylobacter jejuni, possesses a periplasmic formate de

184 eropathogenic E. coli (EPEC), Shigella spp., Campylobacter jejuni, Salmonella enterica, and Aeromonas

185 ae, Treponema pallidum, Helicobacter pylori, Campylobacter jejuni, Synechocystis sp., and Mycobacteri

186 Campylobacter jejuni, the leading cause of human bacteri

187 Campylobacter jejuni, the most common cause of bacterial

188 Campylobacter jejuni, the most frequent cause of food-bo

189 previously identified in invasive strains of Campylobacter jejuni, the most prevalent cause of bacter

190 A bacterial permease, ArsP, from Campylobacter jejuni, was recently shown to confer resis

191 ugh frequent recombination with it, while in Campylobacter jejuni, we find a minority population we p

192 y and kinetic parameters of PglC, a PGT from Campylobacter jejuni, were quickly established using thi

193 that the periplasmic binding protein CeuE of Campylobacter jejuni, which was previously thought to bi

194 zation domain-containing protein 2 (NOD2) in Campylobacter jejuni-induced intestinal inflammation.

195 the contribution of PI3K-gamma signaling in Campylobacter jejuni-induced neutrophil accumulation and

196 involved in biosynthesis of UDP-diNAcBac in Campylobacter jejuni.

197 unosensor for the detection of food pathogen Campylobacter jejuni.

198 ectious neuropathy most frequently caused by Campylobacter jejuni.

199 with the periplasmic binding protein CeuE of Campylobacter jejuni.

200 91% for C. difficile A/B toxins; and 90% for Campylobacter jejuni.

201 the human pathogens Helicobacter pylori and Campylobacter jejuni.

202 IA (ProSpecT), and duplex PCR to distinguish Campylobacter jejuni/C. coli and non-jejuni/coli Campylo

203 According to PCR, Campylobacter jejuni/C. coli infections represented less

204 us GII, rotavirus, and sapovirus), bacteria (Campylobacter jejuni/C. coli, Clostridium difficile, Sal

205 ure alone detected 80/89 (89.9% sensitivity) Campylobacter jejuni/Campylobacter coli-positive cases.

206 oli (STEC), Shigella spp. , Salmonella spp , Campylobacter jejuni/coli , and methicillin-resistant St

207 e 6 bacterial enteric pathogens tested, only Campylobacter jejuni/coli detection was significantly re

208 tructure of a complete bacterial OST enzyme, Campylobacter lari PglB, was recently determined.

209 tructure of a single-subunit OST enzyme, the Campylobacter lari protein PglB, revealed a partially di

210 h as the PglBs from Campylobacter jejuni and Campylobacter lari, catalyze transfer of glycans from me

211 animals serve as a significant reservoir for Campylobacter, limited information is available on antib

212 e intensified along the food chain to reduce Campylobacter load, especially on chicken meat.

213 To induce abortion, orally ingested Campylobacter must translocate across the intestinal epi

214 Campylobacter-negative stool spiked with each of the abo

215 to the pathogens concentration (particularly Campylobacter, Norovirus, and Legionella) and exposure f

216 rmation is available on antibiotic-resistant Campylobacter of bovine origin.

217 ar polysaccharide (CP) biosynthesis, PglB (a Campylobacter oligosaccharyl transferase), and a protein

218 ylobacter jejuni/C. coli and non-jejuni/coli Campylobacter on 432 diarrheal and matched control stool

219 The annual median number of Campylobacter outbreaks increased from 28 in 2004-2006 t

220 a means to evaluate the function of pVir in Campylobacter pathobiology.

221 entative members of this superfamily are the Campylobacter PglCs, which initiate N-linked glycoprotei

222 NA demonstrated to play a functional role in Campylobacter physiology to date.

223 al susceptibility testing suggested that the Campylobacter population developed resistance to several

224 consistently colonized with organisms from a Campylobacter population that adapted to the internal en

225 iotic therapy and long-term excretion on the Campylobacter population.

226 and the effect of antimicrobial treatment on Campylobacter populations in this unusual situation of l

227 e was a substantial reduction in predominant Campylobacter populations proposing that SUCRAM suppleme

228 ith most children (n = 1606; 84.9%) having a Campylobacter-positive stool sample by 1 year of age.

229 The prevalences of Fusobacterium, Campylobacter, Prevotella, Capnocytophaga, Selenomonas,

230 Nineteen additional cases of Campylobacter prosthetic joint infection reported in the

231 icola (94%/74%), Parvimonas micra (86%/62%), Campylobacter rectus (90%/76%), Eubacterium nodatum (64%

232 omitans (Aa), Porphyromonas gingivalis (Pg), Campylobacter rectus (Cr), and Tannerella forsythia (Tf)

233 lis, and Fusobacterium nucleatum, as well as Campylobacter rectus (except for amoxicillin alone).

234 omonas gingivalis, Tannerella forsythia, and Campylobacter rectus (P </=0.05).

235 omonas gingivalis, Streptococcus mutans, and Campylobacter rectus are also reviewed.

236 th groups (both P = 0.043), as were those of Campylobacter rectus in the test group only (P = 0.028).

237 ing association of Prevotella intermedia and Campylobacter rectus with the etiology of peri-implantit

238 terium nucleatum, Prevotella intermedia, and Campylobacter rectus), two red-complex periodontal patho

239 The presence of Campylobacter rectus, Aggregatibacter actinomycetemcomit

240 nomycetemcomitans, Porphyromonas gingivalis, Campylobacter rectus, and Tannerella forsythia) in vascu

241 romonas gingivalis, Treponema denticola, and Campylobacter rectus, were highest in patients with GAgP

242 terium nucleatum, Prevotella intermedia, and Campylobacter rectus.

243 forsythia, as well as Actinomyces viscosus, Campylobacter rectus/showae, Prevotella intermedia, Parv

244 while both Amplon and PAA yielded detectable Campylobacter reductions at all steps.

245 n various C. jejuni strains, suggesting that Campylobacter requires at least one of the two genes for

246 untry settings, the ProSpecT EIA and PCR for Campylobacter reveal extremely high rates of positivity.

247 were only 19/196 (10%) positive cultures for Campylobacter, Salmonella, or Shigella entero-pathogens

248 of 16S rRNA from 53 of these non-jejuni/coli Campylobacter samples showed that it most closely matche

249 ss with the following methods: four types of Campylobacter selective media, four commercial stool ant

250 To better understand how these campylobacters sense nutrient availability, we examined

251 Campylobacter sequence types (STs) were not mutually exc

252 lus, Corynebacterium, Cellulosimicrobium and Campylobacter showed lower abundances in OLP patients, a

253 umors followed by Pseudomonas aeruginosa and Campylobacter sp.

254 imental model for the study of diarrheagenic Campylobacter species and will be useful in exploring th

255 Of these, emerging Campylobacter species have been found to be more prevale

256 lphia region were prospectively analyzed for Campylobacter species other than C. jejuni and C. coli u

257 tool spiked with each of the above-mentioned Campylobacter species revealed reactivity with EIA.

258 vasion, and intracellular survival, emerging Campylobacter species should be investigated as etiologi

259 at least one additional non-jejuni and -coli Campylobacter species that may be missed by routine cult

260 PCR detection of Campylobacter species was strongly associated with diarr

261 ent isolation of C. concisus The majority of Campylobacter species were not clinically significant.

262 Of 225 samples tested, 13 (5.8%) yielded Campylobacter species, with frequent isolation of C. con

263 a to evaluate a PCR assay for distinguishing Campylobacter species.

264 re against infectious microorganisms such as Campylobacter species.

265 but 27.6% were positive for non-jejuni/coli Campylobacter species.

266 sented less than half of all infections with Campylobacter species.

267 95% CI 3.0-7.1), rotavirus (4.8%, 4.5-5.0), Campylobacter spp (3.5%, 0.4-6.3), astrovirus (2.7%, 2.2

268 th diarrhoea in the second year of life were Campylobacter spp (7.9%, 3.1-12.1), norovirus GII (5.4%,

269 oody diarrhoea was primarily associated with Campylobacter spp and Shigella spp, fever and vomiting w

270 bstrates was fabricated for the detection of Campylobacter spp in food matrices.

271 -acquired infections such as Salmonella spp, Campylobacter spp, N gonorrhoeae, and H pylori.

272 bacter pylori, and fluoroquinolone-resistant Campylobacter spp, Neisseria gonorrhoeae, and Salmonella

273 rus 40/41, ST-ETEC, Cryptosporidium spp, and Campylobacter spp.

274 f the most common poultry meat contaminants: Campylobacter spp.

275 l disease agents, particularly norovirus and Campylobacter spp.

276 erichia coli (OR: 1.39; 95% CI: 1.05, 1.83), Campylobacter spp. (OR: 1.46; 95% CI: 1.11, 1.91), heat-

277 Campylobacter spp. are responsible for acute bacterial d

278 ed for the concentrations of PCR amplicon of Campylobacter spp. between 1 and 25 nM with a limit of d

279 million (95% UI 70-251 million) cases, while Campylobacter spp. caused 96 million (95% UI 52-177 mill

280 lification, and electrochemical detection of Campylobacter spp. in raw poultry meat samples.

281 , intestinal enterococci, and thermotolerant Campylobacter spp. in surface waters than that of rDNA-b

282 lmonella (nontyphoidal) spp., Shigella spp., Campylobacter spp. or Yersinia enterocolitica and matche

283 s pathogenic strains of Escherichia coli and Campylobacter spp. that declined in prevalence based on

284 ric pathogens directly from stool specimens: Campylobacter spp., Clostridium difficile (toxin A/B), P

285 llected and tested by enzyme immunoassay for Campylobacter Stool and blood samples were assayed for m

286 port comprehensive performance data for four Campylobacter stool antigen CIDTs versus culture and mol

287 pecificity, and positive predictive value of Campylobacter stool antigen tests were highly variable.

288 Genetic manipulation of diverse Campylobacter strains demonstrated that Cee is not only

289 Campylobacter STs typically reached a peak in prevalence

290 Here we show that, in Campylobacter, the 3D structure of FlgK differs from tha

291 nserved DNA methyltransferase, which we term Campylobacter transformation system methyltransferase (c

292 restingly, the OSTs from Campylobacter coli, Campylobacter upsaliensis, Desulfovibrio desulfuricans,

293 ne copies/swab, respectively; P < .001), and Campylobacter ureolyticus (1.7 x 10(5) and 1.6 x 10(7)16

294 Despite ongoing research on Campylobacter virulence mechanisms, little is known rega

295 Campylobacter was prevalent across diverse settings and

296 26 267 nondiarrheal stool samples tested for Campylobacter We describe a high prevalence of infection

297 Contigs were deposited at the pubMLST.org/campylobacter website and automatically annotated for 1,

298 High concentrations of Campylobacter were found, being the lowest in the lake (

299 health risks associated with the exposure to Campylobacter when harvesting urban stormwater for toile

300 sor would help in the sensitive detection of Campylobacter which can result in reducing pre-enrichmen