ライフサイエンスコーパス: Cdc28 kinase

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1 he accumulation of high levels of the Cln1/2-Cdc28 kinase, a known repressor of mating factor signal

2 Clb1-Cdc28, Clb3-Cdc28, Clb4-Cdc28, and Clb5-Cdc28 kinase activities during meiosis were not affected

3 inding of Cdc45p to chromatin depends on Clb-Cdc28 kinase activity as well as functional Cdc6p and Mc

4 Cln-Cdc28 kinase activity at START is required to repress B-

5 further suggesting that it is the decreased Cdc28 kinase activity in these hsl mutants that causes l

6 , or from reduced histone transcription when Cdc28 kinase activity is compromised.

7 These results could indicate that Cln2-Cdc28 kinase activity is not directly relevant to some C

8 cAMP increases Cln3 protein levels and Cln3-Cdc28 kinase activity.

9 Swe1 kinase, which in turn decreases mitotic Cdc28 kinase activity.

10 n levels, by positive feedback from the Clb2-Cdc28 kinase, and by osmotic stress, but the correspondi

11 ll cells occurs if the gene CLB2, encoding a Cdc28 kinase-associated B-type cyclin, is expressed on a

12 ify components of pathways downstream of the Cdc28 kinase at START by screening for mutations that de

13 Sic1, an inhibitor of C1b-Cdc28 kinases, became phosphorylated at Start, and this

14 Active Clb2-Cdc28 kinase complex was purified from yeast cells after

15 e to the combined activity of Nap1, the Clb2-Cdc28 kinase complex, and the GTP-bound form of Cdc42.

16 quired for both the periodic dissociation of Cdc28 kinase from the CDC20 promoter and the periodic as

17 uit the proteasome to, and/or dissociate the Cdc28 kinase from, the CDC20 promoter, thus facilitating

18 28 consensus sites and is a substrate of the Cdc28 kinase in vitro.

19 The Cln3-Cdc28 kinase is required to activate the Swi4-Swi6 trans

20 e, we find that inactivation of Cln- and Clb-Cdc28 kinases is sufficient to trigger Clb2 proteolysis

21 How Clb/Cdc28 kinases prevent pre-RC assembly is not understood.

22 We propose that Clb/Cdc28 kinases prevent pre-RC reassembly in part by promo

23 licates a decrease in the expression of cdc2/cdc28 kinase subunit Homo sapiens 1 (CKsHs1) and a delay

24 ssion of pheromone signaling pathway by Cln2-Cdc28 kinase takes place at a level around STE20.

25 nts, and the expression and activity of Cln3-Cdc28 kinase that regulates the G1-S boundary of the cel

26 hromatin, S-phase progression as well as Clb/Cdc28 kinases was required for Mcm-GFP export.

27 In the absence of functional Cdc6, Clb/Cdc28 kinases were necessary and sufficient for efficien

28 ating Ste5 recruitment and by inhibiting Cln/Cdc28 kinase, which prevents basal recruitment of Ste5,

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