ライフサイエンスコーパス: ChAT

1 ChAT and CFP expression was evaluated in flat-mounted re

2 ChAT and VAChT are found in the same neurons, including

3 ChAT belongs to a family of CoA-dependent enzymes that a

4 ChAT expression occurs in mucosal-associated lymph tissu

5 ChAT interneurons increased, while calbindin interneuron

6 ChAT was able to detect chromatin signatures previously

7 ChAT, bNOS, glycine, and GAD remain reliable AC markers

8 ChAT-immunoreactive (-ir) perikarya were seen in the olf

9 ChAT-ir fibers were seen throughout the dentate gyrus an

10 ChAT-negative regions lacked retinal waves for the first

11 e differentiated the iPSC clones into ISL-1+/ChAT+ MNs and performed a comparative study during the d

12 s, including GLUR1, GABA-B1a, NMDA1, GAP-43, ChAT, BDNF, nestin, BMP-2, BMP-4, and EGR1, was increase

13 Despite this ability, ChAT(+) B cells were unable to suppress effector T-cell

14 ting, and ON-type choline acetyltransferase (ChAT) ACs in wild-type and ChAT transgenic mice (ChAT- t

15 0-50% decrease in choline acetyltransferase (ChAT) activity in two of three subregions of the hippoca

16 Choline acetyltransferase (ChAT) activity served as a measure of cholinergic differ

17 Choline acetyltransferase (ChAT) and acetylcholinesterase (AChE) are the decisive e

18 ses revealed that choline acetyltransferase (ChAT) and GABA immunoreactive neurons were distributed t

19 munoreactivity to choline acetyltransferase (ChAT) and sent axons into the sciatic nerve as detected

20 directed against choline acetyltransferase (ChAT) and the dopamine D5 receptor.

21 were enriched in choline acetyltransferase (ChAT) and the high-affinity choline transporter (ChT), a

22 visualization of choline acetyltransferase (ChAT) and the low-affinity neurotrophin receptor, p75(NT

23 Choline acetyltransferase (ChAT) and the vesicular ACh transporter (VAChT), as well

24 mical markers for choline acetyltransferase (ChAT) and tyrosine hyroxylase (TH) were also used to qua

25 ein expression of choline acetyltransferase (ChAT) and vesicular acetylcholine transporter (VAChT), w

26 the expression of choline acetyltransferase (ChAT) by immunocytochemistry and Western blot in develop

27 ransmitter enzyme choline acetyltransferase (ChAT) by in vitro motor neurons, likely independent of i

28 In eukaryotes, choline acetyltransferase (ChAT) catalyzes the reversible formation of the neurotra

29 proteins, reduced choline acetyltransferase (ChAT) enzyme expression, fragmented mitochondria, glial

30 mice lacking the choline acetyltransferase (ChAT) enzyme.

31 ned with nNOS and choline acetyltransferase (ChAT) immunocytochemistry, and nicotinamide adenine dinu

32 ing combined with choline acetyltransferase (ChAT) immunohistochemistry in rats.

33 re identified via choline acetyltransferase (ChAT) immunohistochemistry.

34 ths of age, using choline acetyltransferase (ChAT) immunolabeling to identify cholinergic interneuron

35 of the number of choline acetyltransferase (ChAT) immunopositive neurons in the medial septal/diagon

36 ult zebrafish for choline acetyltransferase (ChAT) immunoreactivity.

37 tical density for choline acetyltransferase (ChAT) in both young and aged monkeys treated with estrog

38 s peptide and for choline acetyltransferase (ChAT) in macaque monkeys, in which most preganglionic mo

39 mmunoreactive for choline acetyltransferase (ChAT) in regions of the executive and motor loops of the

40 after MI revealed choline acetyltransferase (ChAT) in stellate sympathetic neurons and vesicular ACh

41 and D2-expressing choline acetyltransferase (ChAT) interneurons express Slc6a15, we examined Slc6a15

42 e distribution of choline acetyltransferase (ChAT) is described in Polypterus and compared with the d

43 ere, we show that choline acetyltransferase (ChAT) is expressed and ACh is produced by B cells and ot

44 linergic based on choline acetyltransferase (ChAT) mRNA expression.

45 were found to be choline acetyltransferase (ChAT) negative, indicating that they are not motor neuro

46 immunolabeled for choline acetyltransferase (ChAT) or p75-neurotrophin receptor (p75(NTR) ).

47 eurons containing choline acetyltransferase (ChAT) or vasoactive intestinal peptide (VIP) share chara

48 he control of the choline acetyltransferase (ChAT) promoter (ChAT-ChR2-EYFP) to dissect cholinergic c

49 Choline acetyltransferase (ChAT) protein was expressed in only 30% of PPNs, but 80%

50 oxylase (GAD) and choline acetyltransferase (ChAT) revealed that all CG neurons are contacted by ChAT

51 the two bands of choline acetyltransferase (ChAT) staining.

52 Choline acetyltransferase (ChAT) synthesizes acetylcholine in neurons and other cel

53 osynthetic enzyme choline acetyltransferase (ChAT) was applied to individual morphologically and elec

54 nthase (nNOS), or choline acetyltransferase (ChAT) were performed in wholemounts of urothelium or det

55 own population of choline acetyltransferase (ChAT)(+) neurons residing in the rodent SVZ neurogenic n

56 albumin (PV)+ and choline acetyltransferase (ChAT)+ cholinergic interneurons in the Cd and the putame

57 and expression of choline acetyltransferase (ChAT), a synthetic enzyme for the neurotransmitter acety

58 lyzed for p75NTR, choline acetyltransferase (ChAT), activating transcription factor 3 (ATF3), and cle

59 with antisera to choline acetyltransferase (ChAT), AMPA receptor subunits GluR1, GluR2/3, or GluR4,

60 yric acid (GABA), choline acetyltransferase (ChAT), and a N-methyl-D-aspartate type 1 receptor protei

61 ial marker F4/80, choline acetyltransferase (ChAT), and syntaxin 1.

62 tent inhibitor of choline acetyltransferase (ChAT), BW813U, on timing behavior in mature (6-10 months

63 histochemistry of choline acetyltransferase (ChAT), calbindin, calretinin, and parvalbumin to mark in

64 nesterase (AChE), choline acetyltransferase (ChAT), choline transporter 1 (CHT1, SLC5A7), vesicular a

65 unoreactivity for choline acetyltransferase (ChAT), contradictory findings, including the expression

66 ts another enzyme choline acetyltransferase (ChAT), leading to enhanced secretion of ACh.

67 e the activity of choline acetyltransferase (ChAT), the enzyme that produces the neurotransmitter ace

68 Choline acetyltransferase (ChAT), the enzyme that synthesizes the neurotransmitter

69 the gene encoding choline acetyltransferase (ChAT), the sole synthetic enzyme for acetylcholine (ACh)

70 ared with that of choline acetyltransferase (ChAT), tyrosine hydroxylase (TH), and serotonin (5HT) by

71 nzymes, including choline acetyltransferase (ChAT), vesicular acetylcholine transporter (VAChT) and t

72 eptide (VIP), and choline acetyltransferase (ChAT), was used to characterize the location and neurotr

73 ytes that express choline acetyltransferase (ChAT), which catalyzes the synthesis of the vasorelaxant

74 campal-projecting choline acetyltransferase (ChAT)-, neuronal nitric oxide synthase (nNOS)-, and parv

75 escent protein in choline acetyltransferase (ChAT)-expressing cells were used to label preganglionic

76 Choline acetyltransferase (ChAT)-immunolabeled cells were detected in the neuroblas

77 ning intensity of choline acetyltransferase (ChAT)-immunopositive cells in the retrodorsal lateral nu

78 4A administration choline acetyltransferase (ChAT)-immunoreactive fibers were extensively damaged on

79 ipolar cells were choline acetyltransferase (ChAT)-immunoreactive.

80 GABA-like and choline acetyltransferase (ChAT)-like immunoreactivity were used as markers for GAB

81 s damage, whereas choline acetyltransferase (ChAT)-positive and glycinergic AC subtypes were unaffect

82 Fos expression in choline acetyltransferase (ChAT)-positive interneurons.

83 the soma size of choline acetyltransferase (ChAT)-positive neurons in the laterodorsal and pedunculo

84 ms of GnRH and to choline acetyltransferase (ChAT).

85 zymatic action of choline acetyltransferase (ChAT).

86 zed by the enzyme choline acetyltransferase (ChAT).

87 roxylase (TH) and choline acetyltransferase (ChAT).

88 lbumin as well as choline acetyltransferase (ChAT).

89 ne transporter or choline acetyltransferase (ChAT).

90 ntibodies against choline acetyltransferase (ChAT).

91 hich both express choline acetyltransferase (ChAT).

92 scent proteins in choline acetyltransferase (ChAT)::Cre(+) transgenic rats, we selectively labeled ch

93 The activity of choline acetyltransferase (ChAT, the enzyme that carries out ACh production) was in

94 ion of p11 in cholinergic acetyltransferase (ChAT) neurons reduces tacrine-induced tremor.

95 urons (expressing choline acetyltransferase; ChAT) and GABAergic interneurons expressing parvalbumin

96 ub structures were choline acetyltrasferase (ChAT)-negative, and preabsorption control tests suggest

97 stimulated by norepinephrine to release ACh, ChAT(+) B cells release ACh after stimulation with sulfa

98 interact with KLC1, and formation of the ACL/ChAT complex is prevented, whereas the disease-associate

99 Analysis of adult ChAT-cre p75(in/in) mice revealed that mutant animals sh

100 insic electrophysiologic properties of adult ChAT-eGFP mouse MNs and classified them into four subtyp

101 is same CNTFRalpha depletion does not affect ChAT labeling in nonlesioned motor neurons, but it signi

102 nment followed by Tree-clustering algorithm (ChAT) employs dynamic programming of combinatorial histo

103 fluorescence demonstrated that virtually all ChAT-ir basal forebrain neurons were also p75(NTR) -posi

104 ges in the localisation of retinal Thy-1 and ChAT immunoreactivities.

105 iform layer strata displaying syntaxin 1 and ChAT.

106 ed by designer drugs (DREADD)' approach, and ChAT-IRES-Cre mice.

107 as assessed by immunostaining with Brn3a and ChAT antibodies, respectively.

108 resulted in a distribution of calbindin and ChAT in spinal gray matter regions where the CST termina

109 led to significant downregulation of CHT and ChAT, and, to a lesser extent, VAChT.

110 high-affinity choline transporter (CHT1) and ChAT.

111 long-chain acyl-CoAs, respectively, CrAT and ChAT display activity toward only short-chain acyl-CoAs.

112 addition to the motor neuron (Nestin-Cre and ChAT-Cre) resulted in the greatest improvement in surviv

113 tor interneurons, the CLIs formed GRASP- and ChAT-positive putative synapses with motoneurons and wer

114 We describe a procedure to measure HACU and ChAT at the same time in cultured cells by simple techni

115 ere described previously to measure HACU and ChAT simultaneously in synaptosomes, but the same for cu

116 le it upregulated ChAT/VAChT mRNA levels and ChAT activity.

117 e cells with BMP9 not only increased p75 and ChAT gene expression in p75- cells, but also augmented t

118 ubunits were found in both ChAT-positive and ChAT-negative cells in MHbV.

119 cortical slow oscillations, but only PV+ and ChAT+ interneurons also fired in time with cortical spin

120 receptors in adult motor neuron survival and ChAT maintenance, independent of developmental functions

121 PCR (n = 40) were Phox2b+, VGlut2+, TH-, and ChAT-, the neurochemical phenotype previously defined fo

122 cetyltransferase (ChAT) ACs in wild-type and ChAT transgenic mice (ChAT- tdTomato).

123 y hemicholinium-sensitive choline uptake and ChAT enzyme activity in a small number of differentiated

124 about 32% of muOR neurons contained VIP and ChAT immunoreactivities.

125 cting to the choroid contained NOS, VIP, and ChAT and were widely distributed in PPG and its pregangl

126 We applied ChAT to genomic maps of 39 histone modifications in huma

127 extension of the nucleus basalis, as well as ChAT(-) cells, release the inhibitory neurotransmitter G

128 Glutamate receptor antagonists fully blocked ChAT inhibition and oxidative stress induced by AbetaOs.

129 All subunits were found in both ChAT-positive and ChAT-negative cells in MHbV.

130 In these mice, brain ChAT activity was reduced by 40-50% relative to the wild

131 evealed that all CG neurons are contacted by ChAT-positive terminals.

132 are a distinct T-cell population defined by ChAT (CD4 TChAT).

133 inal networks became dominated by AChE or by ChAT reactivities.

134 res and functional associations uncovered by ChAT underscore the ability of the algorithm to yield no

135 roduced a significant increase in cerebellar ChAT RNA expression.

136 Cholinergic ChAT-ir neurons were also found within transitional cort

137 owing proteins: AChR subunits, rapsyn, ColQ, ChAT or muscle-specific kinase.

138 We also compared ChAT-immunoreactive (ir) interneuron morphological types

139 Sulprostone severely compromised ChAT activity, dendrite number, axonal length and axonal

140 terminals were also observed that contacted ChAT immunoreactive dendrites.

141 hese ages, and many of these cells contained ChAT or VIP.

142 rend toward amacrine cell loss and decreased ChAT protein levels.

143 Despite these differences, ChAT(+) cells, which have been historically identified a

144 Quantitative immunofluorescence for either ChAT or the low-affinity p75 neurotrophin receptor in th

145 ng the inner antennocerebral tract exhibited ChAT-like immunoreactivity.

146 g proportions of cortical TH neurons express ChAT or VIP developmentally and that a subset of these T

147 Neither of these cell populations expressed ChAT immunoreactivity in adult bladder.

148 the newly established VGAT-ChR2(H134R)-EYFP, ChAT-ChR2(H134R)-EYFP, Tph2-ChR2(H134R)-EYFP and Pvalb(H

149 erebellum (p < 0.005, one-way ANOVA) and for ChAT (choline acetyltransferase) in the male vagal lobe

150 neurons, the receptors become essential for ChAT maintenance when the motor neurons are challenged b

151 bband "d." Applying immunohistochemistry for ChAT and AChE on sections of the chicken retina, we here

152 Cell groups immunoreactive for ChAT were observed in the hypothalamus, the habenula, th

153 tions of TH cell profiles double-labeled for ChAT or VIP significantly increased between P16 and P30.

154 Real-time PCR showed that mRNA levels for ChAT, VAChT, and alpha7 and beta2 nAChR subunits varied

155 cular acetylcholine transporter, but not for ChAT, have enlarged perikarya in epileptic rats.

156 om) unlabeled neurons that were positive for ChAT and CGRP, but negative for GAL.

157 These were positive for ChAT, but negative for nNOS, VIP, SOM, GAL, and CGRP.

158 teen of 26 tumors screened were positive for ChAT.

159 hich were classified in this study, we found ChAT-like immunoreactivity suggesting that they are most

160 gs of fluorescent lumbar MN cell bodies from ChAT-eGFP or superoxide dismutase 1-yellow fluorescent p

161 ified dGs were usually negative for glycine, ChAT (choline acetyltransferase), bNOS (brain-type nitri

162 postnatal day 0 (P0), right after hatching, ChAT-immunoreactivity was present in the ganglion cell l

163 h chx10(+) interneurons and Islet1(+)/Hb9(+)/ChAT(+) motor neurons; the latter were recognized by gre

164 Importantly, precursors mature into HB9/ChAT-expressing functional MNs.

165 ler BFCNs, and overall increased hippocampal ChAT intensity compared with 2N unsupplemented mice.

166 ed in a significant decrease of hipppocampal ChAT and BDNF RNA expression that were unaffected by adu

167 The structure indicates how ChAT is regulated by phosphorylation and reveals an unus

168 However ChAT-Cre alone did not alter the survival of SMA mice by

169 s with non-immunolabeled dendrites; however, ChAT/GABA immunoreactive terminals were also observed th

170 substrate promiscuity of the wild-type human ChAT (hChAT).

171 of choline acetyltransferase-immunoreactive (ChAT) fibres was observed around the SMA and its up-stre

172 bar cord in mice expressing eNpHR or Arch in ChAT(+) or Isl1(+) neurons, depressed motoneuron dischar

173 The changes in ChAT in the SAMP/8 mouse strain were limited to the sept

174 AM-P/8 mice also showed a 40-50% decrease in ChAT activity in the septal region that was maximal by 8

175 Decreases in ChAT activity are associated with a number of disease st

176 There were no differences in ChAT activity in the nucleus basalis of Meynert, nor any

177 ced yellow fluorescent protein expression in ChAT-IRES-Cre mice, we tested the hypothesis that there

178 rhythmically active spinal circuit forms in ChAT mutants, but the duration of each cycle period is e

179 Furthermore, we confirmed the increase in ChAT activity observed upon treatment of cells with stol

180 ing that oxidative damage may be involved in ChAT inhibition.

181 These data demonstrate that p11 located in ChAT or D2R-containing neurons is involved in regulating

182 a number of disease states, and mutations in ChAT cause congenital neuromuscular disorders.

183 enetic stimulation of cholinergic neurons in ChAT-Cre mice, we investigated their effects on hippocam

184 Reduction in ChAT activity instigated by AbetaOs may thus be a releva

185 Slc6a15 protein was unaltered in ChAT interneurons.

186 Pharmacological analysis of the waves in ChAT knock-out regions revealed a requirement for gap ju

187 nists and the eventual emergence of waves in ChAT knock-out regions suggest that homeostatic mechanis

188 Type-I cells had increased ChAT and lost AChE; type-II cells presented less ChAT, b

189 s together with microglial CM also inhibited ChAT activity.

190 acetyltransferase-expressing interneurones (ChAT)(+) of the striatum influence the activity of mediu

191 s2Akita mouse, the morphology of the labeled ChAT-IR and TH-IR amacrine cell somas and dendrites appe

192 Mice lacking ChAT expression in CD4(+) cells have elevated arterial b

193 striatal photoinhibition of ChIs in lesioned ChAT(cre/cre) mice expressing halorhodopsin in ChIs redu

194 and lost AChE; type-II cells presented less ChAT, but some AChE on their surfaces.

195 we used a novel conditional knock-out line (ChAT-cre p75(in/in)) to assess the role of p75(NTR) in t

196 CP2 function from cholinergic neurons (MeCP2 ChAT KO), which recapitulated the cardiac rhythm abnorma

197 ) ACs in wild-type and ChAT transgenic mice (ChAT- tdTomato).

198 with maturation, subband "a" presented more ChAT but less AChE; in subband "d" this pattern was reve

199 keys in having a preponderance of multipolar ChAT-ir interneurons in the caudate nucleus and putamen,

200 by alpha7-, alpha3beta2-, and beta3-nAChRs, ChAT and VAChT pathways.

201 (ChR2) in Choline acetyltransferase neurons (ChAT(+)) or Arch in LIM-homeodomain transcription factor

202 tion or replacement of Smn in motor neurons (ChAT-Cre) significantly alters the functional output of

203 e produces parvalbumin(+) GABAergic neurons, ChAT(+) cholinergic neurons, and oligodendrocytes.

204 ve, spiking type I local interneurons had no ChAT-like immunoreactivity.

205 Ca(2+) -driven spikelets were absent, had no ChAT-like immunoreactivity.

206 In the cat, most nonoculomotor ChAT(+) cells were located in the supraoculomotor area a

207 In the monkey, most nonoculomotor ChAT(+) neurons were found within the EW.

208 About 20% of nonspiking ChAT-lir type IIa1 local interneurons were TKRP-lir.

209 the extracellular space and the activity of ChAT.

210 In a chimera of ChAT promoter-EGFP and mito-mCherry, EGFP efficiently tr

211 Convergence of ChAT-immunoreactive and GABA-immunoreactive transmedulla

212 - and P28-dark-reared retinas the density of ChAT-immunoreactive cells was higher in both the INL and

213 rabbit EW, by contrast, is nearly devoid of ChAT-immunopositive neurons.

214 ight deprivation increases the expression of ChAT, increasing the apparent density of cholinergic neu

215 Thus, haploinsufficiency of ChAT leads to an increased expression of CHT1.

216 , while having no effect on the incidence of ChAT expression.

217 ansmits reinforcement-related information of ChAT interneurons in the mouse neostriatal network.

218 fully prevented AbetaO-induced inhibition of ChAT.

219 We examined the interaction of ChAT and ChT activity using mice heterozygous for a null

220 GC layer, but changed the relative levels of ChAT expression by immunohistochemistry.

221 e was found to express the highest levels of ChAT, and that cell line was chosen for additional studi

222 no effect on the ERG or the localization of ChAT and nNOS immunoreactivities in either the control r

223 , but it significantly increases the loss of ChAT following nerve crush.

224 n: In PTD-treated rats a significant loss of ChAT-immunopositive cells was found only in the MS/DB, b

225 ant, nonredundant role in the maintenance of ChAT in intact adult motor neurons, the receptors become

226 tructural analysis showed that the number of ChAT(+) dendritic profiles per unit area of striatum was

227 tes, no significant changes in the number of ChAT-positive neurons in aged were found comparing to th

228 length and total dendritic segment number of ChAT-positive neurons was detected in both the LDT and P

229 E+ band, indicating the future IPL, pairs of ChAT+ /AChE- /Brn3a- cells appeared between E7/8.

230 Lastly, a small population of ChAT-expressing lymphocytes was identified in the spleen

231 esponsible for the resulting potentiation of ChAT activity.

232 The ratio of ChAT-positive projection neurons to total projection neu

233 rats, but slightly enhanced the recovery of ChAT immunostaining in young rats.

234 The crystal structure of ChAT reported here shows the enzyme divided into two dom

235 o rat choroid, and immunolabeling for NOS or ChAT was used to characterize their neurochemistry.

236 n-D(28k) hypothalamic but not parvalbumin or ChAT containing neurons in select monkey forebrain regio

237 o-localization of ERalpha and parvalbumin or ChAT was not seen in any of the areas of the monkey fore

238 Jurkat T cells overexpressing ChAT (JTChAT) decreased blood pressure when infused into

239 lls maintained their gene expression of p75, ChAT and ChT, while p75-negative (p75-) cells had a low

240 ve deletion of GRK2 in this cell population (ChAT(IRES-cre)Grk2(f/f) KO mice) exhibit reduced behavio

241 As predicted, ChAT-deficient embryos lack both spontaneous and nerve-e

242 fect the ability of microglial CM to promote ChAT activity, treatment of microglia with prostaglandin

243 e choline acetyltransferase (ChAT) promoter (ChAT-ChR2-EYFP) to dissect cholinergic circuit connectiv

244 on cells express the ACh-processing proteins ChAT and VAChT, and reducing their expression impairs le

245 lysis further revealed the dendrites of Q140 ChAT(+) interneurons were significantly fewer and shorte

246 We have generated antibodies that recognize ChAT or VAChT in a model organism, the nematode Caenorha

247 of viability that could account for reduced ChAT activity under these conditions.

248 ChT activity may compensate for the reduced ChAT activity in Chat+/- mice, contributing to the maint

249 88-dependent Toll-like receptor up-regulates ChAT in a transient manner.

250 ntrol, and P14- and P28-dark-reared retinas, ChAT-immunoreactivity showed similar patterns to those i

251 (icv) after AF64A injection increases septal ChAT and AChE activities.

252 lbindin interneurons were located and spared ChAT interneurons.

253 In all species, ChAT labeled a population of small diameter fibers givin

254 c inhibition and stimulation of subependymal ChAT(+) neurons in vivo indicated that they were necessa

255 During slow-wave activity (SWA), ChAT+ interneurons, and some PV+ and NOS+ interneurons,

256 Our studies demonstrate that ChAT-ChR2-EYFP mice show altered cholinergic tone that f

257 These results indicate that ChAT is uniquely essential for the patterning and format

258 uble-immunolabeling techniques revealed that ChAT and GABA were colocalized in axon terminals in the

259 Moreover, Western blotting showed that ChAT protein levels were significantly increased in the

260 Although blinded neuron counts showed that ChAT(+) perikarya were in normal abundance in Q140 mice,

261 ultured cholinergic neurons, suggesting that ChAT is fully inhibited in AbetaO-targeted neurons.

262 The ChAT staining intensity of the axotomized RDLN declined

263 CHT and the cholinergic locus containing the ChAT and VAChT genes.

264 spinal system activity was necessary for the ChAT and calbindin changes.

265 ing and holding the choline substrate in the ChAT active site.

266 induced by hypertonic NaCl was found in the ChAT cells of the MePO.

267 Finally, selective deletion of the ChAT gene from adult sympathetic neurons prevented the i

268 Approximately 30% of the ChAT immunoreactive terminals were also GABA immunoreact

269 Most of the ChAT/GABA immunoreactive terminals formed symmetrical sy

270 of overt double labeling indicated that the ChAT(+) and urocortin(+) cells are separate populations.

271 That these ChAT-positive varicosities represent presynaptic release

272 Ch production is the provision of choline to ChAT.

273 The projections to ChAT-negative (and weakly immunoreactive) cells in AM, V

274 With respect to ChAT immunoreactive postsynaptic profiles, approximately

275 No changes in total ChAT protein were seen.

276 fold serial dilution of in vitro-transcribed ChAT mRNA from 66 to 10(7) copies was discriminated by q

277 ning galanin and choline acetyl transferase (ChAT) and serotonin fibers with fos-immunoreactive (fos-

278 synthase (nNOS), choline acetyl transferase (ChAT), vasoactive intestinal polypeptide (VIP), calciton

279 ed CHT mRNA expression, while it upregulated ChAT/VAChT mRNA levels and ChAT activity.

280 We observed that nicotine upregulated ChAT and VAChT.

281 In this study we used ChAT-Cre;R26R-YFP mice to examine the development of cho

282 We observed a diffuse network of varicose ChAT-positive fibers associated with the nervus terminal

283 ver, all remaining PPNs and motoneurons were ChAT positive at 4 weeks postoperatively.

284 The choroidal SSN neurons were ChAT(+) and appeared largely to correspond to the NOS(+)

285 SSN neurons were ChAT(+), and a subset of them was found to contain NOS.

286 8% of the GABA immunoreactive terminals were ChAT immunoreactive.

287 described in previous studies and which were ChAT immunoreactive.

288 bly, early CST terminations were dense where ChAT interneurons later increased in numbers.

289 calbindin-expressing areas but did so where ChAT interneurons were located.

290 VAChT is found in synaptic regions, whereas ChAT appears to exist in two forms in neurons, a synapse

291 To identify the mechanism by which ChAT activity is increased, we sought to identify the bi

292 table in an embryonic day (E)4 retina, while ChAT appeared 1 day later in the very same cells; at thi

293 same proteins appear to be coexpressed with ChAT by cholinergic neurons in several motor and reticul

294 ritic terminals making synaptic contact with ChAT(+) dendrites per unit area of striatum.

295 erved relaxin-3-immunoreactive contacts with ChAT-, PV-, and glutamate decarboxylase-67-positive neur

296 Contrasting with ChAT+ interneurons, whose firing showed poor brain state

297 omoter-associated signatures discovered with ChAT indicate that complex chromatin signatures, made up

298 dies showed that CXCR4 was co-expressed with ChAT, a marker for cholinergic neurons, and with GAD C38

299 by lymphocytes has specific functions, with ChAT(+) B cells controlling the local recruitment of neu

300 ical TH neurons, the coexpression of TH with ChAT or VIP was examined throughout the neocortex at P16