ライフサイエンスコーパス: ChIP sequencing

1 A sequencing) with genome-wide CHD8 binding (ChIP sequencing).

2 gonucleotide content of a large sample (e.g. ChIP-sequencing).

3 ared occupancy of -15% of p53-bound genes in ChIP sequencing.

4 o far unknown DNA regions identified through ChIP-sequencing.

5 l by quantitative interaction proteomics and ChIP-sequencing.

6 munication during the mitochondrial UPR, via ChIP-sequencing.

7 e genome, which we show is now possible with ChIP-sequencing.

8 tained by merging results from ChIP-chip and ChIP-sequencing.

9 ChIP sequencing analyses displayed enrichment of ZBED6 b

10 its normal down-regulating cues, and NFATc1 ChIP-sequencing analyses reveal a marked enrichment of N

11 mmunoprecipitation-microchip (ChIP-chip) and ChIP-sequencing analyses.

12 Here, we performed SOX9 ChIP sequencing analysis and transcriptome profiling of

13 ChIP sequencing analysis revealed a novel GR binding sit

14 Genome-wide chromatin immunoprecipitation (ChIP) sequencing analysis identified Il2ra and Cd27 as d

15 In this report, ChIP-sequencing analysis in mouse preosteoblasts reveale

16 ChIP-sequencing analysis reveals p53 dissociates from pr

17 In this report, we used ChIP-sequencing analysis to confirm the presence of thes

18 Vdr gene locus in kidney and intestine using ChIP-sequencing analysis, revealing that only one of the

19 mosomal localization mapping of Brachyury by ChIP sequencing and ChIP-exonuclease revealed distinct s

20 Here, we used genome-wide ChIP sequencing and found that the DNA-bound form of cyc

21 lar biology realm remain unanswered, we used ChIP sequencing and loss-of-function strategies to defin

22 Using ChIP sequencing and RNA sequencing analysis, we determin

23 ed the genome-wide association of Lem2 using ChIP sequencing and we found that it binds to the centra

24 Through chromatin immunoprecipitation (ChIP) sequencing and microarray experiments, we further

25 me-wide binding of Tbrain orthologs by using ChIP-sequencing and associates these orthologs with puta

26 cription factor (TF) binding events from 187 ChIP-sequencing and ChIP-on-chip datasets in murine and

27 As assessed by ChIP-sequencing and RNA-sequencing analyses, we found th

28 egard to enhancer structure and contemporary ChIP-sequencing assays, whereby just a small fraction of

29 We use ChIP sequencing (ChIP-seq) and RNA sequencing (RNA-seq)

30 We used ChIP sequencing (ChIP-seq) to identify around 16,000 E2F

31 Here we use ChIP sequencing (ChIP-seq) to identify domains enriched

32 newly performed genomic data sets, including ChIP sequencing (ChIP-seq), genome-wide mRNA profiling,

33 Using ChIP sequencing (ChIP-seq), we compared the ERalpha prof

34 Our chromatin immunoprecipitation (ChIP) sequencing (ChIP-seq) analysis confirmed binding o

35 in vivo, and chromatin immunoprecipitation (ChIP) sequencing (ChIP-seq) approaches to demonstrate th

36 tion with the chromatin immunoprecipitation (ChIP) sequencing (ChIP-Seq) data shows that the domain b

37 ipitation (ChIP)-quantitative PCR (qPCR) and ChIP-sequencing (ChIP-seq) analyses indicated that Ikaro

38 Single-gene ChIP and genome-wide ChIP-sequencing (ChIP-seq) and RNA-seq studies extended

39 ChIP-sequencing (ChIP-Seq) data on other transcription f

40 Integration of a ChIP-sequencing (ChIP-Seq) data set for 10 key stem cell

41 s is detected by ChIP-on-chip (ChIP-chip) or ChIP-sequencing (ChIP-seq).

42 ng chromatin immunoprecipitations (ChIP) and ChIP sequencing (ChIPSeq) of fetal pancreas and islet ch

43 are available under subseries GSE81576; and ChIP sequencing data are available under subseries GSE81

44 lected 108 transcription-related factor (TF) ChIP sequencing data sets in ten murine cell types and c

45 Using ChIP-sequencing data and cell fractionation, we have com

46 For one gene, MuRF1, ChIP-sequencing data identified the location of Bcl-3 an

47 , comparison with three other published EBF1 ChIP-sequencing data sets in B-cells reveals both gene-

48 l for efficiently analyzing large amounts of ChIP-sequencing data to study dynamic changes of gene re

49 By means of bioinformatics analysis of ChIP-sequencing data we found Bcl-3 to be directing tran

50 Using ENCODE LCL transcription factor ChIP-sequencing data, EBNA3C sites coincided (+/-250 bp)

51 ected eleven pairs (H3K4me3 and H3K27me3) of ChIP sequencing datasets in human ES cells and eight pai

52 onent analysis (dPCA) for analyzing multiple ChIP-sequencing datasets to identify differential protei

53 mass spectrometry, X-ray crystallography and ChIP sequencing demonstrate that PHF13 binds chromatin i

54 nd Delta exons 9-14, as well as from a PRDM5 ChIP-sequencing experiment.

55 polymerase II chromatin immunoprecipitation (ChIP)-sequencing experiments from 35 different murine an

56 we conducted chromatin immunoprecipitation (ChIP)-sequencing experiments in lymphoid cells treated w

57 Gene expression and ChIP-Sequencing experiments show that high levels of Myc

58 ChIP-sequencing experiments using an anti-O-GlcNAc antib

59 P, bZIP, EGR, E-Box and NF-kappaB motifs, by ChIP sequencing for a subset of motif corresponding tran

60 We employed ChIP-sequencing for H3K4me3 to examine effects of early

61 xpression, and we use ChIP, validated by p63-Chip sequencing genomewide profiling analysis, and lucif

62 EBNA3C ChIP-sequencing identified >13,000 EBNA3C sites in LCL D

63 Analyzing beta-catenin ChIP sequencing in human cells, we found the 11-bp NREs

64 ential role of GATA3, we performed extensive ChIP-sequencing in unstimulated breast cancer cells and

65 Here we use ChIP-sequencing, integrated with microarray analysis, to

66 m of SD70 that permits its application for a ChIP-sequencing-like approach, referred to as "Chem-seq,

67 decreased Atf5 transcript, and primary islet ChIP-sequencing localized PDX1 to the Atf5 promoter, imp

68 ChIP sequencing of GR showed that corticosterone treatme

69 ChIP sequencing of the major players NUT, ZNF532, BRD4,

70 ChIP sequencing of TNFalpha-stimulated H9c2 cells reveal

71 atin immunoprecipitation (ChIP) analysis and ChIP-sequencing of TP63 binding in differentiated kerati

72 Centromere locations were determined by ChIP-sequencing of two key centromere proteins, Cse4 and

73 ChIP-sequencing provides the first molecular explanation

74 equencing time over any previously published chip sequencing result, with comparable read length and

75 cted by loss of EBF1 in adipocytes, although ChIP-sequencing results suggest that these actions are i

76 Finally, we have combined the ChIP-sequencing results with gene expression microarray

77 ChIP-sequencing revealed that by 24 h after expression,

78 ChIP-sequencing shows that YY1 predominantly binds to pr

79 Genome-wide ChIP-sequencing studies indicate that TAF7L binds to pro

80 We used ChIP sequencing to define genomewide TEAD4 target genes

81 Here, we used ChIP sequencing to identify direct targets of FOXO.

82 Using ChIP sequencing to map sites of GRHL2 binding in the bas

83 rmined cellular MYC levels and used RNA- and ChIP-sequencing to correlate promoter occupancy with gen

84 ChIP sequencing was performed on pro-B cells, revealing

85 Using ChIP-sequencing we found that Bcl-3, an NF-kB transcript

86 EMSA) or genome-wide assays (RNA-sequencing, ChIP-sequencing), we have assembled a comprehensive regu

87 Using shotgun proteomics and ChIP sequencing, we demonstrate that leukodystrophy-caus

88 By combining ChIP sequencing with microarray-based gene profiling, we