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1 we evaluated XRCC1-deficient and -proficient Chinese hamster and human cancer cells for synthetic let
2 ray repair complementing defective repair in Chinese hamster cells 3 (XRCC3), 4 CpGs) birth weight.
5 g in potent inhibition against FR-expressing Chinese hamster cells and human KB tumor cells in cultur
6 in binding in G1-synchronized populations of Chinese hamster cells harboring amplified copies of the
7 t a 2.4-Gb draft genome sequence of a female Chinese hamster, Cricetulus griseus, harboring 24,044 ge
10 type 2, early onset- (BRCA2-) deficient V79 Chinese hamster lung fibroblast cell line derivative (VC
14 ized LDL and acLDL to CR1 on CR1-transfected Chinese Hamster Ovarian cells (CHO-CR1) was tested by fl
17 has been demonstrated from both E. coli and Chinese hamster ovaries (CHO) cell expression platforms;
19 for inhibition of proliferation in isogenic Chinese hamster ovary (CHO) and HeLa cells expressing PC
20 bit the import of spermidine in DFMO-treated Chinese hamster ovary (CHO) and L3.6pl human pancreatic
22 city of the nine NOCs was quantified using a Chinese hamster ovary (CHO) cell assay, and the descendi
25 e human amyloid precursor protein (APP) in a Chinese hamster ovary (CHO) cell line by cleaving APP at
26 s/mL) perfusion culture of an IgG1-producing Chinese hamster ovary (CHO) cell line for 18-25 days.
28 d-type and glycosaminoglycan (GAG)-deficient Chinese hamster ovary (CHO) cell lines and soluble GAGs,
29 al binding and transduction assays on mutant Chinese hamster ovary (CHO) cell lines defective in vari
30 etrotransposition pathway (EN(i)) in certain Chinese hamster ovary (CHO) cell lines that are defectiv
31 vity were evaluated as expressed in a set of Chinese hamster ovary (CHO) cell lines under conditions
32 , we engineered and characterized a panel of Chinese hamster ovary (CHO) cell lines with inducible tr
35 he heavy chain and the light chain (LC) of a Chinese hamster ovary (CHO) cell-expressed monoclonal an
38 om Schistosoma mansoni (Platyhelminthes), in Chinese hamster ovary (CHO) cells and use fluorescence-b
39 etection platform to image endogenous H2S in Chinese hamster ovary (CHO) cells and use the developed
40 fractions, here we show that, when parental Chinese hamster ovary (CHO) cells are briefly exposed to
43 ckdown with small interfering RNA (siRNA) in Chinese hamster ovary (CHO) cells augments M3-MR signali
44 o generate any current when transfected into Chinese hamster ovary (CHO) cells but, surprisingly, exe
45 The McKbac virus entered efficiently into Chinese hamster ovary (CHO) cells constitutively express
47 sibility of a nuclear-translocation assay in Chinese hamster ovary (CHO) cells expressing an NFkappaB
48 the binding of 5,6-EET-EA to membranes from Chinese hamster ovary (CHO) cells expressing either reco
49 shift in neonatal mouse cardiac myocytes or Chinese hamster ovary (CHO) cells expressing the mouse o
50 o demonstrate that somatostatin treatment of Chinese hamster ovary (CHO) cells expressing the wild ty
52 ion of PS on the plasma membrane of isolated Chinese Hamster Ovary (CHO) cells following exposure to
53 hich KCNQ subunits are targeted by AKAP79 in Chinese hamster ovary (CHO) cells heterologously express
54 activation of human DORs stably expressed in Chinese hamster ovary (CHO) cells increased AMPK activit
55 this study, we evaluated the genotoxicity to Chinese hamster ovary (CHO) cells induced by municipal s
56 how that overexpression of TREK-1 in NPE and Chinese hamster ovary (CHO) cells leads to a significant
58 e of exporting the diamine putrescine in the Chinese hamster ovary (CHO) cells selected for resistanc
60 n embryonic kidney (HEK) 293, C6 glioma, and Chinese hamster ovary (CHO) cells stably expressing this
63 ition, compounds 3-6 inhibited the growth of Chinese hamster ovary (CHO) cells that expressed FRs but
64 beta-GlcNAc on the complex N-glycans of Lec8 Chinese hamster ovary (CHO) cells that lack UDP-Gal tran
65 ronic cytotoxicity and acute genotoxicity in Chinese hamster ovary (CHO) cells to compare the toxicit
66 tudy mechanisms, we engineered alphaIIbbeta3 Chinese hamster ovary (CHO) cells to conditionally expre
68 an amphotericin B loss-of-function screen in Chinese hamster ovary (CHO) cells using insertional muta
69 ivator, on hERG channels stably expressed in Chinese hamster ovary (CHO) cells using the patch-clamp
71 LDL increased cytosolic G protein by 350% in Chinese hamster ovary (CHO) cells with genetically induc
72 accomplish glycomic survey of bioengineered Chinese Hamster Ovary (CHO) cells with knock-in/out enzy
76 and duration of reporter gene expression in Chinese Hamster Ovary (CHO) cells, Human Immortalized My
77 ter) obtained from red blood cells (RBC), or Chinese hamster ovary (CHO) cells, were immobilized on t
78 re assayed as luciferase reporter fusions in Chinese hamster ovary (CHO) cells, where the putative ci
79 re caused by EGFR expression, we transfected Chinese hamster ovary (CHO) cells, which lack EGFR expre
80 Here we describe a procedure to vesiculate Chinese hamster ovary (CHO) cells, widely used for the e
102 These analogues inhibited proliferation of Chinese hamster ovary (CHO) sublines expressing folate r
103 alysis was performed on mouse myeloma SP2/0, Chinese hamster ovary (CHO), and human embryonic kidney
104 the first N-glycosylation gene, DPAGT1, from Chinese hamster ovary (CHO), Madin-Darby canine kidney (
106 lecules, and in null versus AQP4-transfected Chinese hamster ovary (CHO)-K1 cells and Fisher rat thyr
109 RNA-based pathway inhibition in recombinant Chinese hamster ovary (CHO)-S1P2 cells as well as human
112 AIRS enzyme by analysis of mutations in the Chinese hamster ovary (CHO-K1) cell that require purines
114 s (c13C6, h-13F6, and c6D8) were produced in Chinese hamster ovary and in whole plant (Nicotiana bent
115 CR3 affects the generation of cAMP, we used Chinese hamster ovary and K562 cells transfected to expr
117 in relation to endogenous Notch receptors of Chinese hamster ovary and murine embryonic stem (ES) cel
118 at were and were not associated with CSBS in Chinese Hamster Ovary and T84 cells and generated a zebr
119 was assayed in vitro by the transduction of Chinese hamster ovary and trabecular meshwork cells.
120 on the stabilities of Escherichia coli- and Chinese hamster ovary cell (CHO)-derived IgG1 Fc high-or
122 1 antibody, generated during production in a Chinese hamster ovary cell culture, was observed in the
125 in a human retinal cell line (ARPE-19) and a Chinese hamster ovary cell line (CHO-K1) to study the fu
126 ath, we generated and characterized a mutant Chinese hamster ovary cell line that is resistant to pal
128 his mutated form of XPF in the XPF-deficient Chinese hamster ovary cell line, UV41, only partially re
129 of human endothelial kidney 293, HepG2, and Chinese hamster ovary cell lines decreases cellular LDL
130 at transduction occurs efficiently in mutant Chinese hamster ovary cell lines deficient in glycosamin
132 promoter trap mutagenesis to generate mutant Chinese hamster ovary cell lines resistant to lipotoxic
135 p91(phox) and p22(phox), we demonstrate in a Chinese hamster ovary cell model system and in RAW 264.7
139 ished potencies based on the comet assay for Chinese hamster ovary cells (assesses the level of DNA s
140 liquids (ILs) on zebrafish (Danio rerio) and Chinese hamster ovary cells (CHO) was investigated with
143 of the three homologs that are expressed in Chinese hamster ovary cells (DPY19L1, DPY19L3, and DPY19
144 fragments [Fab, F(ab')(2)] were expressed in Chinese hamster ovary cells and evaluated in vitro and i
145 acromolecular channel complex in transfected Chinese hamster ovary cells and found a dominant-negativ
146 position tool, and we tested the adhesion of Chinese Hamster Ovary cells and Human Embrionic Kidney c
148 r of calcineurin, increased ASIC currents in Chinese hamster ovary cells and in cortical neurons, sug
149 P9 significantly reduced Abeta generation in Chinese hamster ovary cells and in primary neurons, demo
151 of extracellular signal-regulated kinase in Chinese hamster ovary cells and permits chemokine and pr
152 resistant to c-Cbl-mediated degradation, in Chinese hamster ovary cells and the UMSCC11B human head
155 clamp recordings from channels expressed in Chinese Hamster Ovary Cells at different temperatures (3
156 iggered gating of BKCa channels expressed in Chinese hamster ovary cells at distinct membrane potenti
158 oprecipitated with the I(Ks) channel only in Chinese hamster ovary cells co-expressing AKAP-9, and PD
159 When ADAP was heterologously expressed in Chinese hamster ovary cells co-expressing alphaIIbbeta3,
160 adipose tissue, and expression of GPIHBP1 in Chinese hamster ovary cells confers upon those cells the
161 assays that use human receptors expressed in Chinese hamster ovary cells demonstrate that NDD-713 and
162 epatocytes are induced on cocultivation with Chinese hamster ovary cells engineered to express T-cadh
163 lite trends from a bioreactor cultivation of Chinese hamster ovary cells expressing a recombinant ant
167 was evaluated before and after adsorption to Chinese hamster ovary cells expressing human TSHRs using
173 r Abeta binding and uptake were confirmed in Chinese hamster ovary cells genetically deficient in HSP
174 ibodies as well as significantly enhanced in Chinese hamster ovary cells genetically modified to expr
175 We show that fucosylation-deficient Lec13 Chinese hamster ovary cells have wild type levels of Pof
176 well as in human embryonic kidney cells and Chinese hamster ovary cells heterologously expressing hu
177 demonstrated that ExsE was translocated into Chinese hamster ovary cells in a T3SS-dependent manner.
178 eta3 enhanced IGF-1-induced proliferation of Chinese hamster ovary cells in serum-free conditions (in
179 ultrasound on the intracellular [Ca(2+)] of Chinese hamster ovary cells in the presence of albumin-e
180 adherence of E. histolytica trophozoites to Chinese hamster ovary cells in vitro (P, <0.001 for each
181 t transient expression of kindlin-1 or -2 in Chinese hamster ovary cells inhibits the activation of e
182 ant human TWEAK binding to CD163-transfected Chinese hamster ovary cells is inhibited by the presence
183 etabolism, using RNA interference to deplete Chinese hamster ovary cells of NPC1 alone or in combinat
184 -linked fluorophore to these compartments in Chinese hamster ovary cells or Jurkat lymphocytes, membr
185 n wild-type and D1275N channels expressed in Chinese hamster ovary cells or tsA201 cells in the absen
187 ses formed between primary T lymphocytes and Chinese hamster ovary cells presenting major histocompat
188 TGF-mediated control of beta-AR sensitivity, Chinese hamster ovary cells pretreated with rec-hCTGF di
191 b/IIIa (integrin alphaIIbbeta3) expressed on Chinese hamster ovary cells promoted melanoma cell adhes
192 ene promoter; overexpression of HNF4alpha in Chinese hamster ovary cells re-established transcription
193 mpetition assays at membrane preparations of Chinese hamster ovary cells recombinantly expressing the
194 ivated Teffs with Sn(+) macrophages or Sn(+) Chinese hamster ovary cells resulted in increased cell d
195 at the human beta1-adrenoceptor expressed in Chinese hamster ovary cells revealed negative cooperativ
196 Galpha(q) and Galpha(i) pathways in vitro on Chinese hamster ovary cells stably expressing FFA2.
197 substrate for PKC, and this was confirmed in Chinese hamster ovary cells stably expressing full-lengt
198 50) = 0.2 microM) and whole-cell currents in Chinese hamster ovary cells stably expressing heteromult
199 or binding of fluorescently labeled Abeta to Chinese hamster ovary cells stably expressing human CD36
200 stimulated cAMP accumulation was measured in Chinese hamster ovary cells stably expressing the human
201 agonist 5'-(N-ethylcarboxamido)adenosine in Chinese hamster ovary cells stably transfected with the
202 ophosphate-, and Rap1-independent pathway in Chinese hamster ovary cells stably transfected with the
203 a membrane of intact rabbit erythrocytes and Chinese hamster ovary cells that can be explained by the
204 ection technique to generate a population of Chinese hamster ovary cells that display a global defici
205 horage-independent conditions in transformed Chinese hamster ovary cells that express alphavbeta3 (be
207 ) and selective inhibitory activities toward Chinese hamster ovary cells that expressed FRalpha or FR
208 ulated release of arachidonic acid (AA) from Chinese hamster ovary cells that expressed M(3) muscarin
210 to block the binding of ligands to pgsA-745 Chinese hamster ovary cells that overexpress GPIHBP1.
212 Expression of wild-type hCTR1 in mutant Chinese hamster ovary cells that were unable to initiate
214 pressed mutant forms of full-length JAM-A in Chinese hamster ovary cells to assess reovirus binding a
215 ession of TREM-2-DAP12 enables nonphagocytic Chinese hamster ovary cells to internalize bacteria.
216 lations to bind and/or infect HL-60 cells or Chinese hamster ovary cells transfected to express PSGL-
217 ndent decrease in the luciferase activity in Chinese Hamster Ovary cells transfected with CACNA1C and
218 t using human peripheral blood monocytes and Chinese hamster ovary cells transfected with CD14 cells
219 tent stem cell-derived cardiomyocytes and in Chinese hamster ovary cells transfected with SCN5A, enco
222 to inhibit MO-evoked calcium accumulation in Chinese hamster ovary cells transfected with TRPV1 and T
223 A or fibronectin bound to DC and Mphi and to Chinese hamster ovary cells transfected with VLA-5.
224 Forster resonance energy transfer (FRET) on Chinese hamster ovary cells under total internal refecti
225 nt protein-tagged KCNQ subunits expressed in Chinese hamster ovary cells under total internal reflect
226 ools to analyze proteins in situ in cultured Chinese hamster ovary cells using fluorescence recovery
227 upernatant following transient expression in Chinese hamster ovary cells were analyzed by immunoblot
229 leasing intracellular proteins from adherent Chinese hamster ovary cells while preserving the cell vi
230 imulated 2D diffusion conditions and in live Chinese hamster ovary cells with a GFP-tagged transmembr
231 ropermeabilization of suspended and adherent Chinese hamster ovary cells with green DNA dye SYTOX is
234 tive to E2F1-induced apoptosis compared with Chinese hamster ovary cells with wild-type XRCC1 (AA8).
235 nescence and storage lesion, was assessed in Chinese hamster ovary cells with/without functional GPIb
236 CaK in fusion with enhanced GFP in mammalian Chinese hamster ovary cells' plasma membrane gave rise t
237 ukaryotic cells (up to approximately 36% for Chinese hamster ovary cells) and bacterial cells (up to
238 efficiency for transfection (up to ~71% for Chinese hamster ovary cells) and permeabilization sugges
239 ore, using alphaIIbbeta3 integrin-expressing Chinese hamster ovary cells, a well described model syst
240 cular, in both Xenopus oocytes and mammalian Chinese hamster ovary cells, AaTXKbeta(2)(-)(6)(4), but
241 n sulfate isolated from mutant and wild-type Chinese hamster ovary cells, and select tissues from mut
242 ly localized to the endoplasmic reticulum in Chinese hamster ovary cells, and this intracellular loca
244 of various cell types in culture, including Chinese hamster ovary cells, chicken DF1 fibroblasts, pr
245 etylglucosaminyltransferase I-deficient Lec1 Chinese hamster ovary cells, indicating that N-glycosyla
246 nnels, which are heterologously expressed in Chinese hamster ovary cells, into the depolarizing direc
249 Ibbeta3 integrins by talin head fragments in Chinese hamster ovary cells, nor do I observe affinity i
250 We expressed mutant forms of GPIHBP1 in Chinese hamster ovary cells, rat and human endothelial c
252 capability of our setup on fixed and living Chinese hamster ovary cells, showing the cytoskeleton dy
253 increased plasmalogen synthesis in wild-type Chinese hamster ovary cells, strongly suggesting that Fa
254 tes and its binding to the LDLR expressed on Chinese hamster ovary cells, suggesting a direct interac
255 educes ASIC currents in cortical neurons and Chinese hamster ovary cells, suggesting a role of AKAP15
256 nt Kv3.4 channel heterologously expressed in Chinese hamster ovary cells, supporting our findings in
257 Th2 receptor (1.14 +/- 0.44 nM) expressed in Chinese hamster ovary cells, the binding being reversibl
258 ocell" particles are taken up efficiently by Chinese hamster ovary cells, where, due to a reduced pH
259 -1 K(+) channels heterologously expressed in Chinese hamster ovary cells, which are silent in physiol
282 ans to modify protein glycosylation, we used Chinese hamster ovary ldl-D cells defective in UDP-Gal/U
283 ion and cell retention was confirmed using a Chinese hamster ovary model, in which cellular sphingoli
284 hanced binding to FcgammaRIIIa compared with Chinese hamster ovary or wild-type plant-derived 2G12.
286 which endogenously expresses DRD2) and CHO (Chinese hamster ovary) cell lines, decreasing luciferase
287 e the uptake of silver nanoparticles by CHO (Chinese hamster ovary) cells and their subsequent fate a
288 0), 1A(1)) allele variants expressed by CHO (Chinese hamster ovary) mammalian cells to study their ef
289 ssessed in the arrestin recruitment assay in Chinese hamster ovary-K(1) cells expressing the long iso
292 g to platelets from P1 but absent binding to Chinese hamster ovary-K1 cells expressing variant D304N
293 Ca(2+) upregulation; however, in HEK293T or Chinese hamster ovary-K1 cells overexpressing M3R, piloc
294 single-point mutations were transfected into Chinese hamster ovary-K1 cells, and affinity and functio
297 screening assay using the GeneBridge4 human/Chinese hamster radiation hybrid panel and found to be t
298 rget cells (Salmonella typhimurium TA100 and Chinese hamster V79) of standard mutagenicity tests trea
299 region of the genome, in two cell lines, the Chinese hamster V79-derived G12 and G10 cells, respectiv
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