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1 l as essential for endometrial repair during Chlamydia infection.
2 . trachomatis organism load in human genital chlamydia infection.
3 gulated over the course of the intracellular Chlamydia infection.
4 roduction, revealing that retromer restricts Chlamydia infection.
5 fficacy of azithromycin for the treatment of chlamydia infection.
6 ily (7 days) for the treatment of urogenital chlamydia infection.
7 tor-6alpha (ATF6alpha)-were activated during Chlamydia infection.
8 is heterogeneity in the risk of PID after a chlamydia infection.
9 azithromycin for the treatment of urogenital chlamydia infection.
10 null clones restored full susceptibility to Chlamydia infection.
11 , respectively, as important in facilitating Chlamydia infection.
12 molecule that plays a role in resistance to Chlamydia infection.
13 Golgi apparatus-independent pathway during a Chlamydia infection.
14 d pregnancy outcome in women with persistent chlamydia infection.
15 the incomplete protection provided by prior Chlamydia infection.
16 , and mediators and may reduce pregnancy and chlamydia infection.
17 wer regions of the GT respond differently to Chlamydia infection.
18 sceptibility and immune responses to genital Chlamydia infection.
19 endocarditis and chronicity is a hallmark of Chlamydia infection.
20 on and thereby shape macrophage responses to Chlamydia infections.
21 ng inflammation and adaptive immunity during Chlamydia infections.
22 thological scarring seen as a consequence of Chlamydia infections.
23 participate in the immunopathology seen with Chlamydia infections.
24 innate and adaptive immune responses during Chlamydia infections.
25 nt for the ability of mutant mice to resolve Chlamydia infections.
26 and DQB1*06) to be associated with recurrent Chlamydia infection (adjusted relative odds [RO], >2.0;
27 doxycycline for the treatment of urogenital chlamydia infection among adolescents in youth correctio
29 mmatory mediators by epithelial cells during Chlamydia infection and highlight the contribution of TL
31 quirement for MyD88 in CD4(+) T cells during Chlamydia infection and indicate that the importance of
33 ncy, and tubal factor infertility) following chlamydia infection and repeat infection hampers the des
35 that being African American, having previous chlamydia infection, and having less than a high-school
36 al epithelial cells released IL-1alpha after Chlamydia infection, and increased secretion of the proi
37 both groups, treated baseline gonorrhoea and chlamydia infections, and assessed 9-month gonorrhoea an
38 me has been implicated in the progression of Chlamydia infections, and with chlamydial infections at
39 s estimates for clearance rates of untreated chlamydia infections are important for understanding chl
40 er, these results suggest that P2X7R affects Chlamydia infection by directly inhibiting infection in
41 tested the requirement for these proteins in Chlamydia infection by microinjecting anti-Pls1 and anti
43 did not affect T cell-mediated clearance of Chlamydia infection, consistent with a B cell-specific r
44 endent designs that a concurrent Candida and Chlamydia infection could not accelerate or modulate the
46 g all sexually active adolescent females for chlamydia infection every 6 months, regardless of sympto
48 D8+ T cells, the response of CD8+ T cells to Chlamydia infection has been explored in a number of stu
51 herosclerosis suggests an etiologic role for Chlamydia infection in the development of coronary ather
54 ot reduce the rate of new HIV, gonorrhea, or chlamydia infection in this group of sex workers who use
58 volved in humoral protection against genital chlamydia infection is crucial to development of an effe
59 that the robust type 2 immunity elicited by Chlamydia infection of human genital tissue may analogou
60 genes that were defectively regulated during Chlamydia infection of the TLR3-deficient OE cells, and
62 um is commonly used as a model for ascending Chlamydia infections of the human female genital tract.
63 d cervical cancer may be due to an effect of chlamydia infection on persistence of high-risk HPV.
64 These findings suggest that the effect of a Chlamydia infection on trophoblast secretion of chemokin
65 demonstrated that interleukin-8 induction by Chlamydia infection or PGE2 treatment was dependent on e
69 thelial (OE) cells that is stimulated during Chlamydia infection, resulting in the synthesis of beta
70 transcripts were compared among subgroups of chlamydia infection status and ocular disease presentati
73 g directly observed treatment for urogenital chlamydia infection, the efficacy of azithromycin was 97
74 is no obvious dsRNA molecule associated with Chlamydia infection, the requirement for TLR3 in Chlamyd
76 ydia replication and restored the ability of Chlamydia infection to induce IL-6, CXCL10, and CCL5 exp
77 udies that showed extant upper genital tract Chlamydia infection was associated with increased co-exp
79 uently suffer from ocular diseases caused by Chlamydia infection, we also examined the eye microbiome
80 migrate to the genital tract in response to Chlamydia infection, we generated retrogenic mice that e
81 ow CD4 count (<350 cells/mm(3)) and previous chlamydia infection were associated with an increased ri
82 etween condom use and incident gonorrhea and chlamydia infection were compared between case-crossover
83 niasis, bacterial vaginosis, gonorrhoea, and chlamydia infection were significantly lower in the inte
84 metrial leukocyte (constitutively and during Chlamydia infection), whereas studies with eosinophil-de
85 type had elevated IL-10 concentrations after Chlamydia infection, which may reflect involvement of a
86 ted endocervical specimens for gonorrhea and chlamydia infection with DNA probes, tested for HIV infe
87 IFN-gamma-/- mice have in the resolution of Chlamydia infection, yet IFN-gamma production by CTL is
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