ライフサイエンスコーパス: Chlamydia muridarum

1 did so following intravaginal infection with Chlamydia muridarum.

2 ion with the obligate intracellular pathogen Chlamydia muridarum.

3 growth of the closely related mouse pathogen Chlamydia muridarum.

4 7BL/6 mice were infected intravaginally with Chlamydia muridarum.

5 in the genital tracts of mice infected with Chlamydia muridarum, a model for investigating the human

6 Chlamydia muridarum, a model pathogen for investigating

7 the tc0668 gene of serial in vitro-passaged Chlamydia muridarum, a murine model of human urogenital

8 The native plasmid of both Chlamydia muridarum and Chlamydia trachomatis has been s

9 Chlamydia muridarum and Chlamydia trachomatis mouse mode

10 munity of hysterectomized mice infected with Chlamydia muridarum and Chlamydia trachomatis to determi

11 h CD4 T cells that respond to a common Ag in Chlamydia muridarum and Chlamydia trachomatis Using an a

12 tended this analysis to DHFR originated from Chlamydia muridarum and Listeria grayi We found that the

13 ing, are recruited to Chlamydia trachomatis, Chlamydia muridarum, and Chlamydia pneumoniae inclusions

14 e peritoneal macrophages were performed with Chlamydia muridarum, and the expression of inflammatory

15 ted with attenuated plasmid-cured strains of Chlamydia muridarum are protected from oviduct pathology

16 otective immune responses against intranasal Chlamydia muridarum challenge in 1-day-old C57BL/6 and B

17 and CPAF-mediated responses against genital Chlamydia muridarum challenge.

18 o produced during infection with the species Chlamydia muridarum, Chlamydia psittaci, and Chlamydia c

19 each contains an ortholog of Tarp, although Chlamydia muridarum, Chlamydophila caviae, and Chlamydop

20 t mice exhibit suboptimal late phase vaginal Chlamydia muridarum clearance, greater dissemination, an

21 n of mice with a plasmid-deficient strain of Chlamydia muridarum, CM3.1, does not induce the developm

22 , 27 of 40 mice intravaginally infected with Chlamydia muridarum developed visible hydrosalpinges in

23 loped the surgical methodology of depositing Chlamydia muridarum directly on the endocervix.

24 The cryptic plasmid is essential for Chlamydia muridarum dissemination from the genital tract

25 nfected C57BL/6 mice with two populations of Chlamydia muridarum, each comprised of multiple genetic

26 Plasmid-free Chlamydia trachomatis and Chlamydia muridarum fail to induce severe pathology.

27 ckout (KO) mice exhibit delayed clearance of Chlamydia muridarum genital infection compared to wild-t

28 ction, we examined the course and outcome of Chlamydia muridarum genital infection in mice geneticall

29 the development of oviduct pathology during Chlamydia muridarum genital infection in the mouse model

30 Using the Chlamydia muridarum genital infection model of mice, whi

31 tro coculturing system and a murine model of Chlamydia muridarum genital tract infection.

32 re necessary and sufficient to clear primary Chlamydia muridarum genital tract infections in the mous

33 cells are critical for clearing experimental Chlamydia muridarum genital tract infections.

34 cells are necessary and sufficient to clear Chlamydia muridarum genital tract infections.

35 Although modern Chlamydia muridarum has been passaged for decades, there

36 The mouse chlamydial pathogen Chlamydia muridarum has been used as a model organism fo

37 etrimental to the host during infection with Chlamydia muridarum in both mouse lung and female genita

38 Intravaginal infection with Chlamydia muridarum in mice can ascend to the upper geni

39 eloped model of intracervical infection with Chlamydia muridarum in the mouse to elicit a relatively

40 Infection with Chlamydia muridarum in the mouse urogenital tract can in

41 udy, we examined the replicative capacity of Chlamydia muridarum in the RAW 264.7 murine macrophage c

42 ugh elicited mouse macrophages infected with Chlamydia muridarum in vitro secrete minimal IL-1beta, i

43 with plasmid-competent but not plasmid-free Chlamydia muridarum induces hydrosalpinx in mouse upper

44 ed the role of CD8(+) T cells during genital Chlamydia muridarum infection and oviduct sequelae.

45 le clearance of primary or secondary genital Chlamydia muridarum infection but significantly reduced

46 sess the protective immunity against genital Chlamydia muridarum infection in BALB/c mice.

47 Hydrosalpinx induction in mice by Chlamydia muridarum infection, a model that has been use

48 dilation detected microscopically following Chlamydia muridarum infection.

49 ins Nod1 and Nod2 in epithelial responses to Chlamydia muridarum infection.

50 on with the obligate intracellular bacterium Chlamydia muridarum is commonly used as a model for asce

51 Female mice were first vaccinated with Chlamydia muridarum major outer membrane protein (MOMP)

52 Recent studies have shown immunization with Chlamydia muridarum major outer membrane protein (MOMP)

53 Using the Chlamydia muridarum model of genital infection, we demon

54 In contrast to L2, the mouse pathogen Chlamydia muridarum (MoPn) was consistently inhibited by

55 MOMP protein from the mouse-specific species Chlamydia muridarum (MoPn-MOMP or mMOMP).

56 Studies utilizing the Chlamydia muridarum mouse model have shown that CD4 T ce

57 a new vaccine, mice were immunized with the Chlamydia muridarum native major outer membrane protein

58 pected, oviduct epithelial cells infected by Chlamydia muridarum produced a broad spectrum of chemoki

59 of Th1 immunity and neutrophil influx during Chlamydia muridarum pulmonary infection, but its role du

60 eviously demonstrated that plasmid-deficient Chlamydia muridarum retains the ability to infect the mu

61 anuloma venereum (LGV) and the murine strain Chlamydia muridarum share 99% of their gene content.

62 show that PVs formed by the rodent pathogen Chlamydia muridarum, so-called inclusions, remain free o

63 We have recently shown that Chlamydia muridarum spreads from the genital tract to th

64 We have previously shown that wild-type Chlamydia muridarum spreads to and establishes stable co

65 rs D (UW-3/Cx), E (Bour), or F (IC-Cal-3) or Chlamydia muridarum strain Nigg II using CpG-1826 and Mo

66 the RTR is much more similar to orthologs in Chlamydia muridarum than those in the phylogenetically c

67 kout mice and T cell depletion studies using Chlamydia muridarum that MHC class II and CD4 T cells ar

68 In the murine model of genital disease with Chlamydia muridarum, TLR2 plays a role in both early pro

69 vated signaling pathways, was evaluated in a Chlamydia muridarum urogenital tract infection model.

70 d expression of matrix metalloproteinases in Chlamydia muridarum urogenital tract infection of female

71 ucleotide polymorphisms were identified in a Chlamydia muridarum variant resistant to benzylidene acy

72 Chlamydia muridarum, which naturally infects mice, can i

73 eficient mice would affect host responses to Chlamydia muridarum within the reproductive tract.