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1 ion with the obligate intracellular bacteria Chlamydia trachomatis.
2 -to-culture, obligate intracellular pathogen Chlamydia trachomatis.
3 re compared to Amplicor for detecting ocular Chlamydia trachomatis.
4 ysis of 52 geographically diverse strains of Chlamydia trachomatis.
5 ellular pathogens Listeria monocytogenes and Chlamydia trachomatis.
6 he obligate intracellular bacterial pathogen Chlamydia trachomatis.
7 uitous in many chlamydial species, including Chlamydia trachomatis.
8 lls for the obligate intracellular bacterium Chlamydia trachomatis.
9 fected with a single cervical inoculation of Chlamydia trachomatis.
10 is often found in the absence of detectable Chlamydia trachomatis.
11 munofibrogenic disease process, initiated by Chlamydia trachomatis.
12 xual transmission is probably lower than for Chlamydia trachomatis.
13 eded to prevent the oculogenital diseases of Chlamydia trachomatis.
14 members in the sexually transmitted pathogen Chlamydia trachomatis.
15 nesis of the obligate intracellular bacteria Chlamydia trachomatis.
16 were screened for Neisseria gonorrhoeae and Chlamydia trachomatis.
17 a model for investigating the human pathogen Chlamydia trachomatis.
18 ant role of antibodies in protection against Chlamydia trachomatis.
19 is triggered by diverse bacteria, including Chlamydia trachomatis, a frequent intracellular parasite
23 acid amplification tests (NAATs) that detect Chlamydia trachomatis AC2 also detects Neisseria gonorrh
24 ind that C. muridarum and the human pathogen Chlamydia trachomatis activate not only NLRP3 but also A
25 lesion, and changes in sexual behaviors and Chlamydia trachomatis, an infection with similar epidemi
26 is produced by chronic ocular infection with Chlamydia trachomatis, an obligate intracellular bacteri
27 (Versant CT/GC assay, where "CT" represents Chlamydia trachomatis and "GC" represents Neisseria gono
28 RealTime CT/NG assay (where "CT" stands for Chlamydia trachomatis and "NG" stands for Neisseria gono
29 iable analysis were vaccine status, positive Chlamydia trachomatis and >/=4 partners in the preceding
31 reviewed recently, but the detailed roles of Chlamydia trachomatis and C. pneumoniae in induction of
33 udy was to assess the prevalence of synovial Chlamydia trachomatis and Chlamydia pneumoniae infection
36 The next-generation amplification test for Chlamydia trachomatis and Neisseria gonorrhoeae (Roche c
37 se of infertility and ectopic pregnancy, and Chlamydia trachomatis and Neisseria gonorrhoeae are reco
38 of self-collected swabs for the detection of Chlamydia trachomatis and Neisseria gonorrhoeae by a tra
39 assay (ATV; Gen-Probe) and the prevalence of Chlamydia trachomatis and Neisseria gonorrhoeae coinfect
40 methods exist for the molecular detection of Chlamydia trachomatis and Neisseria gonorrhoeae in clini
41 ilable molecular assays for the detection of Chlamydia trachomatis and Neisseria gonorrhoeae in recta
43 with detectable T. vaginalis, codetection of Chlamydia trachomatis and Neisseria gonorrhoeae occurred
44 utilization of nontraditional locations for Chlamydia trachomatis and Neisseria gonorrhoeae screenin
46 T) has become the preferred method to detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no
47 ing (NAAT) is the preferred method to detect Chlamydia trachomatis and Neisseria gonorrhoeae, but no
48 isease associations, which parallel those of Chlamydia trachomatis and Neisseria gonorrhoeae, the mec
52 ) nonameric epitopes that span the genome of Chlamydia trachomatis and prepared MHC tetramers from ap
54 Female pig-tailed macaques inoculated with Chlamydia trachomatis and Trichomonas vaginalis (n = 9)
55 tigated whether coinfection of macaques with Chlamydia trachomatis and Trichomonas vaginalis decrease
56 e laboratory-detected Neisseria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis infecti
57 ification testing for Neisseria gonorrhoeae, Chlamydia trachomatis, and Trichomonas vaginalis were pe
59 he obligate intracellular bacterial parasite Chlamydia trachomatis are the same as its eukaryotic hos
61 l children, such as Neisseria gonorrhoeae or Chlamydia trachomatis, are due to abusive contact and sh
62 tively restrict growth of the human pathogen Chlamydia trachomatis but fails to control growth of the
63 urethritis (NGU) and cervicitis is aimed at Chlamydia trachomatis, but Mycoplasma genitalium, which
66 female infertility and genital infection by Chlamydia trachomatis (C. trachomatis) is a major cause.
73 sex or symptoms is used to manage anorectal Chlamydia trachomatis (chlamydia) and Neisseria gonorrho
76 The class Ic RNR from the human pathogen Chlamydia trachomatis ( Ct) uses a Mn (IV)/Fe (III) cofa
77 The class Ic ribonucleotide reductase from Chlamydia trachomatis ( Ct) uses a stable Mn(IV)/Fe(III)
78 hose who have not been diagnosed with rectal Chlamydia trachomatis (CT) and/or rectal Neisseria gonor
82 he diagnosis and management of uncomplicated Chlamydia trachomatis (CT) infection in adolescents and
84 study was to estimate the probability that a Chlamydia trachomatis (CT) infection will cause an episo
87 collected for Neisseria gonorrhoeae (GC) and Chlamydia trachomatis (CT) screening of HIV-1-infected M
88 odel of HIV, Neisseria gonorrhoeae (NG), and Chlamydia trachomatis (CT) transmission dynamics among M
89 class Ic ribonucleotide reductase (RNR) from Chlamydia trachomatis (Ct) utilizes a Mn/Fe heterobinucl
92 mSIBA) that allows simultaneous detection of Chlamydia trachomatis (CT), Neisseria gonorrhoeae (NG),
100 ured derivative of the human genital isolate Chlamydia trachomatis D/UW-3/Cx, strain CTD153, which al
103 her probenecid would have a direct effect on Chlamydia trachomatis development through inhibition of
108 bacter baumannii, Burkholderia pseudomallei, Chlamydia trachomatis, Escherichia coli, Klebsiella pneu
109 oring indicates an absence of infection with Chlamydia trachomatis even if FT persists, may be more c
110 erinary relatives, the oculogenital pathogen Chlamydia trachomatis evolved as a commensal organism of
111 o, the obligate intracellular human pathogen Chlamydia trachomatis exhibits elevated expression of a
114 ancient neglected tropical disease caused by Chlamydia trachomatis for which a vaccine is needed.
115 s for detection of Neisseria gonorrhoeae and Chlamydia trachomatis from pharyngeal and rectal specime
116 ances detection of Neisseria gonorrhoeae and Chlamydia trachomatis from rectal and pharyngeal sources
118 oncerning prevalence and disease severity of Chlamydia trachomatis genital infection is whether more
119 finding and treating prevalent, asymptomatic Chlamydia trachomatis genital infection reduces reproduc
120 ancy and infertility observed in women after Chlamydia trachomatis genital infection result from asce
121 nd treatment programs implemented to control Chlamydia trachomatis genital infections and their compl
124 isting of 908 open reading frames encoded in Chlamydia trachomatis genome and plasmid was used to pro
125 fy novel transcriptional regulators from the Chlamydia trachomatis genome by predicting proteins with
127 .09, 0.66), and Neisseria gonorrhoeae and/or Chlamydia trachomatis had 92% lower odds of any adverse
130 tive plasmid of both Chlamydia muridarum and Chlamydia trachomatis has been shown to control virulenc
133 hock proteins of the intracellular bacterium Chlamydia trachomatis have been associated with immune p
136 d infections, such as Neisseria gonorrhoeae, Chlamydia trachomatis, HIV, human papillomavirus (HPV) a
137 atural infection induces partial immunity to Chlamydia trachomatis Identification of chlamydial antig
139 is caused by recurrent ocular infection with Chlamydia trachomatis in childhood, with conjunctival sc
140 he prevalence of trachoma and infection with Chlamydia trachomatis in communities after 3-7 years of
142 nts of the genetically intransigent pathogen Chlamydia trachomatis, in which all mutations have been
146 ned whether TLR variants are associated with Chlamydia trachomatis infection among women with pelvic
147 nces about the true population prevalence of Chlamydia trachomatis infection and disease and the sens
148 A sensing directs IFN-beta expression during Chlamydia trachomatis infection and suggest that effecto
149 ate point-of-care (POC) diagnostic tests for Chlamydia trachomatis infection are urgently needed for
150 rance and confer protective immunity against Chlamydia trachomatis infection but have not been simult
152 witnessed a disturbing increase in cases of Chlamydia trachomatis infection despite enhanced control
153 served signs of trachomatous inflammation or Chlamydia trachomatis infection diagnosed using PCR.
154 ral discharge to diagnose N. gonorrhoeae and Chlamydia trachomatis infection in certain populations b
155 correctly measure the true status of ocular Chlamydia trachomatis infection in individuals, particul
166 w-up studies in a murine model of intranasal Chlamydia trachomatis infection, we analogously found th
180 this study, we show that the human pathogen Chlamydia trachomatis infects the murine respiratory and
207 on with the obligate intracellular bacterium Chlamydia trachomatis is controlled primarily by IFN-gam
211 The obligate intracellular human pathogen Chlamydia trachomatis is the etiological agent of blindi
220 a, caused by repeated infections with ocular Chlamydia trachomatis, is targeted for elimination using
221 aused by the obligate intracellular organism Chlamydia trachomatis, is the world's leading cause of p
224 , increased the growth of the human pathogen Chlamydia trachomatis (L2) in wild-type murine fibroblas
225 pecifically bound and repressed promoters of Chlamydia trachomatis late genes, but not early or mid g
227 he obligate bacterial intracellular pathogen Chlamydia trachomatis leads to the sustained activation
233 assessed the intervention effect on incident Chlamydia trachomatis, Neisseria gonorrhoeae, and Mycopl
234 s, the prevalences of Mycoplasma genitalium, Chlamydia trachomatis, Neisseria gonorrhoeae, and Tricho
235 a, and first-void female urine specimens for Chlamydia trachomatis, Neisseria gonorrhoeae, and Tricho
236 sites were tested for M. genitalium and for Chlamydia trachomatis, Neisseria gonorrhoeae, and Tricho
237 e new insights concerning the concurrence of Chlamydia trachomatis, Neisseria gonorrhoeae, Mycoplasma
238 uently drink alcohol and to be infected with Chlamydia trachomatis, Neisseria gonorrhoeae, or herpes
239 d for M. genitalium and other STI organisms (Chlamydia trachomatis, Neisseria gonorrhoeae, Trichomona
240 ed with those for batches of 24 samples; for Chlamydia trachomatis/Neisseria gonorrhoeae tests, the a
241 e the immunogenicity and vaccine efficacy of Chlamydia trachomatis nMOMP in a nonhuman primate tracho
242 rs) was significantly greater than those for Chlamydia trachomatis or N. gonorrhoeae (27.6 and 25.9 y
243 e a significant association with concomitant Chlamydia trachomatis or N. gonorrhoeae infection overal
246 amples were collected for bacterial culture, Chlamydia trachomatis PCR, and RNA isolation at 1 year.
247 ed on filter paper to test for antibodies to Chlamydia trachomatis pgp3 using a multiplex bead assay.
251 A new R2 subclass, R2c, prototyped by the Chlamydia trachomatis protein was recently discovered.
255 he bacterial obligate intracellular pathogen Chlamydia trachomatis replicates within a membrane-bound
257 cal setting: infection of myeloid cells with Chlamydia trachomatis resulted in the expression of CHOP
258 source, molecular Neisseria gonorrhoeae and Chlamydia trachomatis results, and relative light unit (
259 nd Mn(III)Fe(III) sites in the R2 subunit of Chlamydia trachomatis ribonucleotide reductase using x-r
263 try/mass spectrometry (LC-MS/MS) analyses of Chlamydia trachomatis serovar L2 434/Bu EB, COMC, and Sa
264 cycle of the obligate intracellular pathogen Chlamydia trachomatis serovar L2 is controlled in part b
265 The two distinct lipoic acid ligases in Chlamydia trachomatis serovar L2, LplA1(Ct) and LplA2(Ct
266 .) routes with recombinant MOMP (rMOMP) from Chlamydia trachomatis serovars D (UW-3/Cx), E (Bour), or
267 fections caused by Neisseria gonorrhoeae and Chlamydia trachomatis serovars D to K occur at high inci
274 erive the intact structure of the primordial Chlamydia trachomatis T3SS in the presence and absence o
275 id-based genetic transformation protocol for Chlamydia trachomatis that for the first time provides a
277 or infertility (TFI) that is attributable to Chlamydia trachomatis, the population excess fraction (P
278 nd include the agent of oculogenital disease Chlamydia trachomatis, the respiratory pathogen C. pneum
279 d mice infected with Chlamydia muridarum and Chlamydia trachomatis to determine if there were differe
280 ating between-strain genomic recombinants of Chlamydia trachomatis to facilitate the organism's evolu
282 address the potential for plasmid-deficient Chlamydia trachomatis to serve as a live attenuated vacc
284 haracterization of a previously undocumented Chlamydia trachomatis transcription factor, designated G
285 pants were tested for Neisseria gonorrhoeae, Chlamydia trachomatis, Treponema pallidum (syphilis), he
287 the lifetime was tested for the presence of Chlamydia trachomatis, type-specific human papillomaviru
288 The obligate intracellular human pathogen Chlamydia trachomatis undergoes a complex developmental
289 d as a model organism for the study of human Chlamydia trachomatis urogenital and respiratory tract i
290 ctive antimicrobial chemotherapy, control of Chlamydia trachomatis urogenital infection will likely r
293 nd to a common Ag in Chlamydia muridarum and Chlamydia trachomatis Using an adoptive-transfer approac
294 urine samples) for Neisseria gonorrhoeae and Chlamydia trachomatis using nucleic acid amplification t
297 n a community with a high prevalence of STI, Chlamydia trachomatis was detected in 8.7% and Neisseria
298 An atypical response regulator, ChxR, from Chlamydia trachomatis, was previously reported to form h
300 ndida albicans, Streptococcus agalactiae and Chlamydia trachomatis with a single biochip, enabling a
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