ライフサイエンスコーパス: Citrobacter rodentium

1 e against an epithelium-associated pathogen (Citrobacter rodentium).

2 long with the anti-swarming activity against Citrobacter rodentium.

3 tion of mice by the EPEC-like mouse pathogen Citrobacter rodentium.

4 ease induced by the model bacterial pathogen Citrobacter rodentium.

5 h the enteric pathogens Escherichia coli and Citrobacter rodentium.

6 uced colonization levels of the gut pathogen Citrobacter rodentium.

7 and ILFs in the colon during infection with Citrobacter rodentium.

8 nged orally with the enteric murine pathogen Citrobacter rodentium.

9 hanced resistance to the intestinal pathogen Citrobacter rodentium.

10 onization of the mouse colon by the bacteria Citrobacter rodentium.

11 the gram-negative enteric bacterial pathogen Citrobacter rodentium.

12 on with the gram-negative bacterial pathogen Citrobacter rodentium.

13 ost protection against a bacterial pathogen, Citrobacter rodentium.

14 ted in the A/E-lesion-forming mouse pathogen Citrobacter rodentium.

15 enteropathogenic Escherichia coli (EPEC) and Citrobacter rodentium.

16 more aggressive infectious colitis caused by Citrobacter rodentium.

17 and lethal colitis by the mucosal pathogen, Citrobacter rodentium.

18 infection with the EPEC-like mouse pathogen Citrobacter rodentium.

19 impaired gut colonization resistance against Citrobacter rodentium.

20 onse to the attaching and effacing bacterium Citrobacter rodentium.

21 mpaired clearance of the intestinal pathogen Citrobacter rodentium.

22 of Runx3 in ILCs exacerbated infection with Citrobacter rodentium.

23 with LPS or infected with the enteropathogen Citrobacter rodentium.

24 ssential for the protective immunity against Citrobacter rodentium.

25 specially in the colon during infection with Citrobacter rodentium.

26 Salmonella enterica serovar Typhimurium and Citrobacter rodentium.

27 improves host tolerance of the mild pathogen Citrobacter rodentium.

28 evere colitis and death after infection with Citrobacter rodentium.

29 PEC and EHEC) and the natural mouse pathogen Citrobacter rodentium.

30 istration of dextran sulfate sodium (DSS) or Citrobacter rodentium.

31 ppled responses to intestinal infection with Citrobacter rodentium.

32 nce of the attaching/effacing mouse pathogen Citrobacter rodentium.

33 ir selective depletion during infection with Citrobacter rodentium.

34 itical for the clearance of the A/E pathogen Citrobacter rodentium.

35 ty against an intestinal bacterial pathogen, Citrobacter rodentium.

36 unity to the attaching-and-effacing pathogen Citrobacter rodentium.

37 zene sulfonic-acid (TNBS); or infection with Citrobacter rodentium.

38 In mice infected with Citrobacter rodentium, a model for enteropathogenic Esch

39 at germ-free animals are unable to eradicate Citrobacter rodentium, a model for human infections with

40 reover, mice lacking TACI were able to clear Citrobacter rodentium, a model pathogen for severe human

41 C, we constructed an Stx-producing strain of Citrobacter rodentium, a murine AE pathogen that otherwi

42 s derived from these mice were infected with Citrobacter rodentium, a murine attaching and effacing p

43 Using Citrobacter rodentium, a murine model of attaching and e

44 Citrobacter rodentium, a murine model pathogen for human

45 Citrobacter rodentium, a murine model pathogen that shar

46 C57BL/6 mice were orally gavaged with Citrobacter rodentium, a murine pathogen related to huma

47 Probiotics prevent disease induced by Citrobacter rodentium, a murine-specific enteric pathoge

48 Infection with Citrobacter rodentium, a natural mouse pathogen homologo

49 Citrobacter rodentium, a natural mouse pathogen, has rec

50 cute colitis induced by the enteric pathogen Citrobacter rodentium Adoptive transfer of macrophage-ri

51 killing of the extracellular enteropathogen Citrobacter rodentium after phagocytosis.

52 the murine gut microbiome to infection with Citrobacter rodentium, an attaching-and-effacing bacteri

53 e exhibited impaired intestinal clearance of Citrobacter rodentium, an enteric bacterium that models

54 The lifA/efa1 gene is also present in Citrobacter rodentium, an enteric pathogen that causes a

55 wnregulation of virulence gene expression in Citrobacter rodentium, an enteric pathogen that models h

56 fense against acute bacterial infection with Citrobacter rodentium and Clostridium difficile.

57 Citrobacter rodentium and Escherichia coli O157 triggere

58 P3 activators LPS and ATP, Escherichia coli, Citrobacter rodentium and transfection of LPS, AIM2 acti

59 ike toxin-producing E. coli, E. coli RDEC-1, Citrobacter rodentium, and an EPEC espB insertion mutant

60 hesins of enteropathogenic Escherichia coli, Citrobacter rodentium, and enterohemorrhagic E. coli (EH

61 tary intervention, mice were challenged with Citrobacter rodentium, and pathological responses were a

62 usceptible strain of the pathogenic bacteria Citrobacter rodentium, and we propose a general approach

63 erichia coli, enterohemorrhagic E. coli, and Citrobacter rodentium are classified as attaching and ef

64 erichia coli, enterohemorrhagic E. coli, and Citrobacter rodentium are classified as attaching and ef

65 e surrogate murine infection model for EHEC, Citrobacter rodentium, are all examples of microorganism

66 In this study, we employed Citrobacter rodentium as a physiologic model of pathogen

67 98 in IECs (hCD98 Tg mice) and infected with Citrobacter rodentium as an in vivo model.

68 m the mouse attaching and effacing pathogen, Citrobacter rodentium, as a potential drug target.

69 The attaching and effacing mouse pathogen Citrobacter rodentium associates intimately with the int

70 artonella spp., Lawsonia intracellularis and Citrobacter rodentium) can induce cellular proliferation

71 Citrobacter rodentium causes a similar colitis in mice a

72 Citrobacter rodentium causes an attaching and effacing i

73 Citrobacter rodentium causes epithelial hyperplasia and

74 We demonstrate that after infection with Citrobacter rodentium, CD4(+) LTi cells were a dominant

75 Dextran sodium sulfate (DSS) and Citrobacter rodentium colitis (CC) was induced in adult

76 Citrobacter rodentium colonizes the mouse colon in a sim

77 es after exposure to the intestinal pathogen Citrobacter rodentium Correspondingly, AQP3(-/-) mice sh

78 (EPEC), enterohemorrhagic E. coli (EHEC) and Citrobacter rodentium (CR) infections, are dependent on

79 Citrobacter rodentium (CR) promotes crypt hyperplasia an

80 Utilizing the Citrobacter rodentium (CR)-induced transmissible murine

81 an infection model, using the mouse pathogen Citrobacter rodentium (CR).

82 enteropathogenic Escherichia coli (EPEC) and Citrobacter rodentium during mammalian infections.

83 Lymphocyte inhibitory factor A (lifA) in Citrobacter rodentium encodes the large toxin lymphostat

84 Citrobacter rodentium (formally Citrobacter freundii bio

85 a coli (EPEC)-mediated disease in humans and Citrobacter rodentium (formerly C. freundii biotype 4280

86 Citrobacter rodentium (formerly Citrobacter freundii bio

87 also required for clearance of the bacterium Citrobacter rodentium from the gastrointestinal tract.

88 In mice, susceptibility to Citrobacter rodentium has been shown to be dependent on

89 the control of mouse colonic infection with Citrobacter rodentium in the presence of T cells.

90 onses to the attaching and effacing pathogen Citrobacter rodentium in these.

91 ry response to the colitis-inducing pathogen Citrobacter rodentium in vitro by inhibiting NF-kappaB a

92 n of the TLR7 agonist R848 or infection with Citrobacter rodentium in vivo.

93 activated by the colitis-inducing bacterium Citrobacter rodentium increased NO without affecting iNO

94 ride (LPS) and infection with mouse pathogen Citrobacter rodentium induce translocation of the nuclea

95 We found that the enteric pathogen Citrobacter rodentium induced sequential waves of IL-22-

96 epithelial barrier function in vitro and on Citrobacter rodentium-induced colitis in mice.

97 in IL-22-dependent colitis was confirmed in Citrobacter rodentium-induced disease.

98 ucosa-associated microbiota, and exacerbates Citrobacter rodentium-induced inflammation, effects that

99 Utilizing the Citrobacter rodentium-induced transmissible murine colon

100 Citrobacter rodentium induces transmissible murine colon

101 Citrobacter rodentium induces transmissible murine colon

102 milarly opposing phenotypes were observed in Citrobacter rodentium infection and allergic asthma.

103 22 receptor IL-22RA1 protects against lethal Citrobacter rodentium infection and chemical-induced col

104 sease, whereas expansion of these cells upon Citrobacter rodentium infection exacerbated pathology.

105 lsion, caused by a deficit in ILC2s, whereas Citrobacter rodentium infection is cleared efficiently.

106 Citrobacter rodentium infection of mice induces cell-med

107 ization following microbiome disruption with Citrobacter rodentium infection or antibiotic treatment,

108 nt mice are less able to eradicate a mucosal Citrobacter rodentium infection than wild-type C57BL/6 m

109 The pathogenesis of a Citrobacter rodentium infection was evaluated in mice fe

110 ency increased morbidity and mortality after Citrobacter rodentium infection with decreased secretion

111 During intestinal Citrobacter rodentium infection, a mouse model for enter

112 provides that link for the investigation of Citrobacter rodentium infection, a mouse model for enter

113 testinal epithelial cells after T. gondii or Citrobacter rodentium infection, but also maintained the

114 KKbeta(DeltaIEC) mice efficiently controlled Citrobacter rodentium infection, IKKalpha(DeltaIEC) mice

115 Using mouse model of Citrobacter rodentium infection, we investigated the rol

116 Ahr-deficient mice succumbed to Citrobacter rodentium infection, whereas ectopic express

117 nate inflammatory RelA/NF-kappaB response to Citrobacter rodentium infection, while Nfkb2(-/-) mice s

118 ed responses capable of protecting mice from Citrobacter rodentium infection.

119 cus-degrading bacteria and susceptibility to Citrobacter rodentium infection.

120 tivation during both Helicobacter pylori and Citrobacter rodentium infection.

121 critical inducers of the innate response to Citrobacter rodentium infection.

122 infection, repeated exposure to ketamine and Citrobacter rodentium infection.

123 conferred enhanced mucosal immunity against Citrobacter rodentium infection.

124 cells (DCs) and coexpressed with IL-23 after Citrobacter rodentium infection.

125 duced by dextran sodium sulfate treatment or Citrobacter rodentium infection.

126 ole of epithelial derived ILK in response to Citrobacter rodentium infection.

127 ons that T-helper cell, type 17 responses in Citrobacter rodentium infections are driven by concomita

128 herpes simplex virus, Toxoplasma gondii, and Citrobacter rodentium infections.

129 Infection with Citrobacter rodentium initially was controlled by ILC3,

130 Citrobacter rodentium is a natural mouse pathogen relate

131 Infection of mice with Citrobacter rodentium is a robust model to study bacteri

132 Citrobacter rodentium is an attaching and effacing mouse

133 Citrobacter rodentium is an enteric bacterial pathogen o

134 Citrobacter rodentium is an enteric pathogen which attac

135 Infection with the bacterium Citrobacter rodentium is known to increase epithelial ce

136 Citrobacter rodentium is the causative agent of transmis

137 Citrobacter rodentium is the causative agent of transmis

138 Citrobacter rodentium is the rodent equivalent of entero

139 Citrobacter rodentium is used as an in vivo model system

140 after infection with the intestinal pathogen Citrobacter rodentium, leading to impaired survival.

141 resistance against a murine enteropathogen, Citrobacter rodentium, leading to the death of the anima

142 The Citrobacter rodentium model mimics the pathogenesis of i

143 in a CD4-specific knockout of NLRX1 within a Citrobacter rodentium model of colitis.

144 nvestigated the role of these enzymes in the Citrobacter rodentium model of colitis.

145 hage activation and disease phenotype in the Citrobacter rodentium model of murine infectious colitis

146 that in both the dextran sodium sulfate and Citrobacter rodentium models of colitis, significantly i

147 (EHEC), enteropathogenic E. coli (EPEC), and Citrobacter rodentium Moreover, Salmonella enterica stra

148 he involvement of Map in diarrhoea using the Citrobacter rodentium mouse model of EPEC.

149 ing infection by the murine enteric pathogen Citrobacter rodentium of the family Enterobacteriacea.

150 ute colitis was induced by administration of Citrobacter rodentium or dextran sulfate sodium (DSS) to

151 macrophages infected with Escherichia coli, Citrobacter rodentium or Vibrio cholerae.

152 with EPEC, using the mouse-specific pathogen Citrobacter rodentium Our murine infant model is similar

153 infection with Anaplasma phagocytophilum and Citrobacter rodentium respectively, were used.

154 in the context of intestinal infection with Citrobacter rodentium, resulting in preserved innate imm

155 mice with the intestinal bacterial pathogen Citrobacter rodentium results in colonic mucosal hyperpl

156 ANR homologs of Vibrio cholerae, Citrobacter rodentium, Salmonella enterica and ETEC were

157 egative bacteria including Escherichia coli, Citrobacter rodentium, Salmonella typhimurium, and Shige

158 We also identified an lpf gene cluster in Citrobacter rodentium strain ICC168 (lpf(cr)).

159 We characterized Citrobacter rodentium strains bearing deletions in indiv

160 ies have found the murine bacterial pathogen Citrobacter rodentium to provide a robust, relevant in-v

161 Citrobacter rodentium uses a type III secretion system (

162 Citrobacter rodentium uses virulence factors similar to

163 EPEC), as well as the related mouse pathogen Citrobacter rodentium, utilize a type III secretion syst

164 Citrobacter rodentium was used as a Th17-inducing infect

165 Citrobacter rodentium was used to model human Escherichi

166 rine infection model with one such pathogen, Citrobacter rodentium, was used to elucidate the importa

167 ice with the attaching and effacing bacteria Citrobacter rodentium, we defined the mechanisms and con

168 Using a mouse A/E pathogen, Citrobacter rodentium, we show that interleukin-22 (IL-2

169 d inflammation by the enteric mouse pathogen Citrobacter rodentium, which causes disease similar to t

170 d the murine attaching and effacing pathogen Citrobacter rodentium, which colonizes primarily the sur

171 nse generated to the extracellular bacterium Citrobacter rodentium, which induces a mixed Th1 and Th1

172 activity against the murine enteric pathogen Citrobacter rodentium, which like the related clinically

173 testinal epithelium with the rodent pathogen Citrobacter rodentium, which models human infections wit