ライフサイエンスコーパス: Cl

1 (*)Cl converts to (*)OH.

2 Cl(-) transporters, including KCC2, and extracellular im

3 ported chlorination degrees were -0.9 to 1.0%Cl for SCCP mixtures of 49-71%Cl.

4 y exchange of the OAc(-) ligand for Cl(-) (1-Cl), NO3(-) (1-NO3), and pyridine ([1-py](+)).

5 ein, we report, the discovery of a beta-d-2'-Cl,2'-F-uridine phosphoramidate nucleotide 16, as a nont

6 eneration catalyst precursors [(H2IMes)(py)2(Cl)2Ru horizontal lineCHPh] and [(H2IMes)(3-Br-py)2(Cl)2

7 horizontal lineCHPh] and [(H2IMes)(3-Br-py)2(Cl)2Ru horizontal lineCHPh] is measured to be independen

8 1-silacyclohexanes C5H10Si(Ph,X) (X = F (3), Cl (4)) were studied by gas-phase electron diffraction,

9 by NaClO applied in doses of 10, 20, and 30g Cl/kg of starch, and then acetylated using acetic acid a

10 ific isotope effect ((13)C/(12)C, (37)Cl/(35)Cl) analysis offers a new opportunity to test these hypo

11 major products based on accurate masses, (35)Cl/(37)Cl isotopic patterns, and MS/MS spectra.

12 e its effect on the diffusion of HTO and (36)Cl(-).

13 ce 18 ka using variations in cosmogenic (36)Cl measured on bedrock scarps, mapped using LiDAR and gr

14 of the long-lived beta-emitters (129)I, (36)Cl and the alpha-emitters (154)Dy, (148)Gd, (150)Gd, and

15 Laboratory incubations with (36)Cl as a Cl tracer were performed to test how combination

16 nd-specific isotope effect ((13)C/(12)C, (37)Cl/(35)Cl) analysis offers a new opportunity to test the

17 roducts based on accurate masses, (35)Cl/(37)Cl isotopic patterns, and MS/MS spectra.

18 Zn = -13.7 per thousand), heavy Cl (delta(37)Cl = +15 per thousand), and high U/Pb supports the origi

19 d-specific procedure to measure the delta(37)Cl of a wide array of organic compounds overcoming limit

20 sand for delta(2)H, delta(13)C, and delta(37)Cl values, respectively.

21 tope ratios (delta(13)C, delta(2)H, delta(37)Cl) of its main stereoisomers (alpha-, beta-, delta- and

22 - (i.e., Deltadelta(2)H versus Deltadelta(37)Cl versus Deltadelta(13)C) isotope plot, which opens fur

23 n-chlorine (Deltadelta(13)C vs Deltadelta(37)Cl) isotope patterns for oxidation by thermally activate

24 c interference of the (36)ArH dimer with (37)Cl was minimized by employing dry plasma conditions.

25 gly electron-withdrawing group, such as C(4)-Cl or C(5)-NO2, they rearrange to products 4 in the pres

26 antification in the range of 0.06 (I) to 60 (Cl) mug g(-1).

27 re -0.9 to 1.0%Cl for SCCP mixtures of 49-71%Cl.

28 Cl: -0.04, 5.3); cord/total: beta=22.0 (95% Cl: 3.2, 40.7)] and PDI (cord/maternal: beta=1.7 (95% Cl

29 iated with MDI [cord/maternal: beta=2.6 (95% Cl: -0.04, 5.3); cord/total: beta=22.0 (95% Cl: 3.2, 40.

30 % Cl: -0.5, 3.9); cord/total: beta=15.6 (95% Cl: 0.3, 20.9)).

31 40.7)] and PDI (cord/maternal: beta=1.7 (95% Cl: -0.5, 3.9); cord/total: beta=15.6 (95% Cl: 0.3, 20.9

32 Laboratory incubations with (36)Cl as a Cl tracer were performed to test how combinations of env

33 When Bi as a Cl-storage electrode was coupled with NaTi2(PO4)3 as a N

34 s and potential limitations of using Bi as a Cl-storage electrode.

35 When Sx binds a Cl(-), the protonated Ex moves upward, out of the positi

36 (ICC-IM) by activation of Ca(2+) -activated Cl(-) channels (ANO1, encoded by Ano1) and voltage-depen

37 (ICC-IM) by activation of Ca(2+) -activated Cl(-) channels (encoded by Ano1) and voltage-dependent L

38 physiological importance of Ca(2+)-activated Cl(-) channels (CaCCs) in neurons has been largely overl

39 a Ca(2+) influx that opens Ca(2+)-activated Cl(-) channels, generating the receptor potential.

40 airways not only eliminates Ca(2+)-activated Cl(-) currents, but unexpectedly also abrogates CFTR-med

41 Loss of the AE3 Cl(-)/HCO3(-) exchanger (Slc4a3) in mice causes an impai

42 ese results support a model in which the AE3 Cl(-)/HCO3(-) exchanger, coupled with parallel Cl(-) and

43 the most difficult systems to study, the Am-Cl interaction within AmCl6(3-).

44 tistical shape atlases for the PCa+, Bx- and Cl- prostates.

45 de complexes by Cu K-edge, Cu L2,3-edge, and Cl K-edge X-ray absorption spectroscopy.

46 the extracellular-facing gating Glu (Ex) and Cl(-) binding to the external (Sx) and central (Sc) site

47 icient binding toward the spherical F(-) and Cl(-) anions, and the linear CN(-) (log Kass = 3.46, 3.5

48 impacts on both cell volume homeostasis and Cl(-)-permeable GABAA receptor-dependent membrane excita

49 rt are significantly different for Li(+) and Cl(-) ions, unlike what is expected at infinite dilution

50 iomass under salinity, differences in Na and Cl concentrations in shoots and K/Na ratio were evaluate

51 SISA14-1 (G03) accumulated low Na and Cl under salinity.

52 We also monitored Na(+) and Cl(-) ion concentrations and the temporal dynamics of co

53 red through the group transfer of both P and Cl atoms from chloro-substituted dibenzo-7lambda(3) -pho

54 de-saturated, and exceptionally volatile and Cl-rich SLIP lavas, permitted a massive release of nicke

55 ltaneously determine the changes of pHi and [Cl(-)]i in response to hypercapnia and seizure activity.

56 for the electrochemical detection of anions (Cl(-), SO4(2-), and Br(-)) in a highly diluted water sol

57 (+) -HCO3(-) cotransporter (NBC1) and apical Cl(-) /HCO3(-) exchanger (solute carrier family 26 membe

58 the basolateral K(+) permeability and apical Cl(-) and HCO3(-) permeabilities (CFTR), and reducing th

59 ility to employ a variety of aryl donors (Ar-Cl, Ar-Br, Ar-I, Ar-OTf), amine types (primary and secon

60 nditions (pH = 2.5), photocleavage of the Ar-Cl bond occurred and a phenyl cation chemistry resulted.

61 inobutyric acid (GABAA) receptors, which are Cl(-)-permeable, ligand-gated ion channels.

62 ite and Ca(2+) in the presence of associated Cl(-), which gain prevalence at higher CaCl2 concentrati

63 At Cl:AA = 2.8, the chlorine was found to first react quick

64 anide as well as by depletion of basolateral Cl(-), suggesting that Na(+)/K(+)/2Cl(-) (NKCC1/SLC12A2)

65 and reducing the activity of the basolateral Cl(-) /HCO3(-) exchanger (anion exchanger 2; AE2).

66 For pure MAPbX3 (X = I, Br, Cl) perovskite films, we observe that the cation dynamic

67 Colloidal CsPbX3 (X = Br, Cl, and I) perovskite nanocrystals (NCs) have emerged as

68 ith HOBr by direct bromination leading to Br-Cl-DOM and by bromine substitution of chlorine leading t

69 e Pd-catalyzed amination of 3,2- and 5,2- Br/Cl-pyridines is reported.

70 The ratios Br/Cl and I/Cl in all studied chondrites show a limited ran

71 smic Cl(-) level is dynamically regulated by Cl(-) channels and transporters.

72 The CLC protein fold is shared by Cl(-) channels and 2Cl(-):1H(+) antiporters.

73 e OS-SET (SET to a pi* orbital followed by C-Cl cleavage) in ethenes.

74 r as the predominant pathway enabling dual C-Cl bond formation and contradict an alternative pathway

75 udy investigates, for the first time, dual C-Cl isotope fractionation during anaerobic biodegradation

76 sand), and in CEs when the exergonicity of C-Cl bond cleavage was reduced in an organic solvent (reac

77 bond cleavage in oxidation by persulfate, C-Cl bond cleavage in Fe(0)-mediated reductive dechlorinat

78 itions (pH approximately 12) and reductive C-Cl bond cleavage by cast zerovalent iron, Fe(0).

79 robenzosiloles were further transformed to C-Cl, C-Br, C-I, and C-O bonds in final products.

80 access Ni2(vinylidenoid) intermediates via C-Cl oxidative addition.

81 ons at the two discharging plateaus, while C-Cl bonding on the surface, or edges of natural graphite,

82 ET (SET to a sigma* orbital concerted with C-Cl breakage) in alkanes compared to stepwise OS-SET (SET

83 nical or MRI indications of prostate cancer (Cl-).

84 e to serve as an efficient and high capacity Cl-storage electrode using its conversion to BiOCl was i

85 The cation/Cl(-) cotransporters and ECM metalloproteinases may be p

86 ity in a small subpopulation of F508del-CFTR Cl(-) channels but that the majority remain destabilized

87 ayed deactivation of individual F508del-CFTR Cl(-) channels.

88 s transmembrane conductance regulator (CFTR) Cl(-) channel.

89 brium isotope effect (EIE) for the aryl CH...Cl(-) interaction of anion receptor 1H/1D is reported.

90 CLC proteins transport chloride (Cl(-)) ions across cellular membranes to regulate muscle

91 Intracellular chloride ([Cl(-)]i) and pH (pHi) are fundamental regulators of neur

92 The heavy halogens-chlorine (Cl), bromine (Br) and iodine (I)-are key tracers of accr

93 st reactive sites were consumed by chlorine, Cl-substituted functional groups (Cl-DOM) are reacting w

94 instead measured for Z = OAc, NPhth, CO2Me, Cl, Br, and CN, indicative of alpha-CH2 deactivation wit

95 Intracellular chloride concentration ([Cl(-)]i) in pancreatic beta-cells is kept above electroc

96 Intraneuronal Cl(-) concentration ([Cl(-)]i) regulation impacts on both cell volume homeosta

97 ng the intraneuronal chloride concentration [Cl(-)]i.

98 that ACFL550 had half the chlorine content (Cl%) relative to AC550, which makes ACFL550 a promising

99 onate(-) diminishes the inward tail current (Cl(-) efflux) at a membrane potential of -100 mV due to

100 Cytoplasmic Cl(-) level is dynamically regulated by Cl(-) channels a

101 itical function of CaCC-mediated cytoplasmic Cl(-) homeostasis in controlling the organization of Ptd

102 This newly defined role of cytoplasmic Cl(-) may shed light on the mechanisms of intracellular

103 that TMEM16A-mediated control of cytoplasmic Cl(-) regulates the organization of the major phosphoino

104 loride channel (CaCC), regulates cytoplasmic Cl(-) homeostasis and promotes plasma membrane remodelin

105 However, whether cytoplasmic Cl(-) plays additional roles in animal development and t

106 nnel gated by membrane voltage and cytosolic Cl(-) and Ca(2+).

107 s strain 14DCB1 preferentially dechlorinated Cl that has an ortho H.

108 lorine formal reduction potential E degrees (Cl(*/-)) = 1.87 V vs NHE that is at least 300 meV more f

109 taminated plumes of groundwater that deliver Cl(-) and Na(+) to streams throughout the year.

110 Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N,N-di-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala were ide

111 e stable in water over 10 days except N,N-di-Cl-Phe-Gly.

112 -Gly, N-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala along with their corresponding dipeptides wer

113 N,N-di-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala were identified as the major products based o

114 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N,N-di-Cl-Phe-Gly, N-Cl-Tyr-Al

115 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Ty

116 The reaction of [Zr(Tren(DMBS) )(Cl)] [Zr1; Tren(DMBS) =N(CH2 CH2 NSiMe2 Bu(t) )3 ] with

117 into Slc26a6, a unique cardiac electrogenic Cl(-)/HCO3(-) transporter in ventricular myocytes, linki

118 We show that Slc26a6 mediates electrogenic Cl(-)/HCO3(-) exchange activities in cardiomyocytes, sug

119 dysfunction due to loss of the endolysosomal Cl(-) transporter ClC-b/CLCN7 delayed degradation of int

120 y facilitating the exchange of extracellular Cl(-) for intracellular HCO3(-) .

121 servations that replacement of extracellular Cl(-) with gluconate(-) diminishes the inward tail curre

122 2-), NO3(-), NO2(-), BrO3(-), AsO4(-), F(-), Cl(-), and Br(-) and, to a lesser extent, ClO3(-).

123 e methane (and isotopologues) with atoms (F, Cl, O) and diatoms (OH), with inclusion of also rotation

124 for the first time for further halogens (F, Cl, Br, and I) determination by ion chromatography (IC)

125 ed region, viz., the "boron core" for X = F, Cl, and the surrounding "halogen shell" for X = I, At.

126 ives, including [Tism(Pr(i)Benz)]MgX [X = F, Cl, Br, I, SH, N(H)Ph, CH(Me)Ph, O2CMe, S2CMe].

127 sing Ex proton affinity on binding the first Cl(-) reduces the cost of binding the second Cl(-) at ei

128 n with 9-fluorenylmethyl chloroformate (FMOC-Cl) was determined to be effective for quantitation of f

129 Consequently, the cytosolic constriction for Cl(-) passage is widened in CLC-K such that the kinetic

130 refore that minimizing the driving force for Cl(-) secretion is the main requirement for secreting 14

131 y tuned by exchange of the OAc(-) ligand for Cl(-) (1-Cl), NO3(-) (1-NO3), and pyridine ([1-py](+)).

132 he kinetic barrier previously postulated for Cl(-)/H(+) transporter function would be reduced.

133 s above the Nernst equilibrium potential for Cl(-) and thus can be used as efficient inhibitory tools

134 nd hyperpolarizes the reversal potential for Cl(-).

135 te its applicability to automated functional Cl(-)- and Ca(2+)-imaging with recombinant HEK293 cells

136 conductance regulator (CFTR) is an ATP-gated Cl(-) channel defective in the genetic disease cystic fi

137 chlorine, Cl-substituted functional groups (Cl-DOM) are reacting with HOBr by direct bromination lea

138 y challenging even when good leaving groups (Cl(-) ) are employed.

139 s differed in the following order: Ze-LS >/= Cl-LS > Fluv CWs, reflecting the highest metal adsorptio

140 y substituted 4-X-pyridinium cations (X = H, Cl, Br, I), is reported.

141 Raising the HCO3(-) /Cl(-) permeability ratio of CFTR from 0.4 to 1.0 had lit

142 Zn (delta(66)Zn = -13.7 per thousand), heavy Cl (delta(37)Cl = +15 per thousand), and high U/Pb suppo

143 ation reaction which produces [Cl2Sb(IV)Pd(I)Cl(o-dppp)2], a complex with a covalent Sb-Pd bond.

144 The ratios Br/Cl and I/Cl in all studied chondrites show a limited range, indis

145 lead to the S = 2 five-coordinate HO-Fe(III)-Cl complex with the C(*) of the substrate, pi-oriented r

146 of contaminated groundwater that resulted in Cl(-) loadings to the adjacent stream of 6574 to 40008 k

147 the widely discussed developmental fall in [Cl(-)]i and the role of the K-Cl cotransporter KCC2 in t

148 on, with long-term depletion in incompatible Cl and Pb, and lesser depletion of more-compatible Zn.

149 ClC-4 is an intracellular Cl(-)/H(+) exchanger that is highly expressed in the bra

150 TATEMENT Precise regulation of intracellular Cl(-) levels ([Cl(-)]i) preserves appropriate, often inh

151 hed light on the mechanisms of intracellular Cl(-) signaling events crucial for regulating tissue arc

152 in rat and mouse ducts, raised intracellular Cl(-) and resulted in a lower secreted HCO3(-) concentra

153 as essential in minimizing the intracellular Cl(-) concentration following cAMP stimulation and there

154 Intraneuronal Cl(-) concentration ([Cl(-)]i) regulation impacts on bot

155 we establish that the "neuron-specific" K(+)Cl(-) co-transporter 2 (KCC2, Slc12a5) is expressed in s

156 KCC2 is a neuron specific K(+)-Cl(-) co-transporter that controls neuronal chloride hom

157 KCC2 is a neuron-specific K(+)-Cl(-) cotransporter essential for establishing the Cl(-)

158 The K(+)-Cl(-) co-transporter KCC2 (SLC12A5) tunes the efficacy o

159 (+)-K(+)-2Cl(-) cotransporter 1 and the K(+)-Cl(-) cotransporter 2 (KCC2).

160 ental fall in [Cl(-)]i and the role of the K-Cl cotransporter KCC2 in this process.

161 s to the adjacent stream of 6574 to 40008 kg Cl(-) per winter, depending on winter snowfall.

162 thesized by salt-metathesis reaction of [L2 (Cl)Ge:] 1 with sodium phosphaethynolate [(dioxane)n NaOC

163 e regulation of intracellular Cl(-) levels ([Cl(-)]i) preserves appropriate, often inhibitory, GABAer

164 s as well as pre-edge analysis of the ligand Cl K-edge it is suggested that NKP-1339 remains in its +

165 to the iron center followed by late ligand (Cl(-) or OH(-)) transfer to the substrate.

166 l increases about two-fold in the H2 PO4(-) <Cl(-) approximately Br(-) approximately NO3(-) <ClO4(-)

167 ut unexpectedly also abrogates CFTR-mediated Cl(-) secretion and completely abolishes cAMP-activated

168 stic data are consistent with metal-mediated Cl atom transfer as the predominant pathway enabling dua

169 pression of CFTR stimulated SLC26A6-mediated Cl(-)-oxalate exchange in Xenopus oocytes.

170 Anion exchanger 1 (AE1) mediates Cl(-)/HCO3(-) exchange in erythrocytes and kidney interc

171 oxide production, increases apical membrane Cl(-) permeability approximately 3-5-fold, and increases

172 ermediate, (eta(5)-C5Me5)[N(Et)C(Ph)N(Et)]Mo(Cl)(NHSiMe3) (V), and XOSiMe3 as a co-product.

173 The modeled (*)Cl:(*)OH ratio was approximately 20% for the UV/free chl

174 We propose approaches to modulate [Cl(-)]i that are relevant for two common clinical sequel

175 N-Cl-DCAM can, however, undergo acid-catalyzed decompositi

176 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N,N-di-Cl-P

177 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N-Cl-Tyr-Al

178 Alternatively, N-Cl-DCAM can be produced directly from DCAN chlorination

179 ted N-DBP, N-chloro-2,2-dichloroacetamide (N-Cl-DCAM), was confirmed in this study as the actual DCAN

180 lthough those acidic conditions that favor N-Cl-DCAM degradation are generally atypical for finished

181 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N,N-di-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-

182 N-Cl-Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala along wi

183 -Tyr-Gly, N,N-di-Cl-Tyr-Gly, N-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala along with their corre

184 -Tyr-Gly, N-Cl-Phe-Gly, N,N-di-Cl-Phe-Gly, N-Cl-Tyr-Ala, and N,N-di-Cl-Tyr-Ala were identified as the

185 a family of seven N-chloro-haloacetamides (N-Cl-HAMs).

186 phase extraction (SPE), the occurrence of N-Cl-DCAM and its two brominated analogues (i.e., N-chloro

187 ead to the formation of an equal amount of N-Cl-DCAM by forming a hydrogen bond between hypochlorite

188 The discovery of N-Cl-DCAM or, more broadly speaking, N-Cl-HAMs in chlorina

189 ter pH conditions, and the anionic form of N-Cl-DCAM was found to be very stable in the absence of ch

190 ry of N-Cl-DCAM or, more broadly speaking, N-Cl-HAMs in chlorinated drinking waters is of significanc

191 It is suspected that N-Cl-DCAM has been erroneously identified as DCAM, because

192 Due to its relatively low pKa value, N-Cl-DCAM tends to deprotonate under typical drinking wate

193 the epithelial sodium channel (ENaC), Na(+)/Cl(-) cotransporter (NCC), and with no-lysine-kinase 1 (

194 abundance of total and phosphorylated Na(+)/Cl(-) cotransporter (NCC), claudin-7, and cleaved forms

195 The thiazide-sensitive Na(+)/Cl(-) cotransporter (NCC) is activated by low potassium

196 eostasis and provide evidence that the Na(+)/Cl(-) cotransporter (NCC) compensated for the inactivati

197 then phosphorylate and activate the renal Na-Cl cotransporter (NCC).

198 ted mice, leading to up-regulation of the Na-Cl co-transporter NCC, p-NCC and the development of salt

199 time 60min, temperature 70 degrees C and NBD-Cl concentration 40mM) were systematically investigated.

200 shfeld charges shows that the least-negative Cl discriminates active from nonactive substrates in 14

201 ic inhibition by rapidly adjusting neuronal [Cl(-)]i to GABAAR activity.

202 iotropic molecular determinants of neuronal [Cl(-)]i - cytoplasmic impermeant anions, polyanionic ext

203 e complex regulatory mechanisms of neuronal [Cl(-)]i and how these mechanisms impact on neuronal volu

204 -metalated iminosilane, [DippN=Si(OSiMe3 )Ni(Cl)(NHC)2 ] (3), in a rearrangement cascade.

205 Samples containing 2-3 nmol Cl injected on-column were sufficient to achieve a preci

206 The highest observed %Cl(-) depletion occurred in the submicrometer range, wit

207 tly arise from high tissue concentrations of Cl(-) or Na(+) but were due to changes in the pHapo Most

208 attributed to the higher binding constant of Cl(-) with the anion receptor and the stronger electroni

209 series of benzimidazole-based derivatives of Cl-amidine, hypothesizing that this scaffold would allow

210 -AR for the electrochemical determination of Cl(-) anions was demonstrated using the artificial matri

211 The detection limit for the determination of Cl(-) was found at the 1.0 pM level for both sensing sys

212 d by the differential ligand field effect of Cl(-) versus OH(-) on the Fe center.

213 DRA-mediated exchange of Cl(-) for HCO3(-) was measured by uptake of (125)I.

214 barrier in CLC channels enables fast flow of Cl(-) down its electrochemical gradient.

215 ticles containing chloride (mole fraction of Cl/Na >/= 0.1, 90% chloride depletion).

216 ue to the predominant functional presence of Cl(-) loaders such as the Na(+)K(+)2Cl(-) co-transporter

217 s of activation and kexp for 31 reactions of Cl(*) with organic compounds.

218 ):1H(+) binding stoichiometry is a result of Cl(-) binding to Sx requiring H(+) binding to Ex, wherea

219 The high selectivity of Cl(-) anions can be attributed to the higher binding con

220 ion pathway of electrophilic substitution of Cl at the ortho or para position of the phenol structure

221 ons at both the N-terminus and C-terminus of Cl-amidine result in >100-fold increases in PAD2 potency

222 Parallel measurements of [Cl(-)]i and pHi at the single-cell level in the mouse co

223 that the KCCs are the primary regulators of [Cl(-)]i in SCN neurons.

224 both proposed to be important regulators of [Cl(-)]i Neurons of the reticular thalamic (RT) nucleus e

225 d particles showed the greatest influence on Cl(-) depletion in the submicrometer range.

226 emoved Cl doubly flanked by two Cl or by one Cl and NH2, whereas strain 14DCB1 preferentially dechlor

227 exes with terminal anionic ligands, OH(-) or Cl(-), bound to the lower coordinate metal center are su

228 erivative, activates GABAAR-mediated outward Cl(-) current in the absence of GABA.

229 +)K(+)2Cl(-) co-transporter 1 (Slc12a2) over Cl(-)extruders of unidentified nature.

230 (-)/HCO3(-) exchanger, coupled with parallel Cl(-) and H(+)-extrusion mechanisms and extracellular ca

231 , [Ru(gly)(pdto)]Cl (2), and [Ru(acac)(pdto)]Cl (3), where pdto = 2,2'-[1,2-ethanediylbis-(sulfanediy

232 pounds [Ru(en)(pdto)]Cl2 (1), [Ru(gly)(pdto)]Cl (2), and [Ru(acac)(pdto)]Cl (3), where pdto = 2,2'-[1

233 matrix (ECM) glycoproteins, and plasmalemmal Cl(-) transporters - could help the identification of no

234 Me5 )2 Th[kappa(2) -(C,C')-(CH2 )(CH2 )PPh2 ]Cl.

235 nd carbic anhydride using a (salph)AlCl/[PPN]Cl catalytic pair.

236 The PPy-GOx/PPy-Cl bilayer biosensor was effective in rejecting 98% of a

237 , as an inner layer, and a permselective PPy-Cl film as an outer layer.

238 oxyl radicals (HO(*)) and chlorine radicals (Cl(*)) is an attractive alternative to UV alone or chlor

239 pending on the chlorine to amino acid ratio (Cl:AA).

240 Here, using ratiometric Cl(-) imaging, we have investigated intracellular chlori

241 Strain CBDB1 preferentially removed Cl doubly flanked by two Cl or by one Cl and NH2, wherea

242 ferent substituents on the fused benzo ring (Cl, NO2, NH2, CF3, ureido, amido, heterocycles), were sy

243 the protic substituent and the polarized [Ru-Cl] unit imposes directionality onto the ligand sphere o

244 tively transformed into chloride-green rust (Cl-GR) within 5 min and persisted over 18 days.

245 From the blood samples, Na, P, S, Cl, K, Ca, Fe, Cu, Zn, and Br elemental concentrations w

246 d (Fluv), clinopyroxene-dominated lava sand (Cl-LS) and zeolite-dominated lava sand (Ze-LS), aiming a

247 Cl(-) reduces the cost of binding the second Cl(-) at either Sx or Sc.

248 e cation chloride cotransporters to setting [Cl(-)]i in these SCN neurons and found that the chloride

249 ilyl)nickel(II) complex 3, {[cat((TMS) L)Si](Cl)Ni<--:BH(NHC)2 }, via the cleavage of two B-O bonds a

250 changes in stomatal conductance of the slac1 Cl(-) channel and ost2 H(+)-ATPase mutants, which we ver

251 l model of a local RT network featuring slow Cl(-) extrusion kinetics, similar to those we found expe

252 sion events, which includes the loss of some Cl(-) ions.

253 ss combustion in flaming phase released some Cl-rich-OM/soot particles and cardboard combustion relea

254 Using such isotopes, here we show that Cl, Br and I abundances in carbonaceous, enstatite, Rumu

255 nts in rats (male and female), we find that [Cl(-)]i remains relatively low in RT neurons.

256 Interestingly, we observed that [Cl(-)]i is differentially regulated between AVP+ and VIP

257 neurons and found evidence suggesting that [Cl(-)]i is higher during the day than during the night i

258 be the thermodynamic linkage for binding the Cl(-) to each site and protons to Ex.

259 situ generated HCl as the source of both the Cl(-) and H(+) and is catalyzed by palladium(II) acetate

260 substrate, pi-oriented relative to both the Cl(-) and the OH(-) ligands.

261 e or least negative) aromatic C carrying the Cl to be removed.

262 es were noted at the apex when comparing the Cl- and PCa+ prostates.

263 cotransporter essential for establishing the Cl(-) gradient required for hyperpolarizing inhibition i

264 e of the series decreases gradually from the Cl end to the Br end, indicating that the larger distort

265 sulting in increased Fe(III) contents in the Cl-GR.

266 a reactive secondary group can increase the Cl:AA ratio required for the formation of N,N-dichlorami

267 K(+) channel Kir4.1, and stimulation of the Cl(-) channel ClC-K.

268 racellular alkalization, consistent with the Cl(-)/HCO3(-) exchange activities of Slc26a6.

269 rved both 5f- and 6d-orbital mixing with the Cl-3p orbitals; however, contributions from the 6d-orbit

270 However, the exact nature and origin of this Cl-bearing mineral remained speculative.

271 oscopic reference (an element insensitive to Cl(-) and pH), we show that ratiometric imaging is stron

272 echanisms, one- and two-electron transfer to Cl (strain CBDB1) or H (strain 14DCB1) are compatible wi

273 ide uptake transporter NKCC1 contributes to [Cl(-)]i regulation in SCN neurons, but that the KCCs are

274 s we report a precatalyst, (dppf)Ni(o-tolyl)(Cl) (dppf = 1,1'-bis(diphenylphosphino)ferrocene), for S

275 hways: (i) the precatalyst (dppf)Ni(o-tolyl)(Cl) undergoes comproportionation with the active Ni(0) s

276 centers displayed better selectivity toward Cl(-) anion detection than those based on Cu(II) centers

277 enters, exhibited highest sensitivity toward Cl(-).

278 ntafluorophenyl porphyrin catalyst, Mn(TPFPP)Cl.

279 n meso-tetraphenylporphyrin chloride (Fe[TPP]Cl) complex in 1,1,1,3,3,3-hexafluoropropan-2-ol (HFIP)

280 ferentially removed Cl doubly flanked by two Cl or by one Cl and NH2, whereas strain 14DCB1 preferent

281 arges per cycle across the membrane with two Cl ions moving in the opposite direction of one proton.

282 AC) is the ubiquitously expressed vertebrate Cl(-) /anion channel that is composed of proteins belong

283 ectivity sequence found for both systems was Cl(-) > SO4(2-) > Br(-).

284 to Sx requiring H(+) binding to Ex, whereas Cl(-) binding to Sc does not lead to proton uptake.

285 ruthenium complexes [RuX(CNN)(dppb)] (1, X = Cl; 2, X = H; CNN = 2-aminomethyl-6-tolylpyridine, dppb

286 , I) and C-X...||| (acetylene) contacts (X = Cl, Br).

287 te spatially resolved multicolor CsPbX3 (X = Cl, Br, I, or alloy of two halides) nanowire heterojunct

288 that (+/-)-trans-1,2-dihalocyclohexanes (X = Cl, Br) engage in significant halogen bonding (XB) inter

289 stoichiometric amount of alkyl halides (X = Cl, Br, I) enables a rapid access to a variety of 3,5-di

290 on dispersion forces and weak C-X...pi (X = Cl, Br, I) and C-X...||| (acetylene) contacts (X = Cl, B

291 re by reaction of [(L)2Ni(II)2(mu-X)3]X (X = Cl or Br) with NaOCl in methanol or acetonitrile (where

292 s of an alkylating reagent to UO2 X2 (L)2 (X=Cl, Br, I, OTf; L=triphenylphosphine oxide, 2,2'-bipyrid

293 itrosyl complexes, [CoX2 (NO)(PMePh2 )2 ] (X=Cl, Br), in solution.

294 (pseudo)halide, R3 Si-X (R=aryl, alkyl, H; X=Cl, Br, I, OTf, SPh), cleanly affords (R3 SiO)2 UX2 (L)2

295 However, when X=Cl, only one equivalent of methane is lost with concomit

296 2 with the phosphonium salts [CH3 PPh3 ]X (X=Cl, Br, I) was investigated.

297 reaction energy profiles for [CH3 PPh3 ]X, X=Cl and I showed that in the case of iodide, thermodynami

298 er drug NKP-1339 was studied applying XANES (Cl K- and Ru L2,3-edges) in tumor, kidney and liver tiss

299 The Xe-Br and Xe-Cl bonds are very weakly covalent and can be viewed as s

300 The isostructural chloroxenate anions (Xe-Cl bond lengths, 2.9316(2) to 3.101(4) A) were synthesiz