ライフサイエンスコーパス: Clostridium perfringens

1 Escherichia coli, Bacteroides fragilis, and Clostridium perfringens.

2 ccus aureus, Clostridium acetobutylicum, and Clostridium perfringens.

3 of Clostridium botulinum, and iota toxin of Clostridium perfringens.

4 of Clostridium septicum and epsilon-toxin of Clostridium perfringens.

5 AlgR of Pseudomonas aeruginosa, and VirR of Clostridium perfringens.

6 on toxin secreted by the anaerobic bacterium Clostridium perfringens.

7 nization of specific-pathogen-free chicks by Clostridium perfringens.

8 1 and MprF2) found in the bacterial pathogen Clostridium perfringens.

9 oss firmicutes, including the human pathogen Clostridium perfringens.

10 cherichia coli, 50% GC; calf thymus, 42% GC; Clostridium perfringens, 27% GC) have been employed as t

11 ism isolated from dogs, cats, and horses was Clostridium perfringens (75, 13, and101 isolates, respec

12 The presence and abundance of Clostridium perfringens (8.4%) and Bacteroides dorei (0.

13 Clostridium perfringens, a human pathogen, is one of the

14 cross-react with a homologous sequence of a Clostridium perfringens adenosine triphosphate-binding c

15 idues with 90% homology to a sequence within Clostridium perfringens adenosine triphosphate-binding c

16 be immediate and direct, as in the action of Clostridium perfringens alpha toxin on red cells or plat

17 inescence assay for identifying and assaying Clostridium perfringens alpha toxin.

18 Based on comparisons with two orthologs, Clostridium perfringens alpha-toxin and Bacillus cereus

19 oximately 2 nM) of epsilon toxin produced by Clostridium perfringens and a prominent food toxin.

20 caused by the ubiquitous, anaerobic bacteria Clostridium perfringens and Clostridium septicum.

21 kholderia mallei, Burkholderia pseudomallei, Clostridium perfringens and Entamoeba histolytica.

22 NetB is a pore-forming toxin produced by Clostridium perfringens and has been reported to play a

23 obacterium hafniense, Clostridium novyi, and Clostridium perfringens and increase their activity up t

24 for all corresponding genes of 26 strains of Clostridium perfringens and Streptococcus pneumoniae.

25 erences between the germination of spores of Clostridium perfringens and that of spores of a number o

26 H gene, encoding a hyaluronidase secreted by Clostridium perfringens, and a C. perfringens hyaluronid

27 lfitobacterium hafniense, Clostridium novyi, Clostridium perfringens, and Eggerthella lenta.

28 with enterobacteria, desulfovibrios, type E Clostridium perfringens, and Enterococcus faecalis, wher

29 odborne outbreaks caused by Bacillus cereus, Clostridium perfringens, and Staphylococcus aureus were

30 bacterial IMPDHs from Campylobacter jejuni, Clostridium perfringens, and Vibrio cholerae.

31 e anaerobic sulfatase-maturating enzyme from Clostridium perfringens (anSMEcpe) catalyzes the two-ele

32 These anSME structures from Clostridium perfringens are also the first of an AdoMet

33 s of the common human and livestock pathogen Clostridium perfringens are attributable to a formidable

34 gglutinin upon digestion with sialidase from Clostridium perfringens, Arthrobacter neurofaciens, or S

35 o-beta-galactosidase designated E-ABase from Clostridium perfringens ATCC 10543 capable of liberating

36 mercially available sialidases prepared from Clostridium perfringens ATCC10543 were contaminated with

37 Clostridium perfringens biotype A strains are the causat

38 Clostridium perfringens can obtain sialic acid from host

39 Clostridium perfringens can produce up to three differen

40 -beta-galactosidase (Endo-beta-Gal(GnGa)) in Clostridium perfringens capable of releasing GlcNAcalpha

41 By producing toxins, Clostridium perfringens causes devastating diseases of b

42 Clostridium perfringens causes gas gangrene and gastroin

43 rtex by cortex-lytic enzymes (CLEs), and two Clostridium perfringens CLEs, SleC and SleM, degrade cor

44 tridia in Cluster I, including the pathogens Clostridium perfringens, Clostridium botulinum and Clost

45 y Bacillus anthracis, Staphylococcus aureus, Clostridium perfringens, Clostridium botulinum, and Clos

46 tigated a putative cell-surface adhesin from Clostridium perfringens comprising an N-terminal adhesin

47 sed method developed to specifically measure Clostridium perfringens concentrations.

48 P = .1 and P = .01 for consecutive samples); Clostridium perfringens continued to be more prevalent i

49 te pretreatment with sialidase purified from Clostridium perfringens did (P < 0.05).

50 The genome of the pathogen Clostridium perfringens encodes two proteins, GerO and G

51 y cpb2, is implicated in the pathogenesis of Clostridium perfringens enteritis.

52 Domain I of Clostridium perfringens enterotoxin (cCPE) binds to the

53 A noncytotoxic C-terminal fragment of Clostridium perfringens enterotoxin (cCPE) is a natural

54 Clostridium perfringens enterotoxin (CPE) action starts

55 ious epidemiological studies have implicated Clostridium perfringens enterotoxin (CPE) as a virulence

56 Clostridium perfringens enterotoxin (CPE) causes food po

57 Clostridium perfringens enterotoxin (CPE) causes the gas

58 Clostridium perfringens enterotoxin (CPE) causes the sym

59 Clostridium perfringens enterotoxin (CPE) has a unique m

60 Clostridium perfringens enterotoxin (CPE) has recently b

61 Clostridium perfringens enterotoxin (CPE) induces cytoly

62 After binding, Clostridium perfringens enterotoxin (CPE) initially loca

63 Clostridium perfringens enterotoxin (CPE) is a major cau

64 Clostridium perfringens enterotoxin (CPE) is a pore-form

65 Clostridium perfringens enterotoxin (CPE) is responsible

66 Clostridium perfringens enterotoxin (CPE) is responsible

67 Clostridium perfringens enterotoxin (CPE) is the etiolog

68 d the potential superantigenic properties of Clostridium perfringens enterotoxin (CPE) on human perip

69 The previous model for the action of Clostridium perfringens enterotoxin (CPE) proposed that

70 ost in vitro studies exploring the action of Clostridium perfringens enterotoxin (CPE) utilize either

71 ies performed to investigate the topology of Clostridium perfringens enterotoxin (CPE) when this toxi

72 Clostridium perfringens enterotoxin (CPE), a 35-kDa poly

73 relationship and mechanism of action of the Clostridium perfringens enterotoxin (CPE), a series of r

74 Clostridium perfringens enterotoxin (CPE), a single poly

75 -affinity intestinal epithelial receptor for Clostridium perfringens enterotoxin (CPE), and is suffic

76 y receptors, respectively, for the cytotoxic Clostridium perfringens enterotoxin (CPE), in this study

77 Clostridium perfringens enterotoxin (CPE), the virulence

78 claudins (e.g., Claudin-4) as receptors for Clostridium perfringens enterotoxin (CPE).

79 , claudin-4 and -3 serve as the receptor for Clostridium perfringens enterotoxin (Cpe).

80 Clostridium perfringens enterotoxin causes the gastroint

81 Clostridium perfringens enterotoxin is a common cause of

82 the localization of the genes for the CPE-R (Clostridium perfringens enterotoxin receptor, CPETR1) an

83 CPE (Clostridium perfringens enterotoxin) is the major virule

84 for the sporulation-associated synthesis of Clostridium perfringens enterotoxin, a common cause of f

85 xins, such as botulinum neurotoxins (BoNTs), Clostridium perfringens epsilon toxin (ETX), staphylococ

86 Clostridium perfringens epsilon toxin belongs to the aer

87 the motion of single membrane receptors, the Clostridium perfringens epsilon-toxin (CPepsilonT) recep

88 The Clostridium perfringens epsilon-toxin causes a severe, o

89 The Clostridium perfringens epsilon-toxin is responsible for

90 ntestinal inhabitants: Bacteroides fragilis, Clostridium perfringens, Escherichia coli, Klebsiella pn

91 ia (FIBs; Escherichia coli, enterococci, and Clostridium perfringens) exhibited biphasic decay patter

92 To examine the early effects of Clostridium perfringens exotoxins on tissue perfusion, a

93 Clostridium perfringens food poisoning is caused by type

94 me that the anaerobic gram-positive pathogen Clostridium perfringens forms biofilms.

95 Clostridium perfringens gas gangrene is a fulminant infe

96 Clostridium perfringens gas gangrene is characterized by

97 Clostridium perfringens gas gangrene is characterized by

98 t the site of infection are two hallmarks of Clostridium perfringens gas gangrene.

99 Several Clostridium perfringens genotype E isolates, all associa

100 A published complex of Clostridium perfringens GH125 enzyme with a nonhydrolyza

101 PFO, a virulence factor of the organism Clostridium perfringens, has almost the same molecular m

102 ly, the genome sequences of three strains of Clostridium perfringens have been completed and we ident

103 acillus anthracis, Campylobacter jejuni, and Clostridium perfringens IMPDHs.

104 robiology methods are not suitable to detect Clostridium perfringens in formalin-fixed, paraffin-embe

105 as a competitive exclusion agent to control Clostridium perfringens in poultry.

106 Clostridium perfringens iota-toxin consists of two separ

107 Clostridium perfringens iota-toxin is a binary toxin con

108 Clostridium perfringens is a common cause of food-borne

109 Clostridium perfringens is a Gram-positive anaerobic pat

110 Clostridium perfringens is a Gram-positive, anaerobic sp

111 Clostridium perfringens is a ubiquitous and versatile pa

112 Clostridium perfringens is an anaerobic Gram-positive pa

113 Clostridium perfringens is an anaerobic, Gram-positive b

114 Clostridium perfringens is an extracellular pathogen whi

115 Clostridium perfringens is an important human and animal

116 Clostridium perfringens is capable of producing up to 15

117 turnoff assay for phospholipase C (PLC) from Clostridium perfringens is developed based on the revers

118 Necrotic enteritis (NE) caused by Clostridium perfringens is one of the most detrimental i

119 Although Clostridium perfringens is recognized as an important ca

120 Clostridium perfringens is the cause of several human di

121 Clostridium perfringens is the most common cause of clos

122 Clostridium perfringens is the third most frequent cause

123 logical studies suggested that cpb2-positive Clostridium perfringens isolates are associated with gas

124 s such as norovirus and toxigenic strains of Clostridium perfringens, Klebsiella oxytoca, Staphylococ

125 his model is challenged by the glycosylating Clostridium perfringens large cytotoxin (TpeL toxin) tha

126 and in vitro activity on human substrates of Clostridium perfringens NagJ, a close homologue of human

127 A polyclonal antibody prepared against Clostridium perfringens neuraminidase 1) detected surfac

128 ysis, we were able to identify inhibitors of Clostridium perfringens neuraminidase present in a root

129 C. difficile was codetected with Clostridium perfringens, norovirus, sapovirus, parechovi

130 omic sequences, but only in three bacterial (Clostridium perfringens, Oenococcus oeni, and Leuconosto

131 e monomeric cytolysin secreted by pathogenic Clostridium perfringens, oligomerizes and forms large po

132 ontrast, perfringolysin O (PFO), secreted by Clostridium perfringens, only binds to membranes contain

133 Whether expressed in C. difficile, Clostridium perfringens, or Escherichia coli, TxeR stimu

134 Clostridium perfringens perfringolysin O (PFO or theta-t

135 S. pyogenes gene that closely resembles the Clostridium perfringens pfoR gene, exerts a negative eff

136 Clostridium perfringens phospholipase C (PLC) and perfri

137 ws 60% amino acid sequence identity with the Clostridium perfringens phospholipase C, or alpha-toxin

138 s (G-) and to the transfer protein TcpC from Clostridium perfringens plasmid pCW3 (G+).

139 Spores of Clostridium perfringens possess high heat resistance, an

140 Clostridium perfringens possesses at least two functiona

141 The gram-positive anaerobe Clostridium perfringens produces a large arsenal of toxi

142 ssociated O-acetyl sialic acid--but not from Clostridium perfringens resulted in an increase in RN639

143 Likewise, MazF-cd expression in E. coli or Clostridium perfringens resulted in growth arrest.

144 e crystal structure of ligand-free NanE from Clostridium perfringens reveals a modified triose-phosph

145 Infusion of Clostridium perfringens sialidase to the injury site mar

146 The carbonic anhydrase (Cpb) from Clostridium perfringens strain 13, the only carbonic anh

147 The complete genome sequences of Clostridium perfringens strain ATCC 13124, a gas gangren

148 Clostridium perfringens strains (type A) isolated from a

149 Many Clostridium perfringens strains produce NanI as their ma

150 Clostridium perfringens strains produce severe diseases,

151 2-aminobenzoic acid using neuraminidase from Clostridium perfringens that cleaves sialic acid monomer

152 peL is a recently identified LCT produced by Clostridium perfringens that has received relatively lim

153 Clostridium perfringens, the most broadly distributed pa

154 For example, Clostridium perfringens, the species with the highest va

155 initially attributed to norovirus; however, Clostridium perfringens toxicoinfection was subsequently

156 Clostridium perfringens type A food poisoning is the sec

157 About 5% of Clostridium perfringens type A isolates carry the cpe ge

158 Clostridium perfringens type A isolates carrying an ente

159 Clostridium perfringens type A isolates causing food poi

160 Clostridium perfringens type A isolates producing entero

161 Clostridium perfringens type A strains producing enterot

162 Alpha toxin from Clostridium perfringens type A, a phospholipase C, has b

163 clostridial glucosylating toxins produced by Clostridium perfringens type A, B, and C strains.

164 Culture confirmed the presence of Clostridium perfringens type A.

165 Clostridium perfringens type B and type C isolates, whic

166 Clostridium perfringens type B causes enteritis and ente

167 The important veterinary pathogen Clostridium perfringens type B is unique for producing t

168 Clostridium perfringens type C isolates cause enteritis

169 Clostridium perfringens type C isolates cause enterotoxe

170 Clostridium perfringens type C isolates, which cause ent

171 Clostridium perfringens type C strains are the only non-

172 The ability of Clostridium perfringens type C to cause human enteritis

173 Recombinant beta-toxin from Clostridium perfringens type C was found to increase the

174 usative organism of enteritis necroticans is Clostridium perfringens type C, an anaerobic gram-positi

175 Previous studies showed that Clostridium perfringens type D animal disease strain CN3

176 Clostridium perfringens type D causes disease in sheep,

177 Clostridium perfringens type D enterotoxemias have signi

178 Clostridium perfringens type D isolates are important in

179 Clostridium perfringens type D isolates cause enterotoxe

180 Clostridium perfringens type D strains cause enterotoxem

181 silon-toxin, the primary virulence factor of Clostridium perfringens type D, causes mortality in live

182 Clostridium perfringens type E isolates produce iota-tox

183 iota toxin, a binary enterotoxin produced by Clostridium perfringens type E, were studied by fluoresc

184 ration-initiated hydrolysis catalyzed by the Clostridium perfringens unsaturated glucuronyl hydrolase

185 and in the pathogenesis of cystic fibrosis; Clostridium perfringens VirR, a regulator of virulence f

186 An alpha/beta-type SASP, Ssp2, from Clostridium perfringens was expressed at significant lev

187 ignificant increases in genus Sutterella and Clostridium perfringens when compared to healthy dogs.