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1 nes, was present in all species of the genus Cochliobolus but absent in the filamentous fungus, Penic
2 ion cultures 3 to 5 h after inoculation with Cochliobolus carbonum (Race 1), and then increased rapid
3 determinant for the plant pathogenic fungus Cochliobolus carbonum and an inhibitor of histone deacet
4 Race 1 isolates of the filamentous fungus Cochliobolus carbonum are exceptionally virulent on cert
5 selective pathogenicity (TOX2) in the fungus Cochliobolus carbonum governs production of a cyclic tet
6 PsmES4326 infection, but not in response to Cochliobolus carbonum infection, indicating that PAD4 ha
7 hese include Cochliobolus heterostrophus and Cochliobolus carbonum infection, ultraviolet light treat
10 to confer resistance to the fungal pathogen Cochliobolus carbonum race 1, but the effectiveness of r
11 se inhibitor produced by the fungal pathogen Cochliobolus carbonum race 1, promotes virulence in maiz
12 sed by a previously unknown fungal pathogen, Cochliobolus carbonum race 1, was first described in 193
14 dopsis and leaves inoculated with the fungus Cochliobolus carbonum were fed [35S]cysteine (Cys) and [
15 S2, was isolated from the filamentous fungus Cochliobolus carbonum, a pathogen of maize that makes th
16 ccSNF1, was isolated from the maize pathogen Cochliobolus carbonum, and ccsnf1 mutants of HC toxin-pr
17 chitooligosaccharides, and by infection with Cochliobolus carbonum, Cochliobolus heterostrophus and F
20 and by infection with Cochliobolus carbonum, Cochliobolus heterostrophus and Fusarium verticillioides
21 determinant in the maize (Zea mays) pathogen Cochliobolus heterostrophus and is involved in tolerance
22 genes from the related heterothallic species Cochliobolus heterostrophus can also influence U. botryt
23 Inoculation with the nonpathogenic fungus Cochliobolus heterostrophus drastically reduced the ligh
24 as a virulence factor in the maize pathogen Cochliobolus heterostrophus Hypothesizing that the homol
27 rtional mutants of the fungal maize pathogen Cochliobolus heterostrophus were screened for altered vi
28 bacteria (Ralstonia solanacearum) and fungi (Cochliobolus heterostrophus) results in reduced virulenc
30 controlling the difference between races of Cochliobolus heterostrophus: race T is highly virulent o
32 thologs in the rice (Oryza sativa) pathogen, Cochliobolus miyabeanus, the wheat (Triticum aestivum) p
33 sequences from homothallic and heterothallic Cochliobolus species support the hypothesis that heterot
34 in varying copy number in the genomes of all Cochliobolus spp. examined, giving a distinct fingerprin
37 entification of susceptibility to the fungus Cochliobolus victoriae in Arabidopsis thaliana has enabl
38 and race O of C. heterostrophus on maize, of Cochliobolus victoriae on oats, and of Gibberella zeae o
39 effector produced by the necrotrophic fungus Cochliobolus victoriae), TRX-h5 (a defense-associated th
40 ctorin, the host-selective toxin produced by Cochliobolus victoriae, the causal agent of victoria bli
41 ctorin is a host-selective toxin produced by Cochliobolus victoriae, the causal agent of victoria bli
42 f Avena sativa (oat) is caused by the fungus Cochliobolus victoriae, which is pathogenic because of t
43 rom the filamentous fungus Helminthosporium (Cochliobolus) victoriae that copurifies with mycoviral d
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