ライフサイエンスコーパス: Coxsackie

1 suggests a speciation of PV from a C-cluster coxsackie A virus (C-CAV) ancestor through mutation of t

2 Coxsackie A virus (CAV) serotypes 1, 11, 13, 15, 17, 18,

3 iruses, including PV type 1 (PV1), PV2, PV3, coxsackie A virus 11 (CAV11), CAV13, CAV17, CAV20, CAV21

4 ces closely related to certain cocirculating coxsackie A virus isolates.

5 p that contains enterovirus 71 and 11 of the coxsackie A viruses.

6 Pairwise comparisons suggested that coxsackie A11 and A15 viruses should be classified as st

7 d as strains of the same serotype, as should coxsackie A13 and A18 viruses.

8 cacious by oral administration in treating a Coxsackie A21 infection in CD-1 mice.

9 en 100 and 500 and occurs independent of the coxsackie Ad receptor.

10 fic adapter, sCARhMFE, composed of sCAR [the coxsackie/Ad receptor (CAR) ectodomain] and MFE-23 (a si

11 arget cells depends upon the presence of the coxsackie adenovirus cell surface receptor, CAR, which i

12 The Coxsackie Adenovirus Receptor (CAR) has primarily been s

13 rminal knob of its fiber coat protein to the Coxsackie adenovirus receptor (CAR) protein.

14 ell as binding to native cellular receptors (coxsackie adenovirus receptor (CAR), integrins).

15 tion with antibody for Dynamin 2 but not for Coxsackie adenovirus receptor or for integrin alpha(v).

16 s an integrin binding RGD-4C motif, allowing Coxsackie adenovirus receptor-independent infection of c

17 The coxsackie-adenovirus receptor (CAR) is a transmembrane r

18 digestive or respiratory routes require the Coxsackie-adenovirus receptor (CAR) to infect the epithe

19 ing antibodies, widespread expression of the coxsackie-adenovirus receptor (CAR), and adenovirus sequ

20 ile some serotype D adenoviruses bind to the coxsackie-adenovirus receptor (CAR), the ability of Ad30

21 mediate cell attachment in vitro when using coxsackie-adenovirus receptor (CAR)-containing cell line

22 lian cells via interaction of fiber with the coxsackie-adenovirus receptor (CAR).

23 1, and Ad14) that use receptor(s) other than coxsackie-adenovirus receptor and CD46 were able to trig

24 bstacles is the highly variable level of the coxsackie-adenovirus receptor expression on human primar

25 only used oncolytic adenoviruses targeted to coxsackie-adenovirus receptor or CD46.

26 s is expression of the primary receptor, the coxsackie-adenovirus receptor.

27 ressed glycosylphosphatidylinositol-modified coxsackie-adenovirus receptor.

28 rimary receptor for Ad35 (CD46) but not Ad5 (coxsackie-adenovirus receptor; CAR).

29 T cells, transgenic (Tg) mice expressing the coxsackie/adenovirus receptor (CAR) in their T cell comp

30 o adenovirus infection because they lack the Coxsackie/adenovirus receptor (CAR) needed for virus att

31 sCAR-MFE), which fuses the ectodomain of the coxsackie/adenovirus receptor (sCAR) with a single-chain

32 abeled Ad binding and with the expression of coxsackie/adenovirus receptor but not with the expressio

33 n the expression of fiber receptors, such as coxsackie/adenovirus receptor, and alpha(v) integrins on

34 evels because these cells have low levels of Coxsackie and adenovirus receptor (CAR) and alpha(v) int

35 The presence of coxsackie and adenovirus receptor (CAR) and alpha(v) int

36 per, we reported a significant difference in coxsackie and adenovirus receptor (CAR) from several hum

37 Expression of the Coxsackie and Adenovirus Receptor (CAR) is frequently re

38 The coxsackie and adenovirus receptor (CAR) is identified as

39 Coxsackie and adenovirus receptor (CAR) is not only a hi

40 Coxsackie and adenovirus receptor (CAR) is the primary c

41 to investigate the modulatory effect of the coxsackie and adenovirus receptor (CAR) on ventricular c

42 The coxsackie and adenovirus receptor (CAR) plays key roles

43 Binding of JAML to its ligand Coxsackie and adenovirus receptor (CAR) provides costimu

44 One such receptor is Coxsackie and Adenovirus Receptor (CAR) that binds to Ju

45 oteins with functional binding sites for the coxsackie and adenovirus receptor (CAR) were cleared rap

46 ed cells lost expression of the cell surface coxsackie and adenovirus receptor (CAR) within 24 h post

47 ith domain I of its human cellular receptor, coxsackie and adenovirus receptor (CAR), is presented he

48 r expression of the primary Ad receptor, the coxsackie and adenovirus receptor (CAR), known to be dow

49 termed globin transcription factor 1 (GATA1)-coxsackie and adenovirus receptor (CAR), that expressed

50 higher GFP expression than naked Ad in both coxsackie and adenovirus receptor (CAR)-positive and -ne

51 s its attachment to a cell surface receptor, coxsackie and adenovirus receptor (CAR).

52 ion via binding interactions with epithelial coxsackie and adenovirus receptor (CAR).

53 Recently, a cDNA clone (the coxsackie and adenovirus receptor [CAR]) encoding a 46-k

54 increase was not observed in the absence of Coxsackie and Adenovirus Receptor cell expression, disco

55 cally inhibited by HCBP1 peptide rather than coxsackie and adenovirus receptor specific antibody.

56 CAR (Coxsackie and Adenovirus Receptor) is the primary dockin

57 xpression of related proteins JAM-C and CAR (Coxsackie and adenovirus receptor) was also observed in

58 However, a single-chain anti-CD40/soluble Coxsackie and adenovirus receptor-fusion protein (G28/sC

59 initial cell surface attachment receptor for Coxsackie and group C adenoviruses.

60 The coxsackie- and adenovirus receptor (CAR) functions as th

61 nce of the main receptor for adenovirus, the coxsackie- and adenovirus receptor (CAR).

62 We identified loss-of-function mutations in Coxsackie- and adenovirus receptor-like membrane protein

63 The structures of polio-, coxsackie-, and rhinovirus polymerases have revealed a c

64 All coxsackie B (CB) viruses can initiate infection by attac

65 the inefficiency is due to an absence of the coxsackie B and adenovirus type 2 and 5 receptor (CAR) f

66 l-differentiated human airway epithelia, the coxsackie B and adenovirus type 2 and 5 receptor (CAR) r

67 receptors for the adenovirus fiber protein, coxsackie B and adenovirus type 2 and 5 receptor (CAR),

68 totype laboratory strains suggest that all 6 coxsackie B serotypes interact with a 46-kDa protein rec

69 Many serotypes of echovirus (EV) and Coxsackie B virus (CBV) bind human decay-accelerating fa

70 The coxsackie B virus and adenovirus receptor (CAR) is a mem

71 rmissive hamster cells became susceptible to coxsackie B virus attachment and infection.

72 dy interactions between low-passage clinical coxsackie B virus isolates and the two receptors.

73 g increases risk of chronic pancreatitis (4) Coxsackie B virus may increase severity of alcoholic chr

74 viruses, including parainfluenza virus 5 and Coxsackie B virus, that enter at the plasma membrane.

75 Coxsackie B viruses (CVB) are enteroviruses that have be

76 ole of recombination in the evolution of the coxsackie B viruses (CVB), we determined the partial seq

77 amily (IgSF) and functions as a receptor for coxsackie B viruses (CVBs).

78 human enterovirus B species (HEV-Bs) (e.g., coxsackie B viruses [CVBs] and echoviruses) have been im

79 r (CAR) mediates attachment and infection by coxsackie B viruses and many adenoviruses.

80 ion for heart transplantation worldwide, and coxsackie B viruses are detected in about one-third of i

81 Although many coxsackie B viruses interact with decay accelerating fac

82 Coxsackie B viruses interact with two putative cell surf

83 In this report, we describe experiments with coxsackie B viruses with a cell type-specific propagatio

84 s species B, which contains the echoviruses, coxsackie B viruses, coxsackievirus A9, and enterovirus

85 at iNOS is induced in mice infected with the Coxsackie B3 virus.

86 iated with a moderate rate of diabetes after Coxsackie B4 and vaccinia virus infection.

87 Coxsackie B4 virus is strongly associated with the devel

88 We infected different strains of mice with Coxsackie B4 virus to discriminate between the two possi

89 of LEW*1WR1 rats, whereas H-1, vaccinia, and Coxsackie B4 viruses did not.

90 2 (HSV-2) virus, and enteroviruses (polio-, coxsackie-, echo-, and enteroviruses 68 and 71) from per

91 active opsonophagocytic Ab responses against coxsackie, picorna, and influenza viruses and demonstrat

92 derived from glutamic acid decarboxylase or coxsackie virus (each of which has been associated with

93 mouth disease is caused by a new lineage of Coxsackie virus A6 and is characterised by high fever an

94 ool samples taken on day 6 of illness showed Coxsackie virus A6.

95 they express low levels of the high-affinity Coxsackie virus and adenovirus receptor (CAR).

96 cardiomyocytes were not more susceptible to Coxsackie virus B3 (CVB3) infection than control cells.

97 -altering mutations previously identified in coxsackie virus B3 (CVB3) were mapped onto the nsp12-RdR

98 et al. show that infection of NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus

99 Infection of prediabetic NOD mice with Coxsackie virus B3 or lymphocytic choriomeningitis virus

100 These results show that diabetes induced by Coxsackie virus infection is a direct result of local in

101 complexes did not require expression of the coxsackie virus-Ad receptor (CAR) since similar data wer

102 ovel finding that human erythrocytes present Coxsackie virus-adenovirus receptor (CAR) providing an A

103 ion of the ligand MIP1alpha protects against Coxsackie virus-associated myocarditis.

104 r) is the primary docking receptor for typeB coxsackie viruses and subgroup C adenoviruses.

105 ), Severe acute respiratory syndrome (SARS), coxsackie viruses, and rhinoviruses.