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1 rine infection for the human fungal pathogen Cryptococcus.
2 n >10% of patients with KS, tuberculosis, or Cryptococcus.
3 orphotype required for sexual development in Cryptococcus.
4 o have in-vitro and in-vivo activity against cryptococcus.
5 , 16% (4/25) for tuberculosis, 14% (1/7) for Cryptococcus, 10% (1/10) for Mycobacterium avium complex
10 cally relevant organisms, including Candida, Cryptococcus, and Aspergillus, in subjects with increasi
11 e host defense against Candida, Aspergillus, Cryptococcus, and others, with specific elements of the
13 ansmissions; and prevention and treatment of Cryptococcus-associated immune reconstitution syndrome.
14 e for the exploration of the relationship in Cryptococcus between cellular morphology and pathogenesi
18 of fungal antigens in body fluids, including cryptococcus capsular polysaccharide, histoplasma antige
22 the recruitment of AIC components to nascent Cryptococcus-containing vacuoles (CnCVs) regulates the i
25 Pichia angusta VKM Y-2559 and the oleaginous Cryptococcus curvatus VKM Y-3288 yeast cells were immobi
26 me resequencing of the human fungal pathogen Cryptococcus deuterogattii identified an outbreak lineag
31 he prediction of Cryptococcus neoformans and Cryptococcus gattii in Europe is an important tool to un
33 erapy and management of complications due to Cryptococcus gattii infection in 86 Australian patients
37 es of strains of Cryptococcus neoformans and Cryptococcus gattii isolated from serial episodes of cry
38 lecular types of the Cryptococcus neoformans/Cryptococcus gattii species complex that infect dogs and
39 athway for virulence of the pathogenic yeast Cryptococcus gattii using the highly virulent Vancouver
40 lentulus and Neosartorya udagawae and yeast Cryptococcus gattii VGII (implicated in the outbreak in
41 la-zoster virus, and Cryptococcus neoformans/Cryptococcus gattii We describe a multicenter evaluation
48 Fungal and bacteriological studies showed Cryptococcus in 64 (19.5%) patients, pneumococcus in 8 (
50 accumulation of CD11b(+) DC in the lungs of Cryptococcus-infected mice is primarily attributable to
51 Fungal culture or histopathology confirmed Cryptococcus infection for 20 patients (52.6%), and CrAg
58 Znf2, a master regulator of morphogenesis in Cryptococcus, is necessary and sufficient for the produc
60 th molecular type, but the susceptibility of Cryptococcus isolates from dogs and cats is largely unkn
61 nsistent with the role of Fbp1 in regulating Cryptococcus-macrophage interaction and fungal virulence
62 llular proliferation after phagocytosis in a Cryptococcus-macrophage interaction assay, which likely
63 tory blood immune signature, possibly due to Cryptococcus modulation of the host immune response.
64 erable genetic diversity compared with other Cryptococcus molecular types and could be divided into t
65 nfected with a moderately virulent strain of Cryptococcus neoformans (52D), which resulted in prolong
68 iodiomycete forming an induced DOPA-melanin, Cryptococcus neoformans (CN); and the slow-growing envir
69 ith Pneumocystis jirovecii (pneumocystosis), Cryptococcus neoformans (cryptococcosis), Histoplasma ca
70 Isavuconazole showed good activities against Cryptococcus neoformans (MIC90, 0.12 mug/ml) and other n
71 s presenting with P. marneffei (n = 719) and Cryptococcus neoformans (n = 1598) infection to the Hosp
73 es for melanization of the pathogenic fungus Cryptococcus neoformans also offers unique opportunities
75 Aspergillus fumigatus, Alternaria alternata, Cryptococcus neoformans and Candida albicans) proteins w
76 e of macrophage autophagy in the response to Cryptococcus neoformans and Candida albicans, two import
81 ompare the molecular genotypes of strains of Cryptococcus neoformans and Cryptococcus gattii isolated
84 rabidopsis thaliana, Caenorhabditis elegans, Cryptococcus neoformans and Drosophila melanogaster.
85 erol endoperoxide, which were active against Cryptococcus neoformans and methicillin-resistance Staph
87 in GM-CSF are susceptible to infections with Cryptococcus neoformans and other opportunistic fungi.
89 gh activity toward Aspergillus fumigatus and Cryptococcus neoformans at acidic pH, yet remained nonto
90 e infections with pathogenic species such as Cryptococcus neoformans Because the purine biosynthesis
92 se, and catalytically dead Tps2PD(D24N) from Cryptococcus neoformans bound to trehalose-6-phosphate (
95 ificity of V region-identical IgE and IgA to Cryptococcus neoformans capsular polysaccharide and foun
99 xin, Srx1, in oxidative stress resistance of Cryptococcus neoformans causing fungal meningoencephalit
101 aride capsule of the human pathogenic fungus Cryptococcus neoformans elicit diverse effects on fungal
105 e expulsion of the lethal endosomal pathogen Cryptococcus neoformans from mammalian macrophages, also
107 ed the role of a Th2 bias in pathogenesis of Cryptococcus neoformans H99 infection by comparing inhal
112 nistic fungal pathogens Candida albicans and Cryptococcus neoformans However, the molecular mechanism
113 e mAbs to the capsule of the fungal pathogen Cryptococcus neoformans impaired yeast budding by trappi
116 he development of the protective response to Cryptococcus neoformans in mice with cryptococcal pneumo
118 tes transmigration of the neurotropic fungus Cryptococcus neoformans into the brain parenchyma after
157 ity to progressive infection with the fungus Cryptococcus neoformans is associated with an allergic p
158 pulmonary infection with the fungal pathogen Cryptococcus neoformans is associated with the accumulat
159 ulmonary clearance of the encapsulated yeast Cryptococcus neoformans is associated with the CCR2-medi
162 the disease (cryptococcal disease) caused by Cryptococcus neoformans is incontrovertible, but whether
167 The manifestation of virulence traits in Cryptococcus neoformans is thought to rely on intracellu
169 andida sp., 49 Aspergillus fumigatus, and 33 Cryptococcus neoformans isolates were obtained from infe
170 es, 15 isolates of the Zygomycetes order, 10 Cryptococcus neoformans isolates, 8 Rhodotorula isolates
171 C. glabrata, and 53 C. krusei isolates), 35 Cryptococcus neoformans isolates, and 191 other clinical
172 Numerous virulence factors expressed by Cryptococcus neoformans modulate host defenses by promot
173 rable to infection by the encapsulated yeast Cryptococcus neoformans Most commonly found in the envir
176 of 1201 signature-tagged deletion strains of Cryptococcus neoformans mutants to identify previously u
178 xylomannan (GXM), the major component of the Cryptococcus neoformans polysaccharide capsule, hydrolyz
180 aride capsule of the human pathogenic fungus Cryptococcus neoformans promotes opsonization but also i
181 ulmonary clearance of the encapsulated yeast Cryptococcus neoformans requires a T1 adaptive immune re
184 of C57BL/6 mice with the moderately virulent Cryptococcus neoformans strain 52D models the complex ad
186 ential of these genes by comparing wild-type Cryptococcus neoformans strain H99 with deletant and com
187 of interferon-gamma transgene expression by Cryptococcus neoformans strain H99gamma in abrogating al
188 oducibility strains (4 Candida species and 6 Cryptococcus neoformans strains), and 746 isolates of Ca
189 otective immune responses to highly virulent Cryptococcus neoformans strains, such as H99, are associ
191 ability of the opportunistic fungal pathogen Cryptococcus neoformans to resist oxidative stress is on
192 ignalling pathways to the basal tolerance of Cryptococcus neoformans towards fluconazole, the widely
194 Most cases of cryptococcosis are caused by Cryptococcus neoformans var. grubii (serotype A), which
200 ave directly correlated phenotypic traits of Cryptococcus neoformans with clinical outcome of infecte
202 of lipid droplets during the interaction of Cryptococcus neoformans with macrophages in the presence
203 vestigated the outcome of the interaction of Cryptococcus neoformans with murine macrophages using la
204 rcular (Histoplasma capsulatum) to punctate (Cryptococcus neoformans) to labeling at the bud sites (C
205 a spp., 146 from 9 Aspergillus spp., 84 from Cryptococcus neoformans, 40 from 23 other mold species,
206 trophils have been shown to efficiently kill Cryptococcus neoformans, a causative agent of meningoenc
213 sential for virulence of the fungal pathogen Cryptococcus neoformans, and bacterial P5CDHs have been
214 hly active against a second fungal pathogen, Cryptococcus neoformans, and moderately active against a
215 can is a major component of the cell wall of Cryptococcus neoformans, but its function has not been i
217 n of clinically significant Candida species, Cryptococcus neoformans, Histoplasma capsulatum, and Bla
218 hannel from the model human fungal pathogen, Cryptococcus neoformans, is directly activated by the de
221 e fungal pathogens Aspergillus fumigatus and Cryptococcus neoformans, little was known about their in
222 Pathogens included Balamuthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis vi
225 The cell wall of pathogenic fungi such as Cryptococcus neoformans, provides a formidable barrier t
226 on and analysis of SRP in the human pathogen Cryptococcus neoformans, providing the first description
228 Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, result in more deaths annually
231 pathogens Candida albicans, C. glabrata and Cryptococcus neoformans, the food spoilage organism Zygo
237 r understanding Ab-mediated immunity against Cryptococcus neoformans, where the different isotypes ma
238 sis is important for virulence of the fungus Cryptococcus neoformans, which can cause lethal meningoe
239 ypic diversity in the human pathogenic yeast Cryptococcus neoformans, which is globally distributed a
240 D82 showed rapid and specific recruitment to Cryptococcus neoformans-containing phagosomes compared t
267 an parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryptococcus gattii We describe
268 ction with the opportunistic fungal pathogen Cryptococcus neoformans; however, the role of plasmacyto
270 molecule and the preferred carbon source for Cryptococcus, plays a critical role in fungal developmen
271 h from infection (including tuberculosis and cryptococcus) shortly after the initiation of antiretrov
276 sely known related species of the pathogenic Cryptococcus species complex, and it is non-pathogenic.
278 The yeast genera Kondoa might be protective; Cryptococcus species might also affect asthma severity.
279 etween C. amylolentus and related pathogenic Cryptococcus species provide evidence that multiple chro
281 to bipolar mating systems in the pathogenic Cryptococcus species, as well as its possible link with
282 led that, similar to those of the pathogenic Cryptococcus species, C. amylolentus has regional centro
285 g peptide-MHC class II molecules to identify Cryptococcus-specific Treg cells combined with genetic f
287 ght patients received ISAV for IFD caused by Cryptococcus spp. (n = 9), Paracoccidioides spp. (n = 10
291 cerebrospinal fluid (CSF) clearance rate of cryptococcus, termed early fungicidal activity, measured
292 transporter-like proteins (Hxs1 and Hxs2) in Cryptococcus that share the highest sequence identity wi
295 rationale to support continued investment in Cryptococcus vaccine research, potential challenges that
296 hat an F-box protein, Fbp1, is essential for Cryptococcus virulence independent of the classical viru
298 of the peripheral CD4(+) T-cell response to Cryptococcus was associated with disease severity and ou
300 nce procedures exist for Candida species and Cryptococcus yeasts; however, no standardized methods ha
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