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1 for virulence of the human pathogenic fungus Cryptococcus neoformans.
2 th a similar function in the related species Cryptococcus neoformans.
3 mall-RNA biogenesis in the pathogenic fungus Cryptococcus neoformans.
4 ual development of the human fungal pathogen Cryptococcus neoformans.
5 Candida species, Aspergillus fumigatus, and Cryptococcus neoformans.
6 ntainment following pulmonary challenge with Cryptococcus neoformans.
7 C57BL/6 mice after pulmonary infection with Cryptococcus neoformans.
8 hose for the pathogens, Candida albicans and Cryptococcus neoformans.
9 involved in polysaccharide O-acetylation in Cryptococcus neoformans.
10 haromyces cerevisiae, Malassezia furfur, and Cryptococcus neoformans.
11 illness caused by the opportunistic pathogen Cryptococcus neoformans.
12 ts (MC) arising from phenotypic switching of Cryptococcus neoformans.
13 transgene array in the human fungal pathogen Cryptococcus neoformans.
14 prolongs the survival of mice infected with Cryptococcus neoformans.
15 ghest prevalence of coinfection with HIV and Cryptococcus neoformans.
16 e host survival of the human fungal pathogen Cryptococcus neoformans.
17 sponse to infection with the fungal pathogen Cryptococcus neoformans.
18 tic cells (DC) during their interaction with Cryptococcus neoformans.
19 ssociated trait by the human fungal pathogen Cryptococcus neoformans.
20 nt role in protection against infection with Cryptococcus neoformans.
21 osis induced by intratracheal inoculation of Cryptococcus neoformans.
22 T cell responses to the opportunistic fungus Cryptococcus neoformans.
23 l time of mice infected intracerebrally with Cryptococcus neoformans.
24 nt role in immunity to the pathogenic fungus Cryptococcus neoformans.
25 ergillus isolates, 110 Candida isolates, and Cryptococcus neoformans.
26 Rho1 guanyl-nucleotide exchange factor) from Cryptococcus neoformans.
27 n their susceptibility to a fungal pathogen, Cryptococcus neoformans.
28 e major factor required for the virulence of Cryptococcus neoformans.
29 nd separation in the human pathogenic fungus Cryptococcus neoformans.
30 olog, Gib2, from the human pathogenic fungus Cryptococcus neoformans.
31 or many pathogenic fungal species, including Cryptococcus neoformans.
32 nsing pathway in the human pathogenic fungus Cryptococcus neoformans.
33 t regulates virulence in the fungal pathogen Cryptococcus neoformans.
34 IP5/IP7, which is essential for virulence of Cryptococcus neoformans.
35 central role in regulating the virulence of Cryptococcus neoformans.
36 d with the distantly related fungal pathogen Cryptococcus neoformans.
37 of H3K27 methylation (H3K27me) in the yeast Cryptococcus neoformans.
38 nown whether such an association exists with Cryptococcus neoformans.
41 a spp., 146 from 9 Aspergillus spp., 84 from Cryptococcus neoformans, 40 from 23 other mold species,
42 nfected with a moderately virulent strain of Cryptococcus neoformans (52D), which resulted in prolong
43 trophils have been shown to efficiently kill Cryptococcus neoformans, a causative agent of meningoenc
51 es for melanization of the pathogenic fungus Cryptococcus neoformans also offers unique opportunities
52 ulates not only mating but also virulence in Cryptococcus neoformans, an opportunistic human pathogen
54 of the relative contributions of individual Cryptococcus neoformans and Bacillus anthracis virulence
55 Aspergillus fumigatus, Alternaria alternata, Cryptococcus neoformans and Candida albicans) proteins w
56 e of macrophage autophagy in the response to Cryptococcus neoformans and Candida albicans, two import
61 ompare the molecular genotypes of strains of Cryptococcus neoformans and Cryptococcus gattii isolated
64 poxia-mimetic CoCl2 in the pathogenic fungus Cryptococcus neoformans and demonstrated that CoCl2 lead
65 rabidopsis thaliana, Caenorhabditis elegans, Cryptococcus neoformans and Drosophila melanogaster.
66 Rho-GDI homolog in the human fungal pathogen Cryptococcus neoformans and investigated its impact on p
67 erol endoperoxide, which were active against Cryptococcus neoformans and methicillin-resistance Staph
69 in GM-CSF are susceptible to infections with Cryptococcus neoformans and other opportunistic fungi.
71 AD-box RNA helicase in the pathogenic fungus Cryptococcus neoformans and proposes novel roles for thi
72 pase C1 (ISC1) gene from the fungal pathogen Cryptococcus neoformans and showed that it encodes an en
74 sential for virulence of the fungal pathogen Cryptococcus neoformans, and bacterial P5CDHs have been
75 heromone responses, mating, and virulence in Cryptococcus neoformans, and it also reiterates that the
76 hly active against a second fungal pathogen, Cryptococcus neoformans, and moderately active against a
77 ntamoeba histolytica, Trichomonas vaginalis, Cryptococcus neoformans, and Saccharomyces cerevisiae us
79 ive T cell responses to the pathogenic yeast Cryptococcus neoformans are dependent on heavily mannosy
81 gh activity toward Aspergillus fumigatus and Cryptococcus neoformans at acidic pH, yet remained nonto
82 e infections with pathogenic species such as Cryptococcus neoformans Because the purine biosynthesis
84 se, and catalytically dead Tps2PD(D24N) from Cryptococcus neoformans bound to trehalose-6-phosphate (
85 can is a major component of the cell wall of Cryptococcus neoformans, but its function has not been i
89 rated that the opportunistic fungal pathogen Cryptococcus neoformans can synthesize authentic immunom
90 hree common systemic human fungal pathogens--Cryptococcus neoformans, Candida albicans and Aspergillu
92 ificity of V region-identical IgE and IgA to Cryptococcus neoformans capsular polysaccharide and foun
98 xin, Srx1, in oxidative stress resistance of Cryptococcus neoformans causing fungal meningoencephalit
99 (MAbs) labeled with alpha-emitter 213Bi and Cryptococcus neoformans cells as well as between 213Bi-1
104 tudies, we showed that the pathogenic fungus Cryptococcus neoformans (Cn) produces a specific and uni
105 dismutases (SODs) in the encapsulated yeast Cryptococcus neoformans (Cn) variety gattii to analyse t
106 iodiomycete forming an induced DOPA-melanin, Cryptococcus neoformans (CN); and the slow-growing envir
107 fungal activity against Candida albicans and Cryptococcus neoformans comparable to amphotericin B and
109 D82 showed rapid and specific recruitment to Cryptococcus neoformans-containing phagosomes compared t
110 ith Pneumocystis jirovecii (pneumocystosis), Cryptococcus neoformans (cryptococcosis), Histoplasma ca
112 an parechovirus, varicella-zoster virus, and Cryptococcus neoformans/Cryptococcus gattii We describe
114 aride capsule of the human pathogenic fungus Cryptococcus neoformans elicit diverse effects on fungal
118 e expulsion of the lethal endosomal pathogen Cryptococcus neoformans from mammalian macrophages, also
120 he pathogenicity of a human fungal pathogen, Cryptococcus neoformans, GCS1 is not required for the pa
121 bservation of the altered pathogenicity of a Cryptococcus neoformans glucosylceramide (GlcCer) mutant
124 ed the role of a Th2 bias in pathogenesis of Cryptococcus neoformans H99 infection by comparing inhal
129 n of clinically significant Candida species, Cryptococcus neoformans, Histoplasma capsulatum, and Bla
130 nistic fungal pathogens Candida albicans and Cryptococcus neoformans However, the molecular mechanism
131 ction with the opportunistic fungal pathogen Cryptococcus neoformans; however, the role of plasmacyto
134 e mAbs to the capsule of the fungal pathogen Cryptococcus neoformans impaired yeast budding by trappi
137 he development of the protective response to Cryptococcus neoformans in mice with cryptococcal pneumo
138 ts for different levels of susceptibility to Cryptococcus neoformans in the mouse infection model.
139 (DC) have been shown to phagocytose and kill Cryptococcus neoformans in vitro and are believed to be
143 arization of T cells during murine pulmonary Cryptococcus neoformans infection in the secondary lymph
147 e highly susceptible to intratracheal (i.t.) Cryptococcus neoformans infection relative to BALB/c mic
148 ing the risk of dissemination and outcome of Cryptococcus neoformans infection were assessed in 111 o
149 cterium tuberculosis, Toxoplasma gondii, and Cryptococcus neoformans infection, implicating inflammat
151 tes transmigration of the neurotropic fungus Cryptococcus neoformans into the brain parenchyma after
188 The SREBP ortholog in the pathogenic fungus Cryptococcus neoformans is also activated by high levels
206 ity to progressive infection with the fungus Cryptococcus neoformans is associated with an allergic p
207 pulmonary infection with the fungal pathogen Cryptococcus neoformans is associated with the accumulat
208 ulmonary clearance of the encapsulated yeast Cryptococcus neoformans is associated with the CCR2-medi
211 the disease (cryptococcal disease) caused by Cryptococcus neoformans is incontrovertible, but whether
217 The manifestation of virulence traits in Cryptococcus neoformans is thought to rely on intracellu
218 hannel from the model human fungal pathogen, Cryptococcus neoformans, is directly activated by the de
221 andida sp., 49 Aspergillus fumigatus, and 33 Cryptococcus neoformans isolates were obtained from infe
222 es, 15 isolates of the Zygomycetes order, 10 Cryptococcus neoformans isolates, 8 Rhodotorula isolates
223 C. glabrata, and 53 C. krusei isolates), 35 Cryptococcus neoformans isolates, and 191 other clinical
224 andida isolates, 73 Aspergillus isolates, 53 Cryptococcus neoformans isolates, and 25 other fungal is
226 e fungal pathogens Aspergillus fumigatus and Cryptococcus neoformans, little was known about their in
227 Pathogens included Balamuthia mandrillaris, Cryptococcus neoformans, lymphocytic choriomeningitis vi
228 Isavuconazole showed good activities against Cryptococcus neoformans (MIC90, 0.12 mug/ml) and other n
229 Numerous virulence factors expressed by Cryptococcus neoformans modulate host defenses by promot
230 rable to infection by the encapsulated yeast Cryptococcus neoformans Most commonly found in the envir
234 of 1201 signature-tagged deletion strains of Cryptococcus neoformans mutants to identify previously u
235 s presenting with P. marneffei (n = 719) and Cryptococcus neoformans (n = 1598) infection to the Hosp
239 xylomannan (GXM), the major component of the Cryptococcus neoformans polysaccharide capsule, hydrolyz
240 b, and IgG3 with a 12-mer peptide mimetic of Cryptococcus neoformans polysaccharide revealed a stoich
243 aride capsule of the human pathogenic fungus Cryptococcus neoformans promotes opsonization but also i
245 gulatory networks in human pathogens such as Cryptococcus neoformans provides insights into host-path
246 The cell wall of pathogenic fungi such as Cryptococcus neoformans, provides a formidable barrier t
247 on and analysis of SRP in the human pathogen Cryptococcus neoformans, providing the first description
249 ulmonary clearance of the encapsulated yeast Cryptococcus neoformans requires a T1 adaptive immune re
250 ulmonary clearance of the encapsulated yeast Cryptococcus neoformans requires the development of T1-t
252 Candida albicans, Aspergillus fumigatus and Cryptococcus neoformans, result in more deaths annually
254 meningoencephalitis-causing fungal pathogen Cryptococcus neoformans, sexual development leads to the
258 of C57BL/6 mice with the moderately virulent Cryptococcus neoformans strain 52D models the complex ad
260 s in mice given a pulmonary infection with a Cryptococcus neoformans strain engineered to produce the
261 ential of these genes by comparing wild-type Cryptococcus neoformans strain H99 with deletant and com
262 of interferon-gamma transgene expression by Cryptococcus neoformans strain H99gamma in abrogating al
263 oducibility strains (4 Candida species and 6 Cryptococcus neoformans strains), and 746 isolates of Ca
264 otective immune responses to highly virulent Cryptococcus neoformans strains, such as H99, are associ
266 pathogens Candida albicans, C. glabrata and Cryptococcus neoformans, the food spoilage organism Zygo
272 in plants and animals, but in fungi such as Cryptococcus neoformans they are also associated with vi
274 bilities of 110 isolates of Candida spp. and Cryptococcus neoformans to amphotericin B, caspofungin,
275 nase Ste20, allows the human fungal pathogen Cryptococcus neoformans to grow at high temperature.
277 ability of the opportunistic fungal pathogen Cryptococcus neoformans to resist oxidative stress is on
278 rcular (Histoplasma capsulatum) to punctate (Cryptococcus neoformans) to labeling at the bud sites (C
279 ignalling pathways to the basal tolerance of Cryptococcus neoformans towards fluconazole, the widely
280 that in the pathogenic basidiomycetous yeast Cryptococcus neoformans, two Galpha subunits (Gpa2, Gpa3
283 The basidiomycetous human fungal pathogen Cryptococcus neoformans uniquely uses the stress-activat
284 Most cases of cryptococcosis are caused by Cryptococcus neoformans var. grubii (serotype A), which
288 cies/organism groups were isolated, of which Cryptococcus neoformans was the most common (31.2% of al
292 r understanding Ab-mediated immunity against Cryptococcus neoformans, where the different isotypes ma
293 or virulence factor of the pathogenic fungus Cryptococcus neoformans, which afflicts both immunocompe
294 sis is important for virulence of the fungus Cryptococcus neoformans, which can cause lethal meningoe
296 ypic diversity in the human pathogenic yeast Cryptococcus neoformans, which is globally distributed a
297 ave directly correlated phenotypic traits of Cryptococcus neoformans with clinical outcome of infecte
299 of lipid droplets during the interaction of Cryptococcus neoformans with macrophages in the presence
300 vestigated the outcome of the interaction of Cryptococcus neoformans with murine macrophages using la
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