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1 components of the host response to pulmonary Cryptococcus neoformans infection.
2 ha only during the first week of a pulmonary Cryptococcus neoformans infection.
3 tection was investigated in a mouse model of Cryptococcus neoformans infection.
4 ed immunity is required to clear a pulmonary Cryptococcus neoformans infection.
5 onse polarization using a model of pulmonary Cryptococcus neoformans infection.
6 immune mechanisms for controlling pulmonary Cryptococcus neoformans infection.
7 lpha is expressed in the lungs by day 6 post Cryptococcus neoformans infection and could play a role
8 e investigated the pathogenesis of pulmonary Cryptococcus neoformans infection and passive Ab efficac
10 n is a rational alternative to treatment for Cryptococcus neoformans infections, as these infections
12 body administration to mice with established Cryptococcus neoformans infection has been reported to p
13 cterium tuberculosis, Toxoplasma gondii, and Cryptococcus neoformans infection, implicating inflammat
18 arization of T cells during murine pulmonary Cryptococcus neoformans infection in the secondary lymph
24 e highly susceptible to intratracheal (i.t.) Cryptococcus neoformans infection relative to BALB/c mic
25 ll immunity is required to clear a pulmonary Cryptococcus neoformans infection (via the recruitment a
26 y of murine IgG1 mAb 2H1 to modify pulmonary Cryptococcus neoformans infection was investigated in in
27 ing the risk of dissemination and outcome of Cryptococcus neoformans infection were assessed in 111 o
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