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1 emia due to pituitary corticotroph adenomas (Cushing disease).
2 tary adenomas in 80% of patients with proven Cushing disease.
3 rbances mimicking hypercortisolism caused by Cushing disease.
4 ctive pituitary targeted pharmacotherapy for Cushing disease.
5 gnaling may be a novel strategy for treating Cushing disease.
6 EGFR might provide a therapeutic target for Cushing disease.
7 but the long-term survival of patients with Cushing disease after transsphenoidal surgery has not be
11 pling of the petrosal sinuses to distinguish Cushing disease from the ectopic adrenocorticotropic hor
12 ntral to peripheral ACTH were diagnostic for Cushing disease (> 2 before administration of CRH and >
15 ell as a handful of other cases of infantile Cushing disease in the literature, suggests that feature
18 ors in 43 patients (20 pheochromocytomas, 13 Cushing disease or syndrome, and 10 others), nonfunction
19 rom 16 of 20 patients with surgically proven Cushing disease (sensitivity, 80% [95% CI, 56% to 96%]).
20 rom 19 of 20 patients with surgically proven Cushing disease (sensitivity, 95% [CI, 75% to 99%]).
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