ライフサイエンスコーパス: D cell

1 receptors on the gastric SOM-secreting cell (D cell).

2 e gastrin or G cell, and the somatostatin or D cell.

3 gastrin from G cells, and somatostatin from D cells.

4 se new neurons through immature intermediate D cells.

5 stimulating the release of SOM from gastric D cells.

6 comitant decrease in somatostatin-expressing D cells.

7 ffin (EC) cells; neither was found in antral D cells.

8 itotic entry and catastrophe compared to DDR-D cells.

9 o increase VEGF in the media of cultured T47-D cells.

10 ted by progesterone receptors present in T47-D cells.

11 decrease after cell cycle induction in COMMA D cells.

12 their phenotype such that they resemble AhR-D cells.

13 gene expression differences between EVP and D cells.

14 M2 cells, and is termed here displaced or M-d cells.

15 fically deglutathionylated by GSTO1-1 in T47-D cells.

16 ginosa associated with plastic compared to 3-D cells.

17 hway, showed increased activities in beta1-k/d cells.

18 vbeta3-integrin was detected only in beta1-k/d cells.

19 vity and COL1A2 promoter activity in beta1-k/d cells.

20 enhanced COL1A2 promoter activity in beta1-k/d cells.

21 or the induction of apoptosis in infected M1-D cells.

22 e CLS for SSD1-V cells is twice that of ssd1-d cells.

23 ved by electron microscopy of infected HMVEC-d cells.

24 ssion of Bcl-2, which is not expressed in M1-D cells.

25 he role of LR during KSHV infection of HMVEC-d cells.

26 oxide synthase (nNOS) is present in gastric D-cells.

27 asin mutants and expressed them in 721.220-L(d) cells.

28 mote the hepatocyte differentiation of CD49f(D) cells.

29 NA levels and antral somatostatin-producing (D) cells.

30 le for increased VSV replication in Dicer1(d/d) cells.

31 intracellular Ca2+ release was decreased in DD cells.

32 ly rescue the APP and PSD95 up-regulation in DS cells.

33 actor in the enhanced metabolism of ara-C in DS cells.

34 n the cell cycle distributions of DS and non-DS cells.

35 classes ranging from LGN cells to V1 complex DS cells.

36 ight-evoked spike activity of neighboring ON DS cells.

37 induced Hog1 activation was observed in Pbs2(DD) cells.

38 ntiation of WEHI-3B myelomonocytic leukemia (D(+)) cells.

39 by drifting sinusoidal gratings (negative OS/DS cells); (2) suppressed-by-contrast cells; (3) cells w

40 ck proteins (HSPs) in heat shock-treated T47-D cells, a human breast cancer cell line that expresses

41 promote terminal differentiation of WEHI-3B D(+) cells, a phenomenon accompanied by the recovery of

42 oylphorbol-13-acetate was added to the HMVEC-d cells after 48 h p.i.

43 the sequence of appearance of the different D cells after a 7-day treatment with anti-mitotics.

44 ntrol of a laptop computer and powered by 10 D-cell alkaline batteries.

45 of gamma interferon (IFN-gamma)-activated M1-D cells also resulted in apoptosis but with no evidence

46 alactose supplementation, NPC1-deficient ldl-D cells also transported more cholesterol from lysosomes

47 electron-dense SGL cells, which we call type D cells and are derived from the astrocytes and probably

48 which reached a maximum at 2 h p.i. in HMVEC-d cells and at 8 h p.i. in HFF cells, and significantly

49 released mainly from gastric and pancreatic D cells and enteric neurons, inhibit peripheral cellular

50 ion very early during the infection of HMVEC-d cells and that ROS production was sustained over the o

51 uman dermal microvascular endothelial (HMVEC-d) cells and human foreskin fibroblast (HFF) cells in vi

52 uman dermal microvascular endothelial (HMVEC-d) cells and human foreskin fibroblast (HFF) cells is ch

53 ide the first description of the negative OS/DS cells and demonstrate that the SC segregates cells wi

54 were 2.6-fold higher in DS compared with non-DS cells and may be a factor in the enhanced metabolism

55 eas the dSC is enriched with the negative OS/DS cells and with cells with large RFs, low evoked FRs,

56 ), and have reduced numbers of somatostatin (D-cells) and peptide YY-expressing cells (L-cells).

57 addition to their direction selectivity (OMS-DS cells) and thereby combine sensitivity to two distinc

58 t one blastomere at the four-cell stage, the D cell, and its direct descendants play an important rol

59 focal adhesion significantly more in beta1-k/d cells, and activated alphavbeta3-integrin was detected

60 on secreting A-cells, somatostatin secreting D-cells, and gastrin secreting G-cells are absent from t

61 ON DS cells are coupled indirectly via gap junctions made w

62 The OMS-DS cells are further distinct from standard (non-OMS) DS

63 We propose that OFF inputs to ON DS cells are normally masked by a GABAergic inhibition,

64 Furthermore, although the salamander DS cells are OFF-type, there is a strong analogy to the

65 ationship with DP and found that a subset of DS cells are retained following each hair cycle, exhibit

66 BP-delta gene expression is induced in COMMA D cells arrested in G0 by serum and growth factor withdr

67 nally, we identify OS and two populations of DS cells at the superficial border of the tectal neuropi

68 raft blocked viral gene expression in HMVEC-d cells but not in HUVEC or HFF cells.

69 d mutant FA-D (Fanconi complementation group D) cells but not in mutant FA-A cells.

70 ith the early endosome marker EEA-1 in HMVEC-d cells, but rather with the late endosome marker LAMP1,

71 Introduction of AhR cDNA into AhR-D cells by stable transfection alters these characterist

72 are further distinct from standard (non-OMS) DS cells by their smaller receptive fields and different

73 In contrast to COMMA D cells, C/EBP-beta and C/EBP-delta mRNA levels remain r

74 l effect of cell contacts is to position the D cell cleavage furrow closer to the animal pole, and th

75 ti-biofilm peptide DJK-5 were evaluated on 3-D cells compared to a plastic surface, in medium with an

76 The CEs of 7-d cells contain significant amounts of the protein loric

77 , and the enhanced division asymmetry of the D cell contributes to the suppression of eye development

78 oclonal antibodies to hK2, we found that T47-D cells could be induced with androgens, mineralocortico

79 By contrast, two-dimensional (2-D) cell-counting methods are said to be 'assumption-base

80 icelles were evaluated in vitro in 2-D and 3-D cell culture and in vivo in xenograft models of ovaria

81 lles were not significantly more potent in 2-D cell culture in comparison to paclitaxel; however, the

82 idely developed for efficient and reliable 3-D cell culture research.

83 etection and demonstrated its utility in a 3-D cell culture system.

84 are typically grown using two-dimensional (2-D) cell culture approaches, where the native tumor micro

85 raphy techniques to produce 3 dimensional (3-D) cell culture substrates decorated with micron sized p

86 er, IL-4 stimulated SOM release from primary D cell cultures.

87 The latter population may underlie the type D cell current.

88 Type D cell currents had more hyperpolarized availability and

89 lasses of doublecortin and PSA-NCAM-positive D cells (D1, D2, D3) were observed.

90 The data suggest that IFN-gamma-activated M1-D cell death receptors become susceptible to their ligan

91 osylation, we used Chinese hamster ovary ldl-D cells defective in UDP-Gal/UDP-GalNAc 4-epimerase in w

92 Although the DS cell dendrite received the majority of its synaptic i

93 tly observed synaptic profile consisted of a DS cell dendrite receiving synapses from a cluster of se

94 make a putative inhibitory synapse with the DS cell dendrite.

95 offset of the direct inhibitory input to the DS cell dendrites.

96 nor apoptosis, whereas differentiated M1 (M1-D) cells developed a restricted virus infection and chan

97 Only in H2d/d cells did Ly49C/I dominantly inhibit Ly49D-Dd stimulat

98 majority of genes after UV, whereas pure XP-D cells did not.

99 Although Pbs2(DD) cells did not exhibit high basal levels of Hog1 phos

100 Significantly, both Pbs2(AA) and Pbs2(DD) cells displayed impaired stress resistance and atten

101 Fluorescence microscopy reveals that AhR-D cells do not exhibit an increased rate of death.

102 s provides a secondary mechanism by which ON DS cell ensembles can signal direction of stimulus motio

103 pears key to the direction selectivity of ON DS cells, evoking both an attenuation of spike frequency

104 Our results reveal that AhR-D cells exhibit a different morphology, decreased albumi

105 Because ldl-D cells express endogenous cell surface CD44, the effect

106 Both unactivated and activated M1-D cells expressed TRAIL receptors (R1 and R2), but only

107 Growth arrest is markedly delayed in COMMA D cells expressing a C/EBP-delta antisense construct.

108 appear to interact closely with clusters of D cells forming radial proliferative units.

109 Therefore, we investigated the role of NO in D-cell function and the effects of prolonged exposure of

110 Compared with 2-D, cells grown in 3-D had increased numbers of KSHV late

111 Type D cells had little or no current in 0.3 microM TTX at a

112 Only EVP cells and not TA and D cells had self-renewal capacity as demonstrated by col

113 Although COMMA-D cells harbor mutations in both alleles of p53, they ar

114 Conversely, D cells highly expressed genes related to differentiated

115 uman dermal microvascular endothelial (HMVEC-d) cells, human B (BJAB) cells, and Chinese hamster ovar

116 in FA-A cells but also in FA-B, FA-C and FA-D cells (i.e. in all FA cell lines tested), suggesting a

117 ECs but stimulates PTHrP production by Comma-D cells (immortalized murine mammary cells) and MCF-7 hu

118 at Salmonella established infection of the 3-D cells in a much different manner than that observed fo

119 ic acid increased differentiation of WEHI-3B D(+) cells in a manner that was analogous to the combina

120 d a severe reduction in recipient type (H-2b,d) cells in mice with active GVHD, whereas in protected

121 rong analogy to the systems of ON and ON-OFF DS cells in the mammalian retina.

122 d the kinetics of BeAn virus infection in M1-D cells, in order to temporally relate virus replication

123 ymetrix 430 2.0 arrays) revealed that WT and D(-) cells incubated with 8-CPT-cAMP have similar, but n

124 e in extramitochondrial Ccp1 isolated from 7-d cells, indicating that heme labilization results from

125 1(H) MKPs produced a partial effect on CD49f(D) cells, inducing the formation of hepatoepithelial lay

126 The D cell is activated by either gastrin or CCK and appears

127 One role of PRs in T47-D cells is regulating expression of vascular endothelial

128 uman microvascular dermal endothelial (HMVEC-d) cells, Kaposi's sarcoma-associated herpesvirus (KSHV)

129 Similar to the results for HMVEC-d cells, KSHV infection of HUVEC cells also resulted in

130 strocytes form baskets that hold clusters of D cells, largely insulating them from the hilus.

131 ith normal extracts, but extracts from an XP-D cell line exhibit twice the activity.

132 te Nkx6.3 as a selective regulator of G- and D-cell lineages, which are believed to derive from a com

133 DS cell lines also generated higher levels of ara-CTP co

134 In model B, injection of a anti-b/tol-d cells markedly reduced, but did not deplete, periphera

135 ell locomotion within a three-dimensional (3-D) cell mass, we have undertaken a systematic 3-D analys

136 othesis that enhanced metabolism of ara-C in DS cells may be a contributing factor to the superior su

137 Implanted QNR/D cells migrate only to the retinal ganglion and amacrin

138 normal forces play important roles even in 2-D cell migration.

139 inoglycosides were more efficacious in the 3-D cell model.

140 FBS, colistin was less efficacious in the 3-D cell model.

141 helial cell line Int-407 revealed that the 3-D cells more accurately modeled human in vivo differenti

142 hibition that temporally shifts firing of ON DS cell neighbors, resulting in a desynchronization of s

143 Kit(D)CD45(-)Ter119(-)), the remaining CD49f(D) cells neither differentiated nor survived in vitro.

144 ded on tissue culture-treated surfaces, by 4 d cell numbers and proliferation rates were significantl

145 A threefold decrease in the number of D cells occurred in H. pylori- and A. lwoffii-infected m

146 oreactivity were often in close proximity to D cells of the gastric and intestinal mucosa.

147 array system has the potential to improve 3-D cell oncology models and allow for better-controlled s

148 tional dysregulation compared with "pure" XP-D cells or WT cells.

149 minimum phase differences needed to generate DS cells overlaps that exhibited by non-DS simple cells.

150 However, we find that ON DS cell pairs show robust synchrony for all directions o

151 e distribution of viral capsids at the HMVEC-d cell periphery, and capsids did not associate with mic

152 cromeres in the establishment of the dorsal (D) cell quadrant was examined in C. lacteus by removing

153 logous to the typical spiralian A, B, C, and D cell quadrants.

154 The sustained, uncoupled ON DS cell ramifies completely within the lower cholinergic

155 wever, analysis of cell pairs indicates that DS cells receive cortical input from non-DS simple cells

156 From light micrographic 3-D cell reconstructions, the absolute volume, number, and

157 rythroid precursors and knockdown (KD) in ML-DS cells reduced their erythroid phenotype.

158 ses imply that the slowed growth rate of AhR-D cells reflects prolongation of G1.

159 tely 1100 transcripts were changed in WT and D(-) cells, respectively.

160 stochemistry of junctional markers on A549 3-D cells revealed that these cells formed tight junctions

161 promising profile regarding activity in T47-D cells, selectivity toward ERalpha and ERbeta, inhibiti

162 We conclude that 3-D cell shape information, transduced through tension-ind

163 Moreover, conditioned medium from PC3 PDGF-D cells significantly increased the tube formation of hu

164 Our results suggest that the two classes of DS cells specialize to encode motion direction of local

165 e found that the hK2 in androgen-induced T47-D cell spent media appears to be the pro-form of hK2 rat

166 CRISPR generated, NPC1-deficient ldl-D cells supplemented with galactose accumulated more cho

167 uman microvascular dermal endothelial (HMVEC-d) cell surface heparan sulfate (HS), integrins alpha3be

168 irus was highly restricted, BeAn-infected M1-D cells synthesized and appropriately processed virus ca

169 strate that the expression of GSTO1-1 in T47-D cells that are devoid of endogenous GSTO1-1 resulted i

170 osis in A-cells, and trans-acting factors in D-cells that can overcome this protection, resulting in

171 specifically on the mechanical properties of Dd cells, the results suggest that this behavior will be

172 ilar to the characteristic ON response of ON DS cells, the masked OFF response is also direction sele

173 ted cells suggests that susceptibility of M1-D cells to apoptosis may be controlled, in part, by expr

174 Exposure of canine D cells to tumor necrosis factor alpha in vitro reproduc

175 e considerably more responsive than parental D(+) cells to ATRA as a single agent, supporting the con

176 ion and the effects of prolonged exposure of D-cells to NO.

177 g the mechanisms of increased sensitivity of DS cells to ara-C related to the CBS gene may lead to th

178 apping of adult hair follicle dermal sheath (DS) cells to determine their lineage relationship with D

179 D(+) cells transfected with an expression plasmid contai

180 Unirradiated COMMA-D cells transplanted to mammary fat pads cleared previou

181 ments against an elastic cantilever, and a 2-d cell undergoing wave-like protrusion dynamics.

182 necessary for its efficient entry into HMVEC-d cells via macropinocytosis.

183 le, plausible neuronal circuits that produce DS cells via different mechanisms and tested these circu

184 Androgen induction of hK2 in T47-D cells was dose dependent.

185 PACAP action on D cells was measured by [Ca(2+)](i) and radioimmunoassay

186 howed differential expression between WT and D(-) cells, we found differences in cAMP-mediated regula

187 polymyxins) showed enhanced efficacy when 3-D cells were present.

188 Elevated ara-CTP levels in the DS cells were accompanied by slightly lower levels of en

189 as, thus unresponsive to DBA/2 (a anti-b/tol-d cells), were injected into (B6 x D2)F(1) --> B10.A mix

190 ntly inhibited both mTOR and Akt in PC3 PDGF-D cells, which were correlated with decreased cell proli

191 c or oxidative stress, activate Spc1 in wis1-DD cells, which express Wis1 protein that has the MEKK c

192 al ganglion cell is the direction selective (DS) cell, which responds vigorously to stimulus movement

193 cium signaling and somatostatin release from D cells with almost equal efficacy.

194 ubpopulation of weakly melanopsin-positive M-d cells with similarities to M2 cells.

195 Pretreatment of HMVEC-d cells with the antioxidant N-acetylcysteine (NAC) sign

196 antagonized p21 induction by Nutlin-3a, and (d) cells with high p21 levels were resistant to apoptosi

197 ing: (1) orientation/direction-selective (OS/DS) cells with a firing rate that is suppressed by drift

198 sa formed antibiotic resistant biofilms on 3-D cells without affecting cell viability.