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1 ubstrates (L-glucose, D-mannose, and 2-deoxy-D-glucose).
2 h a 1.7% overall yield from d-cellobiose and d-glucose.
3 T5, as well as a XylE mutant that transports D-glucose.
4 ate cleavage reactivity toward l-fucose over d-glucose.
5 ugar, 4,6-dideoxy-4-(3-hydroxybutanoylamino)-D-glucose.
6 ters that are insensitive to the presence of D-glucose.
7 l tissues were measured using [(3)H]-2-deoxy-D-glucose.
8 sence of the glycosylation inhibitor 2-deoxy-d-glucose.
9  dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl-D-glucose.
10 strate TDP-D-quinovose to TDP-3-keto-6-deoxy-d-glucose.
11 ntly diminished by stimulation of cells with d-glucose.
12 (3-hydroxybutanamido)-4,6-dideoxy-2-O-methyl-d-glucose.
13 utyrate, 4-(3-hydroxybutanamido)-4,6-dideoxy-d-glucose.
14 transporter, and by excess unlabeled 2-DG or d-glucose.
15 m the SpnQ substrate, TDP-4-keto-2,6-dideoxy-D-glucose.
16  C-3 deoxygenation of TDP-4-keto-2,6-dideoxy-D-glucose.
17 cid and phospholipid biosynthesis from (13)C D-glucose.
18 ounds in 16-21 % overall yield starting from D-glucose.
19 dynamic PET imaging of [(18)F]fluoro-2-deoxy-D-glucose.
20 ntial phosphoryl transfer steps using a beta-D-glucose 1,6-bisphosphate (betaG16BP) intermediate.
21 e ribose 5-phosphate and the activator alpha-D-glucose 1,6-bisphosphate (glucose 1,6-bisphosphate), a
22 se (betaPGM) catalyzes isomerization of beta-D-glucose 1-phosphate (betaG1P) into D-glucose 6-phospha
23 e evaluated as potential inhibitors of alpha-D-glucose 1-phosphate thymidylyltransferase (Cps2L), the
24 e phosphorolysis of GDP-D-glucose to GDP and D-glucose 1-phosphate.
25  cultured cells with 2-deoxy-2-[(14)C]carbon-D-glucose ([(14)C]2DG) support at least 35% higher [(18)
26  with PET and (18)F-labeled 6-fluoro-6-deoxy-d-glucose ((18)F-6FDG), a tracer that is transported but
27 ografts in mice using 2-deoxy-2-[F-18]fluoro-D-glucose ((18)F-FDG) PET imaging.
28 er clinical trial using (18)F-fluoro-2-deoxy-d-glucose ((18)F-FDG), (18)F-fluoromisonidazole ((18)F-F
29       The PET tracers 2-(18)F-fluoro-2-deoxy-d-glucose ((18)F-FDG), 3'-deoxy-3'-(18)F-fluorothymidine
30 elling cells with 2'-[(18)F]-fluoro-2'-deoxy-D-glucose ((18)F-FDG).
31 pic imaging (MRSI) and [(18)F]fluoro-2-deoxy-D-glucose ((18)FDG) positron emission tomography (PET) t
32  emission tomography (PET) and [(18)F]fluoro-D-glucose ((18)FDG) to measure brain glucose metabolism
33 d women using PET and 2-deoxy-2[(18)F]fluoro-D-glucose ((18)FDG).
34   The glucose analog [(18)F]fluoro-2-deoxy-2-d-glucose ([(18)F]-FDG) is commonly used in PET/CT that
35 ted macrophages with 2-deoxy-2-[(18)F]fluoro-D-glucose ([(18)F]FDG) has been proposed for identificat
36 MDV2 was superior to 2-[(18)F]fluoro-2-deoxy-d-glucose ([(18)F]FDG) in imaging the BxPC-3 tumors.
37 ured BAT activity by 2-deoxy-2-[(18)F]fluoro-d-glucose ([(18)F]FDG) positron emission tomography/comp
38 d 15% per MBq/ml of 2-deoxy-2-[(18)F]-fluoro-d-glucose ([(18)F]FDG).
39 20 years after Fischer's first synthesis of (D)-glucose (1890), we are witnessing important developme
40 tol (1-[(18)F]FDAM), 2-deoxy-2-[(18)F]fluoro-d-glucose (2-[(18)F]FDG), and 6-deoxy-6-[(18)F]fluoro-d-
41                   The combination of 2-deoxy-D-glucose (2-DG) and UA-4 induced cell cycle arrest in G
42                   The glucose analog 2-deoxy-D-glucose (2-DG) mimics CR effects in several animal mod
43 eted drugs (MTD) in combination with 2-deoxy-d-glucose (2-DG), a compound that inhibits glycolysis.
44                                      2-Deoxy-d-glucose (2-DG), a synthetic glucose analogue that acts
45 traperitoneal-administered 350 mg/kg 2-deoxy-D-glucose (2-DG), on food intake were measured in OBX an
46 apy using periocular carboplatin and 2-deoxy-d-glucose (2-DG).
47 SGLTs and GLUTs; and 2-deoxy-2-[F-18]-fluoro-d-glucose (2-FDG), a substrate for GLUTs.
48 obenz-2-oxa-1, 3-diazol-4-yl) amino]-2-deoxy-D-glucose (2-NBDG) reports on glucose uptake and Tetrame
49 1) pivotal for intestinal mass absorption of d-glucose, 2) triggers the glucose-induced secretion of
50 Cs) treated with DL-Hcy (500 micromol/L) and d-glucose (25 mmol) for 48 h.
51 lysis using the glycolytic inhibitor 2-deoxy-D-glucose (2DG) in experimental models of seizures and e
52 with low doses of the glucose analog 2-deoxy-d-glucose (2DG) on ADPKD progression in orthologous and
53 ycolysis and N-linked glycosylation, 2-deoxy-D-glucose (2DG), potently inhibited surface expression o
54 ples were collected from control and 2-deoxy-d-glucose (2DG)-injected rats for Western blot analysis
55 ivated by glucoprivation, induced by 2-deoxy-D-glucose (2DG).
56 zole (NDI) with glycolytic inhibitor 2-deoxy-d-glucose (2DG).
57 aterally into A1/C1 and responses to 2-deoxy-D-glucose (2DG; 200 mg/kg)-induced glucoprivation were t
58 iosynthetic enzyme (rmlC; TDP-4-keto-6-deoxy-d-glucose 3,5-epimerase), the ATP binding cassette (ABC)
59 substrate for SGLTs; 4-deoxy-4-[F-18]-fluoro-d-glucose (4-FDG), a substrate for SGLTs and GLUTs; and
60 -6-phosphate into mixtures of labeled methyl d-glucose-4,6-phosphates, which were analyzed by (31)P N
61 ical allosteric mechanism in human UDP-alpha-d-glucose 6-dehydrogenase (hUGDH) based on an easily acq
62 of beta-D-glucose 1-phosphate (betaG1P) into D-glucose 6-phosphate (G6P) via sequential phosphoryl tr
63 ent equatorial transamination of 3-oxo-alpha-D-glucose 6-phosphate to form alpha-D-kanosamine 6-phosp
64  (2-[(18)F]FDG), and 6-deoxy-6-[(18)F]fluoro-d-glucose (6-[(18)F]FDG) was studied in EMT6 cells, tumo
65 her non-specific glycolytic proteins such as d-glucose-6-phosphate dehydrogenase.
66 in combination with conversion of the formed d-glucose-6-phosphate into mixtures of labeled methyl d-
67 othelial cell (HUVECs), challenged with high D-glucose (60% inhibition), hydrogen peroxide (80% inhib
68 at the conversion of TDP-4-amino-4,6-dideoxy-D-glucose (8) to TDP-3-keto-4,6-dideoxy-D-glucose (9) is
69 eoxy-D-glucose (8) to TDP-3-keto-4,6-dideoxy-D-glucose (9) is catalyzed by DesII, which is a member o
70 cose (UDP-viosamine) from UDP-4-keto-6-deoxy-D-glucose, a key compound involved also in the biosynthe
71 rystalline form 3-nitro-3,4,6-trideoxy-alpha-D-glucose, a nitro sugar stereochemically homologous to
72 nvolved in the synthesis of 1-O-galloyl-beta-d-glucose, a precursor for the synthesis of hydrolysable
73                           Because intestinal D-glucose absorption is mediated by SGLT1 localized in t
74                In wild-type mice, passage of D-glucose across the intestinal BBM was predominantly me
75     The comparison of growth on L-fucose and D-glucose allows first insights into the genome-wide cha
76              In contrast, in GA cockroaches, D-glucose also stimulated bitter-GRNs and suppressed the
77                     The insulin secretagogue D-glucose also stimulates beta-cell p38 MAPK phosphoryla
78 that varying the position of substitution of d-glucose alters not only the cellular uptake and cytoto
79                                Using 2-deoxy-d-glucose, an inhibitor of glucose uptake, and compound
80 enumber shifts were 50cm(-1)/mM obtained for d-(+)-glucose and 96cm(-1)/mug/mL for Cy5-conjungated Ra
81 e gut microbiota metabolites modulated (14)C-D-glucose and (14)C-deoxy-D-glucose uptake into hepatic
82 bated with (1) normal (5 mM) or high (25 mM) D-glucose and (2) control medium, TNF-alpha (10 ng/mL),
83 s an unbranched heteropolymer with repeating d-glucose and 6-deoxy-l-talose residues in which the 6-d
84 phase diagrams of the food ingredients alpha-d-glucose and citric acid, along with sodium sulfate, we
85 e real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentrations gave unique resu
86 r real-time monitoring of sucrose, sorbitol, d-glucose and d-fructose concentrations is reported.
87 tyl-D-glucosamine, N-acetyl-D-galactosamine, D-glucose and D-galactose, present on the cell surface.
88 ting that the tricyclic core is derived from d-glucose and d-ribose, whereas the tiglyl moiety is der
89 asons hampering simultaneous fermentation of D-glucose and D-xylose, two primary sugars present in li
90 lucose negatively affected transport of both D-glucose and D-xylose.
91 rated the requirement for GDP-d-mannose, UDP-d-glucose and dTDP-l-rhamnose in Psl production and surf
92 ich is selectively fueled by the presence of D-glucose and ethyl butyrate.
93                 Insulin secretion induced by d-glucose and forskolin is amplified by overexpressing i
94  1,3-dioxane derivative was synthesized from d-glucose and found to be a highly stereoselective templ
95 ristic structures in melanoidins formed from d-glucose and l-alanine between 130 and 200 degrees C.
96 ompared by detecting two different analytes: d-glucose and l-cysteine under nonspecific and specific
97                There is no interference from d-glucose and l-glutamic acid, ascorbic acid and o-nitro
98 he elaboration of biosensors for D-sorbitol, D-glucose and L-lactate with using D-sorbitol dehydrogen
99 eating pentasaccharide containing d-mannose, d-glucose and l-rhamnose: [See text].
100 occluded conformations present multiple beta-d-glucose and maltose interaction sites, whereas inward-
101 clearance of non-metabolizable [(3) H]methyl-d-glucose and placental SLC2A8 (glucose transporter 8) g
102 composed of two units of beta-(1-->6)-linked d-glucose and resembles the carbohydrate moiety of lipid
103 hat secrete insulin/C-peptide in response to D-glucose and theophylline.
104 microaerophilic character, it can metabolize D-glucose and/or D-galactose in both oxidative and ferme
105          We monitor environmentally (2-deoxy-D-glucose) and genetically (DeltaPFK2) perturbed Sacchar
106 conditions mimicking diabetes mellitus (high D-glucose) and ischemia-associated starvation (low growt
107 oice between metabolizable sugar (sucrose or D-glucose) and nonmetabolizable (zero-calorie) sugar (su
108 ; and 4) orexin glucosensors detect mannose, d-glucose, and 2-deoxyglucose but not galactose, l-gluco
109 rmer presents overlapping endofacial WZB117, d-glucose, and CB binding envelopes.
110 ncluding L-arabinose, D-fucose, D-galactose, D-glucose, and D-xylose.
111 levels of O-ethyl beta-d-glucopyranoside and d-glucose, and lower levels of malic, quinic and ascorbi
112 d-glucosamine versus 2,3-diamino-2,3-dideoxy-d-glucose, and phosphorylation status all correlated wit
113 A-RFs are filled with different solutions of d-glucose, and the Deltalambdapeak is measured in real t
114 te between lactulose, l-rhamnose, 3-O-methyl-d-glucose, and xylose.
115           For example, metabolisms viable on D-glucose are 1835 times more likely to give rise to met
116 e details of the ROA spectrum of methyl-beta-D-glucose are found to be highly sensitive to solvation
117 o SAM using TDP-[3-(2)H]-4-amino-4,6-dideoxy-D-glucose as the substrate provides strong evidence for
118 lacental uptake and clearance of (3)H-methyl-D-glucose at D19.
119 h dynamic PET imaging of [18F]fluoro-2-deoxy-D-glucose at two occasions with 24-hour interval between
120  epoxidation of d-xylose-based oxepine 1 and d-glucose-based oxepines 2 and 5 reported earlier, suppo
121 fter an injection of [(18)F]2-fluoro-2-deoxy-d-glucose before the OGTT, and the rate of glucose absor
122 ted that the major neutral sugars were alpha-D-glucose, beta-D-glucose, rhamnose and D-glucuronic aci
123       Only the MSPRS was able to detect beta-D-glucose binding to glucose oxidase.
124 ynamic [(15)O]H2O and [(18)F]-fluoro-2-deoxy-d-glucose brain positron emission tomography scans to me
125 ucose metabolism, as measured by [18F] deoxy-D-glucose brain positron emission tomography.
126 s likely that alterations sterically prevent D-glucose but not D-xylose from entering the pocket.
127 observed that platelet exposure to 25 mmol/L d-glucose, but not to iso-osmolar mannitol, 1) reduced t
128 with D-ribo configuration, was prepared from D-glucose by inverting the C-3 stereocenter to introduce
129               PET with [(18)F]fluoro-2-deoxy-D-glucose can be used to image cellular metabolism, whic
130                 PET with [18F]fluoro-2-deoxy-D-glucose can be used to image cellular metabolism, whic
131 accharides (L-arabitol, D-fructose, sucrose, D-glucose, cellotetraose, cellulose, and starch) were tr
132 19, but not D16, transplacental (3) H-methyl-d-glucose clearance was reduced by 33% in corticosterone
133 d increase in food consumption, (3) H-methyl-d-glucose clearance was similar to the controls.
134 ransduction, develop a robust preference for d-glucose compared with isocaloric l-serine independentl
135                                     The beta-d-glucose-containing compound 3, bearing 2-chlorothiophe
136 shown to induce downregulation of the sodium-D-glucose cotransporter 1 (SGLT1) and of the concentrati
137                                        Na(+)-d-glucose cotransporter 1 (SGLT1) is rate-limiting for g
138   To clarify the physiological role of Na(+)-D-glucose cotransporter SGLT1 in small intestine and kid
139 ther L-alanine or a mixture of L-asparagine, D-glucose, D-fructose, and potassium ions.
140                                              D-glucose, D-fructose, saccharin, D-mannitol, and water
141  where transport activities of radiolabelled D-glucose, D-galactose and 2-deoxy-D-glucose were restor
142       The current method has been applied to d-glucose, d-galactose, and d-xylose donors with a nondi
143 biologically relevant underivatized hexoses, d-glucose, d-galactose, d-mannose, and d-fructose, using
144 esis of beta-C-glycopyranosyl aldehydes from D-glucose, D-mannose, and D-galactose.
145 purified protein showed quenching by 2-deoxy-D-glucose, D-mannose, D-glucose or D-galactose in the pr
146 dition of a flavour enhancer solution (FES) (d-glucose, d-ribose, l-cysteine and thiamin) and of sous
147 lactate with using D-sorbitol dehydrogenase, D-glucose dehydrogenase and L-lactate dehydrogenase resp
148                                              d-glucose-dependent suppression of BKCa channel beta1 su
149  aminocyclization/lactamization of d-mannose/D-glucose derived C5-gamma-azido esters as a key step wh
150 hly diastereoselective Barbier reaction on a d-glucose-derived aldehyde.
151 an lead to inaccuracies in amperometric beta-D-glucose determinations.
152                                    Moreover, d-glucose-evoked increases in cytosolic ATP and d-glucos
153 apsed disease, using 2-deoxy-2-[(18)F]fluoro-D-glucose (FDG) and 3-deoxy-3[(18)F]fluorothymidine (FLT
154 ebral metabolism using [(18)F]fluoro-2-deoxy-d-glucose (FDG) in Alzheimer's disease (AD) patients, su
155 ed with the glucose analog, 2-fluoro-2-deoxy-D-glucose (FDG) in vivo.
156 d in real time using 2-deoxy-2-[(18)F]fluoro-d-glucose (FDG) positron emission tomography (PET)/compu
157                 Fluorine-18 2-fluoro-2-deoxy-D-glucose (FDG) positron emission tomography (PET)/compu
158                Although 2[18F]fluoro-2-deoxy-d-glucose (FDG) uptake during positron emission tomograp
159 e origin of the (18)fluorine-labeled 2-deoxy-D-glucose (FdG) uptake signals observed clinically.
160 eled glucose analogue 2[(18)F]fluoro-2-deoxy-D-glucose (FDG).
161 ly allylated sugar derivatives, derived from D-glucose, followed by a sequential ring-closing metathe
162 cultured in 5 mm normal glucose, 25 mm l- or d-glucose for 48 h (osmotic control and high glucose tre
163 ose phosphorylase may function to remove GDP-D-glucose formed by GDP-D-mannose pyrophosphorylase, an
164 ivity of the P3/P4 and propylene-linked beta-d-glucose fragments, stronger in fIIa (15.5 kJ.mol(-1))
165 available in enantiomerically pure form from D-glucose, has given rise to a series of intermediates w
166                                         High D-glucose (HDG) significantly increased PLK2 expression
167         Fungal beta-glucans are comprised of d-glucose homopolymers containing beta-1,3-linked glucos
168 0 M, with a low detection limit of 0.01 M of d-glucose (i.e., 1.80 ppm), a sensitivity of 4.93 nm M(-
169 n using PET imaging of [(18)F]fluoro-2-deoxy-D-glucose in a porcine experimental model of early acute
170 croinjections of 2-deoxy-D-glucose or 5-thio-D-glucose in anesthetized, euglycemic rats.
171 promise for the use of topotecan and 2-deoxy-D-glucose in children.
172 hydrates in ionic liquids, the solubility of d-glucose in four ionic liquids was measured within a te
173 of beta1,4-mannobiose to 4-O-beta-d-mannosyl-d-glucose in mannan metabolism.
174 n-natural phenyl cyclopropanes directly from D-glucose in single-vessel fermentations.
175 oxy-d-glucose to form TDP-3-keto-4,6-dideoxy-d-glucose in the biosynthesis of TDP-d-desosamine.
176                         The concentration of D-glucose in the blood following glucose gavage increase
177 gnificantly, we found an accumulation of GDP-D-glucose in the C10F3.4 mutant worms, suggesting that t
178 -dideoxy-D-glucose to TDP-3-keto-4,6-dideoxy-D-glucose in the desosamine biosynthetic pathway.
179 esized in cells incubated in 5 mM [U-(13) C]-D-glucose in the presence and absence of unlabelled mann
180  dehydrated product, CDP-4-keto-3,6-dideoxy- d-glucose, in the ascarylose biosynthetic pathway.
181 eated with the hexokinase inhibitor, 2-deoxy-d-glucose, indicating that a functional glycolytic pathw
182 ments, the receptor region was identified by D-glucose infusion of isolated regions.
183  before repeating experiments using water or D-glucose infusion.
184 ments performed following [U-(2)H7, U-(13)C]-D-glucose injections.
185                               The effects of d-glucose, insulin, and gonadal and adrenal steroids on
186                               The effects of d-glucose, insulin, and gonadal and adrenal steroids on
187 ard conformations present only a single beta-d-glucose interaction site.
188 natively triggers the inversion of one alpha-d-glucose into a 5-C-acetoxy-beta-l-idose unit possessin
189  dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl-d-glucose into its C-3' nitro derivative.
190 4-(3-hydroxy-3-methylbutanamido)-4,6-dideoxy-d-glucose is found in the side chain of the capsular pol
191                                        Thus, D-glucose is processed as both a phagostimulant and dete
192 T) with FDG-glucose (2-[(18)F]fluoro-2-deoxy-d-glucose) is already being used as a metabolic imaging
193 y of sugars referred to D, as in D-ribose or D-glucose, is not an independent mystery.
194     As a result of this analysis, l-glucose, d-glucose, l-allose, d-allose, d-gulose, d-galactose, an
195 trations and perform steady-state [U-(13) C]-D-glucose labelling.
196                                  Infusion of D-glucose led to 2.9-fold up-regulation in SGLT1 compare
197 tional capacities of ECs cultured under high D-glucose/low growth factors.
198 r) 160 kDa) with its natural substrate (beta-D-glucose, M(r) 180 Da) from its interactions with nonsp
199                              SEC analyses of d-glucose model reactions with and without l-pyroglutami
200                              Heating aqueous d-glucose model reactions with l-glutamine and l-alanine
201 (SS)-catalyzed assembly of (alpha1-4)-linked d-glucose molecules into maltodextrins generally agree t
202 larimetric measurements showed a doubling of d-glucose mutarotation velocity and HPLC analyses of d-f
203  interact with the C6 hydroxymethyl group of D-glucose negatively affected transport of both D-glucos
204 ad a significant effect on [F]fluoro-2-deoxy-D-glucose net uptake rate Ki in high-strain lipopolysacc
205  the glycation process occurs as a result of d-glucose nonenzymatically reacting with proteins such a
206          Cross-reactivity with d-lactose and d-(+)-glucose occurred only at concentrations >10(4)-fol
207 M was elicited by microinjections of 2-deoxy-D-glucose or 5-thio-D-glucose in anesthetized, euglycemi
208 d quenching by 2-deoxy-D-glucose, D-mannose, D-glucose or D-galactose in the presence of sodium ions.
209 ltures grown on rich media supplemented with d-glucose or glycerol produce H2 and simultaneously cons
210 nteers incubated for 60 min with 5-25 mmol/L d-glucose or iso-osmolar mannitol, we evaluated the infl
211 of B6.Sle1Sle2.Sle3 mice with either 2-deoxy-D-glucose or metformin were sufficient to prevent autoim
212                3-Acetamido-3,6-dideoxy-alpha-d-glucose or Quip3NAc is an unusual deoxyamino sugar fou
213                3-Acetamido-3,6-dideoxy-alpha-D-glucose or Quip3NAc is an unusual dideoxy sugar found
214 res and subsequent germination with inosine, d-glucose, or l-valine, respectively, germinate very poo
215 eletion strain was found to totally lack GDP-D-glucose phosphorylase activity; this activity was also
216 10F3.4 mutant worms, suggesting that the GDP-D-glucose phosphorylase may function to remove GDP-D-glu
217                The highest expression of GDP-D-glucose phosphorylase was found in the nervous and mal
218 C15orf58 gene expression products as the GDP-D-glucose phosphorylases of these organisms.
219  the rate of intracellular [F]fluoro-2-deoxy-D-glucose phosphorylation.
220 and is exploited with (18)F-2-fluoro-2-deoxy-d-glucose positron emission tomography ((18)F-FDG-PET) t
221 gnostic role of interim [18F]-fluoro-2-deoxy-D-glucose positron emission tomography (FDG-PET) perform
222 brain glucose concentrations) with 1-[(11)C]-d-glucose positron emission tomography during hyperinsul
223 a phenomenon used in 2-[(18)F]fluoro-2-deoxy-D-glucose positron emission tomography imaging of solid
224 ormal subjects with [(18)F]-2-fluoro-2-deoxy-D-glucose positron emission tomography imaging.
225 he utility of 2-[fluorine-18]-fluoro-2-deoxy-D-glucose positron emission tomography integrated with c
226 ied by FDG-PET-CT (2-[(1)(8)F]fluoro-2-deoxy-D-glucose-positron emission tomography combined with com
227               Early restaging fluoro-2-deoxy-D-glucose-positron emission tomography scans appear to p
228 glucose uptake with [(18) F]2-fluoro-2-deoxy-D-glucose/positron emission tomography, lipolysis (RaGly
229 5,6-dihydrouracil, hesanal, cis-olefin, beta-D-glucose, propanal and some unassigned species.
230 tyl-d-quinovosamine (2-acetamido-2,6-dideoxy-d-glucose, QuiNAc) occurs in the polysaccharide structur
231 fically, we find that high concentrations of D-glucose rapidly signal through AtWNK8 and AtWNK10, whe
232 to the C2 atom of the intermediate to form a d-glucose residue.
233 close analogues of cyclodextrins composed of d-glucose residues and triazole units bound together thr
234 lobiose 2-epimerase (CE) reversibly converts d-glucose residues into d-mannose residues at the reduci
235  Glucoamylases (GAs) from a wild and a deoxy-d-glucose-resistant mutant of a locally isolated Aspergi
236 oduct was formed, whereas the use of 2-deoxy-d-glucose resulted in reduced chemo- and stereoselectivi
237 available in enantiomerically pure form from d-glucose, resulted in the formation of two diastereomer
238 or neutral sugars were alpha-D-glucose, beta-D-glucose, rhamnose and D-glucuronic acid.
239 ion tomography with [(18)F]-2-fluoro-2-deoxy-D-glucose scan in addition to noncontrast computed tomog
240 nical ventilation, dynamic [F]fluoro-2-deoxy-D-glucose scans were acquired to quantify metabolic acti
241  equimolar concentration of L-asparagine and D-glucose showed a significant inhibition of acrylamide
242                              After gavage of d-glucose, small intestinal glucose absorption across th
243 alose, d-(+)-xylose, d-fructose, 1-thio-beta-d-glucose sodium salt, d-(+)-galactose, sorbitol, glycer
244                      V7 is still highly beta-D-glucose specific, highly active with the quinone diimi
245  is an oxidoreductase exhibiting a high beta-D-glucose specificity and high stability which renders g
246 cose-averse (GA) cockroaches, D-fructose and D-glucose stimulated sugar-gustatory receptor neurons (G
247 lucose-evoked increases in cytosolic ATP and d-glucose-stimulated insulin secretion were diminished i
248 hus, our findings are in accord with 2-Deoxy-D-glucose studies performed in V1 of macaques and studie
249 s the synthesis of all positional isomers of d-glucose substitution for platinum warheads with detail
250 In this study, we investigated the effect of d-glucose substitution position on the biological activi
251 zymatic synthesis of TDP-4-amino-4,6-dideoxy-d-glucose that couples a transamination equilibrium to t
252 ibution analysis using 2-deoxy-2-[18F]fluoro-D-glucose that reprogramming of intestinal glucose metab
253 ersion of dTDP-3-amino-2,3,6-trideoxy-4-keto-D-glucose to dTDP-3-amino-2,3,6-trideoxy-4-keto-3-methyl
254 s the deamination of TDP-4-amino-4,6-dideoxy-d-glucose to form TDP-3-keto-4,6-dideoxy-d-glucose in th
255  the C-3 deoxygenation of CDP-4-keto-6-deoxy-D-glucose to form the dehydrated product, CDP-4-keto-3,6
256 lyze C-2 deoxygenation of TDP-4-keto-6-deoxy-D-glucose to form the SpnQ substrate, TDP-4-keto-2,6-did
257 y these enzymes is the phosphorolysis of GDP-D-glucose to GDP and D-glucose 1-phosphate.
258 inated C-linked glycosides of D-galactose or D-glucose to L-cysteine using thiol-ene "click" chemistr
259 s the deamination of TDP-4-amino-4,6-dideoxy-D-glucose to TDP-3-keto-4,6-dideoxy-D-glucose in the des
260 vidual component polyphenols inhibited (14)C-D-glucose transport across differentiated Caco-2/TC7 cel
261 ylococcus epidermidus, homologs of the human D-glucose transporters, the GLUTs (SLC2), provide inform
262 diated cells even in glucose-free or 2-deoxy-D-glucose-treated conditions.
263 2+) uptake in response to d-glucose, whereas d-glucose-triggered cytosolic Ca(2+) oscillations remain
264 1.1.22) catalyze the conversion of UDP-alpha-d-glucose (UDP-Glc) to the key metabolic precursor UDP-a
265 yze the formation of UDP-4-amino-4,6-dideoxy-D-glucose (UDP-viosamine) from UDP-4-keto-6-deoxy-D-gluc
266 lulose oligomers consisting 2, 4, and 6 beta-d-glucose units are examined in explicit solvent using r
267 se-free medium or in the presence of 2-deoxy-D-glucose upon CCCP treatment.
268 nine (Q282A) doubled the Km(app) for 2-deoxy-d-glucose uptake and eliminated cis-allostery (stimulati
269                                    A 2-deoxy-d-glucose uptake assay indicates that depletion of septi
270 es modulated (14)C-D-glucose and (14)C-deoxy-D-glucose uptake into hepatic HepG2 cells.These data ind
271 idal strain enhances local [F]fluoro-2-deoxy-D-glucose uptake primarily by increasing the rate of int
272                          [18F]fluoro-2-deoxy-D-glucose uptake rate was computed for the total lung, f
273                        [(18)F]fluoro-2-deoxy-D-glucose uptake rate was computed for the whole lung, f
274              Overload-induced [(3)H]-2-deoxy-d-glucose uptake was not inhibited by d-fructose, demons
275 s, muscle weights and ex vivo [(3)H]-2-deoxy-d-glucose uptake were assessed.
276 porters (GLUTs) were monitored by 3-O-methyl-D-glucose uptake.
277 dal hyperinflation had [(18)F]fluoro-2-deoxy-D-glucose uptakes similar to controls.
278 te gravitational zones [(18)F]fluoro-2-deoxy-D-glucose uptakes were higher in ventilator-induced lung
279 aboration to their C14-epimers starting from d-glucose using beta-glycosylation and Grubbs olefin cro
280 e was synthesized from the bis(acetonide) of d-glucose using dicyclopentadienylzirconium(0)-mediated
281 st class are defective in both virulence and d-glucose utilization as a result of mutations to residu
282 ptical activity (ROA) spectra of methyl-beta-D-glucose utilizing density functional theory combined w
283 inct hexoses including the key carbon source D: -glucose, various glucose epimers, and several acetyl
284 as performed with injection of 100 mL of 20% d-glucose via the cubital vein.
285 es were lower than maximal rates for 2-deoxy-d-glucose (Vmax of 224 and 32 pmol/min/oocyte for GLUT2
286 sis to produce [2,3,4,6,6-(2)H5, 3,4-(13)C2]-D-glucose was developed to improve the (13)C signal-to-n
287                                         When d-glucose was used as a starting material, only the fura
288 omography (PET) with [(18)F]2-fluoro-2-deoxy-D-glucose was used to measure changes in regional brain
289 channels in patch-clamp experiments, whereas D-glucose was without effect.
290                                Starting from d-glucose, we developed a divergent synthetic route to t
291 and beta-casein formed during glycation with d-glucose were identified and monitored in binary system
292 olabelled D-glucose, D-galactose and 2-deoxy-D-glucose were restored, consistent with the expected sp
293 d mitochondrial Ca(2+) uptake in response to d-glucose, whereas d-glucose-triggered cytosolic Ca(2+)
294 cheap synthesis of 99.4% pure L-glucose from D-glucose which requires purification of neither interme
295 etabolism, and a treatment combining 2-deoxy-D-glucose, which inhibits glucose metabolism, and metfor
296  transporters are competitively inhibited by D-glucose, which is one of the major reasons hampering s
297 lation of hydroxy protected 1,6-anhydro-beta-d-glucose with arylalanes to provide beta-C-arylglucosid
298 ecifically binds the vir gene-inducing sugar d-glucose with high affinity.
299 ileged sites for the nonenzymatic binding of d-glucose with HSA.
300 s were incubated ex vivo with [(3)H]-2-deoxy-d-glucose, with or without insulin or AICAR, before isol

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