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1 d catabolism of the released l-arabinose and d-xylose.
2 as abnormal in 1 of the 2 patients tested by D-xylose.
3 ely affected transport of both D-glucose and D-xylose.
4 DP-D-glucuronic acid to UDP-D-apiose and UDP-D-xylose.
5 by the most stable chair conformation of UDP-D-xylose.
6 thesized in 7-8 steps from easily accessible D-xylose.
7 erodimers has been achieved from inexpensive d-xylose.
8 inose, D-fucose, D-galactose, D-glucose, and D-xylose.
9 y XylR is greatly reduced in the presence of D-xylose.
10 ive species that ferments D-mannitol but not D-xylose.
11 cose, D-fucose, D-quinovose, L-arabinose, or D-xylose.
12           Following CBI, activation of a UDP-D-xylose 4-epimerase gene correlated with increases in a
13 plants are defective in a membrane-bound UDP-D-xylose 4-epimerase.
14  of the terminal ethylene glycol fragment of D-xylose (9.3 kcal/mol) being smaller than that of the p
15 inal carbohydrate absorption, as measured by d-Xylose absorption and fat absorptive capacity as measu
16                                    Water and D-xylose absorption as well as fecal fat studies were ma
17 ood counts, multiphasic biochemical testing, D-xylose absorption testing, and bone mineral density es
18                            Based on rates of D-xylose absorption, GLP-1 receptor blockade did not aff
19 , serum insulin-like growth factor I levels, D-xylose absorption, morphology and DNA proliferation of
20 ration of disease and lower body mass index, D-xylose absorption, serum albumin, CD4/CD45RA cells, CD
21 er plasma glycemia, as was the appearance of d-xylose after the meal.
22 that Gal2-N376F had the highest affinity for D-xylose, along with a moderate transport velocity, and
23 hydroxyecdysone, 20-hydroxyecdysone-3-O-beta-D-xylose and a hydroxyecdysterone derivative.
24 ilabilities of 2% and 6% respectively, while D-xylose and B12 absorption were found to be within norm
25 ear alpha-1,3-linked mannan substituted with D-xylose and D-glucuronic acid.
26 the aldose-ketose isomerization reactions of D-xylose and d-glyceraldehyde 3-phosphate (DGAP), respec
27 olism of hemicellulose, which is composed of d-xylose and l-arabinose.
28  N-oxide), substrates (DL-glyceraldehyde and D-xylose), and a mutation in recombinant aldose reductas
29  g of lactulose, 1 g of L-rhamnose, 0.5 g of D-xylose, and 0.2 g of 3-O-methyl-D-glucose dissolved in
30  motility, raffinose fermentation, glycogen, D-xylose, and methyl-alpha-D-glucopyranoside assimilatio
31   Complexes of the enzyme with D-glucose and D-xylose are presented to resolutions of 1.6 and 1.5 A,
32 tivities in the DMDO-mediated epoxidation of d-xylose-based oxepine 1 and d-glucose-based oxepines 2
33 id sequence identity to the Escherichia coli D-xylose binding periplasmic receptor, XylF, a component
34                                  Strikingly, d-xylose binding to this domain results in a helix to st
35                      However, its N-terminal d-xylose-binding domain contains a periplasmic-binding p
36 nal evidence that T. ethanolicus XylF is the D-xylose-binding protein.
37 zyme catalyzing this reaction also forms UDP-d-xylose by decarboxylation of UDP-d-glucuronate, and ha
38 on of the genes required for l-arabinose and d-xylose consumption is regulated by the sugar-responsiv
39 uding d-(+)-raffinose, sucrose, d-trehalose, d-(+)-xylose, d-fructose, 1-thio-beta-d-glucose sodium s
40            Experiments involved reactions of d-xylose, d-arabinose and d-ribose with glycine, alpha-l
41 ants are not induced by other sugars tested (D-xylose, D-fucose, D-lyxose).
42           All were negative for oxidation of D-xylose, D-mannitol, lactose, sucrose, maltose, and 20
43 tion-fermentation base containing d-glucose, d-xylose, d-mannitol, sucrose, lactose, or maltose.
44  been applied to d-glucose, d-galactose, and d-xylose donors with a nondirecting group incorporated a
45 ubjects consumed a breakfast meal containing D-xylose during fixed hyperglycemia at 5 mmol/l above fa
46 nsumed 50 g oral glucose solution mixed with d-xylose during fixed hyperglycemia at 8 and 10.5 mmol/L
47 binding of the feedback inhibitor, UDP-alpha-d-xylose, elicits a distinct induced-fit response; a bur
48 rations sterically prevent D-glucose but not D-xylose from entering the pocket.
49                    The enzyme that transfers D-xylose from UDP-xylose to the beta-linked mannose of p
50            Thus, the (S)-enone, derived from D-xylose, gave tetrasubstituted pyrrolidines having a de
51 ynthesis of (+)- and (-)-cyclophellitol from d-xylose has been accomplished through utilization of th
52 static agent, fermentation of m-inositol and D-xylose, hydrolysis of urea, and the lack of cytochrome
53 ations have been used to examine the pentose D-xylose in aqueous solution.
54  switch to the metabolism of l-arabinose and d-xylose in the absence of its preferred carbon source,
55      The appearance of postprandial ingested d-xylose in the blood was not affected by Ex-9.
56 freshwater bacterium Caulobacter crescentus, D-xylose induces expression of over 50 genes, including
57         Therefore, we developed the use of a D-xylose-inducible promoter for verification of an essen
58  involved in the isomerization step in which D-xylose is converted to D-xylulose or D-glucose to D-fr
59 ve different sugars, including L-glucose and D-xylose, is described in this issue (Meinert et al., ),
60                                              D-Xylose isomerase (XI) and triosephosphate isomerase (T
61 the location of hydrogen atoms in the enzyme d-xylose isomerase (XI).
62 s xylAB operon, comprising genes that encode D-xylose isomerase and D-xylulose kinase, lies a 1,101-b
63 a S12 strain was obtained by introducing the D-xylose isomerase pathway from Escherichia coli, follow
64 xperimental SAXS profile of the homotetramer D-xylose isomerase.
65 ization states of amino acids in crystals of D-xylose isomerase.
66 on barrier for XI-catalyzed isomerization of D-xylose (k(cat)/K(m) = 490 M(-1) s(-1)) versus that for
67 ylneuraminic acid), 'all-or-none' responses (d-xylose, l-rhamnose) and complex combinations thereof (
68  consisted of four monosaccharides: maltose, D-xylose, mannose, and D-fructose.
69                     By specifically labeling D-xylose molecules with a deuterium atom at the nonexcha
70 ther extreme we find that urea raises Km for D-xylose of the C298A mutant, betaine lowers the Km, and
71 (+)-dehydroisoandrosterone, l-arabinose, and D-xylose on gram scale.
72 cently solved crystallographic models of the D-xylose permease XylE from Escherichia coli and GlcP fr
73                Understanding the l-arabinose/d-xylose regulatory network is key for such biocatalyst
74 s (where Man and Xyl represent d-mannose and d-xylose, respectively), underlying the molecular basis
75 at the 2,6-diamino-2,6-dideoxy-D-glucose and D-xylose rings have restricted motions and are in a stac
76 of the 2,6-diamino-2,6-dideoxy-D-glucose and D-xylose rings, as opposed to the parallel arrangement w
77                             UDP-D-apiose/UDP-D-xylose synthase (AXS) catalyzes the conversion of UDP-
78 nd has therefore been named UDP-d-apiose/UDP-d-xylose synthase.
79                                          The d-xylose test and lactulose-to-rhamnose ratio were used
80 ential for achieving a high biomass yield on D-xylose, the aberrant HexR control appeared to underlie
81              The primary metabolic route for D-xylose, the second most abundant sugar in nature, is v
82 essor is induced to release DNA upon binding D-xylose, thereby freeing the promoter for productive in
83 bacterium Caulobacter crescentus metabolizes D-xylose through a pathway yielding alpha-ketoglutarate,
84  of the terminal ethylene glycol fragment of d-xylose to give DGA results in a large decrease in the
85 e to (i) the barrier to conversion of cyclic d-xylose to the reactive linear sugar (5.4 kcal/mol) bei
86 ity of AXS to catalyze the conversion of UDP-D-xylose to UDP-D-apiose.
87 e defect was traced to the conversion of UDP-D-xylose to UDP-L-arabinose in the microsome fraction of
88  the high-affinity binding-protein-dependent D-xylose transport.
89 l2 deduced from the crystal structure of the D-xylose transporter XylE from Escherichia coli, both re
90                                    All known D-xylose transporters are competitively inhibited by D-g
91 ast growth-based screening system for mutant D-xylose transporters that are insensitive to the presen
92 g simultaneous fermentation of D-glucose and D-xylose, two primary sugars present in lignocellulosic
93 g protein and permease) of the high-affinity D-xylose uptake system are not located in the vicinity o
94 ing of five genes) and a functional combined D-xylose utilization and zeaxanthin biosynthesis pathway
95  assembler can rapidly assemble a functional D-xylose utilization pathway (approximately 9 kb DNA con
96  key element in improving the initially poor D-xylose utilization was the redistribution of 6-phospho
97 growth rate and biomass yield of the evolved D-xylose utilizing P. putida strain.
98                     Previously, an efficient D-xylose utilizing Pseudomonas putida S12 strain was obt
99 e, which is essential for the utilization of D-xylose via the nonoxidative PP pathway.
100 xcellent overall yield of more than 10% from d-xylose, while the heterodimer route led to UT-39 in 19
101 nt-3-enopyranosid-2-ulose) was prepared from D-xylose, while the R analogue was obtained from L-arabi
102 uronate to a mixture of UDP-d-apiose and UDP-d-xylose with a turnover number of 0.3 min-1.
103 ha-D-glucoside (MDG), D-trehalose (TRE), and D-xylose (XYL) by the same isolates was investigated by
104 ic acid (GlcUA), l-iduronic acid (IdoUA), or d-xylose (Xyl).

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