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1 nsitive, calcium dependent, and blocked by a D2 receptor antagonist.
2 t of breast cancer, and (-)-IBZM, a dopamine D2 receptor antagonist.
3 ) and to systemic administration of DA D1 or D2 receptor antagonists.
4 ra-HPC carbachol or NMDA are not reversed by D2 receptor antagonists.
5 vity (S2 group) or with predominant dopamine D2 receptor antagonist activity (D2 group; HBBM, 40 mg,
6 ng task under the influence of either the DA D2 receptor antagonist amisulpride (400 mg), the NMDA re
8 ocker, was ineffective, as were sulpiride, a D2-receptor antagonist, and tertiapin, a G protein-coupl
9 a typical antipsychotic drug haloperidol, a D2 receptor antagonist, at a dose of 0.1 mg/kg, to incre
13 ty, a group of rats received the dopamine D1/D2 receptor antagonist cis-flupenthixol (0.5 mg/kg) 30 m
15 acologic PRL mobilization using the dopamine D2 receptor antagonist domperidone prevented HCC in tumo
17 The action of dopamine was blocked by the D2 receptor antagonist eticlopride, whereas a D1/5 antag
20 the 5-HT1A receptor agonist, 8-OH-DPAT, the D2 receptor antagonist, eticlopride, and the D1 receptor
22 bilateral intra-amygdaloid infusions of the D2 receptor antagonist, eticlopride, on the acquisition
23 anaesthesia) administration of the dopamine D2 receptor antagonist haloperidol, but not raclopride (
27 e antipsychotic drug raclopride, a selective D2 receptor antagonist, increased phosphorylation of DAR
28 D1 receptor antagonist--but not dopaminergic D2 receptor antagonist--inhibited the TSD-mediated incre
30 effects whereas subeffective doses of the DA D2 receptor antagonist, L-741,626, rescued cocaine's abi
31 temic administration of a selective dopamine D2 receptor antagonist partially blocked the effects eli
32 atment for Tourette syndrome and primarily a D2 receptor antagonist, prevented D1 stimulation with SK
33 ctively, given that pretreatment with D1 and D2 receptor antagonists prevents the respective actions
35 llenging them with the preferential dopamine D2 receptor antagonist raclopride and D1 receptor antago
36 usion of 10 microg (but not 2 microg) of the D2 receptor antagonist raclopride facilitated nursing bu
39 Positron emission tomography with the DA D2 receptor antagonist radiotracer [11C]raclopride detec
40 dol (0.2 mg/kg), relatively selective D1 and D2 receptor antagonists, respectively, blocked the AMPH-
42 nist, SR141716A (5 mg/kg, i.p.), or dopamine D2 receptor antagonist, S(-)-raclopride (5 mg/kg, i.p.)
43 ter cAMP levels in other systems); 2) D1 and D2 receptor antagonists (SCH 23390, eticlopride, raclopr
44 rats were injected with the selective D1 or D2 receptor antagonists SCH23390 or eticlopride prior to
45 sing bilateral reverse microdialysis, D1 and D2 receptor antagonists (SCH23390, sulpiride) and the DA
47 This effect was completely reversed by the D2 receptor antagonist sulpiride but not by SCH 23390, a
54 n humans, we combined sulpiride, a selective D2 receptor antagonist, with the dopamine precursor l-DO
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