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1 holinergic interneurons express the dopamine D5 receptor.
2 brain, and diencephalon express the dopamine D5 receptor.
3 ne acetyltransferase (ChAT) and the dopamine D5 receptor.
4 atial object recognition via the dopamine D1/D5 receptor.
5 ether it depends on dopamine acting on D1 or D5 receptors.
6 t depends on hippocampal dopamine D1 but not D5 receptors.
8 of EPSCs, yet slightly reducing release, D1/D5 receptor activation selectively enhanced sustained sy
9 protein synthesis, we show that dopamine D1/D5 receptor activation stimulates local protein synthesi
17 part mediated by dopamine acting through D1/D5 receptors and involves an increase in neuronal synchr
18 -133 phosphorylation of CREB via dopamine D1/D5 receptors and L-type calcium channels in organotypic
19 DRD5 and SLC6A3 which encode dopamine D4 and D5 receptors and the dopamine transporter, respectively.
20 f mature receptors, abrogated binding of the D5 receptor antagonist [(3)H]SCH23390, suggesting that w
23 the binding and functional properties of D1/D5 receptors, as well as effector proteins within their
24 toactivation are sensitive to hippocampal D1/D5 receptor blockade and resistant to adrenoceptor block
26 s reversed by SCH 23390 (an antagonist at D1/D5 receptors), confirming that it was mediated by activa
27 mutagenesis was used to generate a panel of D5 receptors containing mutations in the three predicted
30 Here we tested the hypothesis that dopamine D5 receptors (D5Rs), characterized by a high constitutiv
32 ecently, we have shown that the dopamine D1B/D5 receptor displays binding and coupling properties tha
33 Moreover, dopaminergic projections and D1/D5 receptor distributions display a layer-dependent orga
35 rt the functional importance of the dopamine D5 receptor in the modulation of acetylcholine release t
37 ne the effects of local antagonism of D2 and D5 receptors in the AH on adolescent AAS-induced aggress
40 ce of GAD (a marker for GABA production) and D5 receptors in the lateral subdivision of the AH (LAH).
42 antagonists implicate the involvement of D1/D5 receptors in various pavlovian conditioning tasks in
44 ially overlapping populations, such that the D5 receptor is found in a subpopulation of those spines
46 idate a plausible mechanism for the observed D5 receptor mediation of AAS-induced aggression, brains
50 area 9; rather, dopamine can activate D1 and D5 receptors on the same spines, plus an additional grou
51 the reported association of calcyon with the D5 receptor, or in addition, may suggest that calcyon ha
54 macological antagonism of spinal dopamine D1/D5 receptors reversed priming, whereas D1/D5 agonists in
55 a chronic pain state in both sexes; however, D5 receptors seem to play a critical role in males where
61 istent with this model, the activation of D1/D5 receptors that couple to PKA in dopamine neurons anta
62 a key role in the cell surface expression of D5 receptors, they do not appear to contribute to the re
66 partially prevented by protection of the D1/D5-receptor with the agonist (R)-SKF 38393 or the specif
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