ライフサイエンスコーパス: DC

1 DC and PC Cx26 expression is essential for cochlear ampl

2 DC deficiency and monocytopenia were observed in blood,

3 DC numbers were restored in the spleen of C57BL/6 and pe

4 DC-mediated T cell immune tolerance is an active process

5 DC-RA expression of IL-27 was important to their inducti

6 DC-RAs displayed a mature yet tolerogenic phenotype, exp

7 DC-SIGN is a dendritic cell surface structure which part

8 DC-SIGN is a major receptor for infection of both monocy

9 DCs from these mice show impaired cross-presentation ex

10 We quantified the mRNA level of 17 DC-specific transcription factors and observed that 3 tr

11 This process evolves through a [3 + 2] 1,3-DC when cinnamaldehyde is used in the presence of an azo

12 A DC current is applied through the resonator to induce he

13 tors further validates the human thymus as a DC-poietic organ, which provides selective microenvironm

14 arge established mouse tumors using HMGN1, a DC-activating TLR4 agonist capable of inducing anti-tumo

15 ilure of the monocytes to differentiate to a DC phenotype.

16 y engagement with Clr-g-expressing activated DCs during immune responses.

17 ic lung inflammation were restored in ADAM10(DC)(-/-) with the overexpression of the Notch1-intracell

18 pathy, whereas edema was more frequent after DC.

19 -dependent signalling is inhibited, altering DC outputs and enhancing T-cell responses.

20 e sensitivity of both PPG components (AC and DC) to changes in haemorheology were rigorously investig

21 e collective action of antigen dispersal and DC positioning regulates the extent and quality of T cel

22 S2 limits adaptive anti-tumoral immunity and DC-based priming of T cells in vivo, indicating a critic

23 ytosis by resident pulmonary macrophages and DC.

24 acy and robustness of detecting DC pairs and DC modules.

25 Mutations in dectin-1 (rs7309123) and DC-SIGN (rs11465384 and rs7248637), allogeneic stem cell

26 ment of homeostatic modules in monocytes and DCs from human metastatic melanoma.

27 macrophages, T lymphocytes, neutrophils, and DCs all contribute to liver inflammation.

28 Anti-DC-SIGN monoclonal antibody (MAb) inhibited HHV-8 infect

29 Anti-DC-STAMP-mAb also inhibited pit formation caused by RANK

30 Importantly, local injection of anti-DC-STAMP-mAb also suppressed alveolar bone loss and redu

31 we expected, systemic administration of anti-DC-STAMP-mAb downregulated the ligature-induced alveolar

32 e effects of DC-STAMP in periodontitis, anti-DC-STAMP-monoclonal antibody (mAb) was tested in a mouse

33 LT indication was defined as DC if the Model for End-Stage Liver Disease (MELD) at WL

34 n of osteoclast (OC) cell fusion, as well as DC-mediated immune regulation.

35 e efficient antigen-presenting cells such as DCs.

36 As a result, tumor-associated DCs are no longer able to stimulate adequate CD8 T cells

37 Patients who received CMV-ATCT-DC vaccination experienced a significant increase in the

38 Blood DC analysis can be used to identify ALS patients with an

39 mbrane recognition and demonstrate that BoNT/DC can use a broad range of sialic acid-containing moiet

40 p3 in T cells, trans-presentation of IL-6 by DC-bound IL-6Ralpha (called 'IL-6 cluster signaling' her

41 li to induce secretion of bioactive IL-12 by DCs.

42 own to be dependent on Ag cross-presented by DCs, but they did not inhibit the response to "inflation

43 icity requires IL-2 specifically released by DCs, even when T cell secretion of IL-2 is intact.

44 s known about the molecular pathways used by DCs to sense alum and, in turn, activate T and B cells.

45 ly US$14; 1 PKR = US$0.009543); double cash (DC), a cash transfer of 3,000 PKR; or a fresh food vouch

46 lymph nodes of intestinal migratory CD103(+) DCs carrying oral DNFB, especially the CD103(+)CD11b(+)

47 When exposed to CD11(+) DCs, the mutant virus that lacks the amino terminus of g

48 g oral DNFB, especially the CD103(+)CD11b(+) DC subset expressing the vitamin A-converting enzyme ret

49 n markedly fewer intestinal CD103(+)CD11b(+) DCs and a reciprocal increase in the CD103(-)CD11b(+) de

50 nsfer of HDM-pulsed LRP-1-deficient CD11b(+) DCs into WT mice generated a similar phenotype of enhanc

51 ation of PPARgamma in lung-resident CD11b(+) DCs, which enhances migration to draining lymph nodes an

52 34a provides homoeostatic control of CD1c(+) DC activation via regulation of tyrosine kinase receptor

53 CD1c(+) DCs acquired high langerin and CD1a with granulocyte-mac

54 and nonclassical monocytes, but not CD1c(+) DCs, made foamy macrophages easily in vitro with macroph

55 hat, after lymphoid irradiation, host CD8(+) DCs play a requisite role in tolerance induction through

56 ha(+) subset of dendritic cells (CD8alpha(+) DCs) and is involved in sensing necrotic cells.

57 meostatic maintenance of splenic CD8alpha(+) DCs and in the capacity of these cells to cross-present

58 ells; (iii) interfering with dendritic cell (DC) effector function; and (iv) inhibiting antitumor ada

59 we report that C-type lectin dendritic cell (DC) immunoreceptor (DCIR), a key component in DC homeost

60 typic representatives of the dendritic cell (DC) lineage, they are now considered to be a specialized

61 virulence factor, stimulates dendritic cell (DC) maturation which is dependent on TANK-binding kinase

62 functions that could enhance dendritic cell (DC) maturation.

63 Human dendritic cell (DC) response to alpha-(1,3)-glucan polysaccharide of Asp

64 D4 T cells, macrophages, and dendritic cell (DC) showed chemoattractantDeltadriven vectorial migratio

65 Dendritic cell (DC) subsets with biased capacity for CD4(+) and CD8(+) T

66 ing was used to characterize dendritic cell (DC) transcripts.

67 ) cells on the regulation of dendritic cell (DC)-driven allergic inflammation remain elusive.

68 Dendritic cells (DC) accumulate in the CNS during neuroinflammation, yet,

69 positivity, indicates that dendritic cells (DC) are of crucial importance to pathogenesis by activat

70 Thymic dendritic cells (DC) delete self-antigen-specific thymocytes, and drive d

71 During infection, dendritic cells (DC) mature into antigen-presenting cells that activate T

72 anism of MSC recruitment by Dendritic Cells (DC), hypothesising that it would be mediated by EV.

73 1 for T-cell conjugation to dendritic cells (DC).

74 Dendritic cells (DCs) are activated by pathogens to initiate and shape im

75 Dendritic cells (DCs) are crucial initiators of immune responses, but lit

76 Mast cells (MCs) and dendritic cells (DCs) are essential innate sentinels populating host-envi

77 Dendritic cells (DCs) are thought to form a dendritic network across barr

78 Although dendritic cells (DCs) are vital for the induction of T-cell responses, th

79 preferentially occurred in dendritic cells (DCs) but not in macrophages.

80 ey role in both T cells and dendritic cells (DCs) for development of type-2 immune responses.

81 pend on signals from CD8(+) dendritic cells (DCs) for their activation, we used the mouse model to te

82 to TB, we infected with MTB dendritic cells (DCs) from putatively resistant individuals diagnosed wit

83 Tolerogenic dendritic cells (DCs) have emerged as relevant clinical targets for the t

84 to be produced by CD103(+) dendritic cells (DCs) in T cell-inflamed tumors.

85 Dendritic cells (DCs) initiate immune responses to common aeroallergens,

86 Dendritic cells (DCs) play critical roles in developing immune defenses.

87 Dendritic cells (DCs) promote either tolerogenic or immunogenic T cell re

88 The mechanism of dendritic cells (DCs) recruitment across the blood brain barrier (BBB) du

89 n is a critical function of dendritic cells (DCs) required for induction of antitumor immune response

90 presentation of antigens by dendritic cells (DCs) to naive T lymphocytes.

91 he haptotactic migration of dendritic cells (DCs) toward higher concentrations of immobilized chemoki

92 Trafficking of tissue dendritic cells (DCs) via lymph is critical for the generation of cellula

93 87(+) macrophages, T cells, dendritic cells (DCs), and residential macrophages near high endothelial

94 f noncycling cells, such as dendritic cells (DCs), is impaired due to limited availability of deoxynu

95 ions with a high density of dendritic cells (DCs), macrophages and T cells.

96 The skin hosts a variety of dendritic cells (DCs), which act as professional APC to control cutaneous

97 Targeting newborn dendritic cells (DCs), which integrate vaccine signals into adaptive immu

98 e complexes (Ig-ICs) by gut dendritic cells (DCs).

99 s between T lymphocytes and dendritic cells (DCs).

100 boosted OVA uptake by fetal dendritic cells (DCs).

101 steps of TLR9 activation in dendritic cells (DCs).

102 species with MUTZ-3-derived dendritic cells (DCs).

103 n the cross-presentation of apoptotic cells, DC-specific Vps34-deficient animals developed increased

104 s expected, DC-LMP1/CD40/Ldlr(-/-) chimeras (DC-LMP1/CD40) showed increased antigen-presenting capaci

105 id DC (pDC) and antigen-presenting classical DC (cDC) is controlled by the E protein transcription fa

106 Here, we show that the dorsal column (DC) somatosensory pathway contains projections that conv

107 though uninflamed glomeruli rarely contained DCs, injury with nephrotoxic antibodies resulted in accu

108 In contrast, DCs marked by Zbtb46-GFP or Snx22-GFP were less abundant

109 hese ox-tr-LB, but not LB present in control DCs, covalently bind to chaperone heat shock protein 70.

110 actor 4-dependent dermal type 2 conventional DC subsets and not by epidermal Langerhans cells.

111 inhibitors ablate pDC (but not conventional DC) responses both in vitro and in vivo.

112 ; and circulating progenitor of conventional DCs (cDCs).

113 ble nitric oxide synthase (iNOS) coordinates DC metabolism and function to limit DC-stimulated T-cell

114 immunotherapies, including Tol-DC, Rapa-DC, DC-10, and PGE2-induced myeloid-derived suppressor cells

115 do not develop in mice with IRF-4-deficient DCs (IRF-4(f/f) CD11c-cre).

116 Surprisingly, transferred IRF-4-deficient DCs also effectively prime S. mansoni-specific Th2 respo

117 1 and Jagged 2 single- and double-deficient DCs to induce AAI.

118 IRF4-deficient DCs were impaired for Treg generation in vivo.

119 and humanized TLR8 mouse bone marrow-derived DCs enabled benchmarking of the TLR8 agonist-encapsulati

120 -27 production in murine bone marrow-derived DCs.

121 ession in ES-DCs than in bone marrow-derived DCs.

122 ion of HIV-1 replication in monocyte-derived DCs (MDDCs) is associated with an increased expression o

123 assays using neonatal human monocyte-derived DCs and humanized TLR8 mouse bone marrow-derived DCs ena

124 d that tumour-induced human monocyte-derived DCs exhibited systematic functional deficiencies.

125 ; the percentage of migratory CD1a(+) dermal DCs was significantly decreased in the DSS patients, and

126 hods in accuracy and robustness of detecting DC pairs and DC modules.

127 We developed DC nanozymes with high enzymatic activity and ability to

128 esults offer a promising path for developing DC subset-specific immunotherapies that cannot be provid

129 nsability for normal growth and development, DCs have important biological roles with respect to path

130 These studies suggest that a discrete DC subset both expands Tregs and suppresses CD8 T cells

131 "alpha-(1,3)-Glucan-educated" DCs stimulated the activation of naive T cells and polar

132 In addition, such engineered DCs, when pulsed with a myelin antigen, led to myelin-sp

133 -B, and Runx3) showed lower expression in ES-DCs than in bone marrow-derived DCs.

134 As expected, DC-LMP1/CD40/Ldlr(-/-) chimeras (DC-LMP1/CD40) showed in

135 CDDO-DFPA exposed DCs retained expression of surface ligands and capacity

136 Proteomic profiling of mycolactone-exposed DCs showed that expression of mediators of antigen prese

137 have fewer CD11b(+) migrating DCs and fewer DCs carrying parasite antigens to the lymph nodes.

138 JAG1(+) thymic medullary niche enriched for DC-lineage cells expressing Notch receptors further vali

139 s selective microenvironments permissive for DC development.

140 onectin and galectin-3 in those derived from DCs, whereas T-cell exosomes lacked these molecules.

141 TsSP-induced reduction of TNF secretion from DCs is reversed by receptor antagonists for EP2 and EP4,

142 plasmacytoid DC neoplasia cells and healthy DCs; and circulating progenitor of conventional DCs (cDC

143 This was associated with higher DC and T-cell counts in pericardial AT, which outnumbere

144 racterization and unbiased analysis of human DC subsets in blood, tonsil, spleen, and skin.

145 but its role in endotoxin tolerance in human DCs is still controversial.

146 TLR4-induced tolerogenic phenotype in human DCs, which can help the better understanding of processe

147 dulating Rab7B and P2RX7 expression in human DCs.

148 In vitro differentiation models of human DCs, gene profiling, gene transduction, and immunohistol

149 both porcine and, for the first time, human DCs.

150 Pandemic IAV suppresses this immunogenic DC cell death.

151 ata support a novel concept that immunogenic DCs can be engineered for myelin-specific therapy for MS

152 C) immunoreceptor (DCIR), a key component in DC homeostasis, is required to modulate lung inflammatio

153 genic responses and placental development in DC expanded IL-10(-/-) dams.

154 were used to identify molecules involved in DC subset specification.

155 One key mechanism involved in DC-mediated immunosuppression is the expression of trypt

156 Further, LARG was observed to participate in DC-SIGN mediated internalization of HIV-1 in DCs.

157 d decreased by 37% and 60%, respectively, in DC-LMP1/CD40 chimeras.

158 DC-SIGN mediated internalization of HIV-1 in DCs.

159 creased TACE promoter luciferase activity in DCs.

160 ith conditional Cx26 knock-out (Cx26 cKO) in DCs and PCs that did not influence sensory transduction,

161 a) axis dictates the fate of ingested DNA in DCs for immune evasion.

162 An IFN-I signature was evident in DCs responding to the helminth Schistosoma mansoni or th

163 Akt/mTOR axis and FoxO1 nuclear exclusion in DCs, leading to decreased transcription of the autophagy

164 gh inhibition of the autophagic machinery in DCs in a cytotoxic T-lymphocyte-associated protein 4-dep

165 identify the canonical autophagy pathway in DCs as a molecular target of Foxp3+ Treg-mediated suppre

166 that Sec22b-dependent cross-presentation in DCs is required to initiate CD8(+) T cell responses to d

167 SAMHD1 potently blocks HIV-1 replication in DCs, although the underlying mechanism is still unclear.

168 pairs STAT1-mediated type I IFN signaling in DCs, leading to increased production of IL-12 and increa

169 l through sustaining type I IFN signaling in DCs.

170 with distinct transcriptional signatures in DCs in vivo, reflecting the diverse environments in whic

171 Mice devoid of Smad7 specifically in DCs are resistant to the development of experimental aut

172 of IL-27 (Ebi)(-/-) (ie, IL-27-incompetent) DC-RAs were ineffective in inducing food allergen tolera

173 ligature to a second maxillary molar induced DC-STAMP messenger RNA and protein, along with elevated

174 osed to exosomes derived from HIV-1 infected DCs.

175 required for OVA-induced lung inflammation, DCs have also been shown to be target cells of TSLP.

176 ewly recruited monocyte-derived inflammatory DCs.

177 NO donors (SNAP and DETA-NONOate) inhibited DC antigen presentation.

178 kinase receptor AXL, an important inhibitory DC auto-regulator.

179 treatment reduces the virus-induced PD-L1(+) DC, MDSC, TAM and Treg, as well as co-inhibitory molecul

180 ssion of Ags in LCs but no other langerin(+) DCs.

181 rdinates DC metabolism and function to limit DC-stimulated T-cell responses.

182 Similarly, LRP-1 expression by CD11b(+) lung DCs was significantly reduced in HDM-challenged WT mice.

183 ses elicited by cells of monocyte/macrophage/DC lineages.IMPORTANCE We previously demonstrated that a

184 ontact-dependent cross-talk between the main DC subsets, plasmacytoid and myeloid DCs (mDCs) was nece

185 into the distinct functions of the two major DC subsets in glomerular inflammation.

186 1) in regulating alpha-(1,3)-glucan-mediated DC activation and T-cell responses.

187 CD11c-cre mice have fewer CD11b(+) migrating DCs and fewer DCs carrying parasite antigens to the lymp

188 chived antigens are exchanged with migratory DCs, both directly and through LEC apoptosis, to cross-p

189 phosphate reduced form oxidases (NOXs) in Mo-DC differentiation.

190 an in vitro culture model yielding human mo-DCs and mo-Macs closely resembling those found in vivo i

191 gulators of monocyte differentiation into mo-DCs and mo-Macs, respectively.

192 pressure pulses as delivery method, moderate DC electric fields are used to drive charged molecules o

193 Act-A-iTreg cells (Act-A-iTreg cell-modified DCs) to protect against experimental asthma, and the mec

194 Moreover, DC-LMP1/CD40 chimeras developed inflammatory bowel disea

195 ation of TLR3, TLR4, and TLR9 induces murine DC production of complement components and local product

196 NOX5 was expressed in circulating myeloid DC, and at a lower level in plasmacytoid DC.

197 he main DC subsets, plasmacytoid and myeloid DCs (mDCs) was necessary for increased chemokine and IFN

198 A higher median frequency of myeloid DCs (mDCs) but not plasmacytoid DCs (pDCs) was observed

199 st adult-like maturation profile of neonatal DCs.

200 Our study reveals a new DC subset that shares properties with plasmacytoid DCs (

201 r (TLR) 8 agonists robustly activate newborn DCs but can result in reactogenicity when delivered in s

202 -derived, GM-CSF-instructed, nonconventional DC subsets.

203 he anti-inflammatory cytokine M-CSF, but not DC growth factors, sustains CD33/LAIR-1 expression on bo

204 Depletion of inflammatory monocytes, but not DCs or other myeloid cells, resulted in lower levels of

205 0 substrate, when knocked out of DCs (Notch1(DC)(-/-) ) demonstrated a similar reduction in anaphylax

206 The capacity of DC to stimulate allogenic splenocyte proliferation was a

207 Thus, the residues concentration of DC in eggs after different cooking methods was investiga

208 To evaluate the effects of DC-STAMP in periodontitis, anti-DC-STAMP-monoclonal anti

209 Although a loss of DC autophagy slowed disease, the combined loss of autoph

210 mune dysregulation through the modulation of DC differentiation.

211 ould also modulate the functional profile of DC.

212 The stability of DC in eggs were depended upon the type and time of cooki

213 The ability of DCs conditioned by Act-A-iTreg cells (Act-A-iTreg cell-m

214 wed increased antigen-presenting capacity of DCs and increased T-cell numbers.

215 core chromosomes, the structural features of DCs usually differ for gene density, GC content, houseke

216 ollagen-induced arthritis and interaction of DCs and T cells from these mice are reduced and do not s

217 lacked the capacity to induce maturation of DCs but significantly blocked alpha-(1,3)-glucan-induced

218 ved in the differentiation and maturation of DCs.

219 known ADAM10 substrate, when knocked out of DCs (Notch1(DC)(-/-) ) demonstrated a similar reduction

220 tem allowing to track migratory responses of DCs to precisely controlled immobilized gradients of CCL

221 Here, we evaluated the role of DCs in renal inflammation using a CD11c reporter mouse l

222 new subdivision within the CD1C(+) subset of DCs; the relationship between blastic plasmacytoid DC ne

223 monocytes, lymphocytes and three subsets of DCs from single human CD34(+) progenitor cells, we found

224 he PNVs, we found that the cell viability of DCs was unaffected, up to 200 mug/ml.

225 e on TREM-2 expression in the airways and on DC subsets in the lung and lymph nodes in murine model o

226 examined the effects of Act-A-iTreg cells on DC phenotype, maturation status, and TH2 cell priming po

227 The induction of Notch ligand expression on DC subsets by HDM was quantified by using quantitative r

228 t that expression of Jagged 1 or Jagged 2 on DCs is not required.

229 ect stimulation of complex component CD40 on DCs leads to activation of Akt1, suggesting CD40 involve

230 reas CD40 was required on B cells but not on DCs in the generation of antigen-specific follicular hel

231 ein antigen and adjuvant: B7 was required on DCs but was not required on B cells, whereas CD40 was re

232 Only DC infected with seasonal IAV, but not with pandemic IAV

233 Langerhans cells (LCs) are the only DC subset in the healthy epidermis.

234 We find that the conditions for optimal DC guidance are perfectly provided by the CCL21 gradient

235 counts in pericardial AT, which outnumbered DCs and T cells in lymph nodes.

236 hypothesis, in a subpopulation of patients, DCs recruited at the diseased tissue produce high levels

237 he relationship between blastic plasmacytoid DC neoplasia cells and healthy DCs; and circulating prog

238 oid DC, and at a lower level in plasmacytoid DC.

239 on between interferon-producing plasmacytoid DC (pDC) and antigen-presenting classical DC (cDC) is co

240 y of myeloid DCs (mDCs) but not plasmacytoid DCs (pDCs) was observed in the blood of SR as compared t

241 set that shares properties with plasmacytoid DCs (pDCs) but potently activates T cells, thus redefini

242 ntly, preventing the cross talk by preceding DC depletion decreases MC antigen presenting capacity an

243 Delivery of specific allergen-presenting DC-RAs to half-maximally sensitized mice with ovalbumin

244 r data show that TLR4 ligation in LPS-primed DCs, induced higher levels of both IDO isoforms together

245 the other hand, B cells can directly promote DC maturation and thereby enhance T-cell stimulation.

246 Ischemia-reperfusion injury also promoted DC-dependent cross-presentation of renal antigens to CD8

247 observed that GUWE dose-dependently promoted DC maturation and cytokine secretion through TLR4 signal

248 ndritic cell-specific transmembrane protein (DC-STAMP) plays a key role in the induction of osteoclas

249 er, the calibrated measurements enabled by q-DC should advance applications of cell mechanotype in ba

250 ased immunotherapies, including Tol-DC, Rapa-DC, DC-10, and PGE2-induced myeloid-derived suppressor c

251 Targeting of myeloid-dendritic cell receptor DC-SIGN by numerous chronic infectious agents, including

252 ludes the dendritic cell endocytic receptors DC-LAMP and CD68, as well as LAMP-1 and LAMP-2.

253 ed that MHC-class I acquisition by recipient DCs occurs for at least 1 month following transplantatio

254 ibution of acquired MHC-class I on recipient DCs during the life span of a skin graft.

255 The capacity of renal DCs to present antigen was examined by assessment of pro

256 bodies to deliver vaccine components to skin DC subsets.

257 gnals, as well as the nature of the specific DC subset presenting Ag to T cells.

258 ion of il27p28 expression in vivo in splenic DC following administration of dimethyl PGE2 in conjunct

259 acquired MHC from TEC plus thymic or splenic DC, whereas thymic or splenic B cells were poor donors.

260 When exposed to microbial stimuli, DCs activated nuclear factor (NF)-kappaB, which induced

261 In such DCs, glucose-dependent signalling is inhibited, altering

262 In summary, DC-EV are naturally loaded with chemoattractants and can

263 o inflammatory cytokine induction by SVLP(+) DCs, with adaptive immune response activation ex vivo/in

264 pecimens in high magnetic fields up to 45 T (DC fields) and 60 T (pulsed fields).

265 Here, we report that DC migration from brain parenchyma is dependent upon the

266 Pharmacokinetic study revealed that DC nanozymes significantly prolonged circulation of acti

267 Here we demonstrate that DCs are a principal target of the immune modulating acti

268 We found that DCs lacking SOCS1 expression were functional in driving

269 All the DC-targeting vaccine administrations with poly-ICLC incr

270 unction of LCs in vivo, without altering the DC subset composition in the skin, we have generated tra

271 iently the targeted Ag is endocytosed by the DC.

272 rm and 88.3% (95% CI, 86.1% to 90.1%) in the DC arm.

273 the signal of the OPCPA and the pump of the DC-OPA.

274 l for the induction of T-cell responses, the DC subpopulations that induce Th2 cells in the intestine

275 or cells, we found that specification to the DC lineage occurred in parallel with specification of he

276 ation of an ultrashort pulse after which the DC surface potential is screened with a second optical p

277 vitro and in vivo Targeted depletion of this DC population in Mgl2(DTR) hosts activated cytotoxic lym

278 MHC transfer was donor-cell specific; thymic DC readily acquired MHC from TEC plus thymic or splenic

279 ype DCs (wtDCs) than in TNF knockout (TNFko) DCs.

280 diary processes that link TLR stimulation to DC maturation and the subsequent development of effector

281 er, unlike tissue macrophages and similar to DCs, they homeostatically migrate to lymph nodes and pre

282 led a type-I IFN (IFN-I) signature unique to DCs from Nb-primed mice.

283 as cell-based immunotherapies, including Tol-DC, Rapa-DC, DC-10, and PGE2-induced myeloid-derived sup

284 ways essential for conferring the tolerizing DC phenotype and optimal methods for their induction rem

285 bliographies for articles relating to trauma DC.

286 fficking studies of HIV-1 in cocaine treated DCs revealed increased co-localization of HIV-1 with end

287 ased degradation of HIV-1 in cocaine treated DCs.

288 When we treated DCs with the PNVs, we found that the cell viability of D

289 We present novel evidence that Treg-DC skewed CD4(+) naive T cell polarization toward a regu

290 nd activity of TACE were lower, in wild-type DCs (wtDCs) than in TNF knockout (TNFko) DCs.

291 ndemic IAV, enhance maturation of uninfected DC and T cell proliferation.

292 interactive JavaScript chart framework using DC.js.

293 Electrical response measurements, using DC resistance, AC impedance spectroscopy, and Kelvin Pro

294 ted from Virginia, Maryland, and Washington, DC, to a single level I trauma center.

295 inally Georgetown University, in Washington, DC (PhD), the latter while employed at a commercial biol

296 yclophosphamide (600 mg/m(2)) every 3 weeks (DC) or three cycles of epirubicin (90 mg/m(2)) and cyclo

297 t resident macrophages were sessile, whereas DCs were motile before and after inflammation.

298 reporter mouse line and two mouse lines with DC-specific reporters, Zbtb46-GFP and Snx22-GFP.

299 Similar results were obtained with DCs obtained from malaria-naive US donors and malaria-ex

300 Therefore, persistence within DCs and possibly within other immune cells might contrib