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1 DCMU prevented the normal light response by blocking rea
2 DCMU upregulated transcription from the pea PetE promote
3 DCMU, an inhibitor of photosynthetic electron transport,
5 inear and cyclic electron flows are blocked (DCMU inhibiting PSII and methylviologen accepting electr
6 ough Lhcb mRNA abundance was not affected by DCMU, its polyribosomal loading pattern was altered in m
10 th 3-(3,4)-dichlorophenyl)-1,1-dimethylurea (DCMU) as oxygen uptake in the presence of MV at a rate o
11 out 3-(3,4-dichlorophenyl)-1,1-dimethylurea (DCMU), an inhibitor of photosynthetic electron transport
13 nd 3-(3, 4-dichlorophenyl)-1,1-dimethylurea (DCMU), oxygen-evolving activity was observed in the R342
14 and 3-(3,4-dichlorophenyl)-1,1-dimethylurea (DCMU), specific inhibitors of plastid-located processes
17 sed 3-(3,4-dichlorophenyl)-1,1-dimethylurea (DCMU; a photosystem II inhibitor) to block O2 evolution
18 tially bleached without heat by a herbicide (DCMU, at 24 degrees C) also recovered predominantly with
19 se of gs to ci is similar in darkness and in DCMU-treated leaves, whereas the response in light in no
20 variable fluorescence decay measurements in DCMU-treated cells, charge recombination of Q(A)(-) with
21 onse of gs to ci is insensitive to oxygen in DCMU-treated leaves or in darkness; and (5) stomata resp
22 in the presence of photosystem II inhibitor DCMU were measured as a function of DBMIB concentration
23 W167S was grown in the presence of 20 microM DCMU, the mutant continued to exhibit these defects.
24 leaves, whereas the response in light in non-DCMU-treated leaves is much larger and has a different s
25 ent with norflurazon and lincomycin, but not DCMU, decreased the accumulation of transcripts of pea P
26 luorescence decay kinetics in the absence of DCMU indicated that electron transfer to QB was signific
27 l as genes that responded to the addition of DCMU [3-(3,4-dichlorophenyl)-1,1-N-N'-dimethylurea], a s
30 induced fluorescence rise in the presence of DCMU indicated that in wild type the ratio of PS IIalpha
31 uorescence decay kinetics in the presence of DCMU indicated that the charge recombination between QA-
32 equent 273 K illumination in the presence of DCMU or by dark incubation with low amounts of the one e
33 F(v)) induction and decay in the presence of DCMU showed that all but one of the combinatorial strain
34 wever, fluorescence decay in the presence of DCMU, which monitors primarily Q(A)(-) charge recombinat
36 ion of photosynthesis, either by darkness or DCMU results in a destabilization of the Fed-1 mRNA.
37 taining chimeric PetE constructs showed that DCMU treatment decreased the accumulation of pea PetE an
39 bclustering of subnetworks responding to the DCMU stress identified novel groups of genes that were t
40 with similar spatial patterns in response to DCMU; (2) the response of gs to ci changes slope in conc
41 ibitor of photosynthetic electron transport, DCMU, abolishes the MAMP-induced chloroplastic reactive
44 eroxidase (H2O2 scavenger) or treatment with DCMU (photosynthesis inhibitor) attenuates nuclear H2O2
45 was inhibited by darkness or treatment with DCMU, there appeared a smaller mRNA species that may rep
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