ライフサイエンスコーパス: DD

1 DD affects between 5% and 25% of people of European desc

2 DD GABAergic neurons reverse polarity during larval deve

3 DD mechanisms were developed using an iterative approach

4 DD represents an ideal human model disease for fibrosis

5 DD was considered negative (DD-) if <500 ng/mL.

6 DD, but not VD, MNs reverse their cellular polarity in a

7 for ruling out AAS in patients with ADD-RS=0/DD- or ADD-RS </=1/DD-.

8 f 15.9% (95% CI, 14.3-17.6) for the ADD-RS=0/DD- strategy.

9 In 294 patients with ADD-RS=0/DD-, 1 case of AAS was observed.

10 PGL-1 DD has a novel 13 alpha-helix fold that creates a positi

11 ind RNA and discover unexpectedly that PGL-1 DD is a guanosine-specific, single-stranded endonuclease

12 (DD) or less from the fovea and more than 1 DD away from the optic nerve were included in this study

13 9.9% (95% CI, 47.7-52.2) for the ADD-RS </=1/DD- strategy.

14 In 924 patients with ADD-RS </=1/DD-, 3 cases of AAS were observed.

15 in patients with ADD-RS=0/DD- or ADD-RS </=1/DD-.

16 inal vascularization was delayed (mean, 0.11 DD/week vs. 0.23 DD/week) in those with versus without r

17 m June 2013 to June 2015, we identified 1768 DD livers exported to regional candidates with MELD scor

18 ion was delayed (mean, 0.11 DD/week vs. 0.23 DD/week) in those with versus without recurrence, respec

19 as a doublet of aspartic acid residues ((369)DD(370)) in the membrane-distal end of the L-selectin ta

20 sed (mean, 1.76 disc diameters [DD] vs. 4.48 DD), and the rate of retinal vascularization was delayed

21 s PI3Kbeta binding to Gbetagamma (p110(526KK-DD)).

22 iable method of producing cultures of >/=90% DD Mtb in starved cells.

23 ion that beta-lactamases have evolved from a DD-peptidase ancestor.

24 sitions from initial DU until formation of a DD syndrome.

25 hydrolysis of a beta-lactam while that of a DD-peptidase cannot?

26 transposons that encode a transposase with a DD(E/D) catalytic domain that is topologically similar t

27 tude lower than that of the corresponding AA.DD duplex.

28 ion is observed for homo-sequence 2-mers (AA.DD) where there are no competing folding equilibria.

29 lements present in common databases but also DDs of novel type.

30 Children with ASD (n = 517) and DD (n = 194) were recruited through the California Depar

31 y severe disease, is associated with ASD and DD.

32 polysorbate 80 levels were tested by BMD and DD.

33 Ex vivo dermoscopy and DD with adapted sectioning in a dermatopathology setting

34 usions and Relevance: Ex vivo dermoscopy and DD with adapted sectioning in a dermatopathology setting

35 With EVD and DD, a specific clinicopathologic diagnosis was made in 2

36 opsy specimens examined in 2015 with EVD and DD.

37 opsy specimens examined in 2015 with EVD and DD.

38 cking of self-administration frequencies and DD clinical features.

39 d possible homology, between hippocampal and DD/DL circuitry.

40 were tested as comparator agents for MIC and DD methods, respectively.

41 re established: AD, 0.03 to 0.25 mug/ml, and DD, 33 to 43 mm.

42 A results in increased affinity for NADH and DD-CoA turnover but with a reduction in Vmax for DD-CoA,

43 studies involving KIAA0319 polymorphisms and DD risk.

44 transverse cutting with lesion-specific and DD-guided cutting.

45 ions between KIAA0319 genetic variations and DD has yielded inconsistent results.

46 cts were categorized into +DD ( n = 29) and -DD ( n = 18) groups.

47 tations, we resequenced the gene in 7162 ASD/DD patients (2446 previously reported) and 2169 unaffect

48 R); and two non-arginyl dipeptides: Asp-Asp (DD) and Glu-Asp (ED).

49 the conjugates of the target bacterium-AuNP@DD in the SALDI process.

50 The generated AuNPs@DD has an absorption maximum at approximately 518 nm.

51 ate DD-immobilized gold nanoparticles (AuNPs@DD) from one-pot reactions.

52 The gold ions generated from the AuNPs@DD anchored on the target bacteria were monitored.

53 ets of beta-lactam antibiotics are bacterial DD-peptidases that catalyze the final steps of peptidogl

54 exist for comorbid emotional and behavioral DDs.

55 isotropy compared with youth with behavioral DDs in these WM tracts and (2) that there would be signi

56 with emotional DDs vs those with behavioral DDs, those with both DDs, and the controls (F6,160 = 2.4

57 binding studies of the first double-H-bonded DD.AA-type complexes of a series of aromatic boronic aci

58 s those with behavioral DDs, those with both DDs, and the controls (F6,160 = 2.4; P = .032; all pairw

59 n the absence of C-terminal partner (VirA (C)DD) or a downstream catalytic domain, the N-terminal DD

60 We calculated DD prevalence for each definition and determined the ove

61 FKBP12-derived destabilizing domain to Cas9 (DD-Cas9) enables conditional Cas9 expression and tempora

62 connections between land cover and childhood DD, we compiled a database of 293,362 children in 35 cou

63 larly similar to two of the three culturable DD treponeme phylotypes: specifically, the Treponema med

64 ory motor neuron (MN) classes, the dorsal D (DD) and ventral D (VD) neurons, extend axons along both

65 Mutant IDH1 downregulates the DNA damage (DD) sensor ATM by altering histone methylation, leading

66 led genes was observed in constant darkness (DD) as determined by reverse transcription-quantitative

67 hythmicity under standard constant darkness (DD) conditions.

68 ished activity rhythms in constant darkness (DD) without disrupting the timekeeping mechanism in brai

69 behavioral alterations in constant darkness (DD), including a temporal advance in peak activity.

70 lse (LP) to cultures maintained in darkness (DD).

71 resent study showed that dopamine-deficient (DD) mice, which had received daily L-DOPA injections, co

72 DYRK1A in patients with developmental delay (DD) and autism spectrum disorder (ASD) suggests a differ

73 ding variable levels of developmental delay (DD) and more severe speech and language delay in compari

74 ual disability (ID) and developmental delay (DD) is increasing as next-generation sequencing technolo

75 (ASD) and many forms of developmental delay (DD) originate during fetal development.

76 ldren with ASD (N=415), developmental delay (DD) without ASD (N=188), and general population (GP) con

77 notype that includes ID/developmental delay (DD), characteristic facial features, short stature, and

78 h priority goals for anti-HIV drug delivery (DD) research: (1) to prevent new HIV infections from occ

79 from first drug use (DU) to drug dependence (DD) onset, we estimate a parsimonious set of parameters

80 ess than that of the unmodified drug depots (DDs).

81 tagonist SFRP4, in surgical specimen-derived DD myofibroblasts.

82 DD_DETECTION is able to detect DDs purely from paired-end read alignments.

83 generating such "differentially detectable" (DD) Mtb in vitro would aid studies of the biology and dr

84 asymmetrically position dorsal determinants (DDs) towards the prospective dorsal side.

85 2 and two degradation domains (FKBP-DD, DHFR-DD), in multiple orientations, and determined (i) the fo

86 choroidal melanomas located 1 disc diameter (DD) or less from the fovea and more than 1 DD away from

87 id hemorrhage of more than 3 disc diameters (DD) of various etiologies, attending Tilganga Institute

88 on was decreased (mean, 1.76 disc diameters [DD] vs. 4.48 DD), and the rate of retinal vascularizatio

89 othioamides (ATAs) and 1,2-diaza-1,3-dienes (DDs).

90 d 15 S. pyogenes isolates by disk diffusion (DD) methods.

91 D-dimer (DD) is highly sensitive for AAS but is inadequate as a s

92 chlorinated DD/Fs comprise dibenzo-p-dioxin (DD), 1- and 2-monochlorodibenzo-p-dioxin (1-, 2-MCDD), 1

93 mbers in the development of data disclosure (DD) strategies.

94 Delay discounting (DD), the tendency to discount the value of delayed versu

95 The efforts of discovering DDs in high-throughput sequencing data are currently dom

96 Diarrheal disease (DD) due to contaminated water is a major cause of child

97 Individuals with Dupuytren disease (DD) are commonly seen by physicians and surgeons across

98 Depressive disorders (DDs), anxiety disorders (ADs), obsessive-compulsive diso

99 oromandibular joint (TMJ) disc displacement (DD) and degenerative joint disease (DJD) has never been

100 without (-) bilateral TMJ disc displacement (DD).

101 ion and the recruitment of the death domain (DD) adaptor protein MyD88 into an oligomeric post recept

102 the relevance of the p75(NTR) death domain (DD) and the highly conserved transmembrane residue Cys(2

103 tant mice lacking the p75(NTR) death domain (DD) or a highly conserved transmembrane (TM) cysteine re

104 The assembly of MyD88 death domain (DD) with TRAF3 (anti-viral/anti-inflammatory) and TRAF6

105 TM phosphorylation of the PIDD death domain (DD), which enables RAIDD recruitment to PIDD.

106 ll molecule by using a destabilizing domain (DD).

107 We first identify a PGL dimerization domain (DD) and determine its crystal structure.

108 ated by C- and N-terminal "docking domains" (DDs).

109 Under Share 35, deceased donor (DD) livers are offered regionally to candidates with Mod

110 Ex vivo dermoscopy (EVD) with derm dotting (DD) improves clinicopathologic correlation and the quali

111 Ex vivo dermoscopy (EVD) with derm dotting (DD) improves clinicopathologic correlation and the quali

112 distribution in the CaM-binding site in DR5 DD by the point mutations of W354A, E355K, R359A, L363N,

113 W354A, E355K, R359A, L363N, or E367K in DR5 DD could directly contribute to the experimentally obser

114 e point mutations of the key residues in DR5 DD.

115 between alpha2 helix and alpha3 helix of DR5 DD.

116 Dispersed duplications (DDs) such as transposon element insertions and copy numb

117 A peptide aptamer DVFLGDVFLGDEC (DD) that can recognize S. aureus and methicillin-resista

118 Diastolic dysfunction (DD) is an independent predictor of HFpEF risk, associate

119 is often preceded by diastolic dysfunction (DD).

120 candidate genes for developmental dyslexia (DD) owing to its important role in neuronal migration.

121 of eight cases with developmental dyslexia (DD) revealed several regions shared by the affected indi

122 and those with both behavioral and emotional DDs would show significantly lower fractional anisotropy

123 e hypothesized that (1) youth with emotional DDs and those with both behavioral and emotional DDs wou

124 uncinate fasciculus in youth with emotional DDs vs those with behavioral DDs, those with both DDs, a

125 Empirical DD probabilities observed soon after newly incident use

126 cal approach for comparative epidemiological DD research creates new opportunities in planned studies

127 whereas no effect was observed with RE, ER, DD, or ED.

128 TECTION, a database-free approach to finding DD events in high-throughput sequencing data.

129 with ERT2 and two degradation domains (FKBP-DD, DHFR-DD), in multiple orientations, and determined (

130 wed that Shld1, the chemical inducer of FKBP-DD, does not interfere with stem cell differentiation, w

131 We found that the FKBP-DD confers the PB transposase with a higher transpositio

132 In addition, we found that the FKBP-DD regulates transposon activity in a reversible and dos

133 expression (GAL80-DD) and recombination (FLP-DD).

134 (kappa) was 0.73 (95% CI, 0.64 to 0.83) for DD and 0.76 (95% CI, 0.68 to 0.83) for DJD.

135 ot fully explain the genetic association for DD.

136 nsider this gene as a putative candidate for DD.

137 lts suggest NCAN as a new candidate gene for DD and indicate that NCAN variants affect brain structur

138 39% of the study sample as indeterminate for DD.

139 Our results indicate a requirement for DD signaling by disulfide-linked dimers of p75(NTR) for

140 [SD] age, 12.9 [5.1] years); 17 studies for DD, 10 for AD, 8 for OCD, and 1 for PTSD.

141 search that may lead to novel treatments for DD and fibrosis more generally.

142 oA turnover but with a reduction in Vmax for DD-CoA, impairing overall activity.

143 ere significantly larger (all P < 0.01) for +DD compared to -DD subjects by 1.7-, 2.5-, and 1.9-fold

144 affinity similar to soluble fibrin fragment DD(E) (inhibition constant=0.53-0.83 muM).

145 Mn-FBP binds the soluble fibrin fragment DD(E) with Kd = 110 nM.

146 ith ASD might be etiologically distinct from DD without ASD.

147 hemically controlling gene expression (GAL80-DD) and recombination (FLP-DD).

148 In contrast, thioridazine did not generate DD Mtb from starved cells but killed those generated by

149 ucing agent and protective group to generate DD-immobilized gold nanoparticles (AuNPs@DD) from one-po

150 Greater DD, i.e., a tendency to choose smaller immediate over la

151 he diagnosis of AAS; 8 patients with AAS had DD-.

152 d a middle value (PD50), and eventual higher DD risk asymptotes at higher DU frequencies.

153 F-kB pathway, and with itself, revealing how DD dimerization controls access of intracellular effecto

154 Additionally, we identify three candidate ID/DD-associated genes of which two have an established rol

155 ellectual disability/developmental delay (ID/DD).

156 l disability and developmental disorders (ID/DD) and successfully identify 15 genes with clustering m

157 In DD, individual neuronal oscillators in all circadian sub

158 remodeling of the postsynaptic apparatus in DD and VD neurons using targeted expression of the acety

159 L1/L2 transition, OIG-1 is downregulated in DD neurons by the transcription factor IRX-1/Iroquois, a

160 analysis of PER shows that these dynamics in DD and LP are recapitulated in vivo.

161 Response to placebo is large, especially in DD.

162 under 24-h light-dark (LD) cycles but not in DD.

163 lar ACh levels were significantly reduced in DD mice.

164 ) of 44%, higher than previously reported in DD liposarcoma.

165 orrected the evening activity phase shift in DD.

166 We also showed that 12% of the variance in DD was accounted for by genotype and that the genetic si

167 larger (all P < 0.04) by up to 8.5-fold in +DD compared to -DD subjects.

168 ficantly larger ( P = 0.012) by 1.4-fold in +DD compared to -DD subjects.

169 consensus diagnosis of normal/indeterminate, DD with reduction, or DD without reduction.

170 Individual DD letters were mailed to each participating family, and

171 Forty-seven subjects were categorized into +DD ( n = 29) and -DD ( n = 18) groups.

172 In newly born larvae, DDs receive cholinergic inputs in the dorsal nerve cord.

173 vincentii-like and Treponema phagedenis-like DD spirochetes.

174 sigmoid pattern with one asymptote for lower DD probability (when DU is insubstantial), upturning slo

175 hmic organizer, indicating that FGF and Mek1(DD) initiate qualitatively and/or quantitatively distinc

176 By contrast, Mek1(DD) expression fails to rescue the tectal stem zone and

177 ession of a constitutively active Mek1 (Mek1(DD)), the known ERK activator, restores the tectal stem

178 C, including PlsX and KASII (FA metabolism), DD-transpeptidase and MurB (peptidoglycan synthesis), gl

179 Other genes modulating DD respecification such as lin-14, irx-1, and oig-1 are

180 Furthermore a mutant DD with the phospho-mimetic residue Asp at this position

181 gest that TRAF3/TRAF6 binding sites on MyD88 DD partially overlap, as do IRAK4 and FADD.

182 omains results in a stable fold for VirFG (N)DD and an overall protein-protein complex of unique topo

183 atalytic domain, the N-terminal DD (VirFG (N)DD) exhibits multiple characteristics of an intrinsicall

184 DD was considered negative (DD-) if <500 ng/mL.

185 The detected chlorinated and nonchlorinated DD/Fs comprise dibenzo-p-dioxin (DD), 1- and 2-monochlor

186 RIP2 CARD and RhoGDI bind to p75(NTR) DD at partially overlapping epitopes with over 100-fold

187 The p75(NTR) DD forms non-covalent, low-affinity symmetric dimers in

188 neurons lacking p75(NTR), only the p75(NTR) DD, or just Cys(259) were all equally resistant to prone

189 Application of DD-SRM to human serum and tissue provides precise quanti

190 xample, we predict that, within the class of DD motor neurons, only three (DD04, DD05, or DD06) shoul

191 niform, reliable, and accurate definition of DD for epidemiologic studies is needed.

192 concordance of 4 established definitions of DD in the Coronary Artery Risk Development in Young Adul

193 Four published definitions of DD were studied.

194 heterogeneity in the existing definitions of DD.

195 and clinical factors and the definitions of DD.

196 al health research and in the development of DD strategies for individuals and the community at large

197 ude community partners in the development of DD strategies to enhance translation of the research fin

198 line joint-specific soft tissue diagnoses of DD, 598 (76%) joints showed no change; 109 (14%) demonst

199 als with a surgically validated diagnosis of DD from the UK, with replication in British, Dutch, and

200 whether they contribute to the generation of DD Mtb or kill them.

201 is the first report describing isolation of DD treponemes from a wildlife host, suggesting that the

202 ate significant variability in prevalence of DD and its association with established risk factors acr

203 Prevalence of DD ranged from 2% to 32% across the 4 definitions, with

204 We evaluated the prevalence of DD using previously published definitions in a populatio

205 Prevalence of DD varies markedly in CARDIA by the definition applied.

206 over is associated with lower probability of DD downstream.

207 ), supporting the physiological relevance of DD signaling by disulfide-linked dimers of p75(NTR) in t

208 y genotype and that the genetic signature of DD overlapped with attention-deficit/hyperactivity disor

209 Thus, the active sites of DD-peptidases and serine beta-lactamases are very simila

210 en DU is insubstantial), upturning slopes of DD risk beyond a middle value (PD50), and eventual highe

211 was to assess the longitudinal stability of DD and DJD among 401 subjects.

212 performed a genome-wide association study of DD using 23,127 research participants of European ancest

213 rs (SNRIs), and placebo for the treatment of DD, AD, OCD, and PTSD in children and adolescents.

214 We have identified a new family of DDs in trans-acyl transferase PKSs, exemplified by a mat

215 ceptor activation triggered by separation of DDs.

216 Based on DD connectivity, we illustrate the close similarity, and

217 ffects of specific KIAA0319 polymorphisms on DD risk with greater precision, as they vary across popu

218 The syndrome is characterized by ID and/or DD, malformations of the cerebral cortex, epilepsy, visi

219 ID from mothers of children with ASD-noID or DD suggests that the intellectual disability associated

220 normal/indeterminate, DD with reduction, or DD without reduction.

221 ivity of the subdivisions of dorsal pallium (DD) of an electric gymnotiform fish, Apteronotus leptorh

222 Rbeta, platelet-derived growth factor (PDGF)-DD, opened the possibility of a previously unidentified

223 roduce platelet-derived growth factor (PDGF)-DD, which promotes cellular proliferation, epithelial-me

224 PDGF-DD engagement of NKp44 triggered NK cell secretion of in

225 lignant cell heterogeneity modulated by PDGF-DD signaling in pancreatic neuroendocrine tumors (PanNET

226 delayed in the absence of signaling by PDGF-DD.

227 the dissemination of tumors expressing PDGF-DD more effectively than control mice, an effect enhance

228 Surprisingly, ablation of PDGF-DD did not affect the vasculature or stroma of PanNET; i

229 However, the precise function of PDGF-DD in tumor growth and invasion remains elusive.

230 Thus, while cancer cell production of PDGF-DD supports tumor growth and stromal reaction, it concom

231 istinctive transcriptional signature of PDGF-DD-induced cytokines and the downregulation of tumor cel

232 s and innate lymphoid cells, recognizes PDGF-DD.

233 troma of PanNET; instead, we found that PDGF-DD stimulated bulk tumor cell proliferation by induction

234 characterized by signaling through the PDGF-DD/PDGFRbeta axis.

235 entry involves the predator's peptidoglycan DD-endopeptidases, which decrosslink cell walls and prev

236 s results, suggests a model in which the PGL DD dimer forms a fundamental building block for P-granul

237 tophosphorylated and that the phosphorylated DD is unable to form Myddosomes.

238 We addressed complex formation between PIDD-DD and RAIDD-DD in solution using heteronuclear nuclear

239 ree-layered arrangement containing five PIDD-DDs in one layer, five RAIDD-DDs in a second layer and a

240 A positive DD test result had an overall sensitivity of 96.7% (95%

241 ggest that DacA-1 is V. cholerae's principal DD-carboxypeptidase.

242 apted to co-express, from the same promoter, DD-Cas9 with any other gene of interest without co-modul

243 Of several published DD criteria, it is unclear which, if any, are applicable

244 complex formation between PIDD-DD and RAIDD-DD in solution using heteronuclear nuclear magnetic reso

245 ining five PIDD-DDs in one layer, five RAIDD-DDs in a second layer and an additional two RAIDD-DDs.

246 viest species in solution and that two RAIDD-DDs are loosely attached to the 5:5 core.

247 n a second layer and an additional two RAIDD-DDs.

248 The recognition of a S-DD response to cefepime should alert clinicians to the p

249 reduction in susceptibility, with 1% being S-DD.

250 S) dropped from 77% to 43%, with 34% being S-DD.

251 ation anti-HIV DDS need to address selective DD to the gut mucosa and lymph nodes.

252 owden JA, Kucklick JR, Baatz JE, Spyropoulos DD.

253 loped an antibody-independent deep-dive SRM (DD-SRM) approach that capitalizes on multidimensional hi

254 D MNs, which are divided into two subgroups: DD and VD.

255 > 0.8) with that of the previously suggested DD susceptibility genes KIAA0319, CTNND2, CNTNAP2 and GR

256 ish, Apteronotus leptorhynchus: superficial (DDs), intermediate (DDi) and magnocellular (DDmg) compon

257 downstream catalytic domain, the N-terminal DD (VirFG (N)DD) exhibits multiple characteristics of an

258 ng (AD) and zones by disk diffusion testing (DD).

259 We hypothesize that DD/DL circuitry can implement the same pattern separatio

260 The DD treponemes were isolated from lesional tissues but no

261 The DD(E/D) group, in addition to being the largest and most

262 report structures of complexes formed by the DD of p75 neurotrophin receptor (p75(NTR)) with RhoGDI,

263 cket to facilitate substrate turnover in the DD-CoA binding region of the protein.

264 s of protection observed in mice lacking the DD or only Cys(259) were identical to those of p75(NTR)

265 s that require pre-existing knowledge of the DD elements to be detected.

266 to prevent premature synapse rewiring of the DD motor neurons and, together with the UNC-55 nuclear h

267 to establish a more precise estimate of the DD risk associated with these genetic variations.

268 istered to gauge community perception of the DD strategies.

269 ronal death induced by p75(NTR) requires the DD and TM Cys(259), supporting the physiological relevan

270 In addition, we show that the DD subnuclei have complex reciprocal connections with su

271 sly demonstrated for the complex between the DDs of CD95 and FADD, the NMR data for this system are c

272 Thus, DD-SRM holds great promise for precisely measuring extre

273 with compared to women without bilateral TMJ DD.

274 Of the agents tested, rifamycins led to DD Mtb generation, an effect lacking in a rifampin-resis

275 mpaired DNA repair, increased sensitivity to DD, and reduced HSC self-renewal, independent of TET2.

276 jects strongly to DDi, with lesser inputs to DDs and DDmg.

277 y larger (all P < 0.01) for +DD compared to -DD subjects by 1.7-, 2.5-, and 1.9-fold for day, night,

278 0.04) by up to 8.5-fold in +DD compared to -DD subjects.

279 ( P = 0.012) by 1.4-fold in +DD compared to -DD subjects.

280 ed under light/dark LD conditions than under DD conditions.

281 han that of hydrogel-incorporated unmodified DDs.

282 or well-differentiated/de-differentiated (WD/DD) liposarcomas has been reported to be minimal, howeve

283 ics in patients with retroperitoneal (RP) WD/DD liposarcoma treated at The University of Texas MD And

284 ADD-RS (either ADD-RS=0 or ADD-RS </=1) with DD may be considered to standardize diagnostic rule out

285 ation of pretest probability assessment with DD testing is feasible, but the safety and efficiency of

286 at the bacteria are strongly associated with DD lesions and may therefore be causal.

287 r near the KIAA0319 gene are associated with DD.

288 e used to confirm ASD, whereas children with DD and TD were confirmed by Mullen Scales of Early Learn

289 Results: Use of EVD with DD increased the detection of positive section margins i

290 Use of EVD with DD increased the detection of positive section margins i

291 ical characteristics, usefulness of EVD with DD, and turnaround times (TATs).

292 ical characteristics, usefulness of EVD with DD, and turnaround times (TATs).

293 psy processing with the practice of EVD with DD.

294 psy processing with the practice of EVD with DD.

295 Associations of risk factors with DD varied considerably, with male sex being associated p

296 ers of children with either ASD-noID or with DD had significantly lower levels of the chemokines IL-8

297 arily by the placebo response: patients with DD exhibited significantly larger placebo responses (g =

298 th male sex being associated positively with DD for one definition (odds ratio = 1.4, 95% confidence

299 ant within the NCAN gene co-segregating with DD in the pedigree.

300 of children with either ASD-noID, those with DD, or GP controls.

301 ical trials of SSRIs or SNRIs in youths with DD, AD, OCD, or PTSD were included.