ライフサイエンスコーパス: DG

1 DG ALDLT with ABOi and ABOc graft combination seems to b

2 tudies, either systemic insulin or central 2-DG causes an increase in gastric motility.

3 espond to either Ca(2+) or 2-deoxyglucose (2-DG) stimulation.

4 glycolysis inhibition with 2-deoxyglucose (2-DG).

5 The combination of 2-deoxy-D-glucose (2-DG) and UA-4 induced cell cycle arrest in G2/M phase, pr

6 ose withdrawal or the glycolytic inhibitor 2-DG.

7 s to metformin treatment and impairment of 2-DG-induced motility.

8 activity is stimulated by either Ca(2+) or 2-DG in the soma, dendrites, and axons of hippocampal neur

9 lysoPC(18:1), PE(18:3/16:0), PC(20:1/18:3), DG(24:1/22:6/0:0), PS(18:2/18:0), PI(16:0/16:0), PS(18:0

10 Additionally, 3-deoxyglucosone (3-DG) and 3-deoxygalactosone (3-DGal), both known to be pr

11 methylfurfural (HMF) and 3-deoxyglucosone (3-DG), as well as reduced the formation of overall free fl

12 Methylglyoxal, 3-DG, and glyoxal were the predominant 1,2-dicarbonyl comp

13 dicarbonyl compounds after baking but only 3-DG was significantly reduced by HT.

14 The inhibition of the 3-DG and HMF formation was directly and significantly corr

15 In left CA2/3/DG, we found greater neural differentiation for items th

16 of polyglucose, namely, 4-deoxyglucosone (4-DG) and 3,4-dideoxypentosone (3,4-DDPS), could be identi

17 n increases GABAergic synaptic strength in a DG-dependent manner that mimics homeostatic scaling up i

18 ey update their expectations after playing a DG by reinforcement learning to construct a model that e

19 centrations in the hippocampi of mice with a DG knockout of the GRIN1 gene were not significantly dif

20 In a mouse model with a DG knockout of the GRIN1 gene, we further confirmed that

21 ort- and long-term clinical outcomes of ABOi DG ALDLT.

22 dical records of patients who underwent ABOi DG ALDLT between 2008 and 2014.

23 ily removed, and a one-pot C-H acetoxylation/DG removal protocol was also discovered.

24 link between adiponectin/AdipoR2 activation, DG neuronal excitability and contextual fear extinction,

25 Actomyosin DG transport was unexpected, since filamentous parasite

26 provide insight into how disorders affecting DG, including aging, stress, and depression, influence c

27 nd post-synaptic laminin receptors and alpha-DG and pikachurin in the synaptic space to maintain prop

28 ar dystrophy, acting as a link between alpha-DG and laminin(s).

29 muscular dystrophies characterized by alpha-DG hypoglycosylation and reduced extracellular ligand-bi

30 poglycosylation of alpha-dystroglycan (alpha-DG), a proportion of which show central nervous system i

31 to laminin(s) and alpha-dystroglycan (alpha-DG), an integral part of the dystrophin-glycoprotein com

32 n glycans found on alpha-dystroglycan (alpha-DG), and we recently demonstrated that initiation of cdh

33 te new approaches for restoration of F-alpha-DG in mature muscle fibers with defects in FKRP function

34 , we investigated the restoration of F-alpha-DG in the FKRP mutant muscles and showed that the restor

35 F-alpha-DG in the regenerating fibers reaches up to normal level

36 lly glycosylated alpha-dystroglycan (F-alpha-DG).

37 , a few muscle fibers express strong F-alpha-DG.

38 enerating fibers to produce functional alpha-DG, compensating for the defect in FKRP function.

39 e previously shown to hyperglycosylate alpha-DG.

40 binding of mAgrin to hypoglycosylated alpha-DG on muscle fibers and possibly abrogation of binding f

41 components, including hypoglycosylated alpha-DG, for potential therapeutic applications.

42 muscle tissues, an overall recovery of alpha-DG glycosylation and improved muscle strength, suggestin

43 icient for functional glycosylation of alpha-DG in regenerating fibers, but not in mature fibers.

44 l post-phosphorylation modification of alpha-DG.

45 s that initiate O-Man glycosylation on alpha-DG.

46 nger closely associated with mGluR6 or alpha-DG in the Lamb2-null.

47 Among other genes involved in the alpha-DG glycosylation process, fukutin related protein (FKRP)

48 m a CDP -ribitol present in muscles to alpha-DG, while in vitro it can be secreted as monomer of 60kD

49 cally increased perinatal apoptosis, altered DG cell composition, and impaired learning and memory.

50 ession of NRXN3 mRNA was observed in CA1 and DG regions of FMR1-KO mice.

51 -) hippocampal cultures or in mature CA1 and DG wild-type (Np(+/+)) neurons treated with a function-b

52 imultaneously recorded activity from CA3 and DG of behaving rats when local and global reference fram

53 three multicell layers in the CA1, CA3, and DG regions of the hippocampus.

54 ssociation between periodontal pathogens and DG.

55 mmune/dermatologic disorders can manifest as DG, although the two more common are oral lichen planus

56 mmon FGs (e.g. ketone, alcohol and amine) as DGs has been continuously pursued.

57 major categories: use of ketone carbonyls as DGs, direct beta-functionalization, and alpha-alkylation

58 d temporal rules for information transfer at DG-CA3 connections.

59 Spike transfer at DG-CA3 interneurons recorded in the juxtacellular mode w

60 rules for efficient information transfer at DG-CA3 synaptic connections in the intact circuit.

61 , in the Lamb2-null, beta-dystroglycan (beta-DG) expression is altered, co-localization of beta-DG wi

62 beta-Dystroglycan (beta-DG) is a transmembrane protein with critical roles in ce

63 ta-DG at Tyr(890) is a key stimulus for beta-DG nuclear translocation.

64 pathway has already been described for beta-DG, the intracellular trafficking route by which beta-DG

65 inally, we show that phosphorylation of beta-DG at Tyr(890) is a key stimulus for beta-DG nuclear tra

66 n complex Sec61 mediates the release of beta-DG from the ER membrane, making it accessible for import

67 versity is attributed to the ability of beta-DG to target to, and conform specific protein assemblies

68 pression is altered, co-localization of beta-DG with dystrophin and the glutamate receptor mGluR6 is

69 de intracellular trafficking route that beta-DG follows from PM to the nucleus.

70 In this study, we demonstrated that beta-DG undergoes retrograde intracellular trafficking from t

71 ntracellular trafficking route by which beta-DG reaches the nucleus is unknown.

72 ough pikachurin remains associated with beta-DG, pikachurin is no longer closely associated with mGlu

73 Finally, correlations between DG convolution and neocortical gyrification (or capacity

74 uently, during differentiation of adult-born DG granule cells, Sema7A promotes dendrite growth, compl

75 togenetic place avoidance test, whereas both DG- and BLA-labelled mice that underwent reward conditio

76 ippocampus, and increased NRXN3 mRNA in CA1, DG, and S1 cortex between female WT and FMR1-KO mice.

77 ogether, these data suggest that the CA2/CA3/DG serves to differentiate competing contextual represen

78 and left CA2/3 (r = -0.41; P = .04) and CA4/DG (r = -0.43; P = .03) subfields, and impaired left hip

79 to evaluate the role of Lactobacillus casei DG (LC-DG) and its postbiotic (PB) in modulating the inf

80 whether mature dentate gyrus granule cells (DG GCs) also contribute.

81 Here we characterize DG-CA3 synaptic transmission in vivo using targeted opto

82 examine the DV binding to the CLEC5A-coated DG-AuNP chip.

83 om baseline in vomiting frequency, composite DG Symptom Severity score, GE, and safety.

84 mption, we show two functionally contrasting DG-SOMI-types.

85 Diacylglycerol kinase (DGK) converts DG into phosphatidic acid.

86 eurons, implying that enhancing or dampening DG neuronal excitability may cause resistance to or faci

87 our previous report with fibroblast-derived DG neurons, chronic Li treatment reduced the hyperexcita

88 tidylcholine (PC/Ox-PC), and diacylglycerol (DG) species within implantation sites of p53(d/d) mice a

89 ion as a result of increased diacylglycerol (DG) production in diabetic hyperglycaemia.

90 Diglyceride DG(18:1/20:0)-sodium adduct and protonated octadecanamid

91 However, directed DG transport is dependent on filamentous actin and a uni

92 ics and chemical perturbations that directed DG transport is independent of microtubules and presumab

93 rieties, Thadokkham-11 (TDK11) and Doongara (DG) demonstrated an over-expression of lipids and protei

94 effects of optogenetic inhibition of dorsal DG during context fear conditioning, recall, generalizat

95 al fibrillary acidic protein promoter during DG development produces an increase in the neurogenic pr

96 neuropsychiatric disorders and dysregulated DG neurogenesis is beyond correlation or epiphenomenon,

97 an be improved by interventions that enhance DG function.

98 opose a theoretical hypothesis that enhanced DG-mediated pattern separation leads to "memory flexibil

99 e associated with 13-fold increased odds for DG.

100 s) have been considered to be synonymous for DG-SOMIs.

101 tient-reported outcomes were determined from DG Symptom Severity daily e-diaries, in which patients r

102 red for the SSRI response) specifically from DG GCs and found that the effects of the SSRI fluoxetine

103 In the Dictator game (DG), people expect generous behavior from others even if

104 ifty preschoolers played two dictator games (DG) by deciding how to allocate 10 chocolates between th

105 in-specific/modular (DSM) or domain-general (DG); (2) DSM systems are considered inflexible, built by

106 nessing these developmental cues to generate DG granule neurons from human pluripotent stem cells.

107 , and that the regulation of adult-generated DG neurogenesis merits continued and focused attention i

108 n the literature as desquamative gingivitis (DG).

109 enoylcarnitine, beta-D-glucopyranuronicacid, DG(38:9), MG(20:3), LysoPC(18:2) and LysoPC(16:0).

110 ian cells possess multiple DNA glycosylases (DGs) with overlapping substrate ranges for repairing oxi

111 ABO-incompatible (ABOi) dual-graft (DG) adult living donor liver transplantation (ALDLT) is

112 MF) synapses, which connect dentate granule (DG) neurons to both CA3 and GABAergic neurons.

113 e the impact of the: (a) Pd-directing group (DG) interaction, (b) nature of oxidant, and (c) nature o

114 ic studies confirm that the directing group (DG) oxyacetamide acts as the oxygen source.

115 Directing group (DG) plays a critical role in this transformation.

116 s a new hydrazone-based exo-directing group (DG) strategy developed for the functionalization of unac

117 antage of an exo-imine-type directing group (DG) that can be generated and removed in situ.

118 Most often, directing group (DG)-assisted metallacycle formation serves as an efficie

119 dely employed as effective directing groups (DGs) to control the site-selectivity of C-H activation,

120 The hippocampal dentate gyrus (DG) and CA3 are known to contribute to these functions,

121 ed indirect evidence that the dentate gyrus (DG) and CA3 hippocampal subregions support pattern separ

122 (mf) connections between the dentate gyrus (DG) and CA3 neurons in vivo are still elusive.

123 g-interneurons (SOMIs) in the dentate gyrus (DG) control formation of granule cell (GC) assemblies du

124 like cells in the hippocampal dentate gyrus (DG) granular cell layer.

125 Moreover, we investigated the dentate gyrus (DG) granule cell reactivity and synaptic plasticity in n

126 induction in stress-activated dentate gyrus (DG) granule neurons play a crucial role in these behavio

127 ein levels selectively in the dentate gyrus (DG) in vitro.

128 nis (CA) subfields CA1-4, the dentate gyrus (DG) including a granule cell layer (GCL) and a molecular

129 interneurons of the mammalian dentate gyrus (DG) initiate the therapeutic response to antidepressants

130 that PV-PIIs in rat and mouse dentate gyrus (DG) integrate their intrinsic activity over time and can

131 Adult neurogenesis in the dentate gyrus (DG) is strongly influenced by drug-taking behavior and m

132 The hippocampal dentate gyrus (DG) is thought to be responsible for processing and enco

133 Dentate gyrus (DG) is widely thought to provide a teaching signal that

134 tly reported that hippocampal dentate gyrus (DG) neurons differentiated from induced pluripotent stem

135 in reactive astrocytes in the dentate gyrus (DG) of a mouse model for AD (5xFAD) that results in incr

136 Neurogenesis in the dentate gyrus (DG) of the adult hippocampus is a process regulated by e

137 sions of adiponectin into the dentate gyrus (DG) of the hippocampus in fear-conditioned mice facilita

138 of cells in either the dorsal dentate gyrus (DG) of the hippocampus or the basolateral complex of the

139 nce adult neurogenesis in the dentate gyrus (DG) of the hippocampus, we hypothesized that selective e

140 fewer adult-born cells in the dentate gyrus (DG) overall, reducing populations across different stage

141 evidence that the hippocampal dentate gyrus (DG) plays a critical role in memory, it remains unclear

142 le neurons in the hippocampal dentate gyrus (DG) receive their primary inputs from the cortex and are

143 etween 6.6 +/- 0.7 muM in the dentate gyrus (DG) region of the hippocampus and 22.1 +/- 4.9 muM in th

144 creased NLGN2 mRNA in CA1 and dentate gyrus (DG) regions of the hippocampus, and increased NRXN3 mRNA

145 n and survival in the ventral dentate gyrus (DG) subgranular zone of Ghsr-null mice than in that of w

146 f a disrupted function of the dentate gyrus (DG) subregion of the brain, and they improve with treatm

147 The dentate gyrus (DG) subregion of the hippocampus is widely viewed to rea

148 Synapses from the dentate gyrus (DG) to the CA3 area of the hippocampus are distinctive f

149 The dentate gyrus (DG), a part of the hippocampal formation, has important

150 ampus and particularly in the dentate gyrus (DG), a region of active neurogenesis and a target of ant

151 sed LTP in CA1 but not in the dentate gyrus (DG), although adenosine eliminated potentiation in both

152 u ammonis 2/3 (CA2/3) and CA4/dentate gyrus (DG), as well as impaired hippocampal microstructural int

153 cells (NSCs) in the postnatal dentate gyrus (DG), drastically increased perinatal apoptosis, altered

154 found that, in the developing dentate gyrus (DG), excitatory drive promotes the somatic innervation o

155 and adult neurogenesis in the dentate gyrus (DG), olfactory bulb (OB), and the olfactory epithelium (

156 tors in the adult hippocampal dentate gyrus (DG), one of the select regions of the mature brain where

157 e the subgranular zone of the dentate gyrus (DG), there is continuous production of new neurons.

158 that tonic inhibition in the dentate gyrus (DG), which maintains sparseness of neuronal activation i

159 m, pattern separation, in the dentate gyrus (DG)-CA3 circuit in resolving interference between ambigu

160 asticity, particularly in the dentate gyrus (DG).

161 reduction in the hippocampal dentate gyrus (DG).

162 ctivated granule cells in the dentate gyrus (DG).

163 (PV) interneurons within the dentate gyrus (DG).

164 loss and astrogliosis in the dentate gyrus (DG).

165 (ii) activity in ERC and the dentate gyrus (DG)/CA3 region.

166 anonical trisynaptic circuit (dentate gyrus [DG] to CA3 to CA1), spontaneous GNA in the developing hi

167 Moreover, the transgenic mice show higher DG volume and increased number of mature granule neurons

168 tness levels are associated with hippocampal DG-related memory, which is consistent with literature s

169 microdissection of autopsy human hippocampus DG and qRT-PCR miRNA analyses were combined with immunof

170 tutes the first demonstration that the human DG performs pattern separation.

171 to offer compelling evidence that the human DG supports pattern separation by obtaining critical emp

172 Moreover, the hydrazone DG can be readily removed, and a one-pot C-H acetoxylati

173 from protected primary amines, the hydrazone DGs are shown to site-selectively promote the beta-aceto

174 ed to fluoxetine, indicating that 5HT1ARs in DG GCs are sufficient to mediate an antidepressant respo

175 trieval both involve coordinated activity in DG and CA3.

176 We show changes in DG long-term potentiation (LTP) that parallel behavioral

177 rther confirmed that a selective decrease in DG GluN1 is sufficient to decrease the glutamate concent

178 The functions of some altered genes in DG-8052, totalling 5.7% at acidogenisis and 8.0% at sovl

179 crucial role in FS-induced IEG induction in DG granule neurons and associated behavioral responses.

180 neered to express functional 5HT1ARs only in DG GCs responded to fluoxetine, indicating that 5HT1ARs

181 hich involved activation of Akt signaling in DG.

182 Notably, increasing DG calbindin levels, either by direct virus-mediated exp

183 ns in the medial EC layer II projecting into DG and CA3, rapidly form a distinct representation of a

184 LC-DG and its PB attenuate the inflammatory mucosal respons

185 LC-DG and PB significantly reduced the mRNA levels of pro-i

186 luate the role of Lactobacillus casei DG (LC-DG) and its postbiotic (PB) in modulating the inflammato

187 lthy controls (HC) were treated with LPS, LC-DG and PB.

188 ve effect was more pronounced for PB than LC-DG treatment.

189 uropsids (birds and reptiles), the mammalian DG is larger and exhibits qualitatively different phenot

190 gether, these data indicate that both mature DG GCs and young abGCs must be engaged for an antidepres

191 early neural progenitors in the adult mouse DG and mediates the inhibitory effects of Sema7A on prog

192 contrast, NMDARs subunit composition at mPP-DG synapses is not altered and glycine remains the main

193 xpectedly, glycine is mainly involved at mPP-DG synapses.

194 and medial perforant path-dentate gyrus (mPP-DG) synapses in juvenile and adult rats.

195 Consistently, the mutant DG neurospheres generated fewer NSCs with defects in pro

196 t and a double-generation gold nanoparticle (DG-AuNP) chip, was designed to prove the existence of we

197 solved changes demonstrating accumulation of DG species, depletion of Ox-PC species, and increase in

198 ivo using targeted optogenetic activation of DG granule cells while recording in whole-cell patch-cla

199 lectively target and control the activity of DG granule cells (GCs) while performing whole-cell and j

200 study focused on transcriptional analysis of DG-8052 and its response to CaCO3 treatment via microarr

201 , caloric restriction increased apoptosis of DG subgranular zone cells in Ghsr-null mice, although it

202 expression was unaffected by the deletion of DG D5R neither in the home cage nor upon a shock deliver

203 T. gondii, the molecular motor dependence of DG trafficking is far from certain.

204 ays a key role in the correct development of DG and adult neurogenesis in this region.

205 ices revealed that intrinsic excitability of DG granule neurons was enhanced by adiponectin deficienc

206 ontextual fear and intrinsic excitability of DG granule neurons, implying that enhancing or dampening

207 icit inhibition of intrinsic excitability of DG neurons in AdipoR2 knockout mice.

208 ccompanied by intrinsic hyperexcitability of DG granule neurons.

209 ytes to verify that the hyperexcitability of DG-like neurons is reproduced in this different cohort o

210 produced or exacerbated by the inhibition of DG neurogenesis.

211 In contrast, the levels of DG c-Fos expression was unaffected by the deletion of DG

212 ted at acidogenic and solventogenic phase of DG-8052, respectively.

213 Plasticity of DG-CA3 connections may assist in the encoding of precise

214 ntal principles that regulate the process of DG neurogenesis and discuss recent advances in harnessin

215 Genome resequencing of DG-8052 showed no general regulator mutated.

216 e a long-standing question about the role of DG in memory and provide insight into how disorders affe

217 mory, it remains unclear whether the role of DG relates to memory acquisition or retrieval.

218 mechanisms through which the suppression of DG affects memory performance.

219 All 4 symptoms of DG (composite or individual symptoms) were significantly

220 relin significantly reduced core symptoms of DG and overall composite score compared with placebo, ac

221 tones are an especially attractive choice of DGs and substrates due to their prevalence in various mo

222 Despite the importance of DGs, nothing is known about the mechanisms underlying th

223 ng-term potentiation (LTP) at perforant path-DG synapses in naive rats.

224 However, the increase in Pd-DG interaction alone is not enough to make the mechanist

225 In general, a strong Pd-DG interaction increases the EC iodination barrier and r

226 PCR on pools and individual lines of the Pop-DG population to validate and extend the microarray resu

227 in pools of fruits from siblings of the Pop-DG population with contrasting sensitivity to develop WL

228 nhibits neuronal activity with attenuated PP-DG synapse plasticity, leading to hippocampus-dependent

229 Yet despite this significant progress, DG-assisted selective para-C-H functionalization in aren

230 ing CSDS increased survival of proliferating DG cells, which ultimately developed into mature (NeuN+)

231 1, which reduces aggression in mice, reduced DG granule cell activity during resident-intruder intera

232 fMRI) to map the precise site of age-related DG dysfunction and to develop a cognitive task whose fun

233 ed trial of patients with moderate to severe DG, relamorelin significantly reduced core symptoms of D

234 ach of the other four oxidized base-specific DGs (OGG1, NTH1, NEIL1, and NEIL3), Neil2-null mice show

235 Degenerate Clostridium beijerinckii strain (DG-8052) can be partially recovered by supplementing CaC

236 nd accurate segmentation of the HF subfields DG, CA3, CA2, CA1, prosubiculum, subiculum, presubiculum

237 h moderate to severe gastroparesis symptoms (DG).

238 motions of green fluorescent protein-tagged DGs in intracellular parasites with high temporal and sp

239 ing and optogenetic inhibition, we show that DG contributes to both of these processes.

240 Finally, we show that DG contributes to fear extinction learning, a process in

241 al evidence is needed, however, to show that DG-associated memory decline in otherwise healthy elders

242 Our findings thus suggest that DG contributes to retrieval and extinction, as well as t

243 Some data and models suggest that DG plays no role in retrieval; others encourage the oppo

244 The DG ligand agrin increases GABAergic synaptic strength in

245 ressive loss of NSCs both at the SVZ and the DG of the hippocampus.

246 the number of adult-born neurons in both the DG and OB.

247 l analysis of adult-born neurons in both the DG and the OB showed that dendritic complexity was not s

248 Assisted by the DG, the C-C cleavage of isatins occurs at room temperatu

249 Within hippocampal memory formation, the DG plays a crucial role in pattern separation, which is

250 sis of the generous behavior observed in the DG and also to the wide applicability of reinforcement l

251 ression of IEG induction specifically in the DG and an impaired behavioral immobility response 24 h l

252 vation of granule cell neurons (GCNs) in the DG and produced compulsive-like drug reinstatement.

253 matic inhibition onto GABAergic cells in the DG and project to the medial septum.

254 g mice, which ameliorated neuron loss in the DG despite persistence of ubiquitin accumulation in the

255 Short hairpin RNA knockdown of Chrna7 in the DG enhanced baseline aggression and eliminated the seren

256 ndin expression are inversely related in the DG of individuals with temporal lobe epilepsy (TLE) or A

257 The number of SOX2(+) NSCs in the DG was significantly increased in NDAN individuals as co

258 ffects on theta responses in CA1 than in the DG, and concentrations of ecto-5'-nucleotidase (CD73) we

259 ins sparseness of neuronal activation in the DG, is essential for management of interference.

260 deletion of AdipoR2, but not AdipoR1 in the DG, resulted in augmented fear expression and reduced ex

261 Thus, we found that in the DG, the neurons carrying the memory engram of a given ne

262 ndrite development of newborn neurons in the DG.

263 s associated with learning and memory in the DG.

264 ctivity and promoted global remapping in the DG.

265 owth response protein 1) specifically in the DG.

266 glial numbers and amoeboid morphology in the DG.

267 xplains the main experimental results in the DG.

268 f schizophrenia, particularly indicating the DG as a site of pathology.

269 xons, and coinjection of Atf4 siRNA into the DG reduced the effects of Abeta1-42 in the forebrain.

270 ion dynamics and NSC activation, leaving the DG modified by a functionally integrated, expanded cohor

271 that increased growth and convolution of the DG arose in stem mammals concurrently with nonperiventri

272 Rejuvenation of the DG by enhancing integration of adult-born DGCs in adulth

273 The dysfunction of the DG is accompanied by structural maladaptations, includin

274 ysis to provide compelling evidence that the DG subregion specifically sustains representations of si

275 Our results demonstrate that the DG supports representations of similar scenes that are l

276 ken together, these results suggest that the DG-CA3 glutamatergic pathway is critical for mediating b

277 thereby the flow of information through the DG circuitry.

278 puts and provide dendritic inhibition to the DG circuitry.

279 d from the children immediately prior to the DG tasks.

280 ient types of neuronal inhibition and to the DG, but not the neighboring brain areas, is presented th

281 nformation from the entorhinal cortex to the DG, whereas LTP in HILs may facilitate the temporal coor

282 The feasibility of using the DG component in a catalytic fashion was also demonstrate

283 ide evidence that novelty signals within the DG are stimulus specific rather than generic in nature.

284 ntify a subset of newly born GCNs within the DG that could directly contribute to drug-seeking behavi

285 s remain, available data indicate that these DG traits are present in all orders of mammals, includin

286 To determine the biological role of these DGs, null animal models have been generated.

287 This DG allows the required flexibility to support the format

288 to artichoke leaves and fruits according to DG-SANTE Guidelines.

289 opagates both forward to CA1 and backward to DG.

290 ing for GNA backward propagation from CA3 to DG.

291 filopodia; the structures that give rise to DG-GABA synapses and that regulate feed-forward inhibiti

292 We (DG, PH, and SP) did a survey of professional stakeholder

293 s positively associated with memory, whereas DG/CA3 retrieval activity was negatively associated with

294 (whales, dolphins, and porpoises), in which DG size, convolution, and adult neurogenesis have underg

295 nticum was not statistically associated with DG, whereas, high levels of E. corrodens were associated

296 We performed a study of 393 patients with DG (37.7% male; 9.9% with type 1 diabetes; median age, 5

297 ound in subgingival plaque for patients with DG and pGI.

298 topathogenic microorganisms in patients with DG and to compare it with the microbiologic status of in

299 tically higher in samples from patients with DG than in those with pGI.

300 worse home control hygiene of patients with DG.