ライフサイエンスコーパス: DHA

1 DHA + aspirin had no significant effect on individual pl

2 DHA extensively remodeled the acyl chains of cardiolipin

3 DHA is more effective than EPA in modulating specific ma

4 DHA stimulates the retinoid X receptor, which reportedly

5 DHA upregulated the expression of anti-oxidative enzymes

6 DHA was confined to these lipids, while plastidial lipid

7 DHA, an essential fatty acid with immunomodulatory prope

8 ncrease of total FAs (95% CI: -1.18, -0.05); DHA: beta = -0.24 kg/1% increase (95% CI: -0.45, -0.02);

9 C20:5 n-3 (EPA) and C22:6 n-3 (DHA) showed higher proportions in MM than in DM.

10 n-3, EPA) and docosahexaenoic acid (22:6n-3, DHA).

11 Both total omega-3and DHA serum levels were significantly correlated with micr

12 oups and received either a control diet or a DHA-supplemented diet for 7-8 weeks.

13 Evidence of accelerated DHA catabolism (eg, activation of phospholipases and oxi

14 the relatively innocuous omega-3 fatty acid DHA (docosahexaenoic acid), piracetam, quercetin, vitami

15 yperpolarized [1-(13)C]dehydroascorbic acid (DHA) reduction, which can be measured in vivo using non-

16 o associate with lower docosahexaenoic acid (DHA) (P = 0.052).

17 Oral docosahexaenoic acid (DHA) + aspirin therapy has been shown to reduce periodon

18 sed to concentrate 82% docosahexaenoic acid (DHA) and 11% omega-6 docosapentaenoic acid (DPA-n6) afte

19 fatty acids (n-3 FAs) docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) alter FA metabolism

20 xides are derived from docosahexaenoic acid (DHA) and eicosapentaenoic acid (EPA) to form epoxyeicosa

21 aenoic acid (EPA), and docosahexaenoic acid (DHA) and insulin resistance.

22 taenoic acid (EPA) and docosahexaenoic acid (DHA) and the intermediate and advanced stages of age-rel

23 tudies have shown that docosahexaenoic acid (DHA) attenuates epileptic seizures; however, the molecul

24 (SPMs) generated from docosahexaenoic acid (DHA) can modulate the vascular injury response.

25 taenoic acid (EPA) and docosahexaenoic acid (DHA) content, as well as for oxidative status in a colla

26 ral omega-3 fatty acid docosahexaenoic acid (DHA) has been implicated in protecting patients with vir

27 ed fatty acid (LCPUFA) docosahexaenoic acid (DHA) has proven effective at reducing fat storage in ani

28 taenoic acid (EPA) and docosahexaenoic acid (DHA) in humans have used a mixture of the 2 fatty acids

29 ls of adrenic acid and docosahexaenoic acid (DHA) in testes were significantly reduced in the PFOS tr

30 Docosahexaenoic acid (DHA) is an essential omega-3 fatty acid that is critical

31 Docosahexaenoic acid (DHA) is uniquely concentrated in the brain, and is essen

32 l, whereas lower serum docosahexaenoic acid (DHA) levels were found in ADHD adults.

33 ents is an increase in docosahexaenoic acid (DHA) levels.

34 F) in combination with docosahexaenoic acid (DHA) on corneal nerve regeneration in a mouse model of d

35 Docosahexaenoic acid (DHA) plays important physiological roles in vertebrates.

36 ctating women [1020 mg docosahexaenoic acid (DHA) plus 180 mg eicosapentaenoic acid (EPA)/d together

37 to the rationale that docosahexaenoic acid (DHA) supplementation of preterm infants may improve outc

38 Maternal docosahexaenoic acid (DHA) supplementation prevented the accumulation of macro

39 Circulating levels of docosahexaenoic acid (DHA) were also measured.

40 Here, we show that docosahexaenoic acid (DHA), a key omega-3 polyunsaturated fatty acid in synapt

41 Topical application of docosahexaenoic acid (DHA), a representative omega-3 PUFA, in wild type hairle

42 ns have suggested that docosahexaenoic acid (DHA), an n-3 long-chain polyunsaturated fatty acid, migh

43 pentaenoic acid (EPA), docosahexaenoic acid (DHA), and total omega-3 (n-3) PUFA concentrations were a

44 ved factor (PEDF) plus docosahexaenoic acid (DHA), has been shown to stimulate corneal nerve regenera

45 taenoic acid (EPA) and docosahexaenoic acid (DHA), not least of all given the importance of these fat

46 rived from the omega-3 docosahexaenoic acid (DHA), whose esterified form is transported by the major

47 taenoic acid (EPA) and docosahexaenoic acid (DHA).

48 nd cis-4,7,10,13,16,19-docosahexaenoic acid (DHA).

49 y acids, in particular docosahexaenoic acid (DHA).

50 absence or presence of docosahexaenoic acid (DHA).

51 to efficiently produce docosahexaenoic acid (DHA).

52 the omega-3 fatty acid docosahexaenoic acid (DHA).

53 fatty acids, including docosahexaenoic acid (DHA).

54 taenoic acid (EPA) and docosahexaenoic acid (DHA)/d (4.2 g total omega-3/d; n = 12) with a placebo (4

55 Docosahexaenoic acid (DHA, 22:6 n-3) is abundant in the retina and is enzymati

56 tty acids (FAs) [e.g., docosahexaenoic acid (DHA, 22:6n-3) and eicosapentaenoic acid (EPA, 20:5n-3)],

57 Docosahexaenoic acid (DHA, 22:6n-3) is an omega-3 fatty acid essential for pro

58 atty acids (LC-PUFAs), docosahexaenoic acid (DHA, C22:6) and eicosapentaenoic acid (EPA, C20:5) are u

59 Docosahexanoic acid (DHA) is an important constituent of the brain.

60 id [EPA], + 510 mg/day docosahexaenoic acid [DHA]), or fish oil (1000 mg/day EPA + 500 mg/day DHA) fo

61 The eye acquires DHA from blood, but transporters for DHA uptake across t

62 in the presence of DHA and, in turn, affects DHA structural and aggregative properties.

63 Interactions among DHA, APOE genotype, and stage of AD pathologic changes m

64 Total daily intakes of ALA, AA and DHA were below, whereas LA was above the recommended int

65 ecreasing trend in eicosapentaenoic acid and DHA with PFOS concentrations was observed.

66 ion enhances EPA (eicosapentaenoic acid) and DHA (docosahexaenoic acid) abundance in the skin, compre

67 e confirmed a significant role of BAIAP2 and DHA in the etiology of ADHD exclusively in adults.

68 from 876 twins with 16S microbiome data and DHA, total omega-3, and other circulating fatty acids.

69 EPA and DHA addition to the model systems increased the concentr

70 the totality of current evidence for EPA and DHA and advanced AMD is discordant, though there was no

71 ion enriched chick adipose tissue in EPA and DHA and reduced adiposity by promoting more, but smaller

72 gs highlight the global shortfall of EPA and DHA and the implications this has for the human consumer

73 es (PBLs) and the relation to plasma EPA and DHA concentrations in Alzheimer disease (AD) patients.In

74 P = 0.003, respectively), as did the EPA and DHA content in adipose tissue (P < 0.0001 and P < 0.0001

75 pecial efforts are needed to improve EPA and DHA contents in filets of triploids.

76 lmitate)f).In the omega-3 group, the EPA and DHA contributions to plasma free fatty acids increased (

77 ulated cumulative average intakes of EPA and DHA from FFQs and also computed predicted erythrocyte an

78 Although production of both EPA and DHA has been engineered into land plants, including Arab

79 Fish oil markedly enhanced EPA and DHA in mouse skin within 2 weeks, and this increase plat

80 pite increases in adipose and plasma EPA and DHA in the omega-3 group, there were no significant chan

81 re diploids had higher percentage of EPA and DHA in their muscle tissue (filets) compared to that of

82 ly increased alongside a decrease in EPA and DHA levels.

83 Higher intakes of EPA and DHA may prevent or delay the occurrence of visually sign

84 Nevertheless, the contents of EPA and DHA per mass of the filets in diploid and triploid speci

85 artum weight reduction by increasing EPA and DHA status together with a decreased ratio of omega-6-to

86 ntroversial protective mechanisms of EPA and DHA to better design dietary interventions aimed at redu

87 ted erythrocyte and plasma scores of EPA and DHA yielded slightly stronger inverse associations for i

88 ociation of marine-derived n3 PUFAs (EPA and DHA) with T2D.

89 t in fish oil, the primary source of EPA and DHA, affected adipose development in offspring.

90 oils led to a fast decrease of both, EPA and DHA, and to the development of characteristic volatile c

91 e of specific proportions of omega-3 EPA and DHA, in the modulation of inflammation and oxidative str

92 of manuka honey correlates with its MGO and DHA content.

93 Non-manuka honeys, which lack MGO and DHA, showed significantly less urease inhibition.

94 ion by manuka honey is mainly due to MGO and DHA.

95 herapy using optimized doses of Physcion and DHA as a novel antileukemia treatment without inducing h

96 lly-developing P. falciparum ring stages and DHA-pretreated dormant rings (DP-rings) using a panel of

97 that functional fat-1 and topically applied DHA potentiate cellular defense against UVB-induced skin

98 unt of DHA (0.2-0.3% of total fatty acids as DHA; control group).

99 Between DHA and EPA, changes in CRP (-7.9% +/- 5.0% compared wit

100 We found even stronger associations between DHA and 38 operational taxonomic units (OTUs), the stron

101 owed positively correlated responses between DHA and several channel blockers, suggesting potential s

102 s 2 (Fads2) that enable them to biosynthesis DHA through a more direct route termed the "Delta4 pathw

103 r parental educational level and whole-blood DHA.This study showed associations between rs1535 minor

104 ill oil, do not significantly increase brain DHA, because they are hydrolyzed to free DHA and are abs

105 me amount of free DHA did not increase brain DHA, but increased the DHA in adipose tissue and heart.

106 dietary LPC-DHA efficiently increases brain DHA content and improves brain function in adult mammals

107 ial cells and this release was attenuated by DHA supplementation.

108 enetetrazole was significantly attenuated by DHA, and letrozole completely inhibited this suppressive

109 he elongation of seizure latency elicited by DHA.

110 ed in the prevention of seizures mediated by DHA.

111 hniques, that H50Q and aS4ox are modified by DHA, whereas acetyl-aS is not.

112 g their ability to be chemically modified by DHA.

113 old men had a >2.2-fold higher plasma (13)C-DHA concentration synthesized from (13)C-EPA compared wi

114 The higher plasma (13)C-DHA seen in old men may be a result of slower plasma DHA

115 Hyperpolarized [1-(13)C]DHA can be used, therefore, to assess the capacity of tu

116 ere that the rate of hyperpolarized [1-(13)C]DHA reduction is increased in tumors that have been oxid

117 Here, we first confirmed that cardiac DHA levels are elevated in diabetic humans relative to c

118 rtially remove DPA-n6, further concentrating DHA to 89%.

119 nnon index) after adjusting for confounders (DHA Beta(SE) = 0.13(0.04), P = 0.0006 total omega-3: 0.1

120 As a consequence, DHA is significantly reduced in the serum of SCA38 subje

121 acterize oil product, which mainly contained DHA (15.81 mol%) and EPA (20.23 mol%).

122 rain barrier that transports LPCs containing DHA and other long-chain fatty acids.

123 ), or fish oil (1000 mg/day EPA + 500 mg/day DHA) for 90 days, with monthly study visits.

124 y that oxidized vitamin C, dehydroascorbate (DHA), can induce oxidative stress and cell death in canc

125 ion of oxidized ascorbate (dehydroascorbate, DHA).

126 spontaneous respiration, suggesting dietary DHA also protects the brain.

127 , 7321) with cyclohexane, dihydroanthracene (DHA), and xanthene (Xan), we show here that KIE is a sel

128 Dihydroartemisinin (DHA) was found to disrupt hemoglobin catabolism within 1

129 ls to antimalarial agent dihydroartemisinin (DHA).

130 ART derivatives such as dihydroartemisinin (DHA), a small population of the early ring-stage parasit

131 he parasite responses to dihydroartemisinin (DHA) and various Ca(2+) and Na(+) channel blockers and s

132 acrocycles incorporating two dihydroazulene (DHA) photoswitching subunits, bridged by linkers of vary

133 asure methylglyoxal (MGO), dihydroxyacetone (DHA) and leptosperin simultaneously.

134 le dehydration reaction of dihydroxyacetone (DHA) to methylglyoxal (MGO) in a honey model system has

135 study of the conversion of dihydroxyacetone (DHA) to methylglyoxal (MGO) in maturing New Zealand manu

136 al (MGO) and its precursor dihydroxyacetone (DHA), which are naturally present in manuka honey, were

137 High-dose DHA supplementation in APOE4 carriers before the onset o

138 andomly assigned to receive 400 mg of either DHA or placebo/d from 18 to 22 wk of pregnancy until del

139 , were added to artificial honey with either DHA or MGO and stored at 20, 27 and 37 degrees C.

140 may be a consequence of increased endogenous DHA synthesis in infancy but not at school-age.

141 Enteral DHA supplementation at a dose of 60 mg per kilogram per

142 I], 0.66-0.92; P trend, 0.008) and for EPA + DHA was 0.83 (95% CI, 0.71-0.98; P trend, 0.03).

143 liance with primary oxidation limits and EPA/DHA content by a recently published assessment of fish o

144 microalgae are the primary producers of EPA/DHA and promising alternatives for fish oil.

145 nt marine microalgae that can synthesize EPA/DHA will solve these problems.

146 91% of the products complied with EPA/DHA content claims.

147 noteworthy health effect of 1:1 and 2:1 EPA:DHA proportions over 1:2 EPA:DHA based diets through a d

148 1:1 and 2:1 EPA:DHA proportions over 1:2 EPA:DHA based diets through a down-regulation in the product

149 VHF; VHF = vinylheptafulvene) with the first DHA undergoing isomerization with a similar efficiency a

150 hazard ratio (HR) between the 2 cohorts for DHA comparing the extreme quintiles of intake was 0.78 (

151 monstrated that two alternative pathways for DHA biosynthesis exist in teleosts.

152 IMECs required MFSD2A, which is required for DHA retention and metabolism in the gut vasculature.

153 These findings suggest an important role for DHA metabolism in brain amyloid deposition during the pr

154 cquires DHA from blood, but transporters for DHA uptake across the blood-retinal barrier or retinal p

155 In contrast, the same amount of free DHA did not increase brain DHA, but increased the DHA in

156 -R) leading to the release of DHA; this free DHA led to enhanced docosanoid synthesis and induction o

157 ain DHA, because they are hydrolyzed to free DHA and are absorbed as triacylglycerol, whereas the tra

158 ured by Morris water maze test, whereas free DHA had no effect.

159 rain regions with LPC-DHA, but not with free DHA.

160 lipid vesicles to mitochondria isolated from DHA-fed mice, rescued the major losses in the mitochondr

161 lp to identify the optimal timing for future DHA clinical trials.

162 asked, placebo-controlled trial of daily 2 g DHA or placebo capsules enriched with 81 mg aspirin; 46

163 which 174 AD patients received either 1.7 g DHA and 0.6 g EPA (the n-3 FA group) or placebo daily fo

164 mined a 3.25 A crystal structure of the GLIC-DHA complex in a potentially desensitized conformation.

165 ration of DHA (1% of total fatty acids; high-DHA group) or a standard amount of DHA (0.2-0.3% of tota

166 no evidence of differences between the high-DHA and standard-DHA groups in any of the visual-process

167 with nano-encapsulated fish oil had a higher DHA and EPA contents than yogurt containing free fish oi

168 This study aimed to clarify how DHA suppresses seizures, focusing on the regulation of 1

169 to resist oxidative stress in vivo However, DHA administration resulted in transient respiratory arr

170 polarized [1-(13)C] dehydroascorbic acid (HP DHA), which is reduced to Vitamin C (VitC) rapidly in th

171 ce, we observed a significant decrease in HP DHA to VitC conversion that accompanied hepatic fat depo

172 These findings suggest that HP DHA, a potentially clinically translatable imaging agent

173 cubated with DHA ex vivo produced 17-hydroxy DHA (17-HDHA) and D-series resolvins, as assessed by liq

174 e n-3 FA group displayed marked increases in DHA and EPA plasma concentrations (2.6- and 3.5-fold), a

175 mentation with an n-3 FA preparation rich in DHA on global DNA methylation of peripheral blood leukoc

176 es have associated omega-3 intake, including DHA, with a reduced risk for incident AD.

177 e (LPC) (40 mg DHA/kg) for 30 days increased DHA content of the brain by >2-fold.

178 Instead, DHA increased the concentration of the downstream specia

179 INTERPRETATION: DHA supplementation is a safe and effective treatment fo

180 iched with some essential nutrients (Inulin, DHA & EPA, vitamins B6, K1, and D3) as enhancers of calc

181 We investigated DHA-channel interaction by manipulating both the fatty a

182 clover honey doped with 2000 or 10,000mg/kg DHA and for artificial honey with 2000mg/kg of DHA and e

183 LDL-DHA nanoparticle selectively kills hepatoma cells and re

184 However, rats given LDL-DHA had smaller, pale tumors that were devoid of vascula

185 Injection of LDL-DHA into the hepatic artery of rats selectively deregula

186 We investigated the ability of LDL-DHA to reduce growth of orthotopic hepatomas in rats.

187 Four to 6 days after injection of LDL-DHA, the tumors were 3-fold smaller than those of contro

188 Likewise, DHA treated to JB6 cells inhibited Nrf2 ubiquitination a

189 These studies show that dietary LPC-DHA efficiently increases brain DHA content and improves

190 Moreover, LPC-DHA treatment markedly improved the spatial learning and

191 ctor increased in all brain regions with LPC-DHA, but not with free DHA.

192 We sought to determine whether maternal DHA supplementation during the second half of pregnancy

193 Mechanistically, DHA did not directly target B cells to elevate Ab levels

194 mice as lysophosphatidylcholine (LPC) (40 mg DHA/kg) for 30 days increased DHA content of the brain b

195 Moreover, DHA lowered enzyme activities of respiratory complexes I

196 brain mediated the physiological actions of DHA.

197 Here we show that oral administration of DHA to normal adult mice as lysophosphatidylcholine (LPC

198 itis, with or without oral administration of DHA.

199 profile, availability, and affordability of DHA supplements, refining an omega-3 intervention in APO

200 ids; high-DHA group) or a standard amount of DHA (0.2-0.3% of total fatty acids as DHA; control group

201 erval: 3% to 35%) of the observed benefit of DHA on PD.

202 copherol loaded nanoliposomes as carriers of DHA and EPA and to investigate their physicochemical pro

203 to milk containing a higher concentration of DHA (1% of total fatty acids; high-DHA group) or a stand

204 35mol%), achieving a 2-fold concentration of DHA.

205 the APOE4 allele could alter the delivery of DHA to the brain may be amenable to DHA supplementation

206 A modest effect of DHA + aspirin on Porphyromonas gingivalis counts was ass

207 he role of His-50, we analyzed the effect of DHA on aS-derived species: a naturally occurring variant

208 Little is known about the effects of DHA on established solid tumors.

209 barrier is specific for phospholipid form of DHA.

210 rthermore, there is no significant impact of DHA and EPA production on seed yield in either the green

211 explain the mixed evidence of the impact of DHA supplementation on birth weight.

212 m of this study is to evaluate the impact of DHA with low-dose aspirin therapy on periodontal bacteri

213 and osteoarthritis pain were independent of DHA levels.

214 redisposition to obesity, maternal intake of DHA-rich fish oil during the second half of pregnancy do

215 A and for artificial honey with 2000mg/kg of DHA and either alanine or proline and alanine added.

216 d adolescents with ADHD have lower levels of DHA (seven studies, n=412, g=-0.76, p=0.0002), EPA (seve

217 Levels of DHA-derived epoxides are lower in colon tissues from pat

218 re directly activated by nanomolar levels of DHA.

219 lical secondary structure in the presence of DHA and, in turn, affects DHA structural and aggregative

220 storage temperature accelerated the rate of DHA loss and the initial rate of formation of MGO, but b

221 These results show that the rate of DHA reduction depends not only on the level of reduced g

222 lyzes the glutathione-dependent reduction of DHA.

223 -receptor (PEDF-R) leading to the release of DHA; this free DHA led to enhanced docosanoid synthesis

224 gic changes may explain the mixed results of DHA supplementation reported in the literature.

225 he present study, we evaluated the safety of DHA supplementation, its efficacy for clinical symptoms,

226 Most of the current supplements of DHA, including fish oil and krill oil, do not significan

227 tributes a significant antioxidant effect on DHA and EPA.

228 ebo (ES=-0.39) or DHA (ES=-0.60) and less on DHA than placebo (ES=+0.21); furthermore, EPA-placebo se

229 roved more on EPA than placebo (ES=-0.39) or DHA (ES=-0.60) and less on DHA than placebo (ES=+0.21);

230 ed oil composition to now include EPA and/or DHA.

231 an" AND "dihydroartemisinin-piperaquine" OR "DHA-PPQ".

232 We first confirmed that PEDF + DHA increased nerve regeneration in the mouse cornea.

233 ly, corneal injury and treatment with PEDF + DHA induced transcription of neuropeptide y (npy), small

234 In addition, PEDF+DHA accelerated corneal wound healing, selectively recru

235 right eye and treated in both eyes with PEDF+DHA for 2 weeks, there was a significant increase in cor

236 lts suggest that topical treatment with PEDF+DHA promotes corneal nerve regeneration and wound healin

237 the seizures induced by pentylenetetrazole, DHA significantly prolonged the seizure latency.

238 respectively), but not to changes in plasma DHA concentration, and were not related to apolipoprotei

239 in old men may be a result of slower plasma DHA clearance with age.

240 shed Ab titers whereas the Western diet plus DHA improved titers.

241 on was noted between adipose tissue EPA plus DHA and HOMA-IR.

242 ue n-3 FAs (total n-3 FAs, ALA, and EPA plus DHA) and insulin resistance in healthy adults.

243 Differential responses to prenatal DHA supplementation on the basis of the genetic makeup o

244 tica lipase A (CAL-A) was applied to produce DHA concentrate by controlling the rate and extent of hy

245 receive either an enteral emulsion providing DHA at a dose of 60 mg per kilogram of body weight per d

246 wise, light-induced, ring-opening reactions (DHA-DHA to DHA-VHF to VHF-VHF; VHF = vinylheptafulvene)

247 had reduced levels of inflammation-resolving DHA-derived epoxy metabolites compared to healthy colon

248 e uncyclized parent system while the second (DHA-VHF to VHF-VHF) is significantly slower.

249 Main Outcomes and Measures: Serum DHA levels with cerebral amyloidosis measured using PIB

250 ticipants: Cross-sectional analysis of serum DHA levels together with measures of amyloid deposition

251 clusions and Relevance: In this study, serum DHA levels were associated with pathogenesis of cerebral

252 ifferences between the high-DHA and standard-DHA groups in any of the visual-processing measures.

253 towards C24 PUFA enabling them to synthesise DHA through the Sprecher pathway.

254 expressed in the cytoplasm, and synthesizes DHA and EPA de novo from malonyl-CoA without substantial

255 eeded to determine the effect of a long-term DHA supplementation per se on cardiovascular disease ris

256 ies in rats and rainbow trout confirmed that DHA biosynthesis proceeds through the so-called "Spreche

257 imal and in vitro studies also indicate that DHA prevents amyloid deposition in the brain.

258 The results suggest that DHA and the channel form an ion-dipole bond to promote o

259 These results suggest that DHA delays the onset of seizures by promoting the synthe

260 t randomized, controlled trial suggests that DHA + aspirin therapy improves periodontitis largely by

261 The DHA molecule is bound at the channel-periphery near the

262 The DHA- or eicosapentaenoic acid (EPA)-derived 26 carbon fa

263 n-3DPA (RvD5n-3DPA) and maresin (MaR)-1, the DHA vicinal diol 19,20-dihydroxy-DPA and n-6 PUFA derive

264 from changes in P. gingivalis levels in the DHA + aspirin treatment group.

265 age occurred in 52.3% of the infants in the DHA group and in 46.4% of the infants in the control gro

266 tion occurred in 53.2% of the infants in the DHA group and in 49.7% of the infants in the control gro

267 al cognitive abilities was attenuated in the DHA group compared with in the placebo group (P-interact

268 ificant pre-post clinical improvement in the DHA group versus placebo, using the Scale for the Assess

269 o sign of brain function whereas rats in the DHA-treated group had recurrent seizures and spontaneous

270 id not increase brain DHA, but increased the DHA in adipose tissue and heart.

271 Of the 592 infants assigned to the DHA group, 291 (49.1% by multiple imputation) were class

272 ivery of DHA to the brain may be amenable to DHA supplementation in predementia stages of AD.

273 -induced, ring-opening reactions (DHA-DHA to DHA-VHF to VHF-VHF; VHF = vinylheptafulvene) with the fi

274 ining the susceptibility of P. falciparum to DHA during early blood-stage development.

275 1 and Jagged 1 protein levels in response to DHA supplementation in vivo but similar results were not

276 arkably slow conversion of the second VHF to DHA.

277 increase the half-life of the second VHF-to-DHA conversion from 65 to 202 h at room temperature by s

278 The energy-releasing, VHF-to-DHA, ring closures also occur in a stepwise manner and a

279 ocytes express high levels of Glut1, take up DHA, and reduce it to VC, we tested how erythrocytes mig

280 by an open-label study with overall 40-week DHA treatment.

281 Whether DHA + aspirin therapy influences specific bacterial burd

282 rol and new insight into mechanisms by which DHA promotes brain development and function.

283 While DHA did not affect the duration or scores of the seizure

284 umulated into de novo synthesized TAGs while DHA-TAG species remained rather stable, indicating an in

285 sults show that replacing linoleic acid with DHA lowers select cardiac enzyme activities by potential

286 Moreover, aS forms a covalent adduct with DHA.

287 ent of neurological diseases associated with DHA deficiency, such as Alzheimer's disease.

288 The association of cognitive benefit with DHA supplementation in predementia but not AD dementia s

289 etary supplementation of a Western diet with DHA as a tool to promote cardiac acyl chain remodeling a

290 Artificial honeys doped with DHA and individual perturbants were fitted first, then m

291 in the conversion between clover doped with DHA and manuka honey were observed.

292 ve oil, and a virgin olive oil enriched with DHA, on vitamin D absorption in rats.

293 ffects of long-term treatment of humans with DHA and EPA on various epigenetic factors-such as DNA me

294 Intact human arteries incubated with DHA ex vivo produced 17-hydroxy DHA (17-HDHA) and D-seri

295 experimental values, for the reactions with DHA and Xan (Angew.

296 Dietary supplementation with DHA increased not only the expression of P450arom, but a

297 mice fed standard chow and supplemented with DHA in vitro.

298 lyphenols in the olive oil supplemented with DHA inhibited vitamin D postprandial response in rats (-

299 Combined treatment with DHA and Physcion activates AMP-activated protein kinase,

300 per day or a control (soy) emulsion without DHA until 36 weeks of postmenstrual age.