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1 DMPC (but not lecithin) raises HDL cholesterol and apoA-
2 DMPC in the form of small unilamellar vesicles (SUVs) or
3 DMPC or soy or egg lecithin at 1.0 mg/mL was added to th
4 DMPC-binding assays demonstrate an identical vesicle cle
8 e the apparent melting temperature of apoC-1:DMPC complexes by up to 20 degrees C and decelerate prot
12 ed the expected interaction with ApoE(1-191).DMPC, but surprisingly CR16-18 did not interact with thi
16 4F.DMPC complex is different than in the 2F.DMPC complex as evidenced by the NOE between lipid 2.CH
18 he conformation of the DMPC sn-3 chain in 4F.DMPC complex is different than in the 2F.DMPC complex as
21 , in contrast to 2F in 2F.DMPC, 4F in the 4F.DMPC complex is located closer to the lipid headgroup as
22 heptapeptide anchor (ANCH) in water and in a DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) bilay
23 ucture of the full-length H-ras protein in a DMPC bilayer has been computationally characterized.
24 ower coupling (14%) when the peptide is in a DMPC bilayer suggests a high degree of peptide conformat
25 cs of caveolin-1 (D82-S136; Cav182-136) in a DMPC bilayer using NMR, fluorescence emission measuremen
30 he permeation of Na(+) and Cl(-) ions across DMPC lipid bilayer by computing the corresponding potent
34 reorganization are unfavorable, whereas ANCH-DMPC interactions-especially van der Waals-favor inserti
36 hyl-ammonio]-1-propane sulfonate (Chaps) and DMPC/l-alpha-1,2-dihexanoyl-sn-glycero-3-phosphocholine
39 amic quantities characterizing pure DMPC and DMPC/cholesterol mixtures, thus directly confirming the
40 the volume expansion coefficient of DMPC and DMPC/Cholesterol samples with 13 and 25 mol% cholesterol
43 e of formation of rHDL from rcm apo A-II and DMPC at all FC mole percentages is faster than that of a
44 n the presence of both types of peptides and DMPC vesicles in the presence of nonselective peptides.
46 fect the BaP uptake rate by DMPC-NP-SLBs and DMPC-SUVs, indicating preferential BaP sorption into the
47 viability and growth using DMPC-NP-SLBs and DMPC-SUVs, with and without BaP, as their sole carbon so
48 on cooling from the heat-denatured state and DMPC clearance studies revealed that protein secondary s
50 onolayers of ternary POPC/SM/Chol as well as DMPC/SM/Chol mixtures, which exhibit a surface-pressure-
51 probabilities for direct transitions between DMPC and DHPC were negligible, a third component with in
53 quid crystalline medium, DMPC/DHPC bicelles (DMPC = dimyristoylphosphatidylcholine; DHPC = dihexanoyl
58 tter, does not affect the BaP uptake rate by DMPC-NP-SLBs and DMPC-SUVs, indicating preferential BaP
62 ion we obtained an rHDL structure comprising DMPC, cholesterol, and apolipoprotein AI (423:74:1 mol/m
64 istoylphosphatidylcholine (d54-DMPC) and d54-DMPC/dimyristoylphosphatidylglycerol (DMPG) were used to
65 uterated dimyristoylphosphatidylcholine (d54-DMPC) and d54-DMPC/dimyristoylphosphatidylglycerol (DMPG
66 shorter tail component (either DDPC in DDPC/DMPC mixtures or DMPC in DMPC/DSPC mixtures) extending 2
67 ade NLPs from dimyristoylphospatidylcholine (DMPC) in combination with each of four different apolipo
68 c model of a dimyristoylphosphatidylcholine (DMPC) bilayer at various cholesterol concentrations via
71 s containing dimyristoylphosphatidylcholine (DMPC, di-C(14) tails) and dihexanoylphosphatidylcholine
72 line (DLPC), dimyristoylphosphatidylcholine (DMPC), dioleoylphosphatidylcholine (DOPC), and 1-palmito
73 tituted from dimyristoylphosphatidylcholine (DMPC) and human apolipoprotein C-I (apoC-I, 6 kDa) or it
74 tituted from dimyristoylphosphatidylcholine (DMPC) and selected mutants of human apolipoprotein C-1 (
75 idylcholine (dimyristoylphosphatidylcholine (DMPC) and palmitoyloleoylphosphatidylcholine (POPC)) and
77 id phases of dimyristoylphosphatidylcholine (DMPC) and dilauroylphosphatidylcholine (DLPC) were obtai
78 crystals of dimyristoylphosphatidylcholine (DMPC) and diphytanoylphosphatidylcholine (DPhPC), and th
79 mulations of dimyristoylphosphatidylcholine (DMPC) bilayers to model the creation of bilayer gaps-a c
80 ilization of dimyristoylphosphatidylcholine (DMPC) membranes by apo A-I to give rHDL increases as the
81 ilization of dimyristoylphosphatidylcholine (DMPC) multilamellar vesicles by apolipoprotein A-I (apoA
82 permeabilize dimyristoylphosphatidylcholine (DMPC) and dimyristoylphosphatidylglycerol (DMPG) vesicle
83 osed of pure dimyristoylphosphatidylcholine (DMPC) or palmitoyl-oleoylphosphatidylethanolamine (POPE)
84 n solubilize dimyristoylphosphatidylcholine (DMPC) liposomes and fold into approximately 60% alpha-he
86 neously when dimyristoylphosphatidylcholine (DMPC) large unilamellar vesicles (LUVs) were incubated w
87 DeltaG1 with dimyristoylphosphatidylcholine (DMPC) and 1-palmitoyl-2-oleoyl-phosphatdylcholine bilaye
88 teracts with dimyristoylphosphatidylcholine (DMPC) over a wide range of lipid:peptide ratios from 1:1
90 -PLA(2) with dimyristoylphosphatidylcholine (DMPC) vesicles and found that specific residues 113-120
92 membrane-mimetic dimyristoylphosphocholine (DMPC) or 1-palmitoyl-2-oleoyl-sn-glycerophosphocholine (
93 anism of pore formation and closure in DLPC, DMPC, and DPPC bilayers, with pore formation free energi
94 ster than approximately 10(5) s(-1) in DLPC, DMPC, and POPC bilayers, but the motion is slowed by 2 o
95 P31) and the lipid hydrophobic length (DLPC, DMPC, and DPPC), a wide range of mismatch conditions wer
99 yers with different bilayer thickness, i.e., DMPC and POPC, the intramolecular distance reported by T
102 vesicles (SUVs) or DMPC-NP-SLBs with excess DMPC-SUVs to support colloidal stability, when added to
105 lidate the new unilamellar vesicles data for DMPC, so method 4 is not required for DLPC or future stu
106 ept one of three independent simulations for DMPC and all three DLPC simulations, where the bilayer t
107 ta for the relative form factors F(q(z)) for DMPC were obtained using a combination of four methods.
111 (w/v) phospholipid concentration and a high DMPC/DHPC ratio (q = 2.0) was found to be optimal for no
112 annel model was embedded in a fully hydrated DMPC lipid bilayer, and molecular-dynamics simulations w
113 mopentameric TM2 channel in a fully hydrated DMPC membrane using large-scale computation suggest a mo
114 complexes are similar to those of plasma A-I/DMPC complexes formed under similar conditions: small di
118 either DDPC in DDPC/DMPC mixtures or DMPC in DMPC/DSPC mixtures) extending 2-3 nm away from the prote
123 nal helix has a transmembrane orientation in DMPC bilayers, whereas in POPC bilayers, this domain is
127 tra indicate that the presence of protein in DMPC results in a broad lipid phase transition that is s
128 that opsin can also be directly purified in DMPC/DHPC bicelles to give correctly folded functional o
130 of the on-time distributions of Nile Red in DMPC and SOPC vesicles were significantly different.
133 ly packed transmembrane melittin tetramer in DMPC shows formation of a toroidal pore after 1 mus.
134 ngle of 15 +/- 3 degrees in POPC, whereas in DMPC, 25 +/- 3 degree and 30 +/- 3 degree tilts were obs
135 ssociate with and dissociate from individual DMPC and SOPC vesicles adsorbed on a glass surface, gene
136 arkably, spontaneous insertion of BclXL into DMPC/DHPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine/1
138 Measurements of WALP19 in the ether-linked DMPC analogue ditetradecylphosphatidylcholine (missing t
139 different ratios of the zwitterionic lipid (DMPC, dimyristoyl phosphatidylcholine; DOPC, dioleoyl ph
140 ained in a single liquid crystalline medium, DMPC/DHPC bicelles (DMPC = dimyristoylphosphatidylcholin
144 nd a mutant form apoE4R158M to multilamellar DMPC vesicles was similar and was reduced and eventually
145 or the disk diameter among different mutant-DMPC complexes, the mutations have no significant effect
148 rent concentration on the phase behaviour of DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) multi
149 Here, supported phospholipid bilayers of DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) were
150 at the segmental order parameters (S(CD)) of DMPC approach very large values of approximately 0.35 in
151 ar vesicles indicate that the acyl chains of DMPC are highly disordered in the presence of the peptid
152 led that the volume expansion coefficient of DMPC and DMPC/Cholesterol samples with 13 and 25 mol% ch
153 econstituted vesicle dispersions composed of DMPC, C20BAS/E. coli polar lipid, C20BAS/POPC, C32phytBA
154 ptide bound to membrane bicelles composed of DMPC, DMPG, and DHPC, and compare it to the location of
156 rmotropic phase transition of MLVs formed of DMPC and the DMPC/DMPG (7:3) mixture suggests specific l
159 Specifically, an aqueous phase incubation of DMPC vesicles with purified apolipoprotein A-I results i
160 of the experiment corresponds to the T(m) of DMPC, and domain boundaries between gel and liquid-cryst
161 model is then extended to treat mixtures of DMPC and cholesterol, where small domains of different c
165 -fusion inhibitor, decreased the ordering of DMPC headgroups, whereas arachidonic acid, a membrane-fu
172 to be modulated by the q value, the ratio of DMPC to DHPC, which reflects changes in the bicelle size
173 ated with a slower rate of solubilization of DMPC vesicles by apoE4-mut1 and reduced binding of the p
174 concentration, and the lipid phase state of DMPC, the kinetics varied over 3 orders of magnitude.
176 eract differently with AQP0 than do those of DMPC, but the acyl chains in the EPL and DMPC bilayers o
180 rbidity clearance of unilamellar vesicles of DMPC, is faster at acidic pH values and consistent with
182 otein secondary structure in solution and on DMPC correlates strongly with the maximal temperature of
183 pha-helical content in solution (33%) and on DMPC disks (67%) similar to that of the wild type (WT),
186 ponent (either DDPC in DDPC/DMPC mixtures or DMPC in DMPC/DSPC mixtures) extending 2-3 nm away from t
187 ituted into a TFE/H(2)O mixture or a POPC or DMPC bilayer were estimated to be 10.6 +/- 0.5, 16.8 +/-
188 form of small unilamellar vesicles (SUVs) or DMPC-NP-SLBs with excess DMPC-SUVs to support colloidal
191 ep and immediate weakening, whereas the P294-DMPC binding was slightly strengthened at pH 3.7 and the
193 egrees C did not cause colour change in PCDA/DMPC vesicles for a period of up to 60days of storage.
195 e in membrane fluidity between the gel phase DMPC and the liquid crystal phase POPC for peptide-membr
197 tron density profiles with that of gel phase DMPC provides areas per lipid A, 60.6 +/- 0.5 A(2) for D
201 imyristoyl-sn-glycero-3-phosphatidylcholine (DMPC)/1,2-dimyristoyl-sn-glycero-3-phosphatidylglycerol
202 icelle or a dimyristoyl phosphatidylcholine (DMPC) bilayer, have been used to explore the conformatio
204 plexes with dimyristoyl phosphatidylcholine (DMPC) that resemble nascent HDL were analyzed by density
205 binding to dimyristoyl phosphatidylcholine (DMPC) vesicles and to triglyceride (TG)-rich emulsion pa
208 hospholipid dimyristoyl-phosphatidylcholine (DMPC) the two-state model was sufficient to account for
209 1)) and 1,2-dimyristoyl-phosphatidylcholine (DMPC-d(54)) at different temperatures demonstrates the i
210 yristoyl or dipalmitoyl phosphatidylcholine (DMPC or DPPC), the latter without or with cholesterol, w
211 myristoyl-sn-glycero-3-phosphatidylcholine] (DMPC) interface in the OH stretching mode region of wate
212 ids dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and 1,2-dihexanoyl-sn-glycero-3-phosphocholine (DH
213 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and 1,2-dihexanoyl-sn-glycero-3-phosphocholine (DH
214 1,2-Dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and its mixtures with different amounts of cholest
215 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) and POPC/POPS 3:1 liposomes retain a bilayer macro
216 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) are investigated as constructs for removing PAHs f
217 h a 1,2-dimyristoylglycero-3-phosphocholine (DMPC) bilayer obtained from modeling and all-atom explic
218 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) bilayers in the presence of MSI-78 provides images
219 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) bilayers, the first equivalent of drug bound S31 i
220 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) is a phospholipid that does not exist in nature an
221 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) liposomes, suggesting that lateral gating of the B
222 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC) vesicles and dodecylphosphocholine (DPC) micelles
223 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC)) and phosphatidylglycerols (PGs, such as 1,2-dimyr
224 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC), 1,2-dilauroyl-sn-glycero-3-phosphocholine (DLPC),
225 -dimyristoleoyl-sn-glycero-3-phosphocholine (DMPC), 1,2-dioleoyl-sn-glycero-3-phosphocholine (DOPC),
226 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC), 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC
227 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC), 1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC
230 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC)/1,2-dipalmitoyl-sn-glycero-3-phosphocholine (DPPC)
231 1,2-dimyristoyl-sn-glycero-3-phosphocholine (DMPC)/3-[(cholamidopropyl)dimethyl-ammonio]-1-propane su
233 f 1,2-dimyristoyl-sn-glycero-phosphocholine, DMPC) and the bacterial (liposomes of 1,2-dimyristoyl-sn
234 out in explicit lipid bilayers (DEPC, POPC, DMPC, sphingomyelin), confirming the observed dependence
235 that decreasing bilayer thickness (DEPC-POPC-DMPC) led to an increase in the helix tilt angle from 10
238 thermodynamic quantities characterizing pure DMPC and DMPC/cholesterol mixtures, thus directly confir
243 dramatically improved in the mice receiving DMPC, and there was a significant reduction in aortic le
244 25-50% anionic lipids, and in both saturated DMPC/DMPG (1,2-dimyristoyl-sn-glycero-3-phosphatidylchlo
245 e simulated an excess proton near a solvated DMPC bilayer at 323 K, using a recently developed method
246 lateral mobility of globular actin-supported DMPC (1,2-dimyristoyl-sn-glycero-3-phosphocholine) bilay
252 se transition of MLVs formed of DMPC and the DMPC/DMPG (7:3) mixture suggests specific lipid-peptide
254 ne tension of approximately 10-30 pN for the DMPC edge, in qualitative agreement with experimental es
255 r, transition peak has been observed for the DMPC: cholesterol mixtures suggest that a separate chole
259 e inclusion of Chaps rather than DHPC in the DMPC/Chaps bicelles, however, imparts the greatest stabi
261 volves two factors: 1) calcium ions make the DMPC bilayer partially cationic and thus attractive to t
262 cal thinning and 2 A average thinning of the DMPC (1,2-dimyristoyl-sn-glycero-3-phosphochloline)/DMPG
266 Interestingly, at Tc = 24 degrees C of the DMPC gel-to-liquid crystal transition, the clearance rat
269 According to turbidimetric titrations, the DMPC/apo A-II stoichiometry is 65/1 (moles to moles).
270 ium strengthens Abeta peptide binding to the DMPC bilayer by enhancing electrostatic interactions bet
276 model in which apo A-I and apo A-II bind to DMPC via surface defects that disappear at 20 mol % FC.
278 scopy demonstrated that the peptide binds to DMPC with a high affinity to form at least two sizes of
281 g of the drug molecules in optically trapped DMPC vesicles, the membrane permeability and partitionin
282 inosa demonstrate viability and growth using DMPC-NP-SLBs and DMPC-SUVs, with and without BaP, as the
285 bility, the initial rate of association with DMPC liposomes, and the size of the rHDL particles.
288 ed amantadine, an antiinfluenza A drug, with DMPC bilayers were investigated by solid-state NMR and b
290 ow that LL7-27 is completely integrated with DMPC/DMPG (3:1) liposomes, but induces peptide-rich and
292 teractions of single Nile Red molecules with DMPC and SOPC lipid bilayers were studied by single mole
294 ion of gramicidin D at a 1:20 mol ratio with DMPC results in the formation of protein-lipid hydrogen
295 yl-sn-glycero-3-phosphocholine (DHPC), with [DMPC]/[DHPC] = 2.5, in 10% lipid/aqueous buffer at 25 de
298 elles, and TM2 was disordered in zwiterionic DMPC but was alpha-helical in negatively charged DMPC/DM
299 showed much weaker affinity for zwitterionic DMPC, but had moderate binding affinity to negatively ch
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