ライフサイエンスコーパス: DMSO

1 DMSO causes a drastic decrease in the range of the steri

2 DMSO concentrations >10% (v/v) have recently been report

3 DMSO concentrations higher than 4% (v/v) destabilize the

4 DMSO treatment also improves differentiation into termin

5 DMSO-d6/GL and DMSO-d6/GL-d8 binary mixture solvents see

6 DMSO-d6/GL must be preferred to DMSO-d6/GL-d8 for the st

7 DMSO-d6/GL-d8 is more appropriate for organic compounds

8 th at 48 h compared to vehicle control (0.1% DMSO) when administered 0, 1, 6, and 24 h after addition

9 Even 0.5-1% DMSO alters the KD values, in particular for the tight b

10 At XDMSO >0.1, DMSO enters the lipid interface and restricts the lipid

11 In both cases, 5-10% DMSO decreased stacking interactions and increased local

12 AMG9810 (50 mg/kg) or vehicle (2% DMSO/5% Tween 80/10 ml/kg saline) was injected intraperi

13 mechanism by which low concentrations (2-4% DMSO) induce caspase-3 independent neuronal death that i

14 of CYP142A1 toward unfolding even up to 40% DMSO.

15 e treated daily for 28 days with vehicle (5% DMSO) or rapamycin (0.25 mg/kg, intraperitoneally).

16 d bis(p-nitrophenyl) phosphate (BNPP) in 80% DMSO solution at pH values in the range 8.5-12.0.

17 e dissolved in aqueous solutions of: (1) 90% DMSO, (2) 2 M KOH and (3) 90% DMSO/2 M KOH, and stored f

18 ns of: (1) 90% DMSO, (2) 2 M KOH and (3) 90% DMSO/2 M KOH, and stored for 15 days at approximately 22

19 owed the order: 2 M KOH>90% DMSO/2 M KOH>90% DMSO.

20 crease in Rz followed the order: 2 M KOH>90% DMSO/2 M KOH>90% DMSO.

21 Mw followed the order: 90% DMSO>2 M KOH>90% DMSO/2 M KOH.

22 indicated that starch solubilisation in 90% DMSO/2 M KOH may be a reasonable method for molecular ch

23 Decrease in Mw followed the order: 90% DMSO>2 M KOH>90% DMSO/2 M KOH.

24 A DMSO or PBS vehicle control was included for each experi

25 ant (KD) is up to 10 times higher than for a DMSO-free sample in the case of carbonic anhydrase-chlor

26 s of solvated beta-hematin were grown from a DMSO solution containing the antimalarial drug chloroqui

27 ordination upon the addition of ZnCl2 into a DMSO solution containing the hybrid.

28 e corresponding alpha-ketoaldehydes by new a DMSO-NaBr-H2SO4 oxidation system in yields up to 90% wit

29 rnative exists, researchers compute absolute DMSO final concentrations and include an untreated contr

30 An unprecedented copper acetate-DMSO promoted methylthiolation of arenes and heteroarene

31 e perfusion, mice were randomly administered DMSO, Nec-1 (3.2 mg/kg/day) and Nec-1s (1.6 mg/kg/day) v

32 ctam pseudopeptides conducted in CDCl(3) and DMSO-d(6) solutions using 1D- and 2D-NMR techniques reve

33 dimsyl anion, formed from a strong base and DMSO (solvent), is responsible for inducing the initiati

34 With 20 mol % of DABCO as catalyst and DMSO as the solvent high yields have been achieved for d

35 cetonitrile-d3, acetone-d6, methanol-d4, and DMSO-d6.

36 r of magnitude faster than that of PG-d8 and DMSO-d6.

37 rt intermolecular H-bonding, such as DMF and DMSO, leading to efficient intramolecular photoreaction.

38 nt polarities (i.e., toluene, MeOH, DMF, and DMSO).

39 te, acetone, alcohol, acetonitrile, DMF, and DMSO, identify complex solvent systems, as well as disti

40 Furthermore, EtOH and DMSO can disrupt the main driving forces of these intera

41 DMSO-d6/GL and DMSO-d6/GL-d8 binary mixture solvents seem to be so far

42 s binary solvents, DMSO-d6/glycerol (GL) and DMSO-d6/glycerol-d8 (GL-d8), is reported for the first t

43 on containing glycerol, ethylene glycol, and DMSO at concentrations that approximate the widely used

44 inexpensive and readily available iodine and DMSO with a short reaction time.

45 ectively, have been investigated in MeCN and DMSO by absorption, emission, and (1)H NMR spectroscopie

46 Comparison between MeCN and DMSO clearly shows that in the monoprotonated diamines R

47 (CumO(*)), has been carried out in MeCN and DMSO.

48 s of experiments with radical scavengers and DMSO-d6 and ESI-MS observations.

49 Interestingly, freshly synthesized and DMSO-solubilized 1 was inactive.

50 erties of the hydrazones in both toluene and DMSO were assessed offering insights into the kinetics a

51 used histone H3 fractions from untreated and DMSO-treated Murine ErythroLeukemia (MEL) cells.

52 y competitive H2S and cyanide ion in aqueous DMSO media.

53 molecular iodine and sodium azide in aqueous DMSO providing a general access to geminal diazides.

54 Compounds such as DMSO and hexamethylene bisacetamide (HMBA) have been use

55 observe that higher order glymes as well as DMSO improve the solubility of Mg salts, but only the lo

56 in vitro in a retinal neuronal cell line, at DMSO concentrations >1% (v/v), using annexin V, terminal

57 to a mechanistic understanding of biological DMSO production in surface seawater.

58 n unexpected increase in non-hydrogen-bonded DMSO near the eutectic point (ca. 35 mol %) which also c

59 er network near the lipid membrane at a bulk DMSO mole fraction (XDMSO) of <0.1, regardless of the li

60 alovirus promoter, was markedly increased by DMSO treatment.

61 ving rise to Li(2)CO(3) and Li carboxylates (DMSO and tetraglyme electrolytes).

62 renes, using molecular iodine as a catalyst, DMSO as a stoichiometric oxidant, and different nucleoph

63 unced, especially, in the presence of Chatos DMSO extract.

64 eral structure [4-R-pyH](+)trans-[Ru(III)Cl4(DMSO)(4-R-py)](-), where 4-R-py stands for a 4-substitut

65 nity toward carboxylates in very competitive DMSO/water mixtures.

66 In contrast, DMSO both protects against heme loss and increases the s

67 esence of a very strong H-bonding cosolvent (DMSO), which slowed the observed rates by up to 2 orders

68 n after dissolution into either D2 O or [D6 ]DMSO.

69 O(S2)2picolinate] (Mo-pic) is stable in a d6-DMSO solution after heating at 100 degrees C, in air, re

70 SRS microscopy was used to image D2O, PG-d8/DMSO-d6, and the nail through the O-D, -CD2, and -CH2 bo

71 up in these two solvents, Deltadelta = delta(DMSO) - delta(CDCl3), can be converted into the hydrogen

72 th groups of TAC/DOCA mice (cRbm20(DeltaRRM)-DMSO and cRbm20(DeltaRRM)-raloxifene).

73 TAC/DOCA cRbm20(DeltaRRM)-DMSO mice developed LV hypertrophy and a marked increase

74 oxide (DMSO)-injected mice (cRbm20(DeltaRRM)-DMSO) as the control.

75 pluripotent stem cells in dimethylsulfoxide (DMSO) activates the retinoblastoma protein, increases th

76 canines were divided into dimethylsulfoxide (DMSO) group and 10 mM, 15 mM, and 20 mM A-803467 groups.

77 eveal the key role of the dimethylsulfoxide (DMSO) ligand in controlling this chemoselectivity.

78 r that is required by the dimethylsulfoxide (DMSO) reductase family of enzymes, which includes the ni

79 obial oxidation of DMS to dimethylsulfoxide (DMSO) represents a major sink of DMS in surface seawater

80 iven conversion of DMS to dimethylsulfoxide (DMSO).

81 ne -5.76, sucrose -6.92, dimethylsulphoxide (DMSO) -9.72, mannitol -6.69, trehalose -10.6, NaCl -11.0

82 sions compared to 50% in dimethylsulphoxide (DMSO).

83 nexpectedly, the solvent dimethylsulphoxide (DMSO) alone induced p53 binding to many sites common to

84 w concentrations (5 mul intravitreally dosed DMSO in rat from a stock concentration of 1, 2, 4, and 8

85 t with tetra-n-butylammonium fluoride in dry DMSO.

86 In these experiments, DMSO:acetone (1:40 v/v) solution of 17AAG (500 nmol) was

87 For DMSO, a first-order dependence of decomposition rates on

88 NAP-chol 1 also acts as a super gelator for DMSO.

89 ntrated solutions reduces the available free DMSO molecules that can react with Na and renders the TF

90 micelles upon dialysis of the polymers from DMSO/DMF to aqueous buffer.

91 on constant value of K = 4900 M(-1) in a H2O/DMSO (50:50 v/v) binary solvent mixture.

92 erto unexplored reagent combination PPh3.HBr-DMSO is exemplified with multiple highly diverse one-ste

93 the external morphology of the beta-hematin DMSO solvate crystals is almost indistinguishable from t

94 ine fluorescence spectroscopy to examine how DMSO affects the structure, dynamics, and ligand binding

95 To date, little attention was given to how DMSO influences protein-ligand binding strengths.

96 In DMSO with small amounts of water, the homopolymer PBA sh

97 In DMSO, a retroaldol reaction followed by fast intramolecu

98 In DMSO, the half-lives of nitrone 3 and 4-OOH adducts were

99 DB) showed turn-on response toward Mn(2+) in DMSO and acetonitrile, respectively.

100 s 2a-g and benzylidenemalonates 2h and 2i in DMSO were determined photometrically at 20 degrees C.

101 Dissolution of CP 3 in DMSO favors Co-S bond heterolysis, yielding the diamagne

102 ith 4(2-amino-1H-imidazol-5-yl)phenol (4) in DMSO.

103 onic acids promoted by copper(II) acetate in DMSO provides an attractive alternative to the earlier r

104 were performed using methanesulfonic acid in DMSO and followed by proton NMR.

105 re carried out using methanesulfonic acid in DMSO to study the hyperporphyrin effect across different

106 ase and the pKa of the corresponding acid in DMSO.

107 3- and 4-substituted phenylboronic acids in DMSO is nonlinear, with a small negative slope for elect

108 cid recognizes zwitterions of amino acids in DMSO, and its UV absorption maximum undergoes a signific

109 tives having more tendency of aggregation in DMSO-water mixed solvent showed significant increase of

110 rt a quantitative solvent effect analysis in DMSO/water mixtures for (i) the hydrolysis reaction of d

111 d the assignment in 7 M urea (pH 2.3) and in DMSO.

112 arkable selectivity for the sulfate anion in DMSO, enabling its selective sensing by fluorescence spe

113 ubppm levels (<1.0 ppm) in THF as well as in DMSO-H2O.

114 ns in 3 mM solutions of the azoporphyrins in DMSO was switched between 3.5 and 1.7 s.

115 and nitroarenes using tert-butoxide base in DMSO at room temperature.

116 heptane system requires any external base in DMSO-d6 to afford the corresponding oxime, and no revers

117 the solvent, either complete single bond (in DMSO) or double bond (in cyclohexane) rotation can be in

118 Although catalytic KO(t)Bu in DMSO is sufficient to allow imine generation, stoichiome

119 e to templation with various carboxylates in DMSO.

120 1 chiral phosphoric acid-family catalysts in DMSO were predicted using the SMD/M06-2x/6-311++G(2df,2p

121 However, dissolving cisplatin in DMSO for laboratory-based studies results in ligand disp

122 assembled reactive ester block copolymers in DMSO.

123 The plasma sample simply diluted in DMSO allowed the recovery of various amounts of sunitib,

124 fungin undergoes spontaneous dimerization in DMSO, producing dilomofungin, whose inhibition of MBNL1-

125 and a stoichiometric amount of a disilane in DMSO at room temperature.

126 re of aldehyde and malonic acid dissolved in DMSO allowed the protocol to be performed in continuous

127 , Leu-Tyr, Gly-Tyr, and Ala-Tyr dissolved in DMSO-d6/GL (8:2, v/v) and of an apolar mixture made of b

128 llal, (+)-limonene, and flavone dissolved in DMSO-d6/GL-d8 (5:5, v/v) by means of spin diffusion in h

129 er journals utilizing cisplatin dissolved in DMSO.

130 of ethylenediaminetetraacetic acid (EDTA) in DMSO exerts superior control over wafer coverage and fil

131 es C, and demonstrates greater efficiency in DMSO than in water.

132 by using solution phase electrochemistry in DMSO solutions of Fe(III)-heme plus the tested compounds

133 nd its mono- and dianion were established in DMSO solution by comparing the vicinal proton-proton cou

134 nd B acids were determined experimentally in DMSO solution using proton NMR spectroscopy.

135 uent organoid generation by slow freezing in DMSO supplemented media.

136 issue and from bulk tissues slowly frozen in DMSO supplemented media.

137 the time of labor (20 h compared to 60 h in DMSO-treated controls).

138 asic solutions of alkali-metal hydroxides in DMSO is described.

139 ed synthetic receptors toward acetate ion in DMSO-d6/H2O (99.5%:0.5% m/m).

140 K1/K2 in DMSO (1.3 x 10(7)) was significantly larger than the val

141 Either DHA (16 mg/kg in DMSO) or vehicle DMSO (1 ml/kg) was administered intrape

142 s demonstrated using precipitation of LiF in DMSO as a driving force.

143 3)2C(OH)CH2)2CHOH, 3) by pKa measurements in DMSO and H2O, negative ion photoelectron spectroscopy an

144 erestingly, IMZ is more reactive than MEI in DMSO, compared to water alone, which is attributed to th

145 dipalmitoylphosphatidylcholine membranes in DMSO-water mixtures quantify the hydration- and solvatio

146 in we report the enhanced stability of Na in DMSO solutions containing concentrated sodium trifluorom

147 Treatment of 5 with 1.5 equiv of NaOMe in DMSO at 140 degrees C for 0.5 h gave 6 in good yields.

148 ulation reveal the Na(+) solvation number in DMSO and the formation of Na(DMSO)3 (TFSI)-like solvatio

149 No C-H deactivation was observed in DMSO after addition of Li(+) and Mg(2+).

150 e applied to other ET reactions performed in DMSO.

151 oss-coupling reactions take place rapidly in DMSO in good yields and efficiently proceed in the prese

152 he rate and selectivity of LLB-A reaction in DMSO-D6 is explained with the controlled and online NMR

153 vents, where MK-2 was most easily reduced in DMSO, which may suggest a combination of solvent effect

154 ies of anions as tetrabutylammonium salts in DMSO and MeCN were studied by (1)H and (11)B NMR as well

155 rge data set of (13)C NMR chemical shifts in DMSO are presented with TMS as the calculated reference

156 Furthermore, polymer solutions in DMSO show a reversible response to fluoride ions, which

157 contribute 0.6 pKa units of stabilization in DMSO and 1.1 pKa units in benzene.

158 re, a variable-temperature (1)H NMR study in DMSO-d6 of [2]rotaxane supported the kinetic inertness o

159 detectable form in the NMR spectra taken in DMSO-d(6).

160 dging of a difference of >26 pK(a) units (in DMSO) between a propargylic hydrogen and a protonated te

161 of adjacent dimers hydrogen bond to included DMSO molecules, rather than forming carboxylic acid dime

162 ydrodynamic radius decreases with increasing DMSO concentration up to 10 mol% DMSO.

163 , by competing for hydrophobic interactions, DMSO can have a small but significant effect on RNA stru

164 Interestingly, DMSO platinum complexes show low cytotoxicity in the non

165 ted pyrazoles have been achieved using K2CO3-DMSO.

166 rbonyl compounds or amidines utilizing K2CO3/DMSO at ambient temperature that provides a straightforw

167 ryl pyrazoles to (E)-styryl pyrazoles in KOH/DMSO system.

168 alkynes using a super basic solution of KOH/DMSO has been described.

169 ed out to elucidate the mechanism of the KOH/DMSO superbase catalyzed ketones nucleophilic addition t

170 he R5 or R4 position of the phenyl ring; L = DMSO and P(C6H4CF3-p)3) has been prepared.

171 solution of the reductant Cp2Co, Mn((N,O)L)(DMSO) undergoes a ligand-centered solid-state reduction

172 n(III) affords the chain compound Mn((N,O)L)(DMSO).

173 Measured DMSO pKa values for a series of rigid tricyclic adamanta

174 -4b indicated that weaker donors (THF, MeCN, DMSO, MeOH, and even H2O) likewise promote this pathway,

175 y important anions in a highly polar medium (DMSO + 0.5% H2O).

176 increasing DMSO concentration up to 10 mol% DMSO.

177 ead group and that, from 10 mol% to 20 mol%, DMSO acts to gradually collapse head groups down onto th

178 Moreover, DMSO works as a cryogenic protector avoiding solidificat

179 ation number in DMSO and the formation of Na(DMSO)3 (TFSI)-like solvation structure.

180 Raman spectra of NaTFSI/DMSO electrolytes and ab initio molecular dynamics simul

181 alysis of how lower concentrations (<10%) of DMSO typically used in binding assays affects RNA struct

182 This at least partly explains ability of DMSO to promote ccHBV infection in such cell lines.

183 simple protocol involving the activation of DMSO by chlorotrimethysilane is described for the chemos

184 ss from the adduct position, the addition of DMSO leads to the formation of an insertion complex capa

185 binding affinity decreases upon addition of DMSO.

186 ichalcogenides using an equivalent amount of DMSO as an oxidant, under catalysis by molecular iodine.

187 afforded by the addition of a low amount of DMSO in conjunction with the inherent temporal control e

188 A decrease in the amount of DMSO in the vitrification solution with a corresponding

189 Low amounts of DMSO can reduce in-source dissociation of native protein

190 ough it is known that high concentrations of DMSO (>75%) can significantly affect RNA structure and f

191 fety concerns of using low concentrations of DMSO as a solvent for in vivo administration and in biol

192 rations below 10 mol%, the primary effect of DMSO is to decrease the solvated volume of the PC head g

193 We examined the effect of DMSO on platinum complexes, including cisplatin, carbopl

194 work emphasizes the importance of effects of DMSO as a co-solvent for quantification of protein-ligan

195 We interpret the effects of DMSO as being derived from its enrichment in the electro

196 In this study we investigated the effects of DMSO on different noncovalent protein-ligand complexes,

197 The protective effects of DMSO on labile protein interactions are an important pro

198 Although the effects of DMSO on the membrane structure and the headgroup dehydra

199 In this study, the effects of DMSO on the structure and interactions of avidin and Myc

200 These contrasting kinetic effects of DMSO provide the basis for chemoselective formation of c

201 ) and is driven by exothermic elimination of DMSO.

202 es with angstrom resolution as a function of DMSO concentration from 0 mol% to 20 mol%.

203 is desirable to understand the influence of DMSO concentration on the dissociation or unfolding beha

204 The majority of DMSO molecules solvating Na(+) in concentrated solutions

205 lar hydrogen bonds was ruled out by means of DMSO titration, DOSY experiments, and steric considerati

206 Reactions were performed in a mixture of DMSO/Bu(t)OH (10/90 v/v) at 60 degrees C and catalysed b

207 nations often rely on qualitative notions of DMSO-induced dehydration of lipid head groups.

208 ) were determined in absence and presence of DMSO.

209 mes larger, respectively, in the presence of DMSO.

210 intermediate oxidized by a basic solution of DMSO or atmospheric oxygen led to the desired sp(3) C-H

211 bstantially reduced compared to solutions of DMSO containing the same total CPA concentration.

212 The widespread use of DMSO as a cosolvent, along with its unusual attributes,

213 leukemic T315I cells and avoiding the use of DMSO as solubilizing agent.

214 Given the frequent use of DMSO in biochemical and biophysical assays, it is desira

215 his integrated method was the utilization of DMSO stock solutions of compounds registered in the corp

216 Many studies have focused on DMSO-lipid interactions and the subsequent effects on me

217 t-order dependence of decomposition rates on DMSO concentration was established.

218 on of the latter using either K3[Fe(CN)6] or DMSO/conc.

219 A volume of 0.1 ml of A-803467 or DMSO was injected into the LSG.

220 o an environmentally relevant dose of BPA or DMSO control.

221 diation at a wavelength of 365 nm of MeCN or DMSO solutions of 3-6 results, depending on the expositi

222 The capacity of peach DMSO extracts to inhibit Candida albicans growth was mor

223 thod for the deuteration of pseudoacids (pKa,DMSO = 14-19) with chloroform-d1.

224 pressive value of 1050 M(-1) in highly polar DMSO-d6.

225 mistry models were used to directly quantify DMSO/water hydrogen-bond populations in binary mixtures.

226 revealed no adverse effects of the residual DMSO after the solvent replacement.

227 The slow, DMSO frozen tissue yielded organoids with more accurate

228 pound fluorescein, where the organic solvent DMSO is exchanged against an aqueous buffer.

229 mber of alpha-amino acids in a polar solvent DMSO.

230 ion was performed in polar aprotic solvents (DMSO), the formation of their 5-CF3-substituted isomers

231 e of two new highly viscous binary solvents, DMSO-d6/glycerol (GL) and DMSO-d6/glycerol-d8 (GL-d8), i

232 Specifically, DMSO was found to effectively destabilize the hydration

233 idence for three distinct regimes: 1) strong DMSO-water interactions (<30 mol %), 2) ideal-solution-l

234 cies are dominant at 10 mol %, due to strong DMSO-water interactions.

235 etely abolished DMS oxidation and subsequent DMSO formation.

236 ally involves the use of dimethyl sulfoxide (DMSO) acting as an organic solvent for simultaneous samp

237 can tolerate up to 3.9% dimethyl sulfoxide (DMSO) and up to 10% serum, which shows its compatibility

238 solution containing 10% dimethyl sulfoxide (DMSO) at 21 degrees C.

239 protein C, we found that dimethyl sulfoxide (DMSO) can improve the stability of the noncovalent inter

240 Dimethyl sulfoxide (DMSO) disrupts the hydrogen-bond networks in water.

241 Dimethyl sulfoxide (DMSO) has been broadly used in biology as a cosolvent, a

242 ells was attributable to dimethyl sulfoxide (DMSO) in culture medium, NTCP overexpression, and HBV ge

243 mprised of Ficoll 70 and dimethyl sulfoxide (DMSO) in presence or absence of fetal bovine serum (FBS)

244 Dimethyl sulfoxide (DMSO) is a common solvent and biological additive posses

245 Dimethyl sulfoxide (DMSO) is a promising solvent for this battery but its in

246 Dimethyl sulfoxide (DMSO) is an important aprotic solvent that can solubiliz

247 Dimethyl sulfoxide (DMSO) is widely used as a cosolvent to solubilize hydrop

248 ) in a series of toluene/dimethyl sulfoxide (DMSO) mixtures and find that the experimental values sho

249 ating cosolutes [40% w/v dimethyl sulfoxide (DMSO) or acetonitrile (ACN)].

250 Dimethyl sulfoxide (DMSO) or sulfide ligands have positive and negative role

251 2-propanol (2-PrOH), and dimethyl sulfoxide (DMSO) reveal an internal substrate binding site deep in

252 ilms are obtained from a dimethyl sulfoxide (DMSO) solution via a transitional SnI2.3DMSO intermediat

253 a = 1.6 x 10(4) M(-1) in dimethyl sulfoxide (DMSO)).

254 M) supplemented with 10% dimethyl sulfoxide (DMSO), 15% human serum albumin (HSA) and 0.1% hyaluronan

255 , the assay tolerates 5% dimethyl sulfoxide (DMSO), and it has a Z-score of 0.71, indicating HTS comp

256 ntains a small amount of dimethyl sulfoxide (DMSO), the adduct is able to move to a solvent-exposed c

257 oxylated alcohols and/or dimethyl sulfoxide (DMSO), which can damage cell membranes.

258 taRRM)-raloxifene), with dimethyl sulfoxide (DMSO)-injected mice (cRbm20(DeltaRRM)-DMSO) as the contr

259 the dominant pathway in dimethyl sulfoxide (DMSO).

260 common organic solvent, dimethyl sulfoxide (DMSO).

261 onium persulfate (AP) or dimethyl sulfoxide (DMSO).

262 roxyl radical scavenger (dimethyl sulfoxide, DMSO), and different pH values.

263 on with a polycation in dimethyl sulphoxide (DMSO), the solution was applied underwater to various su

264 ene glycol (PG-d8), and dimethyl sulphoxide (DMSO-d6) were separately applied to the dorsal surface o

265 enes, and guanidine catalyzed by the KOBu(t)/DMSO system leads to 2-aminopyrimidines in up to 80% yie

266 late with the catalytic activity better than DMSO pKa values and appear to be a better measure of aci

267 We recommend that methods other than DMSO are employed for solubilizing drugs but, where no a

268 lity, leading to the general assumption that DMSO-induced structural changes in cell membranes and th

269 Here, we demonstrate that DMSO induces retinal apoptosis in vivo at low concentrat

270 , carboplatin, and oxaliplatin, finding that DMSO reacted with the complexes, inhibited their cytotox

271 ces in multilamellar vesicles, we found that DMSO exclusively weakens the surface water network near

272 We postulate that DMSO acts as an efficient cryoprotectant even at low con

273 Tandem MS/MS experiments showed that DMSO could modify the dissociation pathway of CYP142A1,

274 The DMSO content in the nanoESI buffer was increased systema

275 The DMSO removal performance could be significantly increase

276 olves hydrogen bonding between water and the DMSO aggregate species.

277 s amphipathic helix and that it enhances the DMSO cryopreservation of adherent cell lines.

278 Repeated screening of the DMSO aliquot of synthesized 1 revealed increasing APOBEC

279 ) was significantly smaller than that of the DMSO group (172.80 +/- 13.68%) (P < 0.05).

280 In addition, some of the DMSO platinum complexes effectively inhibit angiogenesis

281 -resistant A2780cisR cells, with most of the DMSO platinum complexes exhibiting IC50 values in the su

282 ssible without a significant decrease of the DMSO removal performance.

283 d to be smaller (121.60 +/- 10.40%) than the DMSO group, though the difference was not statistically

284 de an untreated control group in addition to DMSO vehicle control to check for solvent toxicity.

285 Purified Tmm oxidizes DMS to DMSO at a 1:1 ratio.

286 heterotrophic bacterium, can oxidize DMS to DMSO using trimethylamine monooxygenase (Tmm).

287 We exposed C57BL/6J female mice (F0) to DMSO vehicle, the pharmaceutical obesogen rosiglitazone

288 DMSO-d6/GL must be preferred to DMSO-d6/GL-d8 for the study of biological active compoun

289 alytic reaction, including the ligand, TsOH, DMSO, substrate, and cyclohexenone intermediate.

290 Upon DMSO treatment, global quantitation changes from the MS1

291 mation of electron deficient aldehydes using DMSO as solvent.

292 an oxidation reaction enabled just by using DMSO as the solvent as well as an oxidant.

293 ity through a chemical stimulus, by varying [DMSO]0/[Y(OTf)3]0 ratio from 0 to 30 during the polymeri

294 Either DHA (16 mg/kg in DMSO) or vehicle DMSO (1 ml/kg) was administered intraperitoneally at 5 m

295 +/- 4% of the precontracted area (P<0.01 vs. DMSO control).

296 etting were simultaneously actuated by water-DMSO solvent exchange.

297 extensively studied, the mechanism by which DMSO invokes its effect on lipid membranes and the direc

298 Compared with DMSO, concentration-dependent prolonged action potential

299 Without DMSO and [Fe(II)]0 = 300 muM variation of pH yielded dec

300 superlattice of TiS2/[(hexylammonium)x(H2O)y(DMSO)z], with an in-plane lattice thermal conductivity o